ライフサイエンスコーパス: outflow tract

1 delay in the anterolateral right ventricular outflow tract.

2 displacement into the left ventricular (LV) outflow tract.

3 al arches and the septation of the heart and outflow tract.

4 ling can substitute for the influence of the outflow tract.

5 with a striking increase in the size of the outflow tract.

6 VEGF-C to stimulate vessel growth around the outflow tract.

7 V internal dimension were measured in the RV outflow tract.

8 ied the focal origin in the left ventricular outflow tract.

9 evelops from the slowly conducting embryonic outflow tract.

10 the AV endocardial cushions and the cardiac outflow tract.

11 ions in the pharyngeal apparatus and cardiac outflow tract.

12 particular, for the formation of the cardiac outflow tract.

13 ond heart field cells that contribute to the outflow tract.

14 pattern that was from the basal to apical LV outflow tract.

15 al LVS was earlier than that in the basal LV outflow tract.

16 the great cardiac vein and left ventricular outflow tract.

17 ols for parasternal long-axis view of the RV outflow tract.

18 locity-time integral of the left ventricular outflow tract.

19 in patients with repaired right ventricular outflow tracts.

20 atients with dysfunctional right ventricular outflow tracts.

21 ginally smaller in BAs than in WAs (proximal outflow tract, 30.9+/-5.5 versus 32.8+/-5.3 mm, P<0.001;

22 driveline path (87%), pump pocket (49%), and outflow tract (58%).

23 driveline path (87%), pump pocket (49%) and outflow tract (58%).

24 henotypes, including aortic arch and cardiac outflow tract abnormalities.

25 versus 0 mV); (2) delayed right ventricular outflow tract activation (82+/-18 versus 37+/-11 ms); (3

26 ryological context for understanding cardiac outflow tract alignment and membranous ventricular septa

27 lesion, supporting the idea that AHF-derived outflow tract alignment defects may constitute an embryo

28 function in the AHF results in a spectrum of outflow tract alignment defects ranging from overriding

29 Moreover, we found a range of outflow tract alignment defects resulting from a single

30 ng premature ventricular contractions of the outflow tract alternating with papillary muscle or fasci

31 entricular contractions originating from the outflow tract alternating with the papillary muscle or f

32 cardiovascular malformations that affect the outflow tract and aortic arch arteries with failure of t

33 -CMR data analysis included left ventricular outflow tract and aortic valve segmentation, and extract

34 ht ventricular growth, and interventricular, outflow tract and aortico-pulmonary septation.

35 e arterial and venous poles, leading to both outflow tract and atrial septation defects that can be r

36 the second heart field, pharyngeal endoderm, outflow tract and atrioventricular endocardial cushions

37 ct was the most common CHD observed, whereas outflow tract and atrioventricular septal defects were t

38 myocardial movements that are essential for outflow tract and atrioventricular septation.

39 uber ciliopathy syndromes, including cardiac outflow tract and cochlea defects associated with PCP pe

40 erning of structures formed from NCC such as outflow tract and cranial nerves.

41 acity, regulated by the neighbouring cardiac outflow tract and Hh signalling.

42 ritruncal blood vessels encircle the cardiac outflow tract and invade the aorta, but the underlying p

43 preventing MV systolic displacement into the outflow tract and outflow obstruction.

44 scar most commonly in the right ventricular outflow tract and right ventricle basal regions.

45 age-sensitive role in the differentiation of outflow tract and right ventricle from progenitors of th

46 r region is known to be destined to form the outflow tract and right ventricle.

47 second heart field (AHF), gives rise to the outflow tract and the majority of the right ventricle an

48 e, we were able to detail defects in cardiac outflow tract and valve development associated with Pitx

49 regulates the signaling processes leading to outflow tract and valve morphogenesis and ventricular tr

50 nd the most common areas are the ventricular outflow tracts and left ventricular fascicles.

51 tracellular matrix homeostasis in HDAC3-null outflow tracts and semilunar valves, and pharmacological

52 genetic silencing of Tgf-beta1 in HDAC3-null outflow tracts and semilunar valves.

53 hose that affect the proper alignment of the outflow tracts and septation of the ventricles are a hig

54 ns (3087 [50%] ventricular bodies, 756 [12%] outflow tract, and 162 [3%] epicardial).

55 om the epicardial right ventricular apex, RV outflow tract, and LV free wall, as well as premature at

56 ed absence of cardiac looping, a constricted outflow tract, and no cardiac jelly.

