ライフサイエンスコーパス: oxalate

1 d measured transcellular secretion of [(14)C]oxalate.

2 by making cations unavailable to precipitate oxalate.

3 emperature and ambient pressure catalyzed by oxalate.

4 tite and goethite at pH 7 in the presence of oxalate.

5 lating absorption or endogenous synthesis of oxalate.

6 ilizing vanadinite and precipitating lead as oxalate.

7 on and renal excretion of both phosphate and oxalate.

8 produce butyrate and three taxa that degrade oxalate.

9 ith CaCO(3) due to the multifunctionality of oxalate.

10 o form insoluble Pb minerals, primarily lead oxalate.

11 of alkaline earth carbonates, sulphates and oxalates.

12 mations of the corresponding N-phthalimidoyl oxalates.

13 r forms, precipitating manganese and calcium oxalates.

14 (10.32% vs 9.65%), ash (5.65% vs 5.05%) and oxalates (0.32% vs 0.22%) content, and exhibited lower A

15 (6.2-69.7% by BSW and 10.6-57.3% by BNW) and oxalate (14.7-88.9% by BSW and 14.5-87.3% by BNW) but sa

16 The current study gives oxalate a role as a family of cathode materials and sugg

17 eria that colonize the human gut and degrade oxalate, a component of most kidney stones.

18 is simple but elegant mechanism explains how oxalate, a molecule that humans and most animals cannot

19 ry oxalate results from passive paracellular oxalate absorption as modified by oxalate back secretion

20 autosomal recessive disease characterized by oxalate accumulation in the kidneys and other organs.

21 In the presence of both DFOB and oxalate, oxalate acted synergistically with DFOB to increase the

22 The formation of insoluble oxalate after incubation at 25 degrees C for 30min is a

23 The presence of oxalate alone caused the release of Cr, but not of Fe, f

24 plexed) Ni(2+) in solution suggests that the oxalate also alters Ni adsorption affinity.

25 ility field, with cationic lead(II) and lead oxalate also occurring.

26 pletely suppressed in the presence of 0.01 M oxalate, an organic ligand that can exist in GCS sites.

27 oxalate, and (4) a mixture of 0.2 M ammonium oxalate and 0.1 M ascorbic acid at 96 degrees C) are app

28 stable isotope infusion protocol of (13)C(2)-oxalate and 1-(13)C-glycolate in both healthy individual

29 with kidney stones containing >=50% calcium oxalate and 44 controls matched for age, sex, and race.

30 rapped: the covalent adducts between TPP and oxalate and between TPP and CO2.

31 effect of potassium citrate on urine calcium oxalate and calcium phosphate supersaturation and stone

32 Furthermore, calcium oxalate and calcium phosphate supersaturation were highe

33 d phosphate levels lead to increased calcium oxalate and calcium phosphate supersaturation.

34 adiolabeling was performed by pumping (89)Zr-oxalate and DFO-Bz-trastuzumab into the microfluidic rea

35 Under P deficiency, the root release of oxalate and flavonoids increased.

36 Method precision was good for oxalate and glycolate (coefficients of variation [CV] we

37 d characterized in relation to color change, oxalate and mineral concentration, and protein digestibi

38 .24mgkg(-1)FW, respectively), in addition to oxalate and phytate (14+/-9and0.17+/-0.02mg/100gFW, resp

39 blanching in plain water reduced the tannin, oxalate and phytate contents but showed higher comparati

40 ess copper, excreted approximately 1.9x more oxalate and produced approximately 1.75x less water-solu

41 Oxalate and sulfate are ubiquitous components of ambient

42 monstrate that competition between dissolved oxalate and the mineral surface for Ni overwhelms the en

43 ion causes the accumulation of intracellular oxalate and the primary hyperoxaluria type 1 (PH1).

44 ures and times is important as it can reduce oxalate and thereby prevent its associated problems.

45 in tomato, a model plant for fleshy fruits; oxalate and threonate are accumulated in leaves, as is o

46 enol, phytic acid, tannins, flavonoids, HCN, oxalate and trypsin inhibitor which were observed in the

47 IL-1B following stimulation with ATP, while oxalate and urate crystal-induced IL-1B release was unaf

48 e selectively abrogated ATP-induced, but not oxalate and urate crystal-induced IL-1B release.

