ライフサイエンスコーパス: overeating

1 tiety cues may prevent at-risk children from overeating.

2 t overall energy intake at meals and promote overeating.

3 hysiology of food that may predispose toward overeating.

4 ouths' social network is likely to reinforce overeating.

5 e, physiological satiety signals and promote overeating.

6 vidual differences in the susceptibility for overeating.

7 eating disorders and more moderate forms of overeating.

8 d alpha-MSH secretion in an attempt to limit overeating.

9 susceptibility to weight gain in response to overeating.

10 otential treatment of excessive appetite and overeating.

11 ed asprosin-induced AgRP(ARH) activation and overeating.

12 voke food cravings and may lead to unhealthy overeating.

13 by creating foods that decrease the risk of overeating.

14 ed with lower locomotor activity rather than overeating.

15 increased risk for early- and mid-childhood overeating.

16 dysfunctional reward-seeking behavior, as in overeating.

17 table food cues, which may serve to maintain overeating.

18 g and reward systems differently to moderate overeating.

19 ls in the lateral hypothalamus that promotes overeating.

20 d cues, which might decrease risk for future overeating.

21 in responsivity may result from a period of overeating.

22 ture rather than the induction of compulsive overeating.

23 ve neurons in adult mice caused obesity from overeating.

24 eward region responsivity increases risk for overeating.

25 dency toward certain foods may contribute to overeating.

26 bitory control signalling, which facilitates overeating.

27 vulnerability factor or a result of previous overeating.

28 if it exists, innate or does it develop with overeating?

29 lsive behavior as it applies to pathological overeating: (1) habitual overeating; (2) overeating to r

30 ies to pathological overeating: (1) habitual overeating; (2) overeating to relieve a negative emotion

31 metabolic environment in vivo, resulting in overeating after food deprivation in mice.

32 ributions of hedonic and hormonal factors to overeating after sleep loss are a matter of ongoing cont

33 Overeating and binge eating assessed via questionnaire e

34 arting to binge drink frequently, while both overeating and binge eating predicted starting to use ma

35 isorders, including eating disorders such as overeating and binge-eating disorder, but the brain stru

36 ng, fewer depressive symptoms, lower risk of overeating and certain sexual behaviours.

37 for a common mechanism underlying compulsive overeating and compulsive drug taking.

38 It has been proposed that overeating and concomitant opioid release could downregu

39 cond, stress can affect behavior by inducing overeating and consumption of foods that are high in cal

40 rization of brain circuitry that may promote overeating and contribute to obesity.

41 during prolonged oxidative stress, to avoid overeating and destruction of essential cellular compone

42 fying subpopulations with increased risk for overeating and dietary obesity and detecting early signs

43 nt neurological underpinnings for compulsive overeating and drug addiction.

44 The role of diet composition in response to overeating and energy dissipation in humans is unclear.

45 ric intake and energy expenditure, fostering overeating and further weight gain.

46 everal dysfunctional human behaviors such as overeating and gambling.

47 n of CNTF treatment does not result in binge overeating and immediate rebound weight gain.

48 ng that dietary patterns high in UPF promote overeating and increase the risk of overweight and obesi

49 We all know that overeating and lack of exercise are risk factors for man

50 with food addiction engage more in emotional overeating and less cognitive control and homeostatic pr

51 neurons may contribute to the development of overeating and obesity after chronic high-fat diet.

52 Our results may have implications for why overeating and obesity are common in modern food environ

53 ing the behaviours that lead to pathological overeating and obesity are poorly understood.

54 rochemical pathways involved in pathological overeating and obesity are poorly understood.

55 g problems facing contemporary society, from overeating and obesity to impulsive sexual behavior and

56 elated and addictive disorders contribute to overeating and obesity, and 3) food industry practices a

57 sistent with data on neural underpinnings of overeating and obesity, and makes further experimental p

58 (HF) food over time, which may contribute to overeating and obesity, has not been determined.

59 hich is a factor that contributes to hedonic overeating and obesity.

60 on in this pathway could potentially lead to overeating and obesity.