57 tis, in situ stents in the right ventricular outflow tract, and presence of outflow tract irregularit

58 pression with simultaneous right ventricular outflow tract angioplasty and CA angiography.

59 Outflow tract anomalies identified by micro-CT included

60 l was used for soft tissue structures of the outflow tract, aortic root, and noncalcified valve cusps

61 tive primary repair; their right ventricular outflow tracts are characterized by mild residual obstru

62 tants are of aberrant shape, and the cardiac outflow tracts are short and malformed.

63 LV outflow tract area, indexed stroke volume, and AVA corre

64 Thirty-two patients with idiopathic RV outflow tract arrhythmias and 32 control subjects, match

65 nts with ARVC compared with patients with RV outflow tract arrhythmias and controls.

66 -24 versus 7+/-5 ms/cm) at right ventricular outflow tract borders.

67 y run in parallel along the left ventricular outflow tract, but in the Nkx2-5(+/-)/Sspn(KO) mutant th

68 >=3 mm and the presence of left ventricular outflow tract calcifications under the left coronary cus

69 cemaker dependency included left ventricular outflow tract calcifications under the left coronary cus

70 MAC, AoV, and left ventricular outflow tract calcium (Ca++) scores were quantitated fro

71 tors(2,3) identified Hand2 as a specifier of outflow tract cells but not right ventricular cells, des

72 arteriosus in basal actinopterygians, to an outflow tract commanded by the non-valved, elastic, bulb

73 o difference in E12 in the right ventricular outflow tract compared with the right-left ventricular o

74 In patients with right ventricular outflow tract conduit dysfunction, TPV replacement is as

75 ed as a viable therapy for right ventricular outflow tract conduit dysfunction.

76 function in patients with right ventricular outflow tract conduit dysfunction; the impact of this te

77 median age, 19 years) with right ventricular outflow tract conduit obstruction or regurgitation.

78 implantation in obstructed right ventricular outflow tract conduits in 2010 after a multicenter trial

79 treatment of dysfunctional right ventricular outflow tract conduits in patients >/=30 kg.

80 apy for dysfunctional right ventricular (RV) outflow tract conduits.

81 5 mutation is strongly associated with human outflow tract congenital heart disease (OFT CHD).

82 Surgical right ventricular outflow tract cryoablation was performed in 22 patients

83 sistent truncus arteriosus, a severe cardiac outflow tract defect also seen in human congenital heart

84 ma, inner and outer ear malformations, heart outflow tract defects and craniofacial defects.

85 Damaging GATA6 variants cause cardiac outflow tract defects, sometimes with pancreatic and dia

86 ls that some Isl1 derivatives in the cardiac outflow tract derive from Wnt1-expressing neural crest p

87 All outflow tract-destined cells are intermingled with those

88 e myocyte cell-cell adhesions during cardiac outflow tract development contributes to impaired outflo

89 hich is required for aortic arch and cardiac outflow tract development, and is a known genetic intera

90 TOF in 22q11.2DS and may function in cardiac outflow tract development.

91 (HR, 0.94 per 1%; P=0.02), right ventricular outflow tract diameter (HR, 1.08 per 1 mm; P=0.01), mitr

92 a mean (SD) follow-up of 6.4 (2.5) years, RV outflow tract dimension increased from 35 mm (interquart

93 The RV size was determined by RV outflow tract dimension, and RV and left ventricular (LV

94 myofibres and collagen fibres to the apex-to-outflow-tract direction was consistent with this also be

95 ution involves the transition from a cardiac outflow tract dominated by a multi-valved conus arterios

96 nsformative technology for right ventricular outflow tract dysfunction with the potential to expand t

97 an important treatment of right ventricular outflow tract dysfunction.

98 disease patient with right ventricular (RV) outflow tract dysfunction.

99 treatment of postoperative right ventricular outflow tract dysfunction.

100 The 3-year mean right ventricular outflow tract echocardiographic gradient was 15.7+/-5.5

101 lation without wall motion abnormalities; RV outflow tract ectopy; and exercise-induced T-wave pseudo

102 inding EGF-like growth factor to Jag1-mutant outflow tract explant cultures rescued the hyperprolifer

103 Very few Islet-1(+) cells within the outflow tract expressed the cardiomyocyte marker alpha-a

104 gle device or with strategies to prepare the outflow tract for subsequent device deployment.

