ライフサイエンスコーパス: ovariectomize

1 OMSC, orofacial mesenchymal stem cell; OVX, ovariectomized.

2 e (XE991) (40 mum), in both intact males and ovariectomized, 17beta-estradiol (E2)-treated females.

3 12-week-old Fisher 344 rats were ovariectomized 2 weeks before flight and randomized into

4 hs) and aged (22-23 months) female rats were ovariectomized 7 days prior to a 48-h administration of

5 hs) and aged (22-23 months) female rats were ovariectomized 7 days prior to implantation of silastic

6 Mammary carcinogenesis in ovariectomized ACI rats requires continuous exposure to

7 mary carcinogenesis, six-week-old intact and ovariectomized ACI rats were continuously exposed to low

8 Also, ovariectomized ACI rats were treated with high-dose estr

9 ne, low-dose estrogen plus progesterone, and ovariectomized ACI rats with high-dose estrogen plus pro

10 carcinogenesis induced solely by hormones in ovariectomized ACI rats.

11 male mice or testosterone administration to ovariectomized adult female mice.

12 the right lateral dorsal hippocampus area of ovariectomized adult rat.

13 The response of ovariectomized adults to 17beta-estradiol (E2) and artif

14 Ovariectomized adults were subjected to a hormone replac

15 terotropic and ER-mediated gene responses in ovariectomized AF2ERKI female mice in the same manner as

16 Young adult female Sprague-Dawley rats were ovariectomized and bilaterally implanted via stereotaxic

17 edly protected against bone mass loss in the ovariectomized and dexamethasone treated rat osteoporosi

18 Female C57Bl6/J mice were ovariectomized and implanted with estradiol- or oil-secr

19 IL-6 levels in splenocyte supernatants from ovariectomized and male mice; elevated splenic IL-6 and

20 Ovariectomized and ovariectomized-osteoporosis rats were

21 ntreated (control), laparatomized (sham), or ovariectomized and received a deficient diet (OVX-Diet).

22 Sprague-Dawley adult female rats were ovariectomized and then injected over 2 days with 17beta

23 d aged (about 22-month-old) female rats were ovariectomized and then, 4 weeks later, subcutaneously i

24 Young and middle-aged female monkeys were ovariectomized and treated with conjugated equine estrog

25 ue when subjects were either ovary-intact or ovariectomized and treated with estradiol, estradiol plu

26 e experiment, rats were left ovary-intact or ovariectomized and were then irradiated with 2.5, 5, 10,

27 Cohorts of XX and XO gonadally intact, ovariectomized, and ovariectomized females supplemented

28 Female rats (15 months) were ovariectomized, and, 14 weeks later, adeno-associated vi

29 Cyclic estradiol treatment in aged ovariectomized animals restored MSB frequencies to level

30 polyphenols has translated the findings from ovariectomized animals to postmenopausal osteopenic wome

31 ral stroke and can be modelled using aged or ovariectomized animals.

32 n the DSA task compared to vehicle-implanted ovariectomized animals.

33 levels in the late phase (7 d) of healing in ovariectomized animals.

34 as this was reversed in the estrogen-treated ovariectomized animals.

35 Ovariectomized ApoE(-/-):Ins2(+/Akita) mice presented ch

36 In addition, ovariectomized ARKO hosts with wild-type ovary transplan

37 fferences, subsets of mice were castrated or ovariectomized at 5 weeks of age.

38 Female hamsters were ovariectomized at adulthood and housed in either a stand

39 Female Long Evans hooded rats were ovariectomized at middle age (11-12 months) and placed i

40 In vivo biodistribution studies in female ovariectomized athymic (NCr) nu/nu mice bearing GPR30-ex

41 to stimulate the growth of mammary tumors in ovariectomized athymic nude mice implanted with estrogen

42 implanted subcutaneously in immune competent ovariectomized C57BL/6 mice for a period of 60 days.

