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1 edded in specialized sporophytic structures (ovules).
2 ete precursors from sporophytic cells in the ovule.
3 y cells located in the abaxial region of the ovule.
4 vents multiple pollen tubes from entering an ovule.
5 g pathways for the correct patterning of the ovule.
6 arity and directing pollen growth toward the ovule.
7 initiated at the base (chalazal end) of each ovule.
8 efore being consumed in the production of an ovule.
9  that are carried within a pollen tube to an ovule.
10  pollen tube cell and the sperm cells to the ovule.
11 owth, and delivery of the sperm cells to the ovule.
12 showed embryo sac-specific expression in the ovule.
13  cell (MMC), a single cell in the premeiotic ovule.
14 attraction of the growing pollen tube to the ovule.
15 he angiosperm carpel and anatropous bitegmic ovule.
16 ced, resulting in homeotic transformation of ovules.
17 expression along the funiculi of the primary ovules.
18 o apical and internal tissues, including the ovules.
19 KD plants produced small fruits with aborted ovules.
20 efore mitosis 3, resulting in 45% of sterile ovules.
21 with the predominant NbSACPD-C expression in ovules.
22 e promoting carpelloid traits in transformed ovules.
23  success by distributing pollen tubes to all ovules.
24 bes failed to enter the micropyle of excised ovules.
25 oductive tract as they seek out unfertilized ovules.
26 g tract, but the tubes failed to turn toward ovules.
27  (>90%) still failed to locate and fertilize ovules.
28  genes exhibiting reduced expression in dif1 ovules.
29 el walls of the gynoecium, which enclose the ovules.
30 quired for efficient pollen tube guidance to ovules.
31 ubes through the pistil tissues to reach the ovules.
32 ead of the two integuments seen in wild-type ovules.
33 and pollen tubes and at much lower levels in ovules.
34 tems, and in developing stamens, carpels and ovules.
35  to fertilize the female gametophytes inside ovules.
36                        In the context of the ovule, 11 genes were expressed exclusively in the antipo
37             We hypothesize that in wild-type ovules a physical interaction between ATS and ETT allows
38 VN) and the SCs migrate as a unit toward the ovules, a fundamental but barely understood process.
39  reach the ovary and in most cases penetrate ovules, a phenomenon called late-acting self-incompatibi
40                            Nearly 90% of the ovules aborted when roots were incubated for 12 h in a h
41 less cultivars suggested a potential role in ovule abortion associated with seedlessness.
42 translation in mitochondria often results in ovule abortion before and immediately after fertilizatio
43 ed, the minimum inductive treatment to cause ovule abortion could be determined.
44 terozygous mcm7 mutants resulted in frequent ovule abortion, a phenotype that does not occur in other
45 llen viability, reduced filament elongation, ovule abortion, and failure of flowers to open.
46                                           In ovules, ACC stimulates transient Ca(2+) elevation, and C
47 o the ovules is a potential barrier point to ovule access and waste by inappropriate mates.
48 ed and display small curled leaves, aberrant ovules, altered epidermal cells and reduced numbers of l
49 d some were able to germinate and target the ovules, although the embryos aborted shortly after ferti
50  suggest that common mechanisms may regulate ovule and anther development.
51 istem indeterminancy, and development of the ovule and seed coat.
52 ides the first evidence for a role of GAs in ovule and seed development.
53 CK (STK), a transcription factor controlling ovule and seed integument identity, directly regulates P
54                        In the context of the ovule and seed, AGL61 is expressed exclusively in the ce
55                                   Within the ovule and seed, FEM111/AGL80 is expressed exclusively in
56 CMM) and its derivative tissues, such as the ovule and the septum, resulting in a split gynoecium and
57 ly regulates AGO9 and RDR6 expression in the ovule and therefore indirectly regulates SPL/NZZ express
58 ering plants, diploid sporophytic tissues in ovules and anthers support meiosis and subsequent haploi
59 expression of these genes in domains of both ovules and anthers where miR167 was normally present.
60 f gene expression, and for fertility of both ovules and anthers.