105 in gestation and display defects in cardiac outflow tract formation, atrial and ventricular septatio

106 HAND2, and KDR that with HAND2 orchestrates outflow tract formation.

107 ate atrioventricular canal morphogenesis and outflow tract formation.

108 inear heart tube, resulting in a constricted outflow tract. Furthermore, mutants lacked blood flow an

109 gitation, and 4 had a mean right ventricular outflow tract gradient >/=30 mm Hg.

110 A preoperative left ventricular outflow tract gradient >/=80 mm Hg was a predictor for p

111 , mitral gradient >6 mm Hg, left ventricular outflow tract gradient >20 mm Hg) at 30 days.

112 essure ratio (P<0.001) and right ventricular outflow tract gradient (P=0.004) than those with no tear

113 RV T1 correlated inversely with RV outflow tract gradient (r=-0.28, P=0.02).

114 Residual left ventricular outflow tract gradient after ablation was an independent

115 h significant reduction in right ventricular outflow tract gradient and the RV:Ao ratio when compared

116 g patients with significant (>/=30 mm Hg) RV outflow tract gradient and/or other residual hemodynamic

117 The peak instantaneous left ventricular outflow tract gradient decreased from 75.7+/-28.0 mm Hg

118 Unlike in adults, left ventricular outflow tract gradient had an inverse association, and f

119 ce interval, 1.02-2.30) and left ventricular outflow tract gradient progression (hazard ratio, 1.45;

120 IVSd was not related to left ventricular outflow tract gradient reduction at rest (P=0.883) or du

121 Over time, the left ventricular outflow tract gradient slowly increases and mild aortic

122 estent and lower discharge right ventricular outflow tract gradient were associated with longer freed

123 rial diameter z score, peak left ventricular outflow tract gradient, and presence of a pathogenic var

124 end points include change in postexercise LV outflow tract gradient, New York Heart Association class

125 zation data in 162 consecutive patients with outflow tract gradients (median [interquartile range], 9

126 ressure ratio (P=0.02) and right ventricular outflow tract gradients (P</=0.001).

127 ASA had equal effects on left ventricular outflow tract gradients and symptoms throughout the spec

128 Left ventricular outflow tract gradients are absent in an important propo

129 locity-time integral of the left ventricular outflow tract greater than or equal to 10% during the te

130 iomyopathy was frequently observed (proximal outflow tract >/=32 mm; 45.0% of BAs, 58.5% of WAs).

131 the anterior subepicardial right ventricular outflow tract in 11 patients (group B).

132 diac vein in 9 patients and in the apical LV outflow tract in 2.

133 the derived, monovalvar, bulbar state of the outflow tract in modern actinopterygians.

134 rograms recorded in the epicardium of the RV outflow tract in patients with BrS.

135 t ventricular outflow tract, and presence of outflow tract irregularities at the implant site were as

136 enting of the ventricular septum or systemic outflow tract is feasible and effective in the short ter

137 As a result, full blood momentum in the outflow tract is used to facilitate early ejection.

138 Pacing from the RV outflow tract/lateral RV predicted significantly decreas

139 lmonary left ventricle, and left ventricular outflow tract (LVOT) conduit dysfunction has not been st

140 Cardiac left ventricular outflow tract (LVOT) defects represent a common but hete

141 hteen percent had a resting left ventricular outflow tract (LVOT) gradient >=30 mm Hg.

142 more likely to have dynamic left ventricular outflow tract (LVOT) obstruction (63.3% vs 36.7%, P = 0.

143 e mitral valve could reduce left ventricular outflow tract (LVOT) obstruction and associated mitral r

144 Left ventricular outflow tract (LVOT) obstruction is a leading cause of m

145 rtrophic cardiomyopathy and left ventricular outflow tract (LVOT) obstruction, but without basal sept

146 ajor determinant of dynamic left ventricular outflow tract (LVOT) obstruction.

147 e replacement that prevents left ventricular outflow tract (LVOT) obstruction.