43 evel analyses were investigated in immature, ovariectomized C57BL/6 mice treated with 300 mg/kg o, p'

44 METHODS AND We treated ovariectomized C57BL/6J mice with the ER-beta selective

45 onsiveness ex vivo and in vivo compared with ovariectomized controls.

46 cant reduction in trabecular bone volume but ovariectomized Dmp1-ERalpha(-/-) female mice displayed a

47 (muscimol; 100ng/side) receptor agonists in ovariectomized, estradiol-primed rats.

48 This occurs on a daily basis in ovariectomized, estradiol-treated (OVX+E) mice; GnRH neu

49 study the effect of IL-27 supplementation on ovariectomized estrogen-deficient mice on various immune

50 ment of endometriosis-like lesions in 63% of ovariectomized estrogen-supplemented Tgfb1-null mutant m

51 , hepatosteatosis developed only in male and ovariectomized female aLivGHRkd mice.

52 cts in response to letrozole were similar in ovariectomized female and male mice, with, however, diff

53 (housed individually) or pair housed with an ovariectomized female before induction of stroke, via tr

54 ed from the lungs of E2- and placebo-treated ovariectomized female C57BL/6 mice following infection.

55 We demonstrated that ovariectomized female FBN-ARO-KO mice exhibited signific

56 ated the biliary and gallstone phenotypes in ovariectomized female GPR30(-/-) , ERalpha(-/-) , and wi

57 R, were used to generate tumor xenografts in ovariectomized female immunodeficient mice supplemented

58 ffects whether it is administered to aged or ovariectomized female mice and emphasizes the need to co

59 17beta-Estradiol-treated ovariectomized female mice demonstrated increased lung l

60 found that supplementation of estrogen into ovariectomized female mice enhanced M2 polarization in v

61 te for the first time that E2 replacement in ovariectomized female mice improves stroke-induced perip

62 t recognition and object placement memory in ovariectomized female mice in an ERK-dependent manner, s

63 Immediately after training, ovariectomized female mice received bilateral DH infusio

64 ERalpha-deficient, or ERalpha-AF1-deficient ovariectomized female mice were fed a high-fat diet and

65 ential for vitamin D(3)-mediated protection, ovariectomized female mice were given E(2) or placebo an

66 In sham-operated or ovariectomized female mice, 17beta-E2, P4, 17beta-E2+P4,

67 Ovariectomized female mice, implanted with 17beta-E2 or

68 ced object recognition and spatial memory in ovariectomized female mice, whereas the GPER antagonist

69 EPO treatment also reduced weight gain in ovariectomized female mice, while the effect was abrogat

70 ubstantially suppressed binge-like eating in ovariectomized female mice.

71 d object recognition memory consolidation in ovariectomized female mice.

72 at 1, 6 and 24 h post-ischaemia to aged and ovariectomized female mice.

73 lly suppresses binge-like eating behavior in ovariectomized female mice.

74 periment 1, E2 benzoate-induced LH surges in ovariectomized female monkeys were severely attenuated b

75 ME, also directly influences GnRH release in ovariectomized female monkeys, in which the ovarian sour

76 tal mice were either housed in pairs with an ovariectomized female or socially isolated for the durat

77 nitiated 14 days prior to global ischemia in ovariectomized female rats acts via the IGF-1 receptor t

78 d in bone from 5-wk-old intact and 10-wk-old ovariectomized female rats fed SPI diets.

79 tracerebral injection of 17beta-estradiol to ovariectomized female rats immediately after ischemia re

80 To control estradiol cyclical variability, ovariectomized female rats received empty or estradiol f

81 Ovariectomized female rats were tested for rotational be

82 In ovariectomized female rats, E2 rapidly (within 10 minute

83 signaling cascade to protect CA1 neurons in ovariectomized female rats.

