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1 oenzymatic studies were performed on normal, osteoarthritic, and RA synovium and tonsil with antibodi
2 lockite crystals have been found not only in osteoarthritic articular cartilage but also in normal ca
3 nescence in regenerating skeletal muscle and osteoarthritic articular cartilage.
4 ification and dys-regulated matrix repair in osteoarthritic articular cartilage.
5 iochemistry of the collagenous matrix within osteoarthritic bone has received scant attention.
6 a three- to fourfold increase in TGF-beta in osteoarthritic bone indicates that this might represent
7    In addition, the degradative potential of osteoarthritic bone was considerably higher as demonstra
8 flammatory enzyme expressed in rheumatic and osteoarthritic, but not normal, cartilage.
9 , which is differentially expressed in human osteoarthritic cartilage and after SV40 transformation o
10 -3 has low affinity for the surface layer of osteoarthritic cartilage and has reduced affinity for si
11                  The expression of MMP-13 in osteoarthritic cartilage and its activity against type I
12 mber of snoRNAs with decreased expression in osteoarthritic cartilage and osteoarthritic chondrocytes
13                         Cell death occurs in osteoarthritic cartilage and recent studies suggest auto
14 t Galectin-1 upregulation is associated with osteoarthritic cartilage and subchondral bone histopatho
15  cytokine-induced PGE(2) production by human osteoarthritic cartilage as well as by a synovial sarcom
16 ar chondrocytes and up-regulation of TLR2 in osteoarthritic cartilage chondrocytes in situ.
17 oepitope were significantly more abundant in osteoarthritic cartilage compared with cartilage from he
18 ety of autologous N-TEC for the treatment of osteoarthritic cartilage defects in the knees of two pat
19 eals an intimate relation of glycobiology to osteoarthritic cartilage degeneration.
20                            Chondrocytes from osteoarthritic cartilage exhibited CD44 fragmentation as
21 itogenic effect, and crude extracts of human osteoarthritic cartilage induced a 100% incidence with a
22 ility (up to 25-fold), suggesting that early osteoarthritic cartilage is more vulnerable to higher lo
23 lagen in the damaged extracellular matrix of osteoarthritic cartilage may also be an effective strate
24                                Primary human osteoarthritic cartilage predominantly expressed TCF4 an
25 s derived from single and multiple donors of osteoarthritic cartilage revealed the presence of a dive
26 (top) and deep (bottom) layer of healthy and osteoarthritic cartilage samples from 64 patients (19 co
27 rimary porcine chondrocyte culture and human osteoarthritic cartilage tissue were studied.
28 both cytokine-stimulated cells as well as in osteoarthritic cartilage tissues.
29 tores the inferior compressive properties of osteoarthritic cartilage to that of healthy cartilage, p
30 mine chondroprogenitor numbers in normal and osteoarthritic cartilage where we observed a 2-fold incr
31 icular cartilage, in chondrocyte clusters of osteoarthritic cartilage, and in the zone of proliferati
32                                   Similar to osteoarthritic cartilage, antisense-treated cartilage sl
33  In summary, the -104 CpG is demethylated in osteoarthritic cartilage, correlating with the elevated
34 ype II collagen was strongly up-regulated in osteoarthritic cartilage.
35 llular matrix, including breast tumours, and osteoarthritic cartilage.
36 ated, it was reduced to approximately 30% in osteoarthritic cartilage.
37 display analysis of transcripts expressed in osteoarthritic cartilage.
38 oth types of G1 fragments were isolated from osteoarthritic cartilage.
39 inoma, is expressed by chondrocytes in human osteoarthritic cartilage.
40  the pro-catabolic phenotype associated with osteoarthritic cartilage.
41 ression was significantly increased in human osteoarthritic cartilage.
42 pregulated under genomic control in diseased osteoarthritic cartilage.
43 pregulated under genomic control in diseased osteoarthritic cartilage.
44 y and regenerative potential residing within osteoarthritic cartilage.
45  cartilage sections from older adults and in osteoarthritic cartilage.
