戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 tion of the bone protein matrix (rickets and osteomalacia).
2 hosphataemic rickets, fluorosis, rickets and osteomalacia).
3 he most common heritable form of rickets and osteomalacia.
4 itamin D levels, hypercalciuria, and rickets/osteomalacia.
5  in vivo and in the etiology of drug-induced osteomalacia.
6 lates extracellularly, leading to rickets or osteomalacia.
7 t and C-terminal Fgf23 with hypophosphatemic osteomalacia.
8 an disease X-linked hypophosphatemic rickets/osteomalacia.
9  and low bone mineral density as a result of osteomalacia.
10 th a high risk of fragility fractures due to osteomalacia.
11 atemia, renal phosphate wasting, and rickets/osteomalacia.
12 taflox/y, and hyp-mice manifested comparable osteomalacia.
13 tion of 1,25-dihydroxyvitamin D, and rickets/osteomalacia.
14 ng a new molecular mechanism of drug-induced osteomalacia.
15 iating vitamin D catabolism and drug-induced osteomalacia.
16 nal effects of the hormone and contribute to osteomalacia.
17 g adults and causes the painful bone disease osteomalacia.
18 7.08 microM [0.9-3.7 mg/dL]) and evidence of osteomalacia.
19 in the serum of a patient with tumor-induced osteomalacia.
20 to correct the hypophosphatemia, rickets, or osteomalacia.
21 with osteomalacia and 12 patients with focal osteomalacia.
22 rum fibroblast growth factor-23 (Fgf23), and osteomalacia.
23 phosphatase (TNSALP), results in rickets and osteomalacia.
24 ocalcemia, hyperparathyroidism, rickets, and osteomalacia.
25 malities, secondary hyperparathyroidism, and osteomalacia.
26 omatic, and occasional hypophosphataemia and osteomalacia.
27 t FGF23-mediated hypophosphatemic rickets or osteomalacia.
28 ioactive intact FGF23, hypophosphatemia, and osteomalacia.
29  of secondary hyperparathyroidism and severe osteomalacia.
30  secondary hyperparathyroidism, rickets, and osteomalacia, accompanied by 90% reduction in renal CaBP
31 87.5% of patients, including 9 patients with osteomalacia and 12 patients with focal osteomalacia.
32 milliliter in those with suspected oncogenic osteomalacia and 353+/-510 RU per milliliter (range, 31
33 ated with hypophosphataemia, predisposing to osteomalacia and fractures.
34                  However, the latter, mostly osteomalacia and increased bone resorption, cannot be re
35 amin D deficiency (hypovitaminosis D) causes osteomalacia and poor long bone mineralization.
36 <10 ng/mL, which are concentrations at which osteomalacia and rickets occur).
37 ate kinase (PGK) promoter completely avoided osteomalacia and secondary hyperparathyroidism, and simu
38 er osteodystrophy characterized initially by osteomalacia and, more recently, by adynamic or aplastic
39  in childhood or even adulthood with rickets/osteomalacia and/or osteopenia/osteoporosis, hypophospha
40 ophosphatemia, hyperparathyroidism, rickets, osteomalacia, and alopecia--the last a consequence of de
41 o down-regulate NPT2a, causing P(i) wasting, osteomalacia, and decreased BMD.
42 rfism, decreased bone mineral density (BMD), osteomalacia, and decreased serum phosphate (P(i)).
43  hypophosphatemia, characterized by rickets, osteomalacia, and hypomineralized dentin formation, a ph
44 sociated with bone diseases such as rickets, osteomalacia, and osteoporosis.
45  low bone turnover, and generalized or focal osteomalacia are frequent histologic features in transpl
46                                  Rickets and osteomalacia are seen by day 35; however, as early as da
47 ) in mice, or its mutation in humans, causes osteomalacia believed to reflect renal phosphate wasting
48  5.4 +/- 0.1 mg/dl), and severity of rickets/osteomalacia (bone mineral density: -36, -36, and -30%).
49 mal serum calcidiol may avoid the problem of osteomalacia, but it does not correct malabsorption of c
50 ice, its putative role as an auto-/paracrine osteomalacia-causing factor has not been explored.
51 nant hypophosphatemic rickets, and oncogenic osteomalacia demonstrate that elevated levels of novel c
52 with consecutive hypophosphataemia, rickets, osteomalacia, disproportionate short stature, oral manif
53                       Tumors associated with osteomalacia elaborate the novel factor(s), phosphatonin
54  in children as well as hyperparathyroidism, osteomalacia, enthesopathies, osteoarthritis and pseudof
55                 Four patients with oncogenic osteomalacia had concentrations ranging from 426 to 7970
56 lexural strength, and histologic evidence of osteomalacia; however, cultures of Hyp-derived bone marr
57 otypic changes, including dwarfism, rickets, osteomalacia, hypophosphatemia, increased serum parathyr
58 e rickets and failure to thrive in children, osteomalacia in adults, nephrolithiasis and nephrocalcin
59 s of XLH is based on signs of rickets and/or osteomalacia in association with hypophosphataemia and r
60 CasR deficiency, indicating that rickets and osteomalacia in CasR-deficient mice are not due to an in
61 s disorder are similar to those in oncogenic osteomalacia, in which tumors abundantly express FGF-23
62 he concentrations associated with rickets or osteomalacia influence calcium absorption.
63            The pathogenesis of tumor-induced osteomalacia involves tumor expression of fibroblast gro
64                                Tumor-induced osteomalacia is a rare acquired metabolic disorder chara
65           Vitamin D deficiency, which causes osteomalacia, may also be important in the pathogenesis
66 5 years) and from 17 patients with oncogenic osteomalacia (mean age, 43.0+/-13.3 years) and 21 patien
67 alization defects in the skeleton and teeth (osteomalacia/odontomalacia).
68                                    Thus, the osteomalacia of Akp2-/- mice and the hypomineralized phe
69                   Oncogenic hypophosphatemic osteomalacia (OHO) is characterized by a renal phosphate
70 ether it is elevated in those with oncogenic osteomalacia or X-linked hypophosphatemia, an immunometr
71 be markedly elevated in those with oncogenic osteomalacia or X-linked hypophosphatemia, suggesting th
72 alcemia, hyperparathyroidism, kidney stones, osteomalacia, or sarcoidosis or those who were taking mo
73                   In addition to rickets and osteomalacia, patients with XLH have a heavy disease bur
74         Additional skeletal features include osteomalacia, thinned/porous cortical bone, reduced proc
75 ked hypophosphatemia (XLH) and tumor-induced osteomalacia (TIO).
76                                              Osteomalacia was diagnosed in one patient.
77 al agent rifampin, resulting in drug-induced osteomalacia, which is attributed to vitamin D deficienc
78 pophosphatemic rickets, manifest rickets and osteomalacia with isolated renal phosphate-wasting assoc
79  content, expanded growth plates, and severe osteomalacia, with highly increased bone Fgf23 mRNA (>15