84 prefrontal cortex (dlPFC) of young and aged ovariectomized female rhesus monkeys with and without E

85 ochemical distribution of aromatase in young ovariectomized female rhesus monkeys with or without lon

86 on enhances voluntary alcohol consumption in ovariectomized female rodents and that increased alcohol

87 ochemical effects of chronic E2 treatment of ovariectomized female wild-type and Kal7(KO) mice.

88 Indeed, in ovariectomized females and in male mice, the dominance o

89 couples lifespan from metabolic health, with ovariectomized females having improved survival despite

90 itis (EAE) in intact female mice, but not in ovariectomized females or males.

91 Ovariectomized females supplemented with 17beta-estradio

92 and XO gonadally intact, ovariectomized, and ovariectomized females supplemented with estrogen were e

93 In contrast, ovariectomized females treated with estradiol and proges

94 Ovariectomized females were given daily injections to ap

95 te nucleus increases bone mass in intact and ovariectomized females, confirming the central role of e

96 In ovariectomized females, Kiss1 neurons were scattered thr

97 ellular activity similar to that observed in ovariectomized females, suggesting that estradiol-induce

98 vitamin D(3)-mediated EAE resistance in the ovariectomized females.

99 Two-month-old female Sprague-Dawley rats, ovariectomized for 1 week, were given subcutaneous injec

100 amined in macaques that were ovary-intact or ovariectomized for 3 years living in a relatively natura

101 All mice were ovariectomized; half were estradiol replaced.

102 nterestingly, this effect is not observed in ovariectomized heterozygous BDNF Val66Met females, sugge

103 ter transplantation under kidney capsules of ovariectomized hosts, treated follicles developed to the

104 n a postmenopausal breast cancer model using ovariectomized, immune-competent female mice orthotopica

105 cells grafted subcutaneously into syngeneic ovariectomized immunocompetent mice, we found that E2 po

106 In ovariectomized-induced bone loss mouse model and RANKL-i

107 demonstrated oral efficacy in rat models of ovariectomized-induced thermoregulatory dysfunction and

108 22, were tested in a telemetric rat model of ovariectomized-induced thermoregulatory dysfunction and

109 d oral efficacy in a telemetric rat model of ovariectomized-induced thermoregulatory dysfunction, a m

110 ting (physiological bone loss condition) and ovariectomized (induction of surgical menopause) animals

111 Both intact and ovariectomized Kal7(KO) female mice exhibited decreased

112 e of absent gonadal estrogen, as chronically ovariectomized Kiss1r KO females developed obesity, hype

113 rata oriens, lucidum, and radiatum of CA3 in ovariectomized mice 6 h after administration.

114 n of Phd2 and Phd3 was sufficient to protect ovariectomized mice against bone loss without disrupting

115 rian tissue to restore endocrine function in ovariectomized mice and prevent cell-mediated immune rej

116 ling was increased in female but not male or ovariectomized mice and was associated with increased di

117 rtantly, uterine ILC2s were nearly absent in ovariectomized mice and were increased in wild-type mice

118 Together, our findings establish ovariectomized mice as a model for UTIs in menopausal wo

119 In ovariectomized mice bearing constant-release estradiol i

120 rine weight stimulated by estrone sulfate in ovariectomized mice by 69% and the STS activity in the l

121 Although ovariectomized mice continue to develop high-grade serou

122 Estrogen treatment of ovariectomized mice decreased I(K,total) (46.4+/-3.0 to

123 e surge of estrogen as estrogen treatment of ovariectomized mice did not alter the 26 S proteasome ac

124 7 days post-ischaemia, progesterone-treated ovariectomized mice did not differ significantly in perf

125 compared with shams, whereas vehicle-treated ovariectomized mice displayed a significant functional i

126 As expected, estradiol-implanted ovariectomized mice exhibited increased plasma estradiol

127 rmined morphometrically in male, female, and ovariectomized mice exposed to 6 months of cigarette smo