49 Geochemical modelling of the speciation of oxalates and phosphates in the reaction system confirmed

50 ium dihydrogen phosphate, (3) 0.2 M ammonium oxalate, and (4) a mixture of 0.2 M ammonium oxalate and

51 migenes because of its ability to metabolize oxalate, and its potential contribution to protection fr

52 Urine citrate, phosphate, oxalate, and pH levels were higher and urine calcium lev

53 levels; however, the increases in urine pH, oxalate, and phosphate levels lead to increased calcium

54 g of lithogenic substrates, such as calcium, oxalate, and phosphate, and of inhibitors of crystalliza

55 rphous calcium carbonate cystoliths, calcium oxalates, and silica phytoliths.

56 The R-PO(4) pool agrees well with ammonium oxalate (AO)-extractable phosphorus, but only if measure

57 deoxyfluorination via N-hydroxyphthalimidoyl oxalates are also presented, suggesting that this approa

58 Alkyl oxalates are new bench-stable alcohol-activating groups

59 s of those that produce butyrate and degrade oxalate, associates with perturbations of the metabolome

60 racellular oxalate absorption as modified by oxalate back secretion mediated by the SLC26A6 oxalate t

61 neral resource and indicate the potential of oxalate bioprecipitation as a means for efficient biorec

62 Thus, in order to reduce soluble oxalate, bran samples (wheat, oat and barley) and bean s

63 in a dose-dependent manner the synthesis of oxalate by hepatocytes.

64 plexes with cations, which increases soluble oxalate by making cations unavailable to precipitate oxa

65 etermined by atomic absorption spectroscopy, oxalate by titrimetry, phytate and tannin by colorimetri

66 In the continuous phase, the oxidation of oxalate (C(2)O(4)(2-)) produces a strong reducing agent,

67 ositive for concurrent oxidation of DmFc and oxalate (C(2)O(4)(2-)), blip-type responses are observed

68 sulting in lower supersaturation for calcium oxalate, calcium phosphate, and uric acid.

69 suggest that photodissociation of iron(III) oxalate can lead to the formation of volatile oxidation

70 n the reaction system confirmed that pure Ln oxalates can be formed under a wide range of chemical co

71 The dimerized species of CO2(*-), oxalate, can also be determined quantitatively.

72 We report that calcium oxalate (CaOx) crystal deposition led to rapid tubule di

73 FR2 in human and murine kidneys with calcium oxalate (CaOx) nephrocalcinosis-related CKD compared wit

74 an stimulate calcium phosphate (CaP)/calcium oxalate (CaOx) stone formation.

75 Here we show that multivalent anions such as oxalate, carbonate and sulfite can act as powerful laten

76 be stabilized within the conjugated uranium oxalate-carboxylate sheet.

77 elimination in humans to enhance extrarenal oxalate clearance.

78 d mineral speciation as a function of pH and oxalate closely correlated with experimental conditions

79 unable Zr/Cu ratio have been prepared via an oxalate co-precipitation method, showing excellent perfo

80 ed significant reduction (P<0.05) in soluble oxalate compared to bean samples.

81 s smoothly with CO2 to generate the bridging oxalate complex [(TiX3 )2 (mu2 -C2 O4 -kappaO:kappaO'')]

82 s leads to the stabilization of a bismuth(I) oxalate complex and results in a core ion switch.

83 Importantly, iron(III) oxalate complexes absorb near-UV radiation (lambda > 350

84 on photoreactivity in terms of iron-aqua and oxalate complexes, are not in accordance with our result

85 y the photodissociation of aqueous iron(III) oxalate complexes.

86 ggests that this is dominantly the result of oxalate complexing and solubilizing Ni.

87 Notably, ALN-GO1 reduced urinary oxalate concentration up to 50% after a single dose in t

88 hytase in all samples, but effect on soluble oxalate concentration varied.

89 higher the crust color change, the lower the oxalate concentration, and the higher the amount of some

90 mers due to its tendency to increase urinary oxalate concentration.

91 of calcium ions and this influences soluble oxalate concentration.

92 iated with a significant elevation in plasma oxalate concentration.

93 minerals, protein digestibility and reduces oxalate content, the use of high temperatures and times

94 verage) without affecting vegetables growth, oxalate contents and marketable quality.

95 ntion of starch, PEF treatment reduced tuber oxalate contents by almost 50% in some tissues and could

96 With time, a shift to oxalate coordination of bioaccumulated copper occurred i

97 tiripentol protected kidneys against calcium oxalate crystal deposits in acute ethylene glycol intoxi

98 high oxalate production resulting in calcium oxalate crystal formation and deposition in the kidney a

99 Oxalate overproduction may cause calcium-oxalate crystal formation leading to kidney stones, neph

100 h this, deficiency of RIPK3 or MLKL prevents oxalate crystal-induced acute kidney injury.