61 t their inappropriate expression can lead to overeating and obesity.

62 t led to novel and successful treatments for overeating and obesity.

63 re addictive and whether this contributes to overeating and obesity.

64 te and rich mouthfeel but also contribute to overeating and obesity.

65 genous feeding suppression system to promote overeating and obesity.

66 on in reward circuits may be associated with overeating and obesity.

67 nally representative US sample suggests that overeating and oversleeping can be used to identify an a

68 casing synaptic plasticity in the context of overeating and palatable food exposure.

69 atiety insecurity is an unexamined driver of overeating and partially explains why food insecurity mo

70 osa and its primary symptoms, out-of-control overeating and purging, are poorly understood.

71 enetic conditions, the biological drivers of overeating and reduced activity are unclear.

72 urrent theories consider obesity a result of overeating and sedentary life style and most efforts to

73 s that obesity does not have to be caused by overeating and sedentary life-style but may be the resul

74 sexual cues is associated with indulgence in overeating and sexual activity, respectively, and provid

75 de that unrestricted access to food promotes overeating and that requiring a simple action such as a

76 diets is important for these diets to induce overeating and that requiring mice to perform small amou

77 ivity of reward circuitry increases risk for overeating and that this overeating may further attenuat

78 This may contribute to overeating and the development of obesity.

79 er satiating power and may result in passive overeating and therefore weight gain.

80 gested that insufficient sleep may stimulate overeating and thus play a role in the current epidemic

81 -Acb or intra-vmPFC mu-OR stimulation causes overeating and vigorous responding for food rewards.

82 ptin levels, a consequence which may lead to overeating and weight gain in individuals or populations

83 Palatable high-fat diets reliably cause overeating and weight gain in lab animals.

84 simple action such as a nose-poke can reduce overeating and weight gain in mice.

85 Diets that are high in fat cause overeating and weight gain in multiple species of animal

86 ), interact with the environment and lead to overeating and weight gain; the regulation of cues (ROC)

87 nditure by doubly labeled water prior to the overeating and weight stabilization periods and at weeks

88 se studies can increase our understanding of overeating and, perhaps in some cases, the development o

89 nt of acute energy needs, thereby promoting "overeating" and the consequent development of an energy

90 atment for chronic food cravings, compulsive overeating, and binge eating that may represent a phenot

91 es that are linked to a sedentary lifestyle, overeating, and genetic factors.

92 ed or having little energy, poor appetite or overeating, and having little interest in doing things w

93 es override signals from the body to promote overeating, and indicate OX1R antagonism as a potential

94 oses animals to systemic insulin resistance, overeating, and obesity.

95 t in adipose tissue; increased with feeding, overeating, and obesity; decreased with fasting, exercis

96 triatal D2R, a typical feature of addiction, overeating, and other psychiatric pathologies.

97 epts that obesity is merely a consequence of overeating, and that metabolic health then reflects obes

98 e and treatments for phobias, addiction, and overeating are discussed.

99 These data do not support overeating as a cause of weight gain among breast cancer

100 Dysregulation in both these systems leads to overeating, as seen in genetic syndromes and environment

101 r psychological factors, suggesting that the overeating associated with weight gain might be physiolo

102 fancy, evidence of learning disabilities and overeating behavior due to an abnormal satiety response

103 Emotional overeating behavior may occur in the context of negative

104 he absence of hunger and lead to maladaptive overeating behavior.

105 cated as a putative causal factor in chronic overeating, binge eating, and obesity.

106 lamus are well known for producing long-term overeating, but past research has excluded this site as

107 n diet-induced obese (DIO) mice and promotes overeating by a mechanism that includes inhibiting gut-b

108 ves to maintain energy homeostasis; however, overeating can result in obesity, which is associated wi

109 mpaired emotional response and/or compulsive overeating characteristic of this disease.

110 ific genetic strains or mutations that cause overeating consume a diet enriched with fat, modest amou

111 w the evidence that some forms of obesity or overeating could be considered a food addiction and argu

112 eating, recurrent and persistent episodes of overeating coupled with a lack of control over eating, a

113 relieve a negative emotional state; and (3) overeating despite aversive consequences.

