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1 growth factor treatment or with the state of osteoblast-like activity and appears to determine the na
2  antimigratory effects of zoledronic acid in osteoblast-like and endothelial cells.
3                        The identification of osteoblast-like and osteoclast-like cells in human tissu
4 Sp3 sites act as enhancers in both MC3T3-E1 (osteoblast-like) and J774 (macrophage-like) cell lines,
5 d cell proliferation were analysed to assess osteoblast-like behaviour in the presence of Asc, Dex an
6                These include the presence of osteoblast-like calcifying vascular cells in the artery
7   We have demonstrated that both the UMR-106 osteoblast-like cell line and human osteoblasts in prima
8 bone formation within osteoblasts and in the osteoblast-like cell line MC3T3-E1, a finding consistent
9                                 In the human osteoblast-like cell line MG-63, 1,25-(OH)(2)D(3) and 19
10 estosterone with calcium channels in the rat osteoblast-like cell line ROS 17/2.8 was investigated.
11                           Furthermore, in an osteoblast-like cell line, uroporphyrin-I decreased mine
12                        Three different human osteoblast-like cell lines (MG-63, HOS, and SaOS-2) and
13                                              Osteoblast-like cell lines 2T3 and Oct-1 also show incre
14  well as murine (MC3T3-E1) and human (MG-63) osteoblast-like cell lines displayed all the previously
15 element that drives human SOST expression in osteoblast-like cell lines in vitro and in the skeletal
16       CRISPR-Cas9 editing of CD109 in SaOS-2 osteoblast-like cell lines showed that partial CD109 kno
17 steocalcin expression, we used rat and human osteoblast-like cell lines to generate stably transfecte
18 , whose expression is controlled by CBFA1 in osteoblast-like cell lines.
19 cell processes of Saos-2 when using it as an osteoblast-like cell model.
20              We found that UTY modulates the osteoblast-like cell phenotype in response to nanoscale
21 cale extracellular matrix cues to promote an osteoblast-like cell phenotype.
22  with inflammatory changes and expression of osteoblast-like cell phenotypes, but the cellular mechan
23 lar matrix remodelling and myofibroblast and osteoblast-like cell proliferation.
24 muscle cells to change into a chondrocyte or osteoblast-like cell; high total body burden of calcium
25 tion, RANKL upregulation in human mandibular osteoblast-like cells (HMOBs) were stimulated with PGE2.
26 simple, reproducible method to isolate human osteoblast-like cells (HOBs) and to evaluate in vitro ce
27 y was to examine the expression of CaMKII in osteoblast-like cells (MC4) and to elucidate its role in
28 nesis, nonosseous calcification, and ectopic osteoblast-like cells (that appear to function different
29  and cultured with fetal calf serum (10% for osteoblast-like cells and 2% for osteoclast-like cells).
30 anism of action of applied tensile forces in osteoblast-like cells and have critical implications in
31             The promoters are active in both osteoblast-like cells and in the M12 B-lymphocyte cell l
32 ation of the balance between the activity of osteoblast-like cells and OLCs.
33                    In the present study MG63 osteoblast-like cells and primary bone marrow stromal ce
34    Myofibroblasts further differentiate into osteoblast-like cells and produce calcium nanoparticles,
35 ot analysis, we have demonstrated that human osteoblast-like cells as well as primary human osteoblas
36 is verified the differentiation of DFCs into osteoblast-like cells because clusters of mineralization
37  target gene expression were up-regulated in osteoblast-like cells derived from cortical bone of fema
38 ng vascular cells (CVCs), a subpopulation of osteoblast-like cells derived from the artery wall, were
39 ate that Gli1(+) cells are a major source of osteoblast-like cells during calcification in the media
40                                        These osteoblast-like cells from ALSKO mice failed to induce o
41  signals colocalized with calcified areas or osteoblast-like cells in human vascular lesions.
42           ADAM19 is also highly expressed in osteoblast-like cells in the bone, yet it does not appea
43 ted in the present study using UMR106-01 rat osteoblast-like cells in vitro.
44 at [Ca2+]o-stimulated chemotaxis of MC3T3-E1 osteoblast-like cells involves a G-protein-linked calciu
45 of the murine osteocyte cell line MLO-Y4 and osteoblast-like cells MC3T3-E1 and in primary rat osteob
46 vular leaflets may promote the generation of osteoblast-like cells through enhanced type I IFN signal
47 ses of primary mouse osteoblasts and UMR-106 osteoblast-like cells to a single period of dynamic stra
48      Delivering pediatric human bone-derived osteoblast-like cells to an in vivo murine bone loss mod
49                                         MG63 osteoblast-like cells were cultured on the 3 different s
50            Stably alpha2-silenced MG63 human osteoblast-like cells were used to test whether alpha2be
51 only been shown in some, but not all, clonal osteoblast-like cells, and the molecular mechanisms unde
52 compatibility of the Mg-Zn-Mn alloy on MG-63 osteoblast-like cells, further substantiating its safety
53 AK can target human primary chondrocytes and osteoblast-like cells, in addition to synovial fibroblas
54 al stress and selective differentiation into osteoblast-like cells, offering a promising nanotechnolo
55 sed by stimulation of proliferation of MG-63 osteoblast-like cells, was used to monitor purification
56  human and mouse osteoblasts and mouse MC3T3 osteoblast-like cells, we found that Akt activation by F
57 tiation of vascular smooth muscle cells into osteoblast-like cells, we investigated whether miRs impl
58 ansforming vascular smooth muscle cells into osteoblast-like cells, which can produce a matrix of bon
59 e beta(5) gene expression in macrophages and osteoblast-like cells, with each element exhibiting cell
60 tes in VICs activating to myofibroblasts and osteoblast-like cells.
