ライフサイエンスコーパス: outflow

1 hobic confinement and congests the molecular outflow.

2 BC at two receptor sites of the continental outflow.

3 of the appendage from the right ventricular outflow.

4 amage, which leads to impaired aqueous humor outflow.

5 tonographic outflow facility and uveoscleral outflow.

6 irst, neuroendocrine function, and autonomic outflow.

7 sed by increased resistance to aqueous humor outflow.

8 pulmonary arterial pressure and sympathetic outflow.

9 ty, aqueous humor flow rate, and uveoscleral outflow.

10 excitability, or by decreasing sensory nerve outflow.

11 ificant difference in calculated uveoscleral outflow.

12 er a narrow relativistic jet or an isotropic outflow.

13 hly 16% weakening of subsurface Weddell Gyre outflow.

14 e-specific isotope ratios between inflow and outflow.

15 tion via the sympathetic and parasympathetic outflow.

16 hrough softening of the meshwork to increase outflow.

17 spaces between cells, allowing greater fluid outflow.

18 aocular pressure by regulating aqueous humor outflow.

19 crease in unfiltered MeHg from HCC inflow to outflow.

20 ted five-membered ring in flames and stellar outflows.

21 lues typical of Northern Hemisphere polluted outflows.

22 affected by urban and industrial wastewater outflows.

23 es in cocaine-induced dopamine and glutamate outflow 4 weeks after VU0364572 treatment, without signi

24 tions from atmospheric sources to the Arctic outflow and a higher retention of the long-chain compoun

25 ary arterial pressure influenced sympathetic outflow and baroreflex control of muscle sympathetic ner

26 -lasting elevations in sympathetic vasomotor outflow and blood pressure in healthy humans.

27 inhibition of the RVLM increases sympathetic outflow and blood pressure substantially, providing a me

28 rgic neurones (RVLM-C1) modulate sympathetic outflow and breathing under normal conditions.

29 esponse to manoeuvres increasing sympathetic outflow and by measuring activity-dependent slowing at 2

30 Sympathetic outflow and circulating glucogenic hormones both regulat

31 e relationship between sympathetic vasomotor outflow and either forearm vascular conductance (FVC; re

32 therapeutic interventions to improve aqueous outflow and further advance our understanding of aqueous

33 ed pathogenic link between liver sympathetic outflow and hepatic steatosis and suggest that manipulat

34 on will likely contribute to altered aqueous outflow and intraocular pressure.

35 ecular meshwork is the primary impediment to outflow and its ablation benefits those eyes relatively

36 ed DA neuron properties, reduced striatal DA outflow and motor deficits prior to nigral degeneration.

37 abeculectomy surgery to enhance ocular fluid outflow and reduce intraocular pressure as a treatment f

38 he first evidence for heightened sympathetic outflow and reduced cBRS in RA that can be independent o

39 studies as they may overestimate volumetric outflow and shear stress.

40 t GDF15 was required for hepatic sympathetic outflow and triglyceride metabolism.

41 iative feedback; or by the interplay between outflows and inflows.

42 number of outflows and outflow diameter) of intracerebral haemorrh

43 by alterations in both central (sympathetic outflow) and peripheral (microvascular endothelial) func

44 se pressure nor their role in aqueous humour outflow are understood at the molecular level.

45 ation has been refined to include inflow and outflow based hemodynamic pathways.

46 ting point and resting sympathetic vasomotor outflow both are lower for highland Sherpa compared to a

47 formation manifests as bulging near the cell outflow; bulges that come into contact with the rigid ce

48 ediated activities were detected at the WWTP outflow but diminished downstream.

49 iquid molecules and facilitate the molecular outflow; by contrast, the introduction of ions in the li

50 trated by mapping glaucoma-relevant genes to outflow cell clusters.

51 eight eyes of human donors representing the outflow cell transcriptome.

52 s revealed along the thermal gradients of an outflow-channel and a progressively-drying mineral matri

53 The Martian outflow channels comprise some of the largest known chan

54 separates the downstream reaches of numerous outflow channels from the northern plains.

