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1 bserved Ascaris counts (mean = -0.01 log(10) ova per wet gram of soil, sd = 0.71 log(10)) could be ex
2 trospective analysis of the results of 2,704 ova and parasite (O & P) examinations performed on stool
3 hat by females), sperm become less abundant, ova become relatively less rare, and competition between
4  using intranasal house dust mite or aerosol ova-albumin challenge, and chloroquine or quinine were t
5 e of inactivation increased until nearly all ova were nonviable by day 24.
6  stages until day 16, after which nearly all ova were observed to be nonviable.
7 ated changes in CRF1 expression in brain and ova of stressed female rats and in the brain of their ne
8 ls have started to replace stool culture and ova and parasite exam as a rapid and accurate means of d
9 re physically destroyed during digestion and ova can be inactivated faster if their development cycle
10 oung adult mice and absence of follicles and ova in older mice, along with interstitial stromal cell
11 showed moderate cellular immune reaction and ova (viable and/or calcified).
12 re unchanged in gynogenetic and androgenetic ova; the latter contain two maternal and two paternal ge
13 s: in individual blastomeres of androgenetic ova, both paternal Snrpn alleles were active (Snrpn was
14 et gram, sd = 1.1 log(10)), the high Ascaris ova counts in fecal sludges (prevalence = 88%; mean = 1.
15  result in a steady-state density of Ascaris ova in soils capable of sustaining transmission, given t
16 xtended persistence and viability of Ascaris ova in soils.
17 py to enumerate presumptively viable Ascaris ova in feces, fecal sludges, and soils from compounds (i
18            For internal exposure assessment, ova of parasites in stool samples was detected using wet
19 cell-mediated immunity to provide tumor (B16-ova-Melanoma) protection in around 40% of vaccinated mic
20  in all species with little (sperm) and big (ova) gamete producers.
21     Other obstetric SAE included 14 blighted ova, 9 stillbirths, 1 termination due to hydrops fetalis
22 ected into the perivitelline space of bovine ova.
23               Ovalbumin-specific Treg cells (ova-Tregs) were isolated from patients' peripheral blood
24 ransduced mice maintained stable circulating ova levels without evidence of an immune response.
25                                 In contrast, ova exposed to aerobic conditions did not become dormant
26 rmation of liver granulomas around deposited ova.
27     In addition, only 35% of fully developed ova exposed to the anaerobic environment retained their
28 ression of Snrpn and Igf2r in normal diploid ova was unchanged in individual blastomeres of triploid
29 bility by day 16 compared to 65% for dormant ova.
30 imens were positive for G. lamblia by ELISA, ova and parasite test, and/or direct fluorescent-antibod
31 cquired resistance impairs tick engorgement, ova production, and viability.
32                                     Ep2(-/-) ova could be fertilized in vitro, suggesting that in add
33 erm cells that can navigate to and fertilize ova.
34        The ability to successfully fertilize ova relies upon the swimming ability of spermatozoa.
35 ales will vary in their ability to fertilize ova on the basis of sperm viability alone.
36 esulting from culture of in vitro fertilized ova were transduced with a self-inactivating lentiviral
37 oratories (40% for stool culture and 45% for ova and parasite [O&P] examinations) had restrictions fo
38 s (determined by traditional examination for ova and parasites) preserved in ECOFIX compared to their
39 ultiple stool examinations were negative for ova and parasites.
40 ction of tumors expressing either a foreign (ova) antigen or fully syngeneic tumor antigens (on Panc0
41 tive (Snrpn was not expressed in gynogenetic ova), and in individual gynogenetic and androgenetic bla
42 eing present in the sperm that fertilize her ova.
43 affects CRF1 expression in brain but also in ova, pointing to a possible mechanism of transgeneration
44 creted transgene product (ovalbumin, ova) in ova-specific T-cell receptor (TCR) transgenic mice by he
45 normally present at high levels primarily in ova cytoplasm of developing ovarian follicles, and in th
46            As early as 90min post injection, ova-micelle conjugates were associated with 28% and 55%
47 to form granulomas in response to S. mansoni ova even after T cell activation, suggesting a requireme
48 ve studied the immune response to S. mansoni ova in Stat4- and Stat6-deficient mice, which lack Th1 a
49  IL-10 in animals inoculated with S. mansoni ova, larvae from the filarial helminth Brugia malayi, or
50 iated with the immune response to S. mansoni ova.