128 Anesthetized, ovariectomized mice had optical fibers implanted in the

129 We found that ovariectomized mice had significantly higher bacteriuria

130 Ovariectomized mice heterozygous for the BDNF Met allele

131 In ovariectomized mice IL-17A and IL-23R expression was inc

132 asting; bone marrow-derived macrophages from ovariectomized mice implanted with estrogen exhibited en

133 ol (17-betaE) treatment of latently infected ovariectomized mice induces viral reactivation, as demon

134 In vivo, MPA treatment of latently infected ovariectomized mice inhibited IFN-gamma production and l

135 In uterine tissues of ovariectomized mice injected with P(4), miR-200 expressi

136 vestrant, decreased MCF7 xenograft growth in ovariectomized mice more potently than each drug alone.

137 ort-term effects of estradiol replacement in ovariectomized mice on apoE expression and markers for c

138 in a hydrogel-based capsule and implanted in ovariectomized mice restore ovarian endocrine function i

139 Finally, UPEC-infected ovariectomized mice showed a significant increase in qui

140 ed that 17-beta estradiol supplementation of ovariectomized mice significantly enhanced BM-EPC-induce

141 IL-27 treatment in ovariectomized mice suppressed Th17 cell differentiation

142 omian glands were obtained from young adult, ovariectomized mice that were administered 17beta-estrad

143 s on intravaginal infection of the FRT using ovariectomized mice treated with reproductive hormones.

144 lume ( approximately 20%) in both normal and ovariectomized mice treated with SFN for 5 weeks compare

145 -protein-coupled estrogen receptor (GPER) in ovariectomized mice via the GPER agonist G-1 mimics the

146 homeostasis after infection, was aberrant in ovariectomized mice with defective superficial urothelia

147 ass, and bone strength in normal mice and in ovariectomized mice with established bone loss.

148 re made of GnRH neurons in brain slices from ovariectomized mice with ionotropic GABA and glutamate r

149 e and peripheral and CNS immune responses in ovariectomized mice with or without sustained, controlle

150 In ovariectomized mice, 5-, 10-, and 20-Hz activation of AR

151 In ovariectomized mice, a marked reduction is observed in c

152 In a further study of ovariectomized mice, allowed to survive for 7 days post-

153 onger excited preoptic kisspeptin neurons in ovariectomized mice, an effect that was fully restored b

154 nd functional impacts of E4 on the vagina of ovariectomized mice, and we determined the molecular mec

155 ry-enhancing effects in middle-aged and aged ovariectomized mice, and whether these effects depend on

156 Whereas in ovariectomized mice, at 48 h post-ischaemia, progesteron

157 estored after administration of estradiol in ovariectomized mice, demonstrating that the sex-specific

158 In ovariectomized mice, estrogen treatment resulted in down

159 When injected into ovariectomized mice, the FSH antibody attenuates bone lo

160 Using ovariectomized mice, we first demonstrate that intraperi

161 Using adoptive-transfer experiments and ovariectomized mice, we highlighted the role of female s

162 c disturbances in wt and ERalphaAF-1 degrees ovariectomized mice, whereas these actions were totally

163 ed growth of established MCF-7 xenografts in ovariectomized mice, whereas treatment with the neutrali

164 expression markedly increased in the bone of ovariectomized mice, which was blocked by bisphosphonate

165 endogenous LH pulse parameters measured from ovariectomized mice.

166 xposure to the chemicals was also studied in ovariectomized mice.

167 liver, whereas such benefit was abolished in ovariectomized mice.

168 othalamic cultures from young or aged female ovariectomized mice.

169 ion following cerebral ischaemia in aged and ovariectomized mice.

170 comparing cortical bone in sham-treated and ovariectomized mice.

171 f object recognition and spatial memories in ovariectomized mice.

172 uced cell proliferation is partly reduced in ovariectomized mice.

173 to enhance novel object recognition in young ovariectomized mice.

