戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 fibrosis, increased microvessel density, and osteosclerosis).
2  hematopoiesis, and bone marrow fibrosis and osteosclerosis.
3 al cell-derived osteoprotegerin in promoting osteosclerosis.
4 al osteoclasts resulting in osteopetrosis or osteosclerosis.
5 elopmental increase in bone mass, leading to osteosclerosis.
6 modeling and contributes to the induction of osteosclerosis.
7 mice represent a model for myelofibrosis and osteosclerosis.
8  marrow (BM) cells develop myelofibrosis and osteosclerosis.
9 , resulting in decrease of myelofibrosis and osteosclerosis.
10 adequate for identifying bone resorption and osteosclerosis.
11 teolytic function affects the development of osteosclerosis.
12  associated with a splenomegaly and a marked osteosclerosis.
13  findings included: (1) bone resorption, (2) osteosclerosis, (3) clarity of the mandibular canal, (4)
14  provides automated quantitative analysis of osteosclerosis and collagen fibrosis.
15  JAK2 pathway inhibitors can be effective on osteosclerosis and fibrosis; therefore, accurate grading
16                  In the latter case, diffuse osteosclerosis and H-shaped vertebrae are most typical.
17                                              Osteosclerosis and myefibrosis are complications of myel
18 and 9 mo of age, mutant animals also develop osteosclerosis and osteoarthritis.
19 ic bone lesions that consist of a mixture of osteosclerosis and osteolysis.
20 re the underlying cause of myelofibrosis and osteosclerosis and potential treatments for these diseas
21 ssive osteochondrodysplasia characterized by osteosclerosis and short stature, maps to chromosome 1q2
22 beta antibody also reduces myelofibrosis and osteosclerosis and shows additive effects with ruxolitin
23  secondary intramedullary collagen fibrosis, osteosclerosis, angiogenesis, and extramedullary hematop
24 BMP-6 contributes to prostate cancer-induced osteosclerosis at bone metastatic sites.
25                       Hepatitis C-associated osteosclerosis (HCAO) is a rare disorder characterized b
26                                              Osteosclerosis in patients with PMF is a serious complic
27 peractive WNT signaling mouse model of human osteosclerosis, inhibition of glutamine catabolism or Gc
28                               Intramedullary osteosclerosis is a distinct disorder that typically aff
29 osteoporosis (bone loss) together with focal osteosclerosis (localized bone formation).
30     The phenotype of mice homozygous for the osteosclerosis (oc) mutation includes osteopetrosis, and
31 pe is similar to that of the murine mutation osteosclerosis (oc), which is localized to proximal mous
32 and no pathologic bone abnormalities such as osteosclerosis or myelofibrosis.
33 pecific gain of Notch function causes severe osteosclerosis owing to increased proliferation of immat
34 hese observations indicate that BMPs promote osteosclerosis through VEGF in prostate cancer metastase
35 tected in 123 cases (sensitivity 91.1%), and osteosclerosis was detected in 131 cases (sensitivity 98
36 d to normal levels and the myelofibrosis and osteosclerosis were completely corrected.
37 tal stromal changes, such as BM fibrosis and osteosclerosis, whose occurrence is harbinger of a disma
38  histologically and radiographically evident osteosclerosis with age.
39        Mice lacking Shn3 display adult-onset osteosclerosis with increased bone mass due to augmented
40 essive disorder characterized by generalized osteosclerosis with periosteal bone formation and a dist
41 cular atrophy, platyspondyly and metaphyseal osteosclerosis with relative radiolucency of widened dia