55 for the first time both separate and shared outflow circuitries among skeletal muscle and subcutaneo

56 pressure waveforms, central aortic pressure, outflow conduit pressure gradient, and instantaneous LV

57 Initially, these outflows contain only protons and neutrons; these later

58 omy], surgeries that increase suprachoroidal outflow (Cypass microstent and iStent Supra), and conjun

59 ifferent, nor did the calculated uveoscleral outflow demonstrate any discernible difference.

60 e number of outflows (p = 0.459) and largest outflow diameter (p = 0.298).

61 number of outflows and outflow diameter) of intracerebral haemorrhage due to in

62 The reduction in CSF outflow did not cause an increase in intracranial pressu

63 sing end-organ responsiveness to sympathetic outflow during passive heat stress.

64 organ responsiveness to efferent sympathetic outflow during whole-body heating.

65 further advance our understanding of aqueous outflow dysregulation in the pathogenesis of glaucoma.

66 The finite gyroradius effect of the outflow electrons generates the crescent-shaped agyrotro

67 pulated by varying reservoir height, and eye outflow facility (C) was determined from the pump flow r

68 racranial pressure (ICP) lowers conventional outflow facility (increases aqueous outflow resistance)

69 lity was negatively correlated with baseline outflow facility (r = -0.51; P < 0.001).

70 The effect of ICP elevation on outflow facility and IOP is blocked by TTX.

71 ive effort, which may reflect a reduction in outflow facility and may contribute to the development o

72 ithout any significant effect on tonographic outflow facility and uveoscleral outflow.

73 The reduction in outflow facility correlates with an increase in intraocu

74 Mean outflow facility decreased from 0.23 +/- 0.08 muL/min/mm

75 The 1 iStent showed a greater increase in outflow facility from baseline (0.10+/-0.04 mul/minute p

76 molol, an aqueous humor flow suppressant, on outflow facility in healthy eyes.

77 Timolol reduces outflow facility in healthy human eyes, and this effect

78 ions that reduce aqueous humor production on outflow facility in living human eyes is unclear.

79 chanism driven by ICP regulates conventional outflow facility in rats.

80 After Hydrus placement, the outflow facility increased from 0.23+/-0.03 mul/minute p

81 Neither the tonographic outflow facility nor the uveoscleral outflow was signifi

82 ion with exposure duration, yet conventional outflow facility of implanted eyes was normal (24.1 +/-

83 NAs targeting ZO-1 and tricellulin increased outflow facility significantly.

84 The percent increase in outflow facility was 79+/-21% for the Hydrus and 11+/-16

85 The decrease in outflow facility was independent of blood pressure, reve

86 The tonographic outflow facility was lower in Group 1 compared to the ot

87 Outflow facility was measured before (baseline control)

88 Using a constant pressure perfusion method, outflow facility was measured in paired eyes from human

89 The outflow facility was measured with the participant in th

90 The change in outflow facility was negatively correlated with baseline

91 Outflow facility with 1 iStent (0.38+/-0.07 mul/minute p

92 TM cell markers, and maintained normal IOP, outflow facility, and extracellular matrix.

93 Main outcome measures were tonographic outflow facility, aqueous humor flow rate, and uveoscler

94 lobe, decreased ocular compliance, increased outflow facility, extracellular matrix (ECM) abnormaliti

95 (3-6 attacks) was due to reduced tonographic outflow facility.

96 fect is greater in eyes with higher baseline outflow facility.

97 nt, and correlated with reduced conventional outflow facility.

98 the more SC that it dilates, the greater the outflow facility.

99 rhythm, elucidates organization of circadian outflow from and modulatory input to SCN(VIP) cells, and

100 essure or an otherwise impaired "glymphatic" outflow from eye to brain.

101 ort that a continuous and spontaneous liquid outflow from hydrophobic nanopores with high and stable

102 experimentally induced by disturbing aqueous outflow from the eye, resulting in intraocular pressure

103 cells and, in turn, the aqueous humour (AH) outflow from the eye.

104 owed assessment of microplastics in-wash and outflow from the salt marsh, and its relationship with t

105 st retigabine nearly abolished sensory nerve outflow from the urinary bladder during bladder filling.