61 lar tissues and placenta surface, and in the ovules and developing seed.
62 everal studies have proposed that fertilized ovules and developing seeds initiate signaling cascades
63  in stamen, and in the chalazal seed coat of ovules and developing seeds.
64                  These pollen tubes targeted ovules and fertilized either the egg or the central cell
65 so expressed in vascular tissues, developing ovules and stamens, and in the embryo.
66 he whole pistil, while GID1B is expressed in ovules, and GID1C is expressed in valves.
67 al domain of the gynoecium gives rise to the ovules, and several other structures critical for reprod
68 ing mechanism that determines the spacing of ovule anlagen within the placenta remained unexplored.
69    Expression in the inner integument of the ovule appears to be an ancient expression pattern corres
70                                              Ovules are essential for sexual plant reproduction and s
71                                              Ovules are initiated in a highly regular pattern along t
72                        We found that hua-pep ovules are morphologically sepaloid and show ectopic exp
73 volves pattern formation, which ensures that ovules are regularly arranged in the pistils to reduce c
74                                              Ovules are the female reproductive structures that devel
75 ve hap2(gcs1) or duo1 sperm are delivered to ovules, as many as three additional pollen tubes are att
76 iosis while being up-regulated in apomeiotic ovules at the same stage of development in plants of the
77                          Cooperation between ovule attraction and pollen tube growth acceleration fav
78 enetically identical to somatic cells of the ovule because they are products of mitosis, not of meios
79  in the zygote-embryo transition) and failed ovules (because of a moderate defect in female gametophy
80  illustrates the importance of growth of the ovule before fertilization in determining final size of
81                  These loci are expressed in ovules before fertilization and in the seed coat, embryo
82                                    In mom1-3 ovules, both SUF4 and EC1 genes are down-regulated, and
83 are transported through floral tissues to an ovule by a pollen tube, a highly polarized cellular exte
84 ants, two immotile sperm are delivered to an ovule by a pollen tube.
85 s precise delivery of the sperm cells to the ovule by a pollen tube.
86  brassinosteroid (BR) biosynthesis in cotton ovules by treatment with brassinazole inhibits fiber for
87 zygous fie mutants, an endosperm develops in ovules carrying a maternal fie allele without fertilizat
88 al identity of the AI cell relative to other ovule cell types is unclear.
89 our nuclei, and random groups of sporophytic ovule cells not undergoing these events were collected b
90                              The sporophytic ovule cells were enriched in signaling functions.
91                                   Angiosperm ovules consist of three proximal-distal domains - the nu
92 g plants, the embryo sac embedded within the ovule contains the egg cell, whereas the pollen grain co
93 ngation by the application of GA to cultured ovules corresponds with increased expression of genes th
94 on fiber cell development in immature cotton ovules cultured in vitro.
95  ecotypes, and abnormal gamete precursors in ovules defective for RDR6 share identity with ectopic ga
96                           Regular spacing of ovules depends on EPFL2 expression in the carpel wall an
97 y increases in pace with pollination-induced ovule development and is localized in ovule primordia.
98 apping expression pattern during Arabidopsis ovule development and loss of either gene resulted in co
99 volutionary model with pollination-triggered ovule development and megasporogenesis, a modified embry
100                        Furthermore, arrested ovule development and significantly altered lipid compos
101 however, the roles of auxin and cytokinin in ovule development are largely unknown.
102 ttern in inner integuments in early steps of ovule development as well as in the funiculus, embryo, a
103                                          The ovule development defect was fully complemented by coexp
104 le maintaining membrane lipid composition in ovule development for female fertility in N. benthamiana
105 tative orthologs of the Arabidopsis thaliana ovule development gene INNER NO OUTER (INO) has enabled
106                                              Ovule development in Arabidopsis thaliana involves patte
107 rofiling of these genes during the course of ovule development in seeded and seedless cultivars sugge
108 anding of the molecular mechanisms governing ovule development is far from complete.