61 tometry validated the target specifically in osteoblast-like cells.
62 organized extracellular matrix deposition by osteoblast-like cells.
63 in human microvascular endothelial and human osteoblast-like cells.
64 oses of PTH attenuated P(1)/P(2) activity in osteoblast-like cells.
65 ma cells and in adhesion of myeloma cells to osteoblast-like cells.
66 s of MEK(SP) were specific to Cbfa1-positive osteoblast-like cells.
67 n macrophage (osteoclast precursor)-like and osteoblast-like cells.
68 uent transdifferentiation of PAH-PASMCs into osteoblast-like cells.
69 with TGF-beta 1, PDGF-BB, and bFGF in bovine osteoblast-like cells.
70  PTH-stimulated adenylyl cyclase activity in osteoblast-like cells.
71 tive stress, uremia, and hyperphosphatemia, "osteoblast-like" cells form in the vessel wall.
72  cultures resulted in reacquisition of their osteoblast-like characteristics and lack of LPS responsi
73 l significance of the loss of the PDL cell's osteoblast-like characteristics during inflammation.
74 we observed that IL-1beta down-regulates the osteoblast-like characteristics of PDL cells in vitro.
75 th IL-1beta inhibits the expression of their osteoblast-like characteristics, as assessed by the fail
76 orskolin treatment of CVC for 3 days induced osteoblast-like "cuboidal" morphology, inhibited prolife
77                                  In MC3T3-E1 osteoblast-like cultures, dexamethasone (DEX) activates
78 in vitro vascular calcification by enhancing osteoblast-like differentiation of CVC.
79 nesis in PDL cells while down-regulating its osteoblast-like features and simultaneously reducing the
80 ed the levels of p21 mRNA and protein in the osteoblast-like human osteosarcoma cell line MG63 and st
81 ted gene transcription and mineralization in osteoblast-like MC3T3-E1 cells (WT) via a mechanism invo
82    Immunocytochemical studies performed with osteoblast-like MC3T3-E1 cells and other mammalian cell
83                                              Osteoblast-like MC3T3-E1 cells were allowed to attach to
84 steogenesis, as evidenced by increased human osteoblast-like MG-63 cell proliferation in vitro and ca
85 eaks (DSBs), in human cervix cancer HeLa and osteoblast-like MG-63 cells.
86  have found that a lipid fraction from human osteoblast-like MG63 cell-conditioned medium (MG63CM) co
87 r cells with TNF-alpha for 3 days induced an osteoblast-like morphology.
88             We show here that differentiated osteoblasts, like neurons, express the norepinephrine tr
89 , the MC3T3-E1(MC3T3) pre-osteoblast and rat osteoblast-like osteosarcoma 17/2.8 cell.
90                 The fully differentiated rat osteoblast-like osteosarcoma 17/2.8 cells responded rapi
91 morphogenetic protein-2 [BMP-2]), MG63 human osteoblast-like osteosarcoma cells, and normal human ost
92 ulture undergo a phenotypic conversion to an osteoblast-like pattern of gene expression.
93 e profiles in three separate clones of human osteoblast-like PDL cells.
94 ed at the interface of bone and teeth, where osteoblast-like periodontal ligament (PDL) cells constan
95  a healthy periodontium PDL cells exhibit an osteoblast-like phenotype and are unresponsive to gram-n
96 -Luc assay in HepG2 cells as well as induced osteoblast-like phenotype in C2C12 cells expressing alka
97                          Cells exhibiting an osteoblast-like phenotype in the vessel wall may be impo
98    Furthermore, TGF-beta1 down-regulated the osteoblast-like phenotype of PDL cells, i.e., alkaline p
99 lve associated with the transformation to an osteoblast-like phenotype that is inhibited by atorvasta
100                                           An osteoblast-like phenotype was in part verified for these
101 mal fibroblasts lacking Smad3 can acquire an osteoblast-like phenotype, including activation of Runx2
102 f vascular smooth muscle cells (VSMCs) to an osteoblast-like phenotype.
103 ence on differentiation of the cells into an osteoblast-like phenotype.
104 tion and that this process is mediated by an osteoblast-like phenotype.
105  active regulated process associated with an osteoblast-like phenotype.
106                     To further determine the osteoblast-like properties of PDL cells, human PDL cells
107 erstitial cells increasingly adopt myofibro-/osteoblast-like properties, thereby fostering fibro-calc
108 d, phenotypically unstable chondrocytes with osteoblast-like properties.
109 TGF-L inhibits osteocalcin production in rat osteoblast-like Ros 17/2.8 cells.
110 receptor-mediated increases in ECAR in human osteoblast-like SaOS-2 cells.
111 vity of stretch-activated cation channels of osteoblast-like UMR-106.01 cells.
112 F)-alpha-induced NF-kappaB activation in rat osteoblast-like UMR106 cells.
113               ALDH1A1 inhibition promotes an osteoblast-like VIC phenotype and increases calcium depo

 
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