106 we show that the distribution of population outflow from Wuhan accurately predicts the relative freq

107 d the growth pattern based on the population outflow from Wuhan; the model yields a benchmark trend a

108 Here we report observations of molecular gas outflowing from the centre of our Galaxy.

109 accompanied GW170817 identified(5,6) delayed outflows from a remnant accretion disk formed around the

110 spectrum(2,3), indicating broadly collimated outflows from the centre, directed perpendicular to the

111 h differing responsibilities ensures healthy outflow function and IOP maintenance.

112 e examined the role of LOXL1 in conventional outflow function, the prime regulator of intraocular pre

113 resented with an increased echocardiographic outflow gradient (1 mixed lesion with moderate/severe pu

114 February to April) and MeHg export from the outflow (greatest from September to December) of the HCC

115 he placenta, which is important because this outflow has been largely neglected in the past.

116 oab, Utah, wastewater treatment plant (WWTP) outflow has detected pharmaceuticals, hormones, and estr

117 es (MMPs) contribute to conventional aqueous outflow homeostasis in their capacity to remodel extrace

118 ery induces a reflex increase in sympathetic outflow; however, this has not been examined in humans.

119 ic neurons and for the augmented sympathetic outflow in hypertension.

120 led assessment and quantification of aqueous outflow in real time.

121 r matrix and increased resistance to aqueous outflow in the absence of PrP(C).

122 Multiphase gas has been observed within this outflow, including hot highly ionized(3,4) (temperatures

123 w-induced deformation of the cell blocks the outflow, interrupting (choking) the flow.

124 measurement of the dynamics of human aqueous outflow is difficult to achieve with currently available

125 of the explored environment, and hippocampal outflow is necessary to organize this phenomenon.SIGNIFI

126 o suggest that the mass of the molecular gas outflow is not negligible and could affect the rate of s

127 This multi-phase outflow is probably driven by bursts of star formation,

128 In addition, a persistent radial outflow is seen at the outer edge of the disk that is po

129 for star formation occurring within galactic outflows is still missing.

130 tions of what the reaction rate in supernova outflows is, and how changes affect nucleosynthesis pred

131 lack holes of stellar mass launch collimated outflows (jets) of ionized matter that approach the spee

132 MeHg) and loads at four reservoir inflow and outflow locations through the HCC (2014-2017).

133 been performed, and the results confirm the outflow mechanism in remarkable agreement with simulatio

134 echnique to image and quantify human aqueous outflow noninvasively and in real time.

135 e, which may suggest corticosteroid-mediated outflow obstruction distal to the trabecular meshwork.

136 nuity could predispose to the development of outflow obstruction in young patients with sarcomere mut

137 liocaval venous territory, leading to venous outflow obstruction.

138 diversity amongst the non-uniform inflow and outflow of 'climate migrants'.

139 m and ram2 suggested that FatM increases the outflow of 16:0 fatty acids from the plastid, for subseq

140 dy offers a fundamental understanding of the outflow of confined liquid from hydrophobic nanopores, p

141 with dynamic in vivo imaging that routes of outflow of CSF in mice occur along cranial nerves to ext

142 of olfactory sensory nerves greatly reduced outflow of CSF through the cribriform plate.

143 Outflow of CSF tracers from the spinal subarachnoid spac

144 ction in the brain is balanced by a constant outflow of CSF, the anatomical basis of which is poorly

145 of its floating ice shelves has allowed the outflow of grounded ice to accelerate(1,2).

146 version, yet is challenged by the incomplete outflow of intruded liquid out of nanopores for the syst

147 as two opposing fronts, suggesting a bipolar outflow of material from TYC 2597-735-1.

148 core of cognitive-motor control because the outflow of parietofrontal signaling is conveyed to the s

149 ed that they were broadly consistent with an outflow of radioactive heavy elements; however, there wa

150 carbons via the boundary currents and a net outflow of shorter-chain homologues.

151 The outflow of these pathways is coordinated by various cent

152 These grains condensed in outflows of asymptotic giant branch stars <4.9 Ga ago th

153 e Ga-Cd range in proton-rich neutrino-driven outflows of core-collapse supernovae(3-5).

154 ctions by state and incorporates inflows and outflows of interstate travelers.