109                         Here, we report that ovule development is sensitive to the level of Ribosomal
110 ment of both activities to coordinate proper ovule development strongly argues that the ATS-DELLA com
111  this article shows that cytokinin regulates ovule development through the regulation of PIN1.
112 STA SHAPE (ATS, or KAN4) is necessary during ovule development to maintain the boundary between the t
113 ecium morphogenesis, lateral root emergence, ovule development, and primary branch formation.
114 Z, previously described as key regulators of ovule development, are needed for the auxin and cytokini
115 es the effects of the HD-ZIPIII mutations on ovule development, implicating ectopic WUS expression as
116 sed adaxially in the inner integument during ovule development, independent of ABERRANT TESTA SHAPE (
117 in mediating floral meristem determinacy and ovule development, respectively, in Dendrobium spp. orch
118                                       During ovule development, sexual reproduction initiates with me
119 re, we demonstrate the implication of GAs in ovule development.
120 ot formation, delayed flowering and abnormal ovule development.
121 ns between sporophyte and gametophyte during ovule development.
122 rity were expressed during early Arabidopsis ovule development.
123 an formation during embryo, leaf, carpel and ovule development.
124 genes were proposed to play a unique role in ovule development.
125 nd cytokinin receptors, are expressed during ovule development.
126 that polarity determinants play key roles in ovule development.
127 vision in the integument specifically during ovule development.
128 encodes a homeodomain protein that regulates ovule development.
129 t sites have defective anther dehiscence and ovule development.
130 na, MPK3 and MPK6, share a novel function in ovule development: in the MPK6 mutant background, MPK3 i
131 c features, which may be associated with the ovule developmental program common to both organs.
132 nt of the Arabidopsis (Arabidopsis thaliana) ovule develops asymmetrically, with growth and cell divi
133            However, unpollinated lre-5/lre-5 ovules did not initiate autonomous endosperm development
134 expansion, carpel elongation, and anther and ovule differentiation.
135    Morphological transitions associated with ovule diversification provide unique opportunities for s
136                      In the plants that made ovules, ectopic PFS2 expression blocked megaspore mother
137                A sin2 insertional allele has ovule effects similar to sin2-1, but more pronounced ple
138  that HD2A, HD2B, and HD2C were expressed in ovules, embryos, shoot apical meristems, and primary lea
139 ads to a striking phenotype in which ectopic ovules emerge from nodes of ectopic WUS expression along
140 ely determined by the growth of the maternal ovule, endosperm, and embryo.
141 ose transcripts are down-regulated in sexual ovules entering meiosis while being up-regulated in apom
142 in vitro, they failed to fertilize wild-type ovules even in the absence of competing wild-type pollen
143 lAOC-RNAi lines with strongly reduced AOC in ovules exhibited reduced seed set similarly to the jai1
144                                           In ovules, expression in integument tissues was much higher
145  occurring primarily along the region of the ovule facing the base of the gynoecium (gynobasal).
146 ow that the hap2 sperm that are delivered to ovules fail to initiate fertilization.
147  pollen reaches stigmas links pollination to ovule fertilisation, governing subsequent siring success
148         Following successful pollination and ovule fertilization, heat-stress modified PsACS and PsAC
149           Invasive species produced 3x fewer ovules/flower and >250x more flowers per plant, compared
150 s a pollen tube that carries the sperm to an ovule for fertilization.
151 ransmitting tract (TT) and are guided to the ovule for fertilization.
152 uctive tissues to deliver sperm cells to the ovules for fertilization.
153 precise regulation to deliver sperm cells to ovules for fertilization.
154 opulation-level measurements of Genomosperma ovules for quantitative analysis.
155 vents ectopic expression of class-A genes in ovules for their proper morphogenesis, evoking the class
156 s players in the evolution of the unbranched ovule form in extant angiosperms.
157 le in ovule primordia initiation, inhibiting ovule formation in both Arabidopsis and tomato.
158 ificant transport of Cys into the developing ovule from the mother plant.
