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1 oint disease with synovitis, enthesitis, and osteitis.
6 s on MRI have been confirmed as representing osteitis and cortical bone defects, respectively, adding
7 that tmTNF overexpression in mice results in osteitis and ELS formation in BM, which may account for
9 produce inflammation similar to pulpitis and osteitis and that this mouse model can be used to study
10 iffuse bone marrow oedema (histologically an osteitis) and bone marrow fatty corners detected via mag
11 ance imaging (MRI) scores for joint erosion, osteitis, and synovitis, scores on the Health Assessment
13 to peri-fibrocartilaginous sacroiliac joint osteitis, as well as to spinal peri-entheseal osteitis,
15 ype (GTA) differed significantly between BCG osteitis cases and controls (0.296 vs. 0.184, p = 0.040
19 igh-turnover osteodystrophy characterized by osteitis fibrosa, and a low-turnover osteodystrophy char
21 (synovitis, acne, pustulosis, hyperostosis, osteitis) is an inflammatory disorder of the bone, skin,
22 ied, rs4711998 associated nominally with BCG osteitis; minor allele frequency was 0.215 in 130 BCG os
24 s (33.9%), injection site reactions (35.2%), osteitis/osteomyelitis (27.9%), and distant soft tissue
25 s (33.9%), injection site reactions (35.2%), osteitis/osteomyelitis (27.9%), and distant soft tissue
28 ed that it is a result of bone inflammation (osteitis/osteomyelitis), i.e. replacememt of bone marrow
33 could be fit into distinct groups, including osteitis pubis, adductor compartment injury, rectus abdo
34 (synovitis, acne, pustulosis, hyperostosis, osteitis) syndrome; both had complete clearance of skin
35 , joint pain (but no swelling), and signs of osteitis, synovitis, or tenosynovitis in hand MRI were r
37 model morphologically mimicking pulpitis and osteitis, we tested it for signs of oral pain with an or
39 steitis, as well as to spinal peri-entheseal osteitis, which is often extensive and which provides a