155 Evidence has mounted in recent decades that outflows of matter and energy from the central few parse

156 ronic statin therapy alters skin sympathetic outflow or its relation to cutaneous vascular conductanc

157 nal evidence for star formation triggered by outflows or jets into their host galaxy, as a consequenc

158 ite-induced reflex influences in sympathetic outflow originating from the diaphragm are attenuated in

159 e haemorrhagic group regarding the number of outflows (p = 0.459) and largest outflow diameter (p = 0

160 natomy and physiological function of the CSF outflow pathway along the olfactory sensory nerves throu

161 ulator of TEK signaling in the aqueous humor outflow pathway and identify a new therapeutic target fo

162 stance-generating region of the conventional outflow pathway and locally liberates a controlled dose

163 ance to its outflow through the conventional outflow pathway comprising the trabecular meshwork (TM)

164 The conventional outflow pathway is a complex tissue responsible for main

165 ification of conventional and unconventional outflow pathway structures responsible for IOP homeostas

166 n of fibrillary material in the conventional outflow pathway.

167 ary structural component of the conventional outflow pathway.

168 which together correspond to the uveoscleral outflow pathway.

169 xhibits graded neural damage with unimpaired outflow pathways.

170 The outflow PCO2 was similar regardless of inflow PCO2 and e

171 ered ECM repair/homeostasis and conventional outflow physiology.

172 luminal tissue thickening at the inflow and outflow portion of the frame with no significant alterat

173 These outflows power prompt, brief and intense flashes of gamm

174 During the afterglow phase, the shocked outflow-produced by the interaction between the ejected

175 ealed a potentially-significant finding that outflow properties of rat eyes do not remodel in respons

176 However, the mass outflow rates are found to be insufficient to balance th

177 s 1.36+/-0.15, P<0.0001) and lower RV inflow/outflow ratio (P<0.001), as compared to men.

178 luenced by Mississippi and Atchafalaya River outflow, remained fairly stable over this time period.

179 The cause of the elevated outflow resistance and consequent ocular hypertension ch

180 trices, which has a direct impact on aqueous outflow resistance and IOP.

181 entional outflow facility (increases aqueous outflow resistance) of rat eyes.

182 ct the health of the trabecular meshwork and outflow resistance.

183 Purpose To investigate whether sinovenous outflow restriction (SOR) is more strongly associated wi

184 This excludes the isotropic outflow scenario, which would have produced a larger app

185 of 3726 and 3729 angstroms reveal an ionized outflow spanning 80 by 100 square kiloparsecs, depositin

186 by developmental defects in 2 aqueous humor outflow structures, Schlemm's canal (SC) and the trabecu

187 cytoskeletal dynamics and pressure-dependent outflow suggests potential for a novel therapeutic targe

188 the main Arctic gateway where 80% of in- and outflow takes place), the North Atlantic Waters transpor

189 esponses to increasing sympathetic vasomotor outflow, termed sympathetic neurovascular transduction (

190 NH(4)(+) and DOC, but a source of DIC to its outflow the Ruggles River.

191 that in proton-rich core-collapse supernova outflows, these hitherto neglected processes enhance the

192 ies reduce IOP by facilitating aqueous humor outflow through a vent fashioned from the wall of the ey

193 processes and hydrodynamic resistance to its outflow through the conventional outflow pathway compris

194 sure (IOP) due to insufficient aqueous humor outflow through the trabecular meshwork and Schlemm's ca

195 mprehensive cell atlas of human conventional outflow tissues.

196 lateral medulla (RVLM) determine sympathetic outflow to different territories of the body.

197 umn and reveal the major spinal pathways for outflow to lymphatic vessels in mice.

198 nner core designed to modulate aqueous humor outflow to provide immediate IOP reduction, prevent post

199 ion is associated with increased sympathetic outflow to the bone marrow, we hypothesized that sympath

200 bladder function by blocking afferent nerve outflow to the brain, which is key to sensing bladder fu

201 rial baroreceptors reduced basal sympathetic outflow to the skeletal muscle vasculature and reset vas

202 into surgeries that increase the trabecular outflow [Trabectome, iStent (first and second generation

203 tissues from HCM patients exhibiting severe outflow track obstruction (n = 16) compared to nonfailin

204 driveline path (87%), pump pocket (49%), and outflow tract (58%).