174 ene expression in the hypothalamus in female ovariectomized mice.

175 d a dose-dependent decrease in FSH levels in ovariectomized mice.

176 covery of Opa(+) gonococci does not occur in ovariectomized mice; therefore, the reproductive cycle p

177 Ovariectomized Mig-6(d/d) mice exhibit this hyperplastic

178 Ovariectomized Mig-6(d/d) mice treated with E2 developed

179 Aged ovariectomized monkeys treated with vehicle displayed si

180 In young ovariectomized monkeys without E treatment, presynaptic

181 ulated release of GnRH and E2 in the S-ME of ovariectomized monkeys, (2) electrical stimulation of th

182 nous synapses within layer III dlPFC of aged ovariectomized monkeys.

183 utive and attention processes in middle-aged ovariectomized monkeys.

184 Using an ovariectomized mouse model coupled with micro-computed t

185 Here, we developed an ovariectomized mouse model in which systemic E2 concentr

186 les and confirmed that unlike sham controls, ovariectomized mutant mice showed no increase in self-ad

187 Previously, we reported cyclic ET in aged, ovariectomized NHPs increased spine density on dlPFC neu

188 to address this hypothesis using a long-term ovariectomized non-human primate (NHP) model, the cynomo

189 rogens and leptin modulate gut microbiota in ovariectomized ob/ob (obese) or heterozygote (lean) mice

190 Therefore, we used ovariectomized oil- or E2-treated EGFP (enhanced green f

191 as few PR-labeled profiles were detected in ovariectomized, oil-replaced females.

192 Food-restricted ovariectomized or sham-operated c-fos-GFP rats were trai

193 The mice were ovariectomized or sham-operated, and 5 weeks after surge

194 male Japanese macaques (Macaca fuscata) were ovariectomized or tubal-ligated (n=5/group) and returned

195 deficient, anti-IL-6 receptor alpha-treated, ovariectomized, or male mice; undetectable IL-6 levels i

196 ens of female C57BL/6 mice that were intact, ovariectomized, or ovariectomized with E2 replacement ex

197 ould be an effective enriched milk powder in ovariectomized-osteoporosis and ovariectomized rats as a

198 Ovariectomized and ovariectomized-osteoporosis rats were used as a menopaus

199 ne = ingestion of caffeine/sham surgery); 3) ovariectomized (OVX) = non-ingestion of caffeine/ovariec

200 uate kisspeptin neurons in brain slices from ovariectomized (OVX) and OVX+estradiol (OVX+E) female mi

201 C, the average core temperature (T(CORE)) of ovariectomized (OVX) control rats was significantly elev

202 Eleven ovariectomized (OVX) ewes and four ewes subjected to sha

203 f mice based on the sex and estrogen status [ovariectomized (OVX) female and E2-treated male].

204 n of E(2) on glucose homeostasis in male and ovariectomized (OVX) female control and liver-specific F

205 PKG activity was not enhanced in ovariectomized (OVX) female mice as a result of cardiac

206 1-/-) mice, control Spp1+/+ (C57BL/6J) mice, ovariectomized (OVX) female mice, and estrogen-treated m

207 d the regulation of Hdac4 in the amygdala of ovariectomized (OVX) female mice.

208 onal antibody (Scl-Ab) in aged male rats and ovariectomized (OVX) female rats were used to study the

209 wing acquisition, intact male and intact and ovariectomized (OVX) female rats with and without estrad

210 at is reversed by subsequent E2 treatment in ovariectomized (OVX) female Thy1M-EGFP mice.

211 cement in male, female (freely cycling), and ovariectomized (OVX) females treated with either estroge

212 Ovariectomized (OVX) females were also included to asses

213 Intact males, females, and ovariectomized (OVX) females with and without estradiol

214 ts of mechanical loading to the spine, using ovariectomized (OVX) mice as a model of osteoporosis.

