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1 re, high intracellular calcium, and elevated osmotic fragility.
2 ed by abnormal cell morphology and increased osmotic fragility.
3 l counts as well as decreased red blood cell osmotic fragility.
4 alters spectrin dimer/tetramer stability and osmotic fragility.
5 n and was identified as the top predictor of osmotic fragility.
6 olume (MCV), cell dehydration, and increased osmotic fragility.
7 t was confirmed by documenting a decrease in osmotic fragility and an increase in cell density follow
10 .2) has been proposed based on the increased osmotic fragility and spherocytic morphology of P4.2-def
12 RBC are morphologically normal, have normal osmotic fragility, and mPEG-derivatized murine RBC have
14 ed diabetes-induced increases in erythrocyte osmotic fragility-Diabetes-induced increased oxidative s
17 impaired zinc status, including erythrocyte osmotic fragility, in vitro erythrocyte (65)Zn uptake, a
18 include enhanced sickling rates and altered osmotic fragility, loss of RBC surface area, and hyperve
19 , we have developed a mathematical model for osmotic fragility of infected cells based on diffusional
23 ular volume, cellular dehydration, increased osmotic fragility, reduced deformability, and heterogene
24 gesting that the reported increase in IRBCs' osmotic fragility results from a reduction in surface ar
25 holine-deficient medium had greater membrane osmotic fragility than did cells grown in control medium