205 driveline path (87%), pump pocket (49%) and outflow tract (58%).

206 hteen percent had a resting left ventricular outflow tract (LVOT) gradient >=30 mm Hg.

207 more likely to have dynamic left ventricular outflow tract (LVOT) obstruction (63.3% vs 36.7%, P = 0.

208 Left ventricular outflow tract (LVOT) obstruction is a leading cause of m

209 ajor determinant of dynamic left ventricular outflow tract (LVOT) obstruction.

210 e replacement that prevents left ventricular outflow tract (LVOT) obstruction.

211 cine imaging was performed in short-axis, LV outflow tract (LVOT), and two-, three-, and four-chamber

212 iption factor Nkx2-5 is essential for normal outflow tract (OFT) and right ventricle (RV) development

213 sensitive pathways involved during zebrafish outflow tract (OFT) valve development in vivo.

214 and form during the assembly of the cardiac outflow tract (OFT), a crucial connection between the he

215 mouse identifies common progenitors for the outflow tract (OFT), LV, atrium and SV but not the right

216 The development of the cardiac outflow tract (OFT), which connects the heart to the gre

217 strict the size of the later-differentiating outflow tract (OFT).

218 ve rise to the right ventricle and primitive outflow tract (OFT).

219 o an underdeveloped right ventricle (RV) and outflow tract (OFT).

220 ght ventricle (P=0.037) and left ventricular outflow tract (P<0.001) and higher in left ventricle-rig

221 ioprosthesis (30%), native right ventricular outflow tract (RVOT) (27%) and other (2%).

222 l and according to type of right ventricular outflow tract (RVOT) anatomy.

223 rhythmias originate from the right ventricle outflow tract (RVOT).

224 cardiovascular malformations that affect the outflow tract and aortic arch arteries with failure of t

225 e arterial and venous poles, leading to both outflow tract and atrial septation defects that can be r

226 e, we were able to detail defects in cardiac outflow tract and valve development associated with Pitx

227 >=3 mm and the presence of left ventricular outflow tract calcifications under the left coronary cus

228 cemaker dependency included left ventricular outflow tract calcifications under the left coronary cus

229 MAC, AoV, and left ventricular outflow tract calcium (Ca++) scores were quantitated fro

230 tors(2,3) identified Hand2 as a specifier of outflow tract cells but not right ventricular cells, des

231 Damaging GATA6 variants cause cardiac outflow tract defects, sometimes with pancreatic and dia

232 TOF in 22q11.2DS and may function in cardiac outflow tract development.

233 a mean (SD) follow-up of 6.4 (2.5) years, RV outflow tract dimension increased from 35 mm (interquart

234 The RV size was determined by RV outflow tract dimension, and RV and left ventricular (LV

235 an important treatment of right ventricular outflow tract dysfunction.

236 The 3-year mean right ventricular outflow tract echocardiographic gradient was 15.7+/-5.5

237 HAND2, and KDR that with HAND2 orchestrates outflow tract formation.

238 , mitral gradient >6 mm Hg, left ventricular outflow tract gradient >20 mm Hg) at 30 days.

239 Unlike in adults, left ventricular outflow tract gradient had an inverse association, and f

240 rial diameter z score, peak left ventricular outflow tract gradient, and presence of a pathogenic var

241 end points include change in postexercise LV outflow tract gradient, New York Heart Association class

242 locity-time integral of the left ventricular outflow tract greater than or equal to 10% during the te

243 diac vein in 9 patients and in the apical LV outflow tract in 2.

244 is of Hand2-null embryos revealed failure of outflow tract myocardium specification, whereas right ve

245 sease in adult survivors of left ventricular outflow tract obstruction (n = 164) and ASD (n = 223), w

246 Right ventricular (RV) outflow tract obstruction (RVOTO) was demonstrated to be

247 ling for their higher burden of symptoms and outflow tract obstruction at baseline, reduced ejection

248 r Mitral Leaflet to Prevent Left Ventricular Outflow Tract Obstruction During Transcatheter Mitral Va

249 In 1 patient, left ventricular outflow tract obstruction occurred due to malrotation of

250 Left ventricular outflow tract obstruction occurred more frequently with

251 ated with various types of right ventricular outflow tract obstruction ranging from infundibular narr

