1  of joint cartilage, leading to severe early osteoarthropathy.

2 us on diabetic osteomyelitis and neuropathic osteoarthropathy.

3 loped a prominent chronic severe destructive osteoarthropathy.

4 o probands, excluding secondary hypertrophic osteoarthropathy.

5 is, and the pathogenesis of XLH-degenerative osteoarthropathy.

6 e tissues), and a painful and severe form of osteoarthropathy.

7 f weight-bearing joints, leading to a severe osteoarthropathy.

8 e outward hallmark of pulmonary hypertrophic osteoarthropathy, a clinical constellation that develops

9 n wild type (WT) mice, and mice with genetic osteoarthropathies (AKU) was undertaken to further under

10 KBD) is an endemic, age-related degenerative osteoarthropathy and its cause is hypothesised to involv

11 moptysis, gout, cholelithiasis, hypertrophic osteoarthropathy, and decreased renal function.

12 he pathogenesis of clubbing and hypertrophic osteoarthropathy has hitherto been poorly understood, bu

13 idemark, and greater abundance in a model of osteoarthropathy indicate their formation could be impor

14 le primary (idiopathic) form of hypertrophic osteoarthropathy (PHO) is recognized.

15       Kashin-Beck disease (KBD) is a chronic osteoarthropathy, which manifests as joint deformities a