148 (VAs) originating from the left ventricular outflow tract (LVOT) sometimes require catheter ablation

149 (VAs) originating from the left ventricular outflow tract (LVOT) sometimes require catheter ablation

150 (VAs) originating from the left ventricular outflow tract (LVOT), an alternative approach from the a

151 cine imaging was performed in short-axis, LV outflow tract (LVOT), and two-, three-, and four-chamber

152 ve patients who experienced left ventricular outflow tract (LVOT)/annular/aortic contained/noncontain

153 associated with ascending aortic dilatation, outflow tract malrotation, overriding aorta, double outl

154 activity in the epicardial right ventricular outflow tract may be beneficial in patients with Brugada

155 ow tract development contributes to impaired outflow tract myocardialization and displacement of the

156 ore, impaired NMII-B motor activity inhibits outflow tract myocardialization, leading to mislocalizat

157 is of Hand2-null embryos revealed failure of outflow tract myocardium specification, whereas right ve

158 the free wall of the right ventricular (RV) outflow tract (n=8), lateral RV (n=44), RV apex (n=61),

159 . 24 +/- 6 mm; p < 0.001) and less prevalent outflow tract obstruction (19% vs. 34%; p = 0.015); 2) h

160 thy with severe symptomatic left ventricular outflow tract obstruction (47+/-11 years, 63% male) intr

161 However, left ventricular outflow tract obstruction (LVOTO) has been traditionally

162 y, there was mild residual right ventricular outflow tract obstruction (mean gradient, 24+/-13 mm Hg)

163 sease in adult survivors of left ventricular outflow tract obstruction (n = 164) and ASD (n = 223), w

164 tions, including relief of right ventricular outflow tract obstruction (n=5), pulmonary arterioplasty

165 the use of paroxetine and right ventricular outflow tract obstruction (relative risk, 1.07; 95% CI,

166 Right ventricular (RV) outflow tract obstruction (RVOTO) was demonstrated to be

167 between paroxetine use and right ventricular outflow tract obstruction and between sertraline use and

168 ntly accompanied by dynamic left ventricular outflow tract obstruction and symptoms of dyspnea, angin

169 ling for their higher burden of symptoms and outflow tract obstruction at baseline, reduced ejection

170 sk of Ebstein's anomaly (a right ventricular outflow tract obstruction defect) in infants and overall

171 The prevalence of right ventricular outflow tract obstruction defects was 0.60% among lithiu

172 r Mitral Leaflet to Prevent Left Ventricular Outflow Tract Obstruction During Transcatheter Mitral Va

173 Patients with severe left ventricular outflow tract obstruction had a bisferiens pressure wave

174 g surgery for the relief of left ventricular outflow tract obstruction have low event rates during lo

175 ssociation of symptoms with left ventricular outflow tract obstruction in HCM, there exist paradoxica

176 tral valve was discovered as the cause of LV outflow tract obstruction in the M-mode echocardiography

177 whereas proximally, severe left ventricular outflow tract obstruction is associated with an addition

178 Left ventricular outflow tract obstruction is present at rest in about on

179 ern was evident, which is associated with LV outflow tract obstruction loss and right ventricle systo

180 In 1 patient, left ventricular outflow tract obstruction occurred due to malrotation of

181 Left ventricular outflow tract obstruction occurred more frequently with

182 mic pulmonary hypertension, left ventricular outflow tract obstruction or dilated cardiomyopathy.

183 ated with various types of right ventricular outflow tract obstruction ranging from infundibular narr

184 One delayed left ventricular outflow tract obstruction required elective surgical mit

185 ands were younger with less left ventricular outflow tract obstruction than G- probands, however, had

186 ntry, including 249 in whom left ventricular outflow tract obstruction was absent both at rest and fo

187 r presence and mechanism of left ventricular outflow tract obstruction, and risk stratification for s

188 arction, rehospitalization, left ventricular outflow tract obstruction, device migration, embolizatio

189 lvic, ureteric strictures, stenosis, urinary outflow tract obstruction, hydroureter, hydronephrosis,

190 ac myosin, which has been shown to reduce LV outflow tract obstruction, improve exercise capacity, an

191 ly seen in association with left ventricular outflow tract obstruction, itself part of a spectrum of

192 and risk for either ASD or left ventricular outflow tract obstruction, with effect sizes similar to

193 en and exercise capacity through reducing LV outflow tract obstruction.

194 ilated without evidence of right ventricular outflow tract obstruction.

195 ansvalvular gradient and no left ventricular outflow tract obstruction.

196 for right ventricular dilation and residual outflow tract obstruction.

197 for the surgical relief of left ventricular outflow tract obstruction.

198 dergoing surgical relief of left ventricular outflow tract obstruction.

199 no clinically significant right ventricular outflow tract obstruction.

200 ce of death due to electric abnormalities or outflow tract obstruction.

201 iograms were evaluated for right ventricular outflow tract obstruction.