252 One delayed left ventricular outflow tract obstruction required elective surgical mit

253 r presence and mechanism of left ventricular outflow tract obstruction, and risk stratification for s

254 arction, rehospitalization, left ventricular outflow tract obstruction, device migration, embolizatio

255 lvic, ureteric strictures, stenosis, urinary outflow tract obstruction, hydroureter, hydronephrosis,

256 ac myosin, which has been shown to reduce LV outflow tract obstruction, improve exercise capacity, an

257 and risk for either ASD or left ventricular outflow tract obstruction, with effect sizes similar to

258 (LV) hypertrophy associated with dynamic LV outflow tract obstruction.

259 There was no left ventricular outflow tract obstruction.

260 hy to verapamil in managing left ventricular outflow tract obstruction.

261 en and exercise capacity through reducing LV outflow tract obstruction.

262 ilated without evidence of right ventricular outflow tract obstruction.

263 no clinically significant right ventricular outflow tract obstruction.

264 pears to emerge specifically in the proximal outflow tract of human embryonic hearts and thereafter p

265 caused by anatomical blockage of the bladder outflow tract or by functional impairment of urinary voi

266 In patients referred for left ventricle outflow tract premature ventricular contraction ablation

267 ght consecutive patients with left ventricle outflow tract premature ventricular contraction were inc

268 n evaluated in response to right ventricular outflow tract PVCs with fixed short, fixed long, and var

269 ntractions originating in the left ventricle outflow tract represent a significant subgroup of patien

270 ibutes to critical structures, including the outflow tract septum.

271 index (DI)-the ratio of the left ventricular outflow tract time-velocity integral to that of the aort

272 ed absence of cardiac looping, a constricted outflow tract, and no cardiac jelly.

273 y run in parallel along the left ventricular outflow tract, but in the Nkx2-5(+/-)/Sspn(KO) mutant th

274 slet1-Cre transgene expressed in the cardiac outflow tract, right ventricle and atrium, pharyngeal me

275 oping embryo to give rise to portions of the outflow tract, the valves and the arteries of the heart.

276 pattern that was from the basal to apical LV outflow tract.

277 al LVS was earlier than that in the basal LV outflow tract.

278 the great cardiac vein and left ventricular outflow tract.

279 ols for parasternal long-axis view of the RV outflow tract.

280 inear heart tube, resulting in a constricted outflow tract. Furthermore, mutants lacked blood flow an

281 m the right ventricular and left ventricular outflow tracts (OTs).

282 atients with dysfunctional right ventricular outflow tracts.

283 in patients with repaired right ventricular outflow tracts.

284 Inhibition of global sympathetic outflow using Gi-coupled DREADD (designer receptor exclu

285 (CTA) revealed a large venous aneurysm as an outflow vein of a right-sided pelvic AVM.

286 Venous neointimal hyperplasia (VNH) at the outflow vein of hemodialysis AVF is a major factor contr

287 ion in cell density and proliferation in the outflow veins treated with CorMatrix compared to control

288 In outflow veins treated with CorMatrix, there was an incre

289 Embolisation of the outflow veins was established along with direct percutan

290 eptor hyperactivity and elevated sympathetic outflow via PKC in hypertension.

291 graphic outflow facility nor the uveoscleral outflow was significantly different from baseline.

292 Results Sinovenous outflow was significantly more restrictive (lower median

293 Average outflow water volume from farms was high (60,000 L/day),

294 (PFHpA) to perfluorononanoic acid (PFNA) in outflowing water from the Arctic suggests a higher contr

295 f retigabine altered ex vivo bladder sensory outflow, we measured afferent activity during simulated

296 xports of MeHg and THg out the Ruggles River outflow were consequently very low, but erosion and perm

297 rganic N (DIN) mass reductions (inflow minus outflow) were greater in the Dry Basin than in the Wet B

298 odulated the trabecular component of aqueous outflow whereas another channel, TRPV4, mediated a delay

299 re disrupts normal regulation of sympathetic outflow with effects on cardiovascular parameters.

300 approximately 0.03 solar masses) wind-driven outflow with moderate electron fraction (Ye approximatel