215 e that of GCs, the administration of CpdX to ovariectomized (OVX) mice does not induce a fatty liver

216 Ovariectomized (OVX) mice treated or not with estradiol

217 molecular mechanisms mediating bone loss in ovariectomized (OVX) mice, a model of human menopause, u

218 Ovariectomized (ovx) mice, but not AIB1 x ERalpha-/- mic

219 increased in the exosomes of the bone-losing ovariectomized (OVX) mice, while it was significantly de

220 We have shown that ovariectomized (Ovx) monkeys treated with estradiol (E)

221 ir in an estrogen-deprived animal model, the ovariectomized (Ovx) mouse, principally by dampening the

222 Two-month-old rats were ovariectomized (OVX) or had maxillary molars removed fro

223 Ovariectomized (OVX) rats (n=5-6/group) were treated wit

224 Ovariectomized (OVX) rats 24 h (but not 6 or 72 h) after

225 trus vs estrus), (2) pLTF would be absent in ovariectomized (OVX) rats and in physiological condition

226 ue inflammation, and the cecal microbiota in ovariectomized (OVX) rats bred for low-running capacity

227 es aldosterone binding, in male rats, and in ovariectomized (OVX) rats given estradiol benzoate (EB)

228 Treatment of ovariectomized (OVX) rats with estradiol benzoate (EB) c

229 xperimental periodontitis was established in ovariectomized (OVX) rats, and the OVX-periodontitis rat

230 Following implantation in ovariectomized (ovx) rats, the Sr(++)-cross-linked const

231 nucleus of the solitary tract (NTS) of lean ovariectomized (OVX) rodents.

232 However, in ovariectomized (OVX) WT and in ERbeta(-/-) mice, there w

233 ps: 1) young intact, 2) old intact, 3) young ovariectomized (OVX), 4) old OVX, 5) young OVX plus estr

234 Mice were ovariectomized (OVX), and a subset was treated with estr

235 studies were undertaken in brain slices from ovariectomized (OVX), diestrous, and proestrous kisspept

236 After task acquisition, animals were ovariectomized (OVX).

237 owth also increased mammary tumorigenesis in ovariectomized polyomavirus middle T-antigen mice.

238 program even in the absence ovarian E in an ovariectomized, progesterone-supplemented pregnant mouse

239 enting hot flushes in the morphine-dependent ovariectomized rat model and results in the restoration

240 Long-term ovariectomized rats also displayed a robust hyperinducti

241 14 T magnetic field exposure was compared in ovariectomized rats and ovariectomized rats with estradi

242 lk powder in ovariectomized-osteoporosis and ovariectomized rats as a model of menopause-osteoporosis

243 chronic 17beta-estradiol (E2) replacement of ovariectomized rats enhanced Kal7 expression in the hipp

244 ase in the trabecular bone formation rate in ovariectomized rats following oral administration.

245 In ovariectomized rats irradiated with 15 Gy, estradiol inc

246 diol or progesterone into the hippocampus of ovariectomized rats on serotonin (5-HT) clearance, as we

247 E(2)-treated ovariectomized rats received an injection of antisense o

248 We show in ovariectomized rats receiving estradiol or control treat

249 nt of low rates of responding (DRL) tasks in ovariectomized rats that were given chronic estradiol vi

250 e was no difference between ovary-intact and ovariectomized rats that were irradiated at lower doses.

251 RANK was significantly expressed in ovariectomized rats treated with etidronate.

252 mia protected the hippocampus from damage in ovariectomized rats via phosphorylation of cyclic-AMP re

253 of spaceflight on bone microarchitecture in ovariectomized rats was bone-and bone compartment-specif

254 evant insult, as the CA3 region of long-term ovariectomized rats was profoundly hypersensitive to the

255 ity in hippocampal neurons of young and aged ovariectomized rats were determined.