51 es of larval miracidia within the S. mansoni ova.
52       The development of Schistosoma mansoni ova-induced granulomas is regulated by cytokines secrete
53 -term selection ovulates on average 6.7 more ova than its randomly selected control line.
54 simultaneous birth of siblings when multiple ova are released, is an evolutionary paradox.
55 mice have impaired Th2 responses in a murine ova-induced asthma model, while Th1 responses are normal
56 MI (97/103) and MII (6/103) nondisjunctional ova were strikingly different.
57 ific expression of Snrpn and Igf2r in normal ova were unchanged in gynogenetic and androgenetic ova;
58 ion scenarios capable of explaining observed ova counts in soils.
59                         Approximately 65% of ova were able to retain their viability for up to 16 day
60             In particular, administration of ova from the pig whipworm Trichuris suis (T. suis; TSO)
61 e results of serologic tests or detection of ova) and 17 healthy controls were included in the study.
62  assessment methods supports the efficacy of ova-Tregs; this immune therapy approach warrants further
63 ingle male will compete for fertilization of ova with spermatozoa from another male when present in t
64 ered the "gold standard" for the fixation of ova and parasites in the preparation of permanently stai
65                                Injections of ova-Tregs were well tolerated, with 54 adverse events (2
66  supporting immune-suppressive mechanisms of ova-Tregs.
67 tinal inflammation and reduces the number of ova within the gut.
68 eated stool examinations for the presence of ova and parasites, in practice this test is not sensitiv
69 ic B cells (P = 0.003) and the production of ova-specific antibodies (P = 0.03) after immunization.
70 les must compete to fertilize a given set of ova.
71 were randomly assigned to receive placebo or ova treatment.
72 ization with the soluble antigen, ovalbumin (ova), administered i.p. or orally without adjuvant, acti
73  to a secreted transgene product (ovalbumin, ova) in ova-specific T-cell receptor (TCR) transgenic mi
74 hine evolution resulted in multiple ovulated ova with each cycle, and that these changes triggered ad
75 evere inflammation directed against parasite ova, whereas IL-4Ralpha expression by non-BM-derived cel
76  extent of hepatic damage caused by parasite ova during natural infection with Schistosoma mansoni, b
77 croscopy.IMPORTANCEGastrointestinal parasite ova and parasite (O&P) detection from stools is a manual
78 GF-beta mAbs had little effect upon parasite ova-induced intestinal pathology or development of alter
79 istosoma mansoni infection in which parasite ova induce granulomas whose cellular content is 50% eosi
80 osomiasis mansoni, schistosome worms produce ova that incite focal Th2-type granulomatous inflammatio
81         Following adoptive transfer into RIP-ova(high) recipients, PD-1(-/-) OT-I T cells expanded in
82 the control of the rat insulin promoter (RIP-ova(high)).
83 lls adoptively transferred into the same RIP-ova(high) recipient mouse.
84 , a recombinant Listeria expressing OVA (rLM-ova) was generated.
85 resolved discrepant results by using routine ova and parasite examination (O&P) and on immunofluoresc
86 ferent males compete to fertilize a female's ova, is a widespread and fundamental force in the evolut
87 r more males compete to fertilize a female's ova.
88 of being transmitted to each of the mother's ova and, therefore, would appear to have equal interests
89                                  Schistosoma ova were identified exclusively among African refugees a
90 normal granulomatous response to schistosome ova during the course of natural infection.
91 host cellular immune response to schistosome ova is a risk factor for urinary tract morbidity in area
92  local granulomatous response to schistosome ova nor the systemic response to soluble egg Ag switches
93                    Results suggest that some ova are physically destroyed during digestion and ova ca
94 RL were proportional to the numbers of sperm/ova and are indicative of the stage of the reproductive
95  specimens) that were tested by the standard ova and parasite (O&P) examination as the "gold standard
96 n stains, which are not part of the standard ova-and-parasite examination.