114 dipose tissue occurs in response to cold and overeating (diet induced), and there is an inverse relat

115 for therapeutic interventions for compulsive-overeating disorder.

116 The growing prevalence of overeating disorders is a key contributor to the worldwi

117 es hunger state and pathways associated with overeating disorders.

118 New experiments demonstrate that this overeating doesn't occur when mice work to earn the food

119 l studies showing gross nutrient imbalances; overeating driven by high energy density, hyper-palatabi

120 used to study maladaptive behaviors, such as overeating, drug taking, or relapse.

121 Overeating during mealtimes and unscheduled snacking dis

122 Emotional overeating (EOE) has been associated with increased obes

123 rol is an important indicator of severity of overeating episodes.

124 e commonly accepted explanation is pervasive overeating: ever-increasing energy intake as the populat

125 somatosensory brain regions, contributing to overeating, few studies have examined the relative role

126 f substance use and relapse and is linked to overeating, gambling, and other maladaptive behaviors.

127 Hyperphagia continued in maturity, with overeating greatest during the mid- to late-dark cycle.

128 The food addiction model of overeating has been proposed to help explain the widespr

129 whether there is a genetic susceptibility to overeating high-fat diets has not been resolved.

130 ely, unraveling the neurobiological basis of overeating holds promise for addressing the pressing pub

131 gs suggest that FF commercials contribute to overeating in adolescents through reward mechanisms.

132 Is starvation in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

133 accurately recognize hunger signals, whereas overeating in bulimia nervosa could represent an exagger

134 to restricted eating in anorexia nervosa and overeating in bulimia nervosa.

135 Alcohol intake associates with overeating in humans.

136 ested that higher levels of binge eating and overeating in males at age 13 y likely cause higher BMI

137 t and thrombospondin receptor, in triggering overeating in mice with central BDNF depletion.

138 The neurobiological mechanisms underlying overeating in obesity are not understood.

139 mals affording a potential target to control overeating in people.

140 eural network that mediates this cue-induced overeating in sated rats include lateral hypothalamus, a

141 ll activity is essential for ethanol-induced overeating in the absence of societal factors, in single

142 s of the posterodorsal amygdala (PDA) elicit overeating in their own right.

143 ad necessary for the genetic and environment overeating-induced increase of the gut absorptive capaci

144 This overeating is a clinical concern, but its causes are puz

145 Impulsive overeating is a common, disabling feature of eating diso

146 Overeating is a complex behavioral phenotype in terms of

147 seful for short periods when difficulty with overeating is anticipated, such as during vacations.

148 The pain associated with acute overeating is illustrated by the firsthand experience of

149 highly palatable food is readily available, overeating is often driven by motivational, rather than

150 Increasing childhood overeating is prospectively associated with an adverse c

151 eward seeking become dysregulated to promote overeating is unknown.

152 sivity of these regions may increase risk of overeating, it is unclear whether this is an initial vul

153 ct appetite in children, which could lead to overeating later in the day.

154 Thus, we tested whether overeating leads to reduced striatal responsivity to pal

155 Mounting evidence suggests that overeating may be conceptualized within the same behavio

156 ing worldwide trend in obesity, suggest that overeating may be the largest avoidable cause of cancer

157 rthermore, higher levels of binge eating and overeating may cause higher BMI in later life.

158 decreased reward sensitivity, implying that overeating may contribute to reduced striatal responsivi

159 increases risk for overeating and that this overeating may further attenuate responsivity of reward

160 uggest that initiating a prolonged period of overeating may increase striatal responsivity to food cu

161 Reward deficits, resulting from repeated overeating, may in turn contribute to the perpetuation o

162 e inhibitory inputs to the VP might exist in overeating mice even before exposure to HFHS, thus makin

163 Neither overeating nor binge eating was associated with starting

164 e explore the intricate relationship between overeating, obesity, and the underlying neurobiological

165 tor subtypes in obesity induced by voluntary overeating of a palatable diet.

