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1 hypertension, hyperlipidemia, and polycystic ovarian disease.
2 ons of gall stones such as pancreatitis, and ovarian diseases.
3 trols (147.1 ng/mL), 46 patients with benign ovarian disease (254.4 ng/mL), and 47 patients with othe
4 n in insulin-resistant women with polycystic ovarian disease and enhances the effects of insulin in t
5 g s-OHSS but also in ruling out other cystic ovarian diseases and to determine the underlying etiolog
6 e prostate disease, obesity, kidney disease, ovarian disease, and parturition (birth) abnormalities.
7 rant to the peptide; they develop autoimmune ovarian disease (AOD) and autoantibody response 5 weeks
8 ay 3 after birth (d3tx) developed autoimmune ovarian disease (AOD) and autoimmune disease of the lacr
9 ectomized on day 3 (d3tx) develop autoimmune ovarian disease (AOD) and dacryoadenitis.
10                                   Autoimmune ovarian disease (AOD) and oocyte autoantibody response o
11 (ZP3) epitope was found to induce autoimmune ovarian disease (AOD) and premature ovarian failure in n
12                          Based on autoimmune ovarian disease (AOD) in day 3 thymectomized (d3tx) mice
13                                   Autoimmune ovarian disease (AOD) is a probable cause of human prema
14             In striking contrast, autoimmune ovarian disease (AOD) was suppressed equally by male and
15 sting a role of environmental epigenetics in ovarian disease etiology.
16 health problems, such as obesity, polycystic ovarian disease, hyperlipidaemia, hypertension and ather
17 dy to murine zona pellucida 3 induces severe ovarian disease in neonatal, but not adult, mice (neonat
18  adult NK cells inhibits neonatal autoimmune ovarian disease induction.
19                        The murine autoimmune ovarian disease is induced by immunization with the ZP33
20 ncrease in cysts resembling human polycystic ovarian disease (PCO) and a decrease in the ovarian prim
21  abnormalities, testis disease, obesity, and ovarian disease (primary ovarian insufficiency and polyc
22  Epigenetic transgenerational inheritance of ovarian disease states was induced by all the different
23 maternal autoantibodies (neonatal autoimmune ovarian disease); thus, neonatal but not adult NK cells
24 the F1 and F3 generation progeny adult onset ovarian disease was assessed.
25  epigenetic transgenerational inheritance of ovarian disease was investigated with the use of a fungi