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1 e in S. equi pathogenicity by stimulating an overzealous and inappropriate Th1 response that may inte
2                                              Overzealous antibody responses pit the immune system aga
3                                              Overzealous bronchial smooth muscle constriction is thou
4 is viewpoint will help temper enthusiasm for overzealous conversion of guidelines into performance me
5 response induced by endotoxin is mediated by overzealous cytokine production.
6 tion (AT2 AT1) was critical for mounting the overzealous host immune response in fatal disease; ALO m
7  of N. aromaticivorans, and the promotion of overzealous IFN-gamma- and IL-17-dominated T cell respon
8 ion, suggesting that intestinal damage or an overzealous immune response is not the primary mediator
9 ead damage in the lungs in the setting of an overzealous immune response whose origin remains unclear
10 ed regulatory apparatus to prevent or dampen overzealous immune responses.
11     However, it is equally critical to quell overzealous immune signaling to limit collateral damage
12  or disease susceptibility due to a muted or overzealous immune system, with a primary focus on the c
13 o respond to injury with what is arguably an overzealous immunological response; thus setting the sta
14  that the response the TLR triggers might be overzealous in its attempt to counter the attack by the
15  The immunopathophysiology of sepsis entails overzealous inflammation causing acute organ dysfunction
16 s an inflammatory condition characterized by overzealous inflammation that leads to joint damage and
17     Thus, NLRX1 functions as a checkpoint of overzealous inflammation.
18 dwide, involves concomitant expression of an overzealous inflammatory response and inefficient bacter
19                                           An overzealous inflammatory response is an important cause
20 4) Could unintended medical harm result from overzealous intervention?
21    We also emphasized overt complications of overzealous needling of such a vascular lesion for histo
22 ation, suggesting that it may act to balance overzealous neutrophilic responses.
23  that vulnerable subjects are protected from overzealous or overreaching researchers.
24 ry, together explain human susceptibility to overzealous, pathology-inducing cytokine responses.
25  Acute myocardial infarction (MI) results in overzealous production and infiltration of neutrophils t
26 ng inflammatory responses via suppression of overzealous production of inflammatory cytokines/chemoki
27                                          The overzealous production of proinflammatory cytokines in s
28 erapy was associated with suppression of the overzealous production of tumor necrosis factor alpha, i
29 umoniae pulmonary infection by preventing an overzealous proinflammatory response mediated by TNF-alp
30  cytokines are known to promote fibrosis, an overzealous repair response, but their contribution to h
31 ation must be tightly controlled to preclude overzealous responses that cause self-damage.
32 nflammatory cell recruitment and limiting an overzealous TNF-alpha response.
33          Accumulating evidence suggests that overzealous use of oxygen, leading to hyperoxia, is asso
34                                          The overzealous use of radical nephrectomy for the T1 renal