46 ize with MMP13 protein and activity in human osteoarthritic cartilage.
47 ed kinase (ERK) around chondrocytes in human osteoarthritic cartilage.
48 lage matrix synthesis is reduced in aged and osteoarthritic cartilage.
49 truction of extracellular matrix proteins in osteoarthritic cartilage.
50 ed at a specific 'aggrecanase' site in human osteoarthritic cartilage; this cleavage can be performed
51 ecan and small proteoglycans from normal and osteoarthritic cartilages were analyzed for chain intern
52  approximately 10% of chains from normal and osteoarthritic cartilages, respectively.
53 d population, as well as prevention of early osteoarthritic changes in the injured athletic populatio
54 ssociation between cartilaginous lesions and osteoarthritic changes was calculated.
55                  All ACLT knees demonstrated osteoarthritic changes.
56 nced MMP13 by galactose-replacement in human osteoarthritic chondrocyte cultures inversely paralleled
57  which has many of the characteristics of an osteoarthritic chondrocyte in terms of the cytokines, ch
58 ated during cellular stress, is decreased in osteoarthritic chondrocytes and this reduction in autoph
59 d cartilage, through phenotype modulation of osteoarthritic chondrocytes in order to stimulate growth
60 tro, the lectin was secreted and it bound to osteoarthritic chondrocytes inhibitable by cognate sugar
61 n RNA, protein extracts, and nuclei of human osteoarthritic chondrocytes left untreated or treated wi
62       A dysfunctional TRPV4-GSK3B pathway in osteoarthritic chondrocytes rendered the cells unable to
63                                              Osteoarthritic chondrocytes secrete matrix metalloprotei
64                                           In osteoarthritic chondrocytes, the antioxidants Mn(III) te
65                                              Osteoarthritic chondrocytes, which are deficient in auto
66                              In contrast, in osteoarthritic chondrocytes, which possessed elevated ba
67 stress relaxation restricting it) but not in osteoarthritic chondrocytes.
68 d medium from normal adult porcine and human osteoarthritic chondrocytes.
69 lation of its cognate receptor expression in osteoarthritic chondrocytes.
70 d expression in osteoarthritic cartilage and osteoarthritic chondrocytes.
71 ave undergone dedifferentiation in vitro and osteoarthritic chondrocytes.
72 fic CpG dinucleotides within its promoter in osteoarthritic compared to normal cartilage, which corre
73 een a tendency to equate cartilage loss with osteoarthritic degeneration.
74  with age, some of which are consistent with osteoarthritic degeneration.
75 y 6 months of age, mutant condyles displayed osteoarthritic degradation with apical/mid-zone separati
76  for cartilage homeostasis and prevention of osteoarthritic disease.
77 l linear modeling to accelerometry data from osteoarthritic dogs participating in a cross-over non-st
78        Previous efforts to quantify sleep in osteoarthritic dogs using accelerometry have not demonst
79 hich no oral or parenteral disease-modifying osteoarthritic drug (DMOAD) is currently available.
80 and that nidogen-2-null mice exhibit typical osteoarthritic features.
81 eposited collagen in the subchondral zone of osteoarthritic femoral heads, supporting a greater propo
82 bone collagen metabolism is increased within osteoarthritic femoral heads, with the greatest changes
83 ited in the metabolism of bone collagen from osteoarthritic hips might exacerbate the processes invol
84    We compared IGF-I signaling in normal and osteoarthritic human articular chondrocytes and investig
85 id shear stress (20 dyn/cm(2))-activated and osteoarthritic human chondrocytes, however, the precise
86                       We analyzed normal and osteoarthritic human knee cartilage, and found HS biosyn
87  vitro, we validate extraction of HC*HA from osteoarthritic human synovial fluid as a model complex b
88  Local delivery of StemJEL to post-traumatic osteoarthritic jaw and knee joints in rabbit, rat, and m
89 s upregulated in middle zone chondrocytes in osteoarthritic joint cartilage (where hypertrophic marke
90 been shown to be differentially expressed in osteoarthritic joint cartilage.