202 There was no left ventricular outflow tract obstruction.

203 (LV) hypertrophy associated with dynamic LV outflow tract obstruction.

204 hy to verapamil in managing left ventricular outflow tract obstruction.

205 95% CI 3.60-25.91%), while left ventricular outflow tract obstruction/mid-ventricular obstruction (L

206 Associations between left ventricular outflow tract obstructions and nitrogen dioxide and betw

207 strategy consisting of relief of inflow and outflow tract obstructions, resection of endocardial fib

208 l defects, conotruncal, and left ventricular outflow tract obstructive lesions are underway.

209 ization are present in the right ventricular outflow tract of BrS patients.

210 pears to emerge specifically in the proximal outflow tract of human embryonic hearts and thereafter p

211 d connect the dorsal head vasculature to the outflow tract of the heart.

212 ally as the floor of the mandibular arch and outflow tract of the heart.

213 niofacial structures, pigment cells, and the outflow tract of the heart.

214 red for patterning of the great arteries and outflow tract of the heart.

215 ifferentiation, cardiomyocyte proliferation, outflow tract (OFT) and atrioventricular septation, and

216 iption factor Nkx2-5 is essential for normal outflow tract (OFT) and right ventricle (RV) development

217 uding cells in the atrioventricular (AV) and outflow tract (OFT) cushions.

218 play key roles in development of the cardiac outflow tract (OFT) for establishment of completely sepa

219 vious genetic studies in mice indicated that outflow tract (OFT) formation requires Dvl1 and 2, but i

220 Defects of the outflow tract (OFT) make up a large percentage of human

221 Outflow tract (OFT) malformation accounts for approximat

222 ecades, the mechanisms underlying RA-induced outflow tract (OFT) malformations are not understood.

223 In mammals, the outflow tract (OFT) of the developing heart septates int

224 pharyngeal arch arteries (PAAs). and cardiac outflow tract (OFT) requires multipotent neural crest ce

225 sensitive pathways involved during zebrafish outflow tract (OFT) valve development in vivo.

226 and form during the assembly of the cardiac outflow tract (OFT), a crucial connection between the he

227 mouse identifies common progenitors for the outflow tract (OFT), LV, atrium and SV but not the right

228 The development of the cardiac outflow tract (OFT), which connects the heart to the gre

229 ular myocardium and in three lineages in the outflow tract (OFT).

230 strict the size of the later-differentiating outflow tract (OFT).

231 ve rise to the right ventricle and primitive outflow tract (OFT).

232 o an underdeveloped right ventricle (RV) and outflow tract (OFT).

233 Flow velocities in the LV outflow tract on the pre-SAM frame 1 and 2 mm from the t

234 PC-CMR of aortic valve and left ventricular outflow tract on the same day.

235 caused by anatomical blockage of the bladder outflow tract or by functional impairment of urinary voi

236 The proximity of the outflow tracts (OTs) frequently results in an overlap in

237 m the right ventricular and left ventricular outflow tracts (OTs).

238 ght ventricle (P=0.037) and left ventricular outflow tract (P<0.001) and higher in left ventricle-rig

239 act compared with the right-left ventricular outflow tract (P=0.75) pairs.

240 plified continuity equation=left ventricular outflow tract peak flow rate/aortic peak velocity.

241 00 ventricular extrasystoles (or >500 non-RV outflow tract) per 24 h; and symptoms, ventricular tachy

242 lve replacement in dilated right ventricular outflow tracts, permitting lower risk, nonsurgical pulmo

243 ignaling is required for EMT in the proximal outflow tract (pOFT) but not atrioventricular canal (AVC

244 In patients referred for left ventricle outflow tract premature ventricular contraction ablation

245 ght consecutive patients with left ventricle outflow tract premature ventricular contraction were inc

246 Cranial pSHF cells also contribute to the outflow tract: proximal and distal at 4 somites, and dis

247 perior imaging of the right ventricular (RV) outflow tract, pulmonary arteries, aorta, and aortopulmo

248 n evaluated in response to right ventricular outflow tract PVCs with fixed short, fixed long, and var

249 ects in children requiring right ventricular outflow tract reconstruction typically involves multiple

250 Within the heart outflow tract, reduced proliferation of myocardial and e

251 conduction slowing in the right ventricular outflow tract region.

