戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 lation (EFS)-enhanced osteogenic response in osteoprogenitor cells.
2 een the proliferation and differentiation of osteoprogenitor cells.
3 nd pericytes were postulated to be potential osteoprogenitor cells.
4 tion and differentiation of chondrocytes and osteoprogenitor cells.
5  in regulating osteogenic differentiation of osteoprogenitor cells.
6 d molecular mechanisms, we studied C3H10T1/2 osteoprogenitor cells.
7 us mRNA in mouse pluripotent mesenchymal and osteoprogenitor cells.
8 lial cells can induce the differentiation of osteoprogenitor cells.
9 may directly regulate the differentiation of osteoprogenitor cells.
10 new blood vessels can serve as a conduit for osteoprogenitor cells.
11 quire communication between immune cells and osteoprogenitor cells.
12 ion of the nearby periosteum and a source of osteoprogenitor cells.
13  PYK2 regulates the differentiation of early osteoprogenitor cells across species and that inhibitors
14  accounted for by increased proliferation of osteoprogenitor cells and bone formation resulting from
15 ghlight studies that illustrate the roles of osteoprogenitor cells and cranial suture-derived stem ce
16 ession is suppressed in nonosseous cells and osteoprogenitor cells and during the early proliferative
17                                              Osteoprogenitor cells and endosteal-lining osteopontin(+
18 feration and osteoblastic differentiation of osteoprogenitor cells and enhancing angiogenesis while m
19 ication is a regulated process that involves osteoprogenitor cells and frequently complicates common
20 ne marrow (BMT) leads to engraftment of both osteoprogenitor cells and hematopoietic cells; however,
21 is highly expressed in chondroprogenitor and osteoprogenitor cells and in vitro experiments suggest t
22 is observed in mutant mice combining reduced osteoprogenitor cells and increased osteoclast numbers.
23 veolar bone was preserved by recruiting more osteoprogenitor cells and increasing the rate of bone fo
24 e marrow derived MSC differentiation towards osteoprogenitor cells and inhibited Notch signaling in a
25 d demonstrate that its functions map to both osteoprogenitor cells and mature osteoblasts.
26  chitosan potentiates the differentiation of osteoprogenitor cells and may facilitate the formation o
27 ein fingerprints from single native MC3T3-E1 osteoprogenitor cells and MC3T3-E1 cells transfected wit
28  also demonstrate that RSPO3 is expressed in osteoprogenitor cells and osteoblasts and that osteoblas
29 ) transcription and delay differentiation in osteoprogenitor cells and patient-derived bone.
30 rix mineralization and calcium deposition by osteoprogenitor cells and primary mesenchymal stem cells
31 /B-catenin signaling in early mesenchymal or osteoprogenitor cells and provide evidence that bone def
32 o apoptosis, leading to decreased numbers of osteoprogenitor cells and subsequently reduced bone form
33 iated with significantly more Wnt-responsive osteoprogenitor cells and their progeny as compared with
34 ration and differentiation of Wnt-responsive osteoprogenitor cells and their progeny.
35 tase subunits was confirmed in human primary osteoprogenitors cells, and a significant increase in en
36 idics enables efficient sorting of committed osteoprogenitor cells, as distinct from these mesenchyma
37 ncy enhances differentiation and activity of osteoprogenitor cells, as does expressing a PYK2-specifi
38 d halt the recruitment or the advancement of osteoprogenitor cells at the sites where sutures should
39 cytes with the growth and differentiation of osteoprogenitor cells by simultaneously modulating Bmp4
40  for their effects on the differentiation of osteoprogenitor cells (C2C12) and the proliferation and
41                           In line with this, osteoprogenitor cell cultures from the Sirt1(DeltaOsx1)
42 ed the role of p27 during differentiation of osteoprogenitor cells derived from the bone marrow (BM)
43 me of our data differ from current models of osteoprogenitor cell differentiation and emphasize compo
44 nd molecular evidence for Dlx3 in regulating osteoprogenitor cell differentiation and for both positi
45 hosphatase activity suggests that effects on osteoprogenitor cell differentiation are the result of a
46  of the biology of bone graft remodeling and osteoprogenitor cell differentiation.