101 Oxalate crystal-induced renal inflammation is associated

102 Established contributors to oxalate crystal-induced renal necroinflammation include

103 Also in human oxalate crystal-related acute kidney injury, dying tubul

104 -calcium inclusions (CCaI) either as calcium oxalate crystals (CaOx) or amorphous calcium carbonate c

105 he urine results in the formation of calcium oxalate crystals and subsequent kidney stone formation.

106 Calcium oxalate crystals are widespread among animals and plants

107 In combination, calcium oxalate crystals in leaves can act as a biochemical rese

108 Mice with acute oxalosis displayed calcium oxalate crystals inside distal tubular epithelial cells

109 mononucleated cells stimulated with calcium-oxalate crystals, monosodium urate crystals, or ATP lead

110 Most kidney stones are made of calcium oxalate crystals.

111 ellular matrix (ECM) from the fungus, and Ca oxalate crystals.

112 ited progression of kidney injury induced by oxalate crystals.

113 roxaluria) promotes the formation of calcium oxalate crystals.

114 y; and the metabolic pathways for vitamin D, oxalate, cysteine, purines and uric acid.

115 e aggregates which were identified as cerium oxalate decahydrate (Ce(2) (C(2) O(4) )(3) .10H(2) O) an

116 (2) (C(2) O(4) )(3) .10H(2) O) and lanthanum oxalate decahydrate (La(2) (C(2) O(4) )(3) .10H(2) O).

117 Oxalate decarboxylase (OxDC) catalyzes the Mn-dependent

118 ti-walled carbon nanotubes (MWCNT-COOH), and oxalate decarboxylase enzyme (OxDc) immobilized onto a c

119 anced expression level of Cmoxdc1 coding for oxalate decarboxylase.

120 Results show that oxalate decreases particle yield in solution from the re

121 cidity-mimicking phenotypes such as improved oxalate degradation and increased antifungal activity at

122 reas plays a repressor role in regulation of oxalate degradation and possibly antifungal activity of

123 Improved efficacy in oxalate degradation by DeltaCmpacC-29 was consistent wit

124 Representation of genes involved in oxalate degradation showed no significance difference am

125 The oxalate-degrading bacterial taxa identified as decreased

126 All species examined have abundant calcium oxalate deposits around the veins.

127 ents revealed severe retinal alterations and oxalate deposits, including macular crystals and hyperpi

128 The natural history of (sub)retinal oxalate deposits, the pathogenesis of subretinal fibrosi

129 te-minus mutants, whereas supplementing with oxalate did not.

130 ia, and extensive precipitation of magnesium oxalate dihydrate (glushinskite, Mg(C(2) O(4) ).2H(2) O)

131 elial pellets and transformed into manganese oxalate dihydrate (lindbergite) and calcium oxalate dihy

132 oxalate dihydrate (lindbergite) and calcium oxalate dihydrate (weddellite).

133 late resulting in precipitation of magnesium oxalate dihydrate which also accumulated within the myce

134 As a result, the hydrogenation of dimethyl oxalate (DMO) to ethylene glycol, an important reaction

135 Aspergillus niger also precipitated lead oxalate during growth in the presence of lead carbonate,

136 similar mechanisms contribute to intestinal oxalate elimination in humans to enhance extrarenal oxal

137 on of CFTR stimulated SLC26A6-mediated Cl(-)-oxalate exchange in Xenopus oocytes.

138 Increased urinary oxalate excretion (hyperoxaluria) promotes the formation

139 es CM significantly reduced (>32.5%) urinary oxalate excretion and stimulated (>42%) distal colonic o

140 24 of 26 patients had steatorrhea and urine oxalate excretion averaged 69 mg/day, with a positive co

141 of the study was to determine whether urine oxalate excretion correlates with elements of fat balanc

142 eceived Stiripentol for several weeks: urine oxalate excretion decreased by two-thirds.

143 The reduction in urinary oxalate excretion in hyperoxaluric mice treated with O.

144 l administration reduced significantly urine oxalate excretion in rats.

145 Twenty-four hour urine oxalate excretion is an inaccurate measure for endogenou

146 d treated with Stiripentol had a lower urine oxalate excretion than control patients.

147 Average urine oxalate excretion was 61 mg/day; there was no correlatio

148 Fecal oxalate excretion was enhanced in wild-type mice with CK

149 Fat balance and urine oxalate excretion were measured simultaneously in 26 sev

150 oxalate secretion, thereby reducing urinary oxalate excretion, via an unknown secretagogue.

151 inhibitor (LDH5) stiripentol reduces urinary oxalate excretion.

152 th primary hyperoxaluria reduced the urinary oxalate excretion.