159  vital meristematic structure that generates ovules from the medial domain of the gynoecium, the fema
160                                Therefore, in ovules, GA perception is mediated by GID1A and GID1B, wh
161 red with those expressed in prefertilization ovules, germinating seedlings, and leaves, roots, stems,
162               Texas Marker-1 (TM-1) immature ovules (GH_TMO).
163 a1 mutants are semisterile and show aberrant ovule growth, whereas double eif4a1 eif4a2 homozygous mu
164                We show that an oversupply of ovules has two opposing influences on pollen limitation
165 is demonstrates that the mpk3(+/-) mpk6(-/-) ovules have abnormal integument development with arreste
166 ied, and genes controlling the initiation of ovules have been identified.
167                             We show that the ovule identity gene, SEEDSTICK, directly regulates AGO9
168                              In Arabidopsis, ovule identity is determined by homeotic MADS-domain pro
169 VAL) and VERDANDI (VDD), both targets of the ovule identity MADS-box complex SEEDSTICK-SEPALLATA3, in
170 tants reduced homeotic conversions, rescuing ovule identity while promoting carpelloid traits in tran
171 s FBP7 and FBP11 are not essential to confer ovule identity.
172 ntained normal seeds and remnants of aborted ovules in a 1:1 ratio.
173  division in the leaf, the gynoecium and the ovules in A. fimbriata.
174 etophyte (egg) competition within individual ovules in addition to male gametophyte (sperm) competiti
175 Here, we demonstrate that stigma, style, and ovules in Arabidopsis pistils stimulate pollen germinati
176 locus analysis, we found that the spacing of ovules in the developing gynoecium and fruits is control
177                                   Angiosperm ovules include one, or more commonly two, integuments th
178 e LPRi epialleles revealed many unfertilized ovules, increased numbers of aborted seeds, and decrease
179 enta mutants resulting in a complete loss of ovule initiation and a reduction of the structures deriv
180  that BRs also participate in the control of ovule initiation in tomato, by promoting an increase on
181 -regulation by GAs and BRs of the control of ovule initiation indicate that two different mechanisms
182 evelopmental stages that immediately proceed ovule initiation, the earliest stages of seed developmen
183 ion mutant has reduced seed set due to outer ovule integument development arrest, leading to female s
184 onse regulators, act as positive factors for ovule integument development in a mechanism that involve
185 er (HD-ZIPIII) genes leads to aberrations in ovule integument development.
186 tor could transmit to leaves, roots, and the ovule integument from which fibers originate.
187 ent to maintain the boundary between the two ovule integuments and to promote inner integument growth
188                                 As a result, ovule integuments had arrested growth, and anthers grew
189 wth of the male gametophytes and pollen tube-ovule interaction.
190 sed by the paracrine RALF34 peptide from the ovule interfering with this signaling pathway.
191 , a single somatic, sub-epidermal cell in an ovule is selected to differentiate into a megaspore moth
192 e step taken by pollen tubes en route to the ovules is a potential barrier point to ovule access and
193 ditionally, over-expression of PHB or PHV in ovules is not sufficient to repress ATS expression, and
194 ing to apomixis initiation in Hieracium spp. ovules is scarce and the functional identity of the AI c
195 opsis female gametophyte in the unfertilized ovule, leading to multinucleate central cells at high fr
196 failure is caused by aberrant development of ovules, leading to gamete degeneration.
197 on in the integuments surrounding mnt mutant ovules, leading to the formation of enlarged seed coats.
198 valuate the hypothesis that an oversupply of ovules leads to increased pollen limitation.
199  found that AGL6-like genes are expressed in ovules, lodicules (second whorl floral organs), paleas (
200                                           In ovules, LORELEI is expressed during pollen tube receptio
201                                 In wild-type ovules, MMC differentiation requires SPOROCYTELESS/NOZZL
202 ide an unique sensitized background to study ovule morphogenesis when C- and D-functions are simultan
203 S expression to the nucellus and maintaining ovule morphology.
204                        In the context of the ovule, MYB98 is expressed exclusively in the synergid ce
205 growth is stunted, limiting fertilisation to ovules nearest the stigma.