47 feration and differentiation in CNCC-derived osteoprogenitor cells during intramembranous bone format
48 e also active in the programming of arterial osteoprogenitor cells during vascular and valve calcific
49 n periosteal (HPO) cells were chosen because osteoprogenitor cells found in bone repair typically com
50 iferation versus osteoblast differentiation, osteoprogenitor cells from the skulls of Tgfbr2(f/f) emb
51                        In contrast, p53-null osteoprogenitor cells have increased proliferation, incr
52 egulate the proliferation and recruitment of osteoprogenitor cells; however, CTGF is down-regulated a
53 as the use of concentrated blood products or osteoprogenitor cells in conjunction with grafts, have b
54 unction in the developing limb mesenchyme or osteoprogenitor cells in mice resulted in shortened and
55 o affected the proliferation and survival of osteoprogenitor cells in osteogenic condensations, leadi
56                   The paucity of PDL-derived osteoprogenitor cells in osteoporotic mice was associate
57 TGFbeta IIR is required for proliferation of osteoprogenitor cells in the CNC-derived frontal bone an
58 as accompanied by a reduction in the pool of osteoprogenitor cells in the PDL.
59  suggest that the endothelium is a source of osteoprogenitor cells in vascular calcification that occ
60  but also to modulate the differentiation of osteoprogenitor cells in vitro and in vivo.
61 naling affected the functional activities of osteoprogenitor cells, including the RUNX2-mediated tran
62                       The differentiation of osteoprogenitor cells, indicated by alkaline phosphatase
63  and blocks BMP2-mediated differentiation of osteoprogenitor cells into osteoblasts.
64 /progenitor cells (MSCs)--rather than mature osteoprogenitor cells--into osteoblasts, resulting in ne
65   In addition, the differentiation of marrow osteoprogenitor cells is regulated by leptin.
66                          Deletion of Pten in osteoprogenitor cells led to increased numbers of osteob
67 rized the response of 2T9 cells, an immature osteoprogenitor cell line derived from the calvariae of
68        In this study we examined an immature osteoprogenitor cell line for its potential utility in m
69       Microarray analysis of MC3T3 cells, an osteoprogenitor cell line, revealed that EGFR signaling
70 were initiated using MC3T3-E1 cells, a mouse osteoprogenitor cell line.
71                                        These osteoprogenitor cells may be derived from the circulatio
72                       This increased pool of osteoprogenitor cells may be susceptible to additional t
73 ted from combinations of such materials with osteoprogenitor cells or osteoinductive factors such as
74       There is evidence that the size of the osteoprogenitor cell population determines the rate of c
75 an active process that can originate from an osteoprogenitor cell population in the adventitia.
76  biocompatible, with defects showing repair, osteoprogenitor cell presence, and vascular channel form
77  the developing bone, consequently promoting osteoprogenitor cell proliferation and decreasing differ
78 ng that allows for independent regulation of osteoprogenitor cell proliferation and differentiation.
79                Moreover, Panx3 also inhibits osteoprogenitor cell proliferation and promotes cell cyc
80             In calvarial cultures we reduced osteoprogenitor cell proliferation; however, we did not
81 n osteoblasts, and overexpression of Dlx3 in osteoprogenitor cells promotes, while specific knock-dow
82 , the presence of Runx2 in actively dividing osteoprogenitor cells suggests that the protein may also
83 row cells can serve as an abundant source of osteoprogenitor cells that are capable of repairing cran
84 lial cells but required for specification of osteoprogenitor cells that differentiate into preosteobl
85              Significantly, PTEN is found in osteoprogenitor cells that give rise to bone-forming ost
86     Fgfr2 is expressed only in proliferating osteoprogenitor cells; the onset of differentiation is p
87 ically caused by impaired differentiation of osteoprogenitor cells, they also suggest that increased
88 te rate of osteoblastic differentiation from osteoprogenitor cell to terminally differentiated osteoc
89 cular endothelium has a role in contributing osteoprogenitor cells to the calcific lesions.
90 lates the vascular endothelium to contribute osteoprogenitor cells to the vascular calcification.
91                       The outcome of various osteoprogenitor-cell transplantation protocols was asses