252 rs), 32 patients underwent right ventricular outflow tract reintervention for obstruction (n=27, with

253 ntractions originating in the left ventricle outflow tract represent a significant subgroup of patien

254 f the Melody TPV to patients with nonconduit outflow tracts (right ventricular outflow tract [RVOT])

255 slet1-Cre transgene expressed in the cardiac outflow tract, right ventricle and atrium, pharyngeal me

256 In hearts with outflow tract rotation defects, misplaced stems were ass

257 ioprosthesis (30%), native right ventricular outflow tract (RVOT) (27%) and other (2%).

258 l and according to type of right ventricular outflow tract (RVOT) anatomy.

259 placed epicardially on the right ventricular outflow tract (RVOT) before video-assisted thoracoscopic

260 Due to recurrent right ventricular outflow tract (RVOT) dysfunction, patients with complex

261 The shape of right ventricular outflow tract (RVOT) has been assumed to be circular.

262 atients with postoperative right ventricular outflow tract (RVOT) obstruction or pulmonary regurgitat

263 siological sequelae of the right ventricular outflow tract (RVOT) reconstruction.

264 and cardiomyopathy-related right ventricular outflow tract (RVOT) ventricular arrhythmias (VAs) is cr

265 he anterolateral region, 8 (17.7%) in the RV outflow tract (RVOT), and 8 (17.7%) in the apex.

266 imings across the right ventricle (RV) body, outflow tract (RVOT), and left ventricle were calculated

267 e usually localized to the right ventricular outflow tract (RVOT), presumably below the pulmonic valv

268 rhythmias originate from the right ventricle outflow tract (RVOT).

269 nonconduit outflow tracts (right ventricular outflow tract [RVOT]) has the potential to vastly expand

270 an isolated subepicardial right ventricular outflow tract scar serving as a substrate for fast VT in

271 Larger preoperative RV outflow tract scar was associated with a smaller improve

272 haryngeal arch artery remodeling and cardiac outflow tract septation during vertebrate development.

273 ibutes to critical structures, including the outflow tract septum.

274 The RV outflow tract, septum, and apex were mapped during left

275 diac phenotype (119-113 Ma) and suggest that outflow tract simplification in actinopterygians is comp

276 defects that are preceded by a reduction in outflow tract size and loss of caudal pharyngeal arch ar

277 or early primary repair by right ventricular outflow tract stenting (stent).

278 Right ventricular outflow tract stenting of symptomatic tetralogy of Fallo

279 ) (r = 0.880; p < 0.0001), right ventricular outflow tract stroke volume (r = 0.660; p < 0.0001), and

280 oping embryo to give rise to portions of the outflow tract, the valves and the arteries of the heart.

281 were placed into the right ventricular apex/outflow tract through a subclavian vein puncture with a

282 ng the left ventricle is reversed toward the outflow tract through rotating reversal flow around the

283 index (DI)-the ratio of the left ventricular outflow tract time-velocity integral to that of the aort

284 la: LVEI=indexed LV end-systolic diameter/LV outflow tract time-velocity integral.

285 the ventricular septum or subvalvar systemic outflow tract, using 1 of the following 3 delivery appro

286 as ([LA emptying fraction x left ventricular outflow tract-velocity time integral] / [indexed LA end-

287 dle branch block excluding right ventricular outflow tract ventricular tachycardia.

288 uctural heart disease, most left ventricular outflow tract ventricular tachycardias (VTs) have a foca

289 ent of specific anatomical structures (e.g., outflow tract, ventricular septum, and atrial septum) th

290 irect transcriptional target of MEF2C in the outflow tract via an AHF-restricted Tdgf1 enhancer.

291 maximal Doppler velocity in left ventricular outflow tract (VmaxAo) measured using either approach, a

292 hologies (MMs) of inducible left ventricular outflow tract VT may indicate a scar-related VT that can

293 patients referred for ablation of sustained outflow tract VT without overt structural heart disease,

294 tural heart disease, 24 had left ventricular outflow tract VT, 10 had MM VT, and 14 had a single VT (

295 l pole morphogenesis, identifying defects in outflow tract wall and cushion morphology that preceded

296 terior right free wall and right ventricular outflow tract, which increased after flecainide from 17.

297 erved that VEGF-C is widely expressed in the outflow tract, while cardiomyocytes develop specifically

298 that CXCL12 is present at high levels in the outflow tract, while peritruncal endothelial cells (ECs)

299 bserved exclusively in the right ventricular outflow tract with the following properties (in comparis

300 ection flow velocity in the left ventricular outflow tract, with consequent loss of flow momentum.