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1 fects on glucose homeostasis during high-fat overfeeding.
2 reased by caloric restriction and reduced by overfeeding.
3 imize nutrition and protect individuals from overfeeding.
4 port in obese, diabetic patients rather than overfeeding.
5 hosphatase in the liver following short term overfeeding.
6 insulin and leptin resistance by short-term overfeeding.
7 balance of approximately 275 g after 96 h of overfeeding.
8 ptin and insulin ensued after just 3 days of overfeeding.
9 c rate or in thermogenesis from meals during overfeeding.
10 2 g/d (control) to a maximum of 10 g/d after overfeeding.
11 may contribute to the hypophagic response to overfeeding.
12 sue from humans gaining weight (4-7 kg) with overfeeding.
13 y underfeeding and to avoid complications of overfeeding.
14 4; 2243 +/- 253 mg.kg-1.min-1) carbohydrate overfeeding.
15 dictions for overweight subjects may lead to overfeeding.
16 d less calories (r = -0.91, P = 0.03) during overfeeding.
17 ctor (TNF), was changed in adipose tissue by overfeeding.
18 ts receiving nutrition on CVVH may result in overfeeding.
19 ain containing (ADIPOQ)] were changed by SFA overfeeding.
20 t oxidation were reciprocally related during overfeeding.
21 pEE did not increase during the first day of overfeeding.
22 he metabolic chamber during the last week of overfeeding.
23 izer under conditions of acute refeeding and overfeeding.
24 to estimate EI during weight gain induced by overfeeding.
25 ot the increase of IHCLs induced by fructose overfeeding.
27 gy imbalance in six lean men on mixed diets (overfeeding: 16.5 MJ/d, +33%, n = 3; underfeeding: 3.5 M
30 eight gain over time can be assessed by 24-h overfeeding a low-protein diet and measurements of plasm
37 was observed during underfeeding relative to overfeeding and during vancomycin treatment compared wit
38 uld increase with underfeeding compared with overfeeding and increase during oral vancomycin treatmen
39 d 3.1 +/- 2.1 (mean +/- SD) kg body fat with overfeeding and lost 2.4 +/- 1.7 kg body fat with underf
40 ise, FGF21 is produced from the liver during overfeeding and mitigates peripheral insulin resistance.
41 epEE) were measured on days 1, 14, and 56 of overfeeding and on day 57 while consuming the baseline d
42 aintaining normal blood glucose, by avoiding overfeeding and providing insulin therapy when necessary
43 rease in response to extra energy intake.SFA overfeeding and PUFA overfeeding induce distinct epigene
44 abolic phenotype prone to weight gain during overfeeding and resistant to weight loss during caloric
45 ase in 24-h energy expenditure (24EE) during overfeeding and smaller decrease in 24EE during fasting
46 r avidly accumulates fat during carbohydrate overfeeding and support a role for DNL in the pathogenes
47 ophagic signaling in response to involuntary overfeeding and support the hypothesis that MCs are impo
48 OSM is involved in the metabolic response to overfeeding and that hematopoietic cell-derived OSM can
49 unteracted most of the effects of short-term overfeeding and under-activity at the whole-body level a
52 e to dietary fat intake (150 and 20 g/d, for overfeeding and underfeeding, respectively), rather, its
55 seems to normalize the anabolic response to overfeeding and, if these findings are confirmed, may be
57 asting, a smaller EE response to low-protein overfeeding, and a larger response to high-carbohydrate
58 istinct responses of different fat depots to overfeeding, and fat-cell number increases in certain de
59 termine whether such responses to fasting or overfeeding are associated with future weight change.
60 xacerbates obesity by provoking compensatory overfeeding as one way to restore reward sensitivity.
62 nese honeys suggests product adulteration by overfeeding bee colonies with industrial sugars syrups d
63 aling pathway may help understand hedonistic overfeeding behavior observed due to modern AGE-rich die
65 obial community structure with under- versus overfeeding but a more widespread change in community st
67 n random order: energy balance (control) and overfeeding by 50% of energy requirements with fat (O(fa
68 vo lipogenesis was 2- to 3-fold higher after overfeeding by 50% than after the control treatment in a
69 novo lipogenesis was measured during 96 h of overfeeding by 50% with either sucrose or glucose and du
73 We hypothesized that 4 weeks of carbohydrate overfeeding (COF) with +25% energy would unmask key T2D
74 t is poorly recognized and may contribute to overfeeding complications.We aimed to quantify the syste
75 rectly predicted resistance to fat gain with overfeeding (correlation coefficient = 0.77, probability
76 inpatient individuals, calculated EI during overfeeding determined from DeltaES in FM and FFM was (m
77 free-living individuals, estimated EI during overfeeding determined from DeltaES in FM and FFM was 41
81 ttenuates the effects of short-term fructose overfeeding, dietary conditions known to increase intrah
83 energy balance, fasting, and four different overfeeding diets with 200% energy requirements was meas
84 hreefold only after the two low-protein (3%) overfeeding diets, one high in carbohydrate (75%) and th
89 eers underwent 6 wk of 150% low-protein (2%) overfeeding followed by another wk of weight-maintaining
91 Adjusting changes in REE with under- and overfeeding for the corresponding changes in the anatomi
92 ses in total energy expenditure (TEE) during overfeeding have been small (0.9 +/- 0.8 MJ/d) and accou
93 at, 20% carbohydrate), and high-carbohydrate overfeeding (HCOF) (75% carbohydrate, 5% fat) diets.