91                                              Osteoarthritic joint damage may be associated with clini
92                                           In osteoarthritic joint disease, expression of bFGF and MMP
93 in angiogenic RA compared with nonangiogenic osteoarthritic joint fluid.
94 hate (BCP) crystals are common components of osteoarthritic joint fluids and tissues.
95 are aberrantly activated in human and murine osteoarthritic joint tissues.
96 gain importance, like reducing loading in an osteoarthritic joint, but people often do not change the
97 es to target the sites of inflammation in an osteoarthritic joint, thereby reducing the inflammation-
98 s and the ATP-dependent P2XR channels in the osteoarthritic joint.
99 secreted protein pattern of explants from 12 osteoarthritic joints (knee, hip, and shoulder) and 14 n
100 ed in vitro to inflammatory stimuli found in osteoarthritic joints and favorably altered the inflamma
101 ms of calcium crystals that are found within osteoarthritic joints continues to challenge and confoun
102  nanofibre into rats with surgically induced osteoarthritic joints led to cartilage regeneration and
103  turnover in intact, moving, human joints or osteoarthritic joints treated by HA injections.
104 bited the production of catabolic enzymes in osteoarthritic joints, and suppressed immune cells.
105 ficiency hyper-activates NF-kappaB in murine osteoarthritic joints, which impairs bone formation and
106 variety of inflammatory disorders and 9 from osteoarthritic joints.
107 sential role in the remodeling and repair of osteoarthritic joints.
108 opment, and their expression is increased in osteoarthritic joints.
109 n directly contribute to cartilage repair in osteoarthritic joints.
110 tered the inflammatory profile of cells from osteoarthritic joints.
111  snoRNAs in old sham versus old experimental osteoarthritic joints.
112 ols to the chronicity and destruction of the osteoarthritic joints.
113 on of complement is abnormally high in human osteoarthritic joints.
114 ht hyaluronan (LMW-HA) is often increased in osteoarthritic joints; however, its biological function
115  mean rate of joint space narrowing (JSN) in osteoarthritic knees by 26% (P = not significant).
116 ilage: cartilage adjacent to the site of the osteoarthritic lesion and cartilage distal from the lesi
117                                              Osteoarthritic mice also exhibited an elevated ratio of
118             We show that in young, older and osteoarthritic mice, hierarchical changes in tissue stru
119  the hyper responsiveness of knee loading in osteoarthritic mice.
120 ilage through crosstalk of bone-cartilage in osteoarthritic mice.
121           Whole femoral heads extracted from osteoarthritic (n = 5) and healthy controls (n = 5) unde
122  chondrocyte hypertrophy develops in situ in osteoarthritic (OA) articular cartilage and promotes dys
123 o be transcriptionally up-regulated in human osteoarthritic (OA) articular cartilage.
124 zed by immunohistochemistry using normal and osteoarthritic (OA) cartilage and by immunoblotting of c
125 WISP-3/CCN6 by determining its expression in osteoarthritic (OA) cartilage and by investigating its e
126       Pathologic mineralization is common in osteoarthritic (OA) cartilage and may be mediated by ext
127     S100A4 has been shown to be increased in osteoarthritic (OA) cartilage and to stimulate chondrocy
128 drocyte hypertrophy that commonly develop in osteoarthritic (OA) cartilage can promote dysregulated m
129                                    Aging and osteoarthritic (OA) cartilage commonly demonstrate enhan
130 zed by immunohistochemistry using normal and osteoarthritic (OA) cartilage from young and old monkeys
131 icular chondrocytes, to determine changes in osteoarthritic (OA) cartilage, and to address the functi
132 ge from mice, and in chondrocytes from human osteoarthritic (OA) cartilage.
133 genes was also investigated in preserved and osteoarthritic (OA) cartilage.
134  assess the role of SirT1 in the survival of osteoarthritic (OA) chondrocytes in humans.
135                                              Osteoarthritic (OA) degeneration of the lumbar facet joi
136                                              Osteoarthritic (OA) dogs are good translational models,
137 s of inhibition of each enzyme in normal and osteoarthritic (OA) explants.