206 st, in Arabidopsis both GAs and BRs regulate ovule number independently of the activity levels of the
207                                 BRs regulate ovule number through the downregulation of GA biosynthes
208  (AtHEMN1) adversely affects silique length, ovule number, and seed set.
209  pollen grains/anther, pollen viability, and ovule number.
210 at all four meiotically derived cells in the ovule of Arabidopsis are competent to differentiate into
211 n the inner, sexual whorl, within developing ovules of female flowers and anther primordia of male fl
212 se, CitRWP, is expressed at higher levels in ovules of polyembryonic cultivars.
213 est growth and to rupture after entering the ovules of quintuple loss-of-function EN mutants, indicat
214     In this study, three cDNA libraries from ovules of radish before and after fertilization were seq
215 As showed overall a higher representation in ovules of sexual plants at late premeiosis.
216                                              Ovules of the haploinsufficient er-105 erl1-2 erl2-1/+ m
217                                The branching ovules of the mutant resemble those of some fossil gymno
218 mote fiber elongation in cultured fertilized ovules of the upland cotton variety Coker 312.
219 and genetic mapping was taken to compare the ovules of the Xuzhou 142 wild type (WT) with its fuzzles
220      Moreover, the central tissue within the ovules of Trimenia, through which the embryo sacs grow,
221 these lines rules out alternatives involving ovule or seed mortality and points to a truly meiotic me
222 gynoecia and fruits and irregular spacing of ovules or even ovule twinning.
223 where, following fertilization, cells of the ovule outer integument differentiate into a unique cell
224    As a result of these opposing influences, ovule oversupply has only a modest effect on the degree
225                                          The ovule oversupply hypothesis states that, in response to
226                                       First, ovule oversupply increases the likelihood that pollen re
227                                      Second, ovule oversupply increases the proportion of pollen grai
228                                              Ovule oversupply is not the cause of the pollen limitati
229 Together, EPFL2 and EPFL9 help to coordinate ovule patterning and thereby seed number with gynoecium
230                            Genes controlling ovule patterning have been identified and studied in det
231 analyses presented here, PFS2 affects either ovule patterning or differentiation.
232  a set of ERECTA-family receptors coordinate ovule patterning with fruit growth.
233 in developing ovules, which accounts for the ovule phenotype in pfs2 mutants.
234 ons recapitulated mARF6 and mARF8 anther and ovule phenotypes, indicating that MIR167a is the main mi
235 n grains received that are used to fertilize ovules ('pollen use efficiency').
236 h such features as short siliques with fewer ovules, pollen and seed sterility, altered Megaspore Mot
237                                              Ovule primordia formation is a complex developmental pro
238 ation in tomato, by promoting an increase on ovule primordia formation.
239 entical developmental processes even, as for ovule primordia initiation, if the same set of hormones
240 rellins (GAs) also play an important role in ovule primordia initiation, inhibiting ovule formation i
241  of DELLA proteins that will finally promote ovule primordia initiation.
242 itiation and regular, equidistant spacing of ovule primordia, which may serve to minimize competition
243 icted ARF3 expression to the medio domain of ovule primordia.
244 nduced ovule development and is localized in ovule primordia.
245                               The pattern of ovule production in Impatiens requires the meristem to b
246 elivery of more than one pair of sperm to an ovule, provides a means of salvaging fertilization in ov
247 ypes, including those comprising Arabidopsis ovules, replums and stamen abscission zones.
248 erpretations, including the possibility that ovules represent meristematic axes with their own type o
249 evation, and Ca(2+) influx in octuple mutant ovules rescues LURE1.2 secretion.
250 B-RGL2 interactions only occur in valves and ovules, respectively.
251 down-regulation of SUF4 in homozygous suf4-1 ovules results in reduced EC1 expression and delayed spe
252                                We hybridized ovule RNA probes with Affymetrix ATH1 genome arrays and
253 he vegetative cell, but it is also active in ovules, roots, and guard cells.
254 functioning of the pollination drop, a vital ovule secretion at the pollination stage.