153 reduce hepatic oxalate production and urine oxalate excretion.

154 s no correlation between fecal fat and urine oxalate excretion.

155 ysis and lead oxalate formation depending on oxalate excretion.

156 tive correlation between fecal fat and urine oxalate excretions (r = 0.71, P < .001).

157 te acetylhydrolase gene in H915-1 eliminated oxalate formation but neither influence on pH decrease n

158 formation reaction commonly anticipated for oxalate formation by reductive coupling of CO2 on low-va

159 ing on organic phosphate hydrolysis and lead oxalate formation depending on oxalate excretion.

160 olubilization, element release and secondary oxalate formation, relevant to the biogeochemical cyclin

161 ent oxalate oxidoreductase (OOR) metabolizes oxalate, generating two molecules of CO2 and two low-pot

162 Isotopic enrichments of plasma oxalate, glycolate, and glyoxylate were measured on a ga

163 hich in addition to iron redox activity, the oxalate group itself also shows redox behavior enabling

164 were defined as fecal fat >7 g/day and urine oxalate >40 mg/day.

165 e coupled in a similar fashion through their oxalate half esters.

166 A state of oxalate homeostasis is maintained in patients with healt

167 ophiles such as ethyl glyoxalate and diethyl oxalate in aqueous medium leads to the formation of benz

168 Soluble oxalate in foods is major concern for kidney stone forme

169 addition to the well-characterized C2O4(2-) oxalate in Li2C2O4 viable covalent CO2-based nets emerge

170 is critical in decreasing the body burden of oxalate in murine CKD models.

171 The presence of iron oxalate in seed aerosol is found to inhibit aerosol grow

172 ading to the deposition of insoluble calcium oxalate in the kidney.

173 Excessive excretion of oxalate in the urine results in the formation of calcium

174 Thus, oxalate increases the relative reactivity ratio of bioti

175 umulation of weathered calcium with secreted oxalate, increasing significantly in sequence: quartz, g

176 understand the molecular pathophysiology of oxalate-induced AIK, we conducted in vitro studies in mo

177 necrosis and necroptosis both contribute to oxalate-induced AKI, identifying PPIF as a potential mol

178 permeability transition displayed attenuated oxalate-induced AKI.

179 te defects in growth, stress resistance, and oxalate/insecticidal compound production, only a small d

180 Before and after RYGB, high oxalate intake contributed to the severity of hyperoxalu

181 ed activity of oxalate oxidase that converts oxalate into CO2 Similar results were also observed unde

182 bon radicals from tert-alkyl N-phthalimidoyl oxalates is proposed that is based on earlier pioneering

183 icrobial communities and early-onset calcium oxalate kidney stone disease is unknown.

184 upstream determinants of early-onset calcium oxalate kidney stone disease.

185 1) in two unrelated individuals with calcium oxalate kidney stones.

186 tial contribution to protection from calcium oxalate kidney stones.

187 (AGT), which allows glyoxylate oxidation to oxalate leading to the deposition of insoluble calcium o

188 The oxalate leaving group of oxaliplatin is not required for

189 ecretion, Cftr(-/-) mice had serum and urine oxalate levels 2.5-fold greater than those of wild-type

190 Serum oxalate levels suddenly increase with certain dietary ex

191 e relationship between succinate and citrate/oxalate levels, we assessed blood and urine levels of me

192 bers and if PEF treatment could reduce tuber oxalate levels.

193 -beyerane scaffold is required along with an oxalate-like group at C-18/C-19 or a sugar residue at C-

194 ilibrated with aqueous solutions of acetate, oxalate, malonate, or citrate at 50 degrees C and 90 bar

195 The oxalate-minus mutants accumulated fumaric acid, produced

196 Here, we generated oxalate-minus mutants of S. sclerotiorum using two indep

197 The oxalate-minus mutants retained pathogenicity on plants,

198 ng the host tissue enhanced virulence of the oxalate-minus mutants, whereas supplementing with oxalat

199 catalyzes the Mn-dependent conversion of the oxalate monoanion into CO2 and formate.

200 Some calcium oxalate monohydrate (whewellite) was formed with natural

201 Here we report that crystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydra

202 Crystals of calcium oxalate, monosodium urate, or calcium pyrophosphate dihy

203 This investigation shows the oxalate motif is maintained when the concentration of li

204 eased incidence of hyperoxaluria and calcium oxalate nephrolithiasis.

205 ion mechanism in kidneys in a mouse model of oxalate nephropathy accompanying lysosomal damage.

206 lene glycol intoxication and chronic calcium oxalate nephropathy models.