94 (STOF) (50% carbohydrate, 30% fat), high-fat overfeeding (HFOF) (60% fat, 20% carbohydrate), and high
98 ently by dietary fat composition and general overfeeding in a randomized trial.We studied the effects
99 is, and regional fat responses to short-term overfeeding in BMI-matched overweight/obese individuals
101 g a period of weight gain as a result of 40% overfeeding in individuals who are inpatients or free-li
109 (photomicrographs) and number in response to overfeeding in upper- and lower-body s.c. fat depots of
110 e of the author with acute weight gain while overfeeding, in the context of current obesity research,
114 extra energy intake.SFA overfeeding and PUFA overfeeding induce distinct epigenetic changes in human
116 -cell hyperplasic effect in a mouse model of overfeeding-induced obesity (OIO) based on direct gastri
119 of food intake that accompanies involuntary overfeeding is an effective regulatory response to posit
120 tin M (OSM) in the metabolic consequences of overfeeding is debated, at least in part, because prior
121 responses to oleic acid following short term overfeeding is likely to contribute to the rapid onset o
122 m a dietary intervention show that 5 days of overfeeding is sufficient to increase the odds of lipid
123 ts for the variability in weight gain during overfeeding is the energy expended in physical activity.
125 re during fasting and smaller increases with overfeeding, lose less weight during caloric restriction
127 Controlling hyperlipidemia and preventing overfeeding may improve outcomes more than tight control
128 increase in 24EE during 200% normal-protein overfeeding measured at baseline (r = 0.91, P = 0.005).
130 The decrease in walking that accompanied overfeeding occurred to a similar degree in the lean (-1
131 also affected by many other factors, such as overfeeding of bees with sucrose, harvesting prior to ma
133 ved in the obese human gut microbiome, where overfeeding of the microbiome creates a dysbiosis detrim
134 ond what is expected with weight gain during overfeeding (OF), has been reported but also refuted.
136 tion potentiates the effects of a short-term overfeeding on intrahepatocellular lipid (IHCL) concentr
138 he 2 groups to examine the overall effect of overfeeding on the DNA methylation in adipose tissue.The
144 Ten healthy subjects were exposed to a 6-d overfeeding period (130% daily energy needs, with 15% ex
145 sessed at baseline, at wk 1, 3, and 6 of the overfeeding period, and 1 wk following overfeeding throu
147 ture during fasting and a larger response to overfeeding predicted more weight loss over 6 weeks, eve
148 sly reported in young lean participants that overfeeding PUFA compared with SFA induced greater lean
152 fat accumulation and body composition during overfeeding saturated fatty acids (SFAs) or polyunsatura
153 o investigate if the differential effects of overfeeding SFA and PUFA on lean tissue accumulation cou
155 During the oral glucose tolerance test, overfeeding significantly increased endogenous glucose p
158 24-h RQ from EBL during fasting and standard overfeeding (STOF) (50% carbohydrate, 30% fat), high-fat
161 ALERIE) weight-loss study, the Bouchard Twin overfeeding study, and the Minnesota Starvation Experime
163 f the overfeeding period, and 1 wk following overfeeding through the use of dual-energy X-ray absorpt
164 n subjects enrolled in a trial of 8 weeks of overfeeding to promote fattening, adipocyte expansion re
167 xposed during our early evolution, including overfeeding, underactivity, aging, artificial lighting a
170 erage weight gain during 6 wk of low-protein overfeeding was 3.8 kg (6.1%, min: +2.5%, max: +8.0%).
175 responses to fasting and 200% normal-protein overfeeding were measured in a whole-room indirect calor
176 gy expenditure responses to fasting and 200% overfeeding were measured in a whole-room indirect calor
180 ) and macronutrient oxidation in response to overfeeding with different types of dietary carbohydrate
181 difference in fat balance during controlled overfeeding with fat, fructose, glucose, or sucrose.
184 at de novo lipogenesis would increase during overfeeding, would vary depending on the type of carbohy