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1 ensitivity of SHR movement to treatment with oryzalin.
2 oderate levels of the microtubule inhibitor, oryzalin.
3 hen the microtubules were depolymerized with oryzalin.
4 r sensitivity to depolymerization by cold or oryzalin.
5 ations of the microtubule-destabilizing drug oryzalin.
6  to the microtubule-depolymerizing herbicide oryzalin.
7 emoved upon depolymerizing microtubules with oryzalin.
8 tivity to the microtubule destabilizing drug oryzalin.
9         Analogs of the antimitotic herbicide oryzalin (3,5-dinitro-N4,N4-di-n-propylsulfanilamide) we
10                                 In contrast, oryzalin, an agent that depolymerizes microtubules, seve
11 roxyurea, whereas it was induced by sucrose, oryzalin, and auxin, thereby revealing expression charac
12 by the antimicrotubule drugs propyzamide and oryzalin, and right skewing is exacerbated by cold treat
13 ith the anti-microtubule drugs, propyzamide, oryzalin, and trifluralin also failed to open under the
14                 Docking studies predict that oryzalin binds with an average affinity of 23 nM to a si
15 by short-term treatments with latrunculin B, oryzalin, brefeldin A (BFA) or wortmannin--all of which
16 the controls, was increasingly variable with oryzalin concentration, meaning that microfibril orienta
17 ubule defects, including hypersensitivity to oryzalin, defects in cell division, cortical array organ
18 s of Bos taurus alpha-tubulin indicated that oryzalin did not interact with this site and had a signi
19                                              Oryzalin disrupted guard-cell microtubules and prevented
20  the G142S mutation are largely dependent on oryzalin for viable growth in culture.
21 oaniline herbicides (such as trifluralin and oryzalin) have been developed for the selective control
22                             By screening for oryzalin hypersensitivity, we identified several mutants
23                                      Neither oryzalin nor latrunculin affected the speed of CESA comp
24 e (dichlorobenylnitrile; DCB), microtubules (oryzalin), or actin (latrunculin B).
25 ce of the microtubule organization inhibitor oryzalin, suggesting a central role for cytoskeleton reo
26 n the growth zone for all treatments, but on oryzalin the distributions became broad, indicating poor
27                                              Oryzalin-treated kinesin-14d1 mutant cells had kinetocho
28 subcellular scale, cellulose microfibrils in oryzalin-treated roots were as well aligned as in contro
29                                              Oryzalin treatments disrupt the frequency and increase t
30 putational studies, a common binding site of oryzalin, trifluralin, and GB-II-5 on apicomplexan and k
31                             Dinitroanilines (oryzalin, trifluralin, ethafluralin) disrupt microtubule
32 arkably sensitive to dinitroanilines such as oryzalin, which disrupt plant but not animal microtubule