255  histochemical techniques were used to study ovule/seed development and germination of Austrobaileya.
256                                 Genomosperma ovules show significant variation in integumentary lobe
257 understood upstream regulation of SPL/NZZ in ovules, showing that the RdDM pathway is important to re
258 pression in the carpel wall and in the inter-ovule spaces, where it acts through ERL1 and ERL2.
259 hed seed formation in jai1 together with the ovule-specific accumulation of the JA biosynthesis enzym
260 f extant angiosperms, one event produced the ovule-specific D lineage and the well-characterized C li
261 for male gametophyte development, as well as ovule specification and function.
262 -1 function in sporophytic tissues to affect ovule structure and impede embryo sac development, there
263 pecific marker is absent in the multiple ahk ovules, suggesting that disruption of cytokinin signalin
264 th in vitro or in the pistil, but it reduces ovule targeting by twofold.
265 transmitting tract cells before reaching its ovule targets.
266  availability, plants evolve to produce more ovules than they expect to be fertilized, and that this
267 ood that pollen receipt limits the number of ovules that can be fertilized ('prezygotic pollen limita
268 ovides a means of salvaging fertilization in ovules that have received defective sperm, and ensures m
269                              The lre-5/lre-5 ovules that remain undeveloped due to defective pollen t
270  male gametes, while the carpels contain the ovules that when fertilized will produce the seeds.
271                           After targeting an ovule, the pollen tube bursts, releasing two sperm that
272 t tubes appeared to grow far enough to reach ovules, the vast majority (>90%) still failed to locate
273 mitosis up to pollen tube penetration in the ovule, thereby revealing the dynamics of vacuole morphol
274 oth auxin distribution and patterning of the ovule; this process required the homeodomain transcripti
275 which nonmotile sperm cells are delivered to ovules, thus allowing fertilization to occur.
276 ial domain-derived structures, including the ovules, thus validating our approach.
277 e embryo sac is embedded within the maternal ovule tissue, we have utilized the Arabidopsis (Arabidop
278 ne expression profile analysis of Li1 mutant ovule tissues, the gene remains uncloned and the underly
279  previously published microarray data of Li1 ovule tissues.
280 rted and delivered by the pollen tube to the ovule to achieve double fertilization.
281 acting synergid cells persists, enabling the ovule to attract more pollen tubes for successful fertil
282 ident bursting of the pollen tube inside the ovule to release the sperm.
283 s and multiple sets of sperm within a single ovule to show that Arabidopsis efficiently prevents mult
284 ormation and promotes homeotic conversion of ovules to carpels when ectopically expressed in flowers,
285 rains often arrive on stigmas than there are ovules to fertilize, resulting in pollen competition.
286                  Comparative analysis of the ovule transcriptomes of Li1 and WT reveals that a number
287                   Pollen tube arrival at the ovule triggers the accumulation of nitric oxide at the f
288  nearly all pollen tubes failed to reach the ovules; tube growth was arrested at the apex of the ovar
289 uits and irregular spacing of ovules or even ovule twinning.
290 stricted to the gynobasal side of developing ovules via negative regulation by the transcription fact
291 n homozygous-heterozygous plants, 50% of the ovules were arrested at different stages according to th
292 growth to deliver two nonmotile sperm to the ovule where they fuse with an egg and central cell to ac
293 long distances through the carpel toward the ovules, where double fertilization is executed.
294 in signaling localized in the chalaza of the ovule, which is enhanced by the asymmetric localization
295 transcripts were most abundant in developing ovules, which accounts for the ovule phenotype in pfs2 m
296  expression in determinant infertile1 (dif1) ovules, which lack female gametophytes.
297 ort integuments 2-1 (sin2-1) mutant produces ovules with short integuments due to early cessation of
298 ty of different floral tissues to access the ovules within the pistil.
299 thaliana) mutant sporocyteless that produces ovules without embryo sacs, together with the ATH1 Arabi
300 nting multiple pollen tubes from entering an ovule would ensure that only two sperm are delivered to

 
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