97 umber of unnecessary stool cultures (STCUL), ova/parasite (O&P) examinations, and Giardia/Cryptospori
98 overed STHs; and viability assessment of STH ova.
99 ion supports further investigation of T suis ova in patients with immune-mediated diseases, particula
100 okine responses were not affected by T. suis ova treatment.
101 ion of T. suis-specific cytokines in T. suis ova-treated participants, allergen-specific cytokine res
102 b-cohort of randomized participants (T. suis ova-treated, n = 12, Placebo-treated, n = 10).
103 ed three weekly doses of 2500 Trichuris suis ova (n = 45) or placebo (n = 44) over 6 months.
104                               Trichuris suis ova (TSO) have been tested for therapeutic application i
105 ess the safety or efficacy of Trichuris suis ova in allergies, inflammatory bowel diseases, multiple
106        Patients received 2500 Trichuris suis ova or placebo orally at 2-week intervals for 12 weeks.
107 sts that cytokines induced by Trichuris suis ova treatment do not alter allergic reactivity to pollen
108                               Trichuris suis ova were obtained from the US Department of Agriculture.
109     Eggs of the pig whipworm (Trichuris suis ova) have been shown to be safe in multiple studies.
110 t difference in inactivation of Ascaris suum ova in digesters operated at different solids residence
111 f embryonated and unembryonated Ascaris suum ova in six laboratory-scale mesophilic (35 degrees C) an
112 idual blastomeres of triploid and tetraploid ova.
113                                          The ova of an imperiled fish, the robust redhorse (Moxostoma
114  subsequent mating events from accessing the ova.
115 es at the second meiotic division, after the ova has been fertilized by a C57BL/6 sperm bearing a Y c
116 ype hypersensitivity reaction induced by the ova.
117                    During fertilization, the ova face a similarly challenging task of SNBP eviction a
118 erminus of MBP with six amino acids from the ova peptide, and replacement of the lysine side chain in
119 nd organic contaminants were measured in the ova and embryonic tissues and compared to concentrations
120 ernal DDK Om allele in only a portion of the ova of F1 females.
121 ain symptomatic after a negative result, the ova and parasite examination and special stains for othe
122 ntly cycled and ovulated normally, and their ova could be fertilized; however, the embryos did not pr
123 ning TCR+CD4+ cell population was anergic to ova antigen in vitro and enriched for CD25+ cells.
124 lop an exaggerated granulomatous response to ova trapped in the bladder wall, with associated urinary
125 ells and splenocytes were hypo-responsive to ova as early as day 10 after gene transfer.
126 racytoplasmic sperm injection into wild-type ova and implantation of the fertilized eggs into foster
127 able to complete fertilization of uninfected ova, but a rescue function allows infected eggs to devel
128  with age, even after adjustment for urinary ova excretion.
129  eosinophils, epithelial erosion, and viable ova.
130                 In contrast, concomitant VSV-ova infection induced OT-I-mediated epithelial cell dest
131 dogenous CD8 cells were nonresponsive to VSV-ova infection.
132                   Morphologically, T. vulpis ova resemble those of the human whipworm (T. trichiura)
133 ther vaccinia virus expressing ovalbumin (VV-ova) or peptide-pulsed dendritic cells.
134  we used an adoptive transfer system wherein ova-specific T cells were infused into a syngeneic host,
135 e occurred in 13 of 30 patients (43.3%) with ova treatment compared with 4 of 24 patients (16.7%) giv
136  inhibited IFN-gamma in mice inoculated with ova.
137 , neither E- nor P-selectin was found within ova.
138                                         Worm ova accumulation in the intestines of Arg I-deficient bo
139 t lethal intestinal pathology caused by worm ova in mice infected with the human parasite Schistosoma
140 imeras develop a marked accumulation of worm ova within the ileum but impaired fecal egg excretion co

 
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