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1 growth factor treatment or with the state of osteoblast-like activity and appears to determine the na
4 Sp3 sites act as enhancers in both MC3T3-E1 (osteoblast-like) and J774 (macrophage-like) cell lines,
5 d cell proliferation were analysed to assess osteoblast-like behaviour in the presence of Asc, Dex an
7 We have demonstrated that both the UMR-106 osteoblast-like cell line and human osteoblasts in prima
8 bone formation within osteoblasts and in the osteoblast-like cell line MC3T3-E1, a finding consistent
10 estosterone with calcium channels in the rat osteoblast-like cell line ROS 17/2.8 was investigated.
14 well as murine (MC3T3-E1) and human (MG-63) osteoblast-like cell lines displayed all the previously
15 element that drives human SOST expression in osteoblast-like cell lines in vitro and in the skeletal
17 steocalcin expression, we used rat and human osteoblast-like cell lines to generate stably transfecte
22 with inflammatory changes and expression of osteoblast-like cell phenotypes, but the cellular mechan
24 muscle cells to change into a chondrocyte or osteoblast-like cell; high total body burden of calcium
25 tion, RANKL upregulation in human mandibular osteoblast-like cells (HMOBs) were stimulated with PGE2.
26 simple, reproducible method to isolate human osteoblast-like cells (HOBs) and to evaluate in vitro ce
27 y was to examine the expression of CaMKII in osteoblast-like cells (MC4) and to elucidate its role in
28 nesis, nonosseous calcification, and ectopic osteoblast-like cells (that appear to function different
29 and cultured with fetal calf serum (10% for osteoblast-like cells and 2% for osteoclast-like cells).
30 anism of action of applied tensile forces in osteoblast-like cells and have critical implications in
34 Myofibroblasts further differentiate into osteoblast-like cells and produce calcium nanoparticles,
35 ot analysis, we have demonstrated that human osteoblast-like cells as well as primary human osteoblas
36 is verified the differentiation of DFCs into osteoblast-like cells because clusters of mineralization
37 target gene expression were up-regulated in osteoblast-like cells derived from cortical bone of fema
38 ng vascular cells (CVCs), a subpopulation of osteoblast-like cells derived from the artery wall, were
39 ate that Gli1(+) cells are a major source of osteoblast-like cells during calcification in the media
44 at [Ca2+]o-stimulated chemotaxis of MC3T3-E1 osteoblast-like cells involves a G-protein-linked calciu
45 of the murine osteocyte cell line MLO-Y4 and osteoblast-like cells MC3T3-E1 and in primary rat osteob
46 vular leaflets may promote the generation of osteoblast-like cells through enhanced type I IFN signal
47 ses of primary mouse osteoblasts and UMR-106 osteoblast-like cells to a single period of dynamic stra
51 only been shown in some, but not all, clonal osteoblast-like cells, and the molecular mechanisms unde
52 compatibility of the Mg-Zn-Mn alloy on MG-63 osteoblast-like cells, further substantiating its safety
53 AK can target human primary chondrocytes and osteoblast-like cells, in addition to synovial fibroblas
54 al stress and selective differentiation into osteoblast-like cells, offering a promising nanotechnolo
55 sed by stimulation of proliferation of MG-63 osteoblast-like cells, was used to monitor purification
56 human and mouse osteoblasts and mouse MC3T3 osteoblast-like cells, we found that Akt activation by F
57 tiation of vascular smooth muscle cells into osteoblast-like cells, we investigated whether miRs impl
58 ansforming vascular smooth muscle cells into osteoblast-like cells, which can produce a matrix of bon
59 e beta(5) gene expression in macrophages and osteoblast-like cells, with each element exhibiting cell
72 cultures resulted in reacquisition of their osteoblast-like characteristics and lack of LPS responsi
73 l significance of the loss of the PDL cell's osteoblast-like characteristics during inflammation.
74 we observed that IL-1beta down-regulates the osteoblast-like characteristics of PDL cells in vitro.
75 th IL-1beta inhibits the expression of their osteoblast-like characteristics, as assessed by the fail
76 orskolin treatment of CVC for 3 days induced osteoblast-like "cuboidal" morphology, inhibited prolife
79 nesis in PDL cells while down-regulating its osteoblast-like features and simultaneously reducing the
80 ed the levels of p21 mRNA and protein in the osteoblast-like human osteosarcoma cell line MG63 and st
81 ted gene transcription and mineralization in osteoblast-like MC3T3-E1 cells (WT) via a mechanism invo
82 Immunocytochemical studies performed with osteoblast-like MC3T3-E1 cells and other mammalian cell
84 steogenesis, as evidenced by increased human osteoblast-like MG-63 cell proliferation in vitro and ca
86 have found that a lipid fraction from human osteoblast-like MG63 cell-conditioned medium (MG63CM) co
91 morphogenetic protein-2 [BMP-2]), MG63 human osteoblast-like osteosarcoma cells, and normal human ost
94 ed at the interface of bone and teeth, where osteoblast-like periodontal ligament (PDL) cells constan
95 a healthy periodontium PDL cells exhibit an osteoblast-like phenotype and are unresponsive to gram-n
96 -Luc assay in HepG2 cells as well as induced osteoblast-like phenotype in C2C12 cells expressing alka
98 Furthermore, TGF-beta1 down-regulated the osteoblast-like phenotype of PDL cells, i.e., alkaline p
99 lve associated with the transformation to an osteoblast-like phenotype that is inhibited by atorvasta
101 mal fibroblasts lacking Smad3 can acquire an osteoblast-like phenotype, including activation of Runx2
107 erstitial cells increasingly adopt myofibro-/osteoblast-like properties, thereby fostering fibro-calc