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1 LC) of the lesion formation process to avoid overexposure.
2 de-efferentation without purposeful acoustic overexposure.
3 t normally occurs with aging and/or acoustic overexposure.
4 n the mediation of protection after auditory overexposure.
5 ze changes in auditory receptors after noise overexposure.
6 ge, resulting in "silent" chronic tacrolimus overexposure.
7 ommonly in instances of accidental radiation overexposure.
8 nt role in protecting the cochlea from noise overexposure.
12 esults underscore the need to minimize noise overexposure and for strategies to personalize diagnosis
14 dent and human cardiomyocytes upon palmitate overexposure, and appeared as an early lipid-induced eve
15 monitor eculizumab effectiveness, avoid drug overexposure, and save money considering the extremely h
17 ch-perception problems associated with noise overexposure are pervasive in today's society, even with
18 fetuses to model excess prenatal androgenic overexposure associated with conditions such as polycyst
22 pite their integral role in sustaining life, overexposure can lead to deleterious neurological outcom
23 ICANCE STATEMENT A single incidence of noise overexposure causes damage at the hair cell synapse that
25 ing of the inner ear in mouse, that acoustic overexposures causing moderate, but completely reversibl
27 tacrolimus seem a promising tool to prevent overexposure directly posttransplantation in patients on
29 A neurotransmission, in-utero glucocorticoid overexposure had a modest or no effect on a range of con
31 (IHCs): following ototoxic drugs or acoustic overexposure, IHC death is rapid whereas SGN degeneratio
32 t earlier ages, implying that glucocorticoid overexposure in LP fetuses occurs via 11beta-HSD2-indepe
33 l to detect the pathological legacy of noise overexposure in mass stranded cetaceans as a key to unde
35 P450 (CYP) 3A4 and could lead to tacrolimus overexposure in patients genetically lacking the alterna
36 idence for the presence of hidden tacrolimus overexposure in patients with mild diarrhea while on tre
37 to assess the degree of unnoticed tacrolimus overexposure in renal transplant patients with mild diar
39 essing deficits.SIGNIFICANCE STATEMENT Noise overexposure is a major cause of central auditory proces
42 nese (Mn) is an essential trace element, but overexposure is characterized by Parkinson's like sympto
43 ulates skin tissue injury and pain after UVB overexposure, it is discussed whether TRPV4 downregulati
46 anganese accumulation in these structures on overexposure may be common to all primates, including hu
47 ental lactate production induced by cortisol overexposure may contribute to the adverse effects of ma
48 gs support the hypothesis that acute ethanol overexposure may increase the risk of infection and inhi
50 of white adipose tissues is associated with overexposure of lean organs to circulating triglycerides
51 ysiology and cognition in humans, and either overexposure or (more rarely) insufficiency can cause ne
55 a channel's sensitivity is reduced following overexposure to adaptation stimuli that selectively stim
58 ory decrements can lead to food poisoning or overexposure to environmentally hazardous chemicals that
59 d fetal growth, both involving fetoplacental overexposure to glucocorticoids but from distinct source
60 The inner ear can be permanently damaged by overexposure to high-level noise; however, damage can be
63 ethics of using a placebo, the potential for overexposure to iodine, and the possibility of community
71 of outer hair cells would be compatible with overexposure to underwater noise, affecting the region w
72 onal trauma, contact with bleaching phenols, overexposure to UV, and mechanical injury can lead to pr
73 iption of the sterile inflammation caused by overexposure to UVB irradiation (i.e., sunburn) in the m
76 vertheless, it has long been recognized that overexposure to vitamin A or retinoic acid causes widesp