256 Ovariectomized rats were given ten injections of E2 at 4

257 In another experiment, ovariectomized rats were treated continuously with three

258 Adult (6 mo) and aged (22 mo) ovariectomized rats with (OP) and without (Ovx) 17- exis

259 sure was compared in ovariectomized rats and ovariectomized rats with estradiol replacement.

260 Leptin did not alter LSNA or HR in ovariectomized rats, but its effects were normalized wit

261 agonism manifests as tissue-selectivity: in ovariectomized rats, Cl-4AS-1 mimics DHT while TFM-4AS-1

262 In ovariectomized rats, osteoclasts were noticed in the roo

263 In ovariectomized rats, there was more c-Fos expressed in t

264 By testing different classes of compounds in ovariectomized rats, we established that ligands that tr

265 st cancer cells and in the mammary glands of ovariectomized rats.

266 ng, memory and ischemic neuronal survival in ovariectomized rats.

267 and apposition sides of the periodontium in ovariectomized rats.

268 to inhibit serotonin transporter function in ovariectomized rats.

269 normal cycling rats and in estrogen-treated ovariectomized rats.

270 mammary carcinogenesis in intact but not in ovariectomized rats.

271 loped a standardized oral osteotomy model in ovariectomized rats.

272 responses in a dose-dependent manner in the ovariectomized rats.

273 in a 3 mm femur osteoporotic defect model in ovariectomized rats.

274 cently stained femur samples from normal and ovariectomized rats.

275 oreover, endocrine function was recovered in ovariectomized recipients, including elevated levels of

276 , and decreased bone turn over markers in an ovariectomized rodent model for postmenopausal osteoporo

277 e-dependent manner, to treat osteoporosis in ovariectomized rodents because of an isolated increase i

278 Estradiol treatment of ovariectomized rodents is known to affect the morphology

279 of young (4-5 months) and old (22-24 months) ovariectomized Sprague-Dawley female rats.

280 To test this, ovariectomized, steroid-primed rats were injected (i.p.)

281 inistration and extinction, female rats were ovariectomized to isolate estrogen effects on reinstatem

282 bone loss phenotype was seen in young adult ovariectomized transgenic mice by microcomputed tomograp

283 m unmoved objects after a 60-min delay while ovariectomized vehicle-treated females and gonadally int

284 However, adult ovariectomized Vgat-Cre;Lepr(lox/lox) mice displayed sig

285 he XE991-sensitive M-current by threefold in ovariectomized (vs oil-treated) female mice.

286 ta in regulating transcription and learning, ovariectomized wild-type (WT) and ERalpha and ERbeta kno

287 the development of preneoplastic lesions in ovariectomized wild-type (WT) and ERbeta knockout (ERbet

288 gene responses were increased in D3-treated ovariectomized wild-type animals, in a manner similar to

289 en-like effects of ICI in different tissues, ovariectomized wild-type mice and mice with mutations in

290 pithelial and stromal cells of the uterus in ovariectomized wild-type mice, but not in PRKO mice.

291 In ovariectomized wild-type mice, tamoxifen significantly r

292 anxiety, motor, and nociceptive behavior of ovariectomized, wildtype (WT), and ERss knockout (ssERKO

293 To achieve this goal, 2-month-old ovariectomized Wistar rats were injected bilaterally, in

294 Young adult female FVB/NJ mice were ovariectomized with (OVX+E, n=6) or without (OVX, n=8) e

295 /6 mice that were intact, ovariectomized, or ovariectomized with E2 replacement exhibited no differen

296 rmones are driving these sex differences, we ovariectomized WT and Npas2 mutant females and confirmed

297 olism, and glucose intolerance compared with ovariectomized WT females.

298 Infarct sizes were equivalent between ovariectomized XX and XO mice, between intact XX and XO

299 d XO mice, and between estrogen-supplemented ovariectomized XX and XO mice.

300 bserved in the cholinergic neurons in either ovariectomized young or middle-aged monkeys.