166 d cause overeating or would be the effect of overeating of carbohydrate and fat.

167 aberrant eating, which posits that recurrent overeating of palatable food is similar to addictive beh

168 ntermittent access to palatable food exhibit overeating of palatable food upon renewed access and an

169 creased propensity to develop uncontrollable overeating of palatable food.

170 may play a significant role in promoting the overeating of palatable food.SIGNIFICANCE STATEMENT In m

171 induced HP food motivation and engagement in overeating of such foods to enhance risk of weight gain

172 baseline intake or food restriction-induced overeating of the standard chow diet.

173 often narrows to the question of whether the overeating of these foods causes the same long-term neur

174 For instance, perceived overeating or 'uncontrolled eating' (UE) is the most com

175 This suggests that behaviors, such as overeating or drug taking, might be especially prone to

176 Discontinuing unhealthy behaviors, such as overeating or drug use, depends upon an individual's abi

177 l variant frontotemporal dementia identified overeating or increased sweet food preference in 80 (78%

178 re a result of maladaptive eating behaviors (overeating or undereating) and are associated with life-

179 aired with behavioral traits associated with overeating or undereating.

180 a relatively low protein intake would cause overeating or would be the effect of overeating of carbo

181 erventions for disorders such as drug abuse, overeating, or pathological gambling.

182 s in glucose levels might reduce the risk of overeating, particularly in environments inundated with

183 adult brain induced a hitherto unrecognized overeating phenotype and caused overweight that was prev

184 to be fixed in cave populations in which the overeating phenotype is present.

185 Overeating produced significantly less weight gain in th

186 may not be initial vulnerability factors, as overeating reduces D2 receptor density, D2 sensitivity,

187 ogical factors necessary for ethanol-induced overeating remain unclear, and societal causes have been

188 zards associated with calorie restriction or overeating, respectively.

189 ognized that some lifestyle patterns such as overeating result in metabolic syndrome, which may play

190 In conclusion, overeating SFAs promotes hepatic and visceral fat storag

191 den of disease is associated with behaviors--overeating, smoking, excessive alcohol consumption, and

192 erved between perceived stress and emotional overeating; stronger among urban (r = 0.32), female (r =

193 eward region responsivity increases risk for overeating, suggesting qualitatively distinct reward sur

194 ble foods may explain, at least in part, why overeating tasty foods is so irresistible even in the fa

195 gion responsivity to food, which may lead to overeating that produces blunted dopamine signaling and

196 en processing of palatable foods can promote overeating, the neurobiological mechanisms that underlie

197 Despite overeating, they weighed less and had significantly redu

198 ht- and eating-related phenotypes (e.g. BMI, overeating), three phenotypes related to caregiver somat

199 , and low mu-opioid availability may promote overeating to compensate decreased hedonic responses in

200 cal overeating: (1) habitual overeating; (2) overeating to relieve a negative emotional state; and (3

201 ciate longitudinal trajectories of childhood overeating, undereating, and fussy eating with metabolic

202 is increasing evidence that the pathological overeating underlying some forms of obesity is compulsiv

203 One potentially important difference between overeating versus drug abuse refers to the sensory stimu

204 fully adjusted models, binge eating, but not overeating, was associated with incident overweight/obes

205 ely causes higher levels of binge eating and overeating, weight and shape concerns, and weight-contro

206 ponsiveness, enjoyment of food and emotional overeating) were positively associated with infancy and

207 od insecurity promotes opportunistic eating (overeating when food is available), contributing to obes

208 rceived hedonic qualities of food as driving overeating, whereas subliminal signals arising from the

209 d to a chronic food restriction challenge by overeating, which resulted in prerestriction, rather tha

210 crease their susceptibility to opportunistic overeating while at the same time making food intake les

211 lation causing DRD2-dependent inhibition and overeating, while stronger stimulation causing DRD1-depe

212 Although any overeating, with or without loss of control, predicted t

213 Infrequent binge eating or purging or overeating without a loss of control were reported by 31