138 anizations, and they commonly aggregate near osteoarthritic (OA) fissures.
139                                   Normal and osteoarthritic (OA) human articular cartilage was used f
140 ry cell cultures established from normal and osteoarthritic (OA) human knee articular cartilage were
141 l protein 1 (HMGB-1), which are increased in osteoarthritic (OA) joints, drive procatabolic chondrocy
142      Calcium deposition occurs frequently in osteoarthritic (OA) joints.
143 mal ankle cartilage of organ donors and from osteoarthritic (OA) knee tissue obtained from patients u
144 ens from adult human donors with and without osteoarthritic (OA) lesions were stained by immunohistoc
145 a rat model of monoiodoacetate (MIA) induced osteoarthritic (OA) pain as evaluated by hindlimb grip f
146 ls of pathological pain, including models of osteoarthritic (OA) pain.
147 tracellular matrix, which can progress to an osteoarthritic (OA) phenotype.
148  active cellular process and was enhanced in osteoarthritic (OA) tissue.
149 terns of MUC1, MUC2, MUC3, and MUC5AC in RA, osteoarthritic (OA), and normal human ST.
150 s available for small and large experimental osteoarthritic pain animal models.
151 h factor (NGF) is a key regulator of chronic osteoarthritic pain, but the exact targets of NGF action
152  of joint nociceptors, which is critical for osteoarthritic pain, is also dependent on Piezo2, and ta
153 ing to relief of neuropathic, orofacial, and osteoarthritic pain-like behaviors via 3 different route
154 d capsaicin-induced mechanical allodynia and osteoarthritic pain.
155  connectivity among these regions in chronic osteoarthritic patients (n = 54) and healthy controls (n
156 ly identified HTRA1 as being up-regulated in osteoarthritic patients and as having the potential to r
157                          Up to 8-10% of poly-osteoarthritic patients are predicted and observed to re
158         Similar to BMMSC, CPSC isolated from osteoarthritic patients express stem cell markers and ha
159 atoid arthritis, but it was not detected for osteoarthritic patients from either clinic.
160                          Cartilage repair in osteoarthritic patients remains a challenge.
161                                           In osteoarthritic patients, as in other patient groups, eff
162 in levels were elevated in chondrocytes from osteoarthritic patients, consistent with a down-regulati
163 recan at several sites detected in joints of osteoarthritic patients.
164  of SirT1 in human chondrocytes derived from osteoarthritic patients.
165 e tissues of arthritic mice as well as human osteoarthritic patients.
166 rizes to activin A) were produced by all the osteoarthritic samples and half of the normals.
167 d, with increased levels of 6-O-sulfation in osteoarthritic samples, which correlated with increased
168 ot blot was within the normal range for most osteoarthritic samples, with only 5 of 24 displaying ele
169  snoRNAs in old sham versus old experimental osteoarthritic serum.
170     As example, in the superior femoral head osteoarthritic specimens exhibited a more heterogeneous
171 ic capacity and propensity to a procatabolic osteoarthritic state.
172 tilage exhibited regenerative capacity under osteoarthritic stress, offering a model for a regenerati
173 e generalized and greater increase in BMD in osteoarthritic subjects seen in previous studies of unre
174  synovial fluid as compared with healthy and osteoarthritic subjects.
175 rthritis, and vascular growth is enhanced in osteoarthritic synovia when infiltrating macrophages gen
176 c acid (mRNA) was compared between RASFs and osteoarthritic synovial fibroblast (OASFs) using quantit
177 essed by chondrocytes) and their presence in osteoarthritic synovial fluid confirmed.
178 died in chondrocytes using interleukin-1 and osteoarthritic synovial fluid.
179  phosphate crystals are common components of osteoarthritic synovial fluids and define subsets of pat
180 icromasses, ions with or without IL-1beta or osteoarthritic synovial fluids for 48 h.
181 or AAT abrogated the effects of IL-1beta and osteoarthritic synovial fluids on anabolic gene expressi
182 med joints, considerably higher than that in osteoarthritic synovial fluids.

 
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