207 lay progressive forms of nephrocalcinosis in oxalate nephropathy, such as primary hyperoxaluria.

208 les as a fundamental initiating mechanism of oxalate nephropathy.

209 in renal pathology are nephrocalcinosis and oxalate nephropathy.

210 ons with fumaric and muconic acids show that oxalate (not sulfate) and pH are determining factors in

211 could potentially aid the development of low oxalate oca-based foods.

212 E were efficiently precipitated as Ce and La oxalates of high purity, and did not contain Nd, Pr and

213 the effects of organic ligands (acetate and oxalate) on biotite dissolution and surface morphologica

214 uconic acids with Fe(III) in the presence of oxalate or sulfate.

215 Feeding a diet high in soluble oxalate or weekly injections of aristolochic acid induce

216 Oxalate overproduction may cause calcium-oxalate crystal

217 In the presence of both DFOB and oxalate, oxalate acted synergistically with DFOB to incr

218 Several forms of InsP(n) in the oxalate-oxalic acid extracted sediment were identified.

219 )) in sediment samples was tested, utilizing oxalate-oxalic acid extraction followed by determination

220 Tomato waste was treated with ammonium oxalate/oxalic acid by conventional extraction (CE), und

221 An oxalate oxidase assay measured fecal and P(ox) concentra

222 l decomposition and by increased activity of oxalate oxidase that converts oxalate into CO2 Similar r

223 riginating enzymes, i.e., glycolate oxidase, oxalate oxidase, and catalase.

224 Thiamine pyrophosphate (TPP)-dependent oxalate oxidoreductase (OOR) metabolizes oxalate, genera

225 However, as GFR declines, plasma oxalate (P(ox)) concentrations start to rise.

226 Cl), only produced by P. javanicus, and lead oxalate (PbC2O4), produced by A. niger and P. javanicus.

227 ated with higher urinary citrate, magnesium, oxalate, phosphate, uric acid, volume, and pH, and lower

228 cium than other GLVs, because of low fibres, oxalate, phytate and tannin content.

229 toxic substances (nitrate, nitrite, cyanide, oxalate, phytate, and trypsin inhibitor) in tubers of Je

230 identify a means for biorecovery of REE via oxalate precipitation the bioleaching and bioprecipitati

231 simple tertiary alcohol-derived homoallylic oxalate precursors.

232 Alkyl oxalates, prepared from their corresponding alcohols, ar

233 ether calcium would combine with the soluble oxalate present in the spinach.

234 izosphere are able to protect plants against oxalate-producing pathogens by a combination of actions

235 otrophic bacteria can protect plants against oxalate-producing pathogens by a combination of actions

236 n UV-induced mutants that concomitantly lost oxalate production and pathogenicity.

237 Stiripentol would potentially reduce hepatic oxalate production and urine oxalate excretion.

238 tion is an inaccurate measure for endogenous oxalate production in PH patients and not applicable in

239 demonstrate the ability of ALN-GO1 to reduce oxalate production in preclinical models of PH1 across m

240 ency in this enzyme leads to abnormally high oxalate production resulting in calcium oxalate crystal

241 s isoenzyme is also the last step of hepatic oxalate production, we hypothesized that Stiripentol wou

242 m characterized by an increase in endogenous oxalate production.

243 5 and 14.13 percent in phytin phosphorus and oxalate respectively.

244 (Mg(2+) ), the latter reacting with excreted oxalate resulting in precipitation of magnesium oxalate

245 Net intestinal absorption of dietary oxalate results from passive paracellular oxalate absorp

246 When fed an oxalate-rich diet, wild-type mice developed progressive

247 We demonstrate that compounds S9 and its oxalate salt S9OX interfere with FOXO3 target promoter b

248 loropurine provided crystalline 2AMTA as the oxalate salt.

249 While many metal oxalate salts are known, few are known to form zeolite-t

250 es were readily prepared and isolated as the oxalate salts in high yield and high purity.

251 eolite types by reporting a family of indium oxalate salts with multiple zeolite topologies, includin

252 f researchers have described augmentation of oxalate secretion in the colon in models of CKD, but the

253 Slc26a6-mediated enteric oxalate secretion is critical in decreasing the body bur

254 to test the hypothesis that SLC26A6-mediated oxalate secretion is defective in cystic fibrosis.

255 We conclude that defective intestinal oxalate secretion mediated by SLC26A6 may contribute to

256 e exhibited significantly less transcellular oxalate secretion than intestinal tissue of wild-type mi

257 ation with the profound defect in intestinal oxalate secretion, Cftr(-/-) mice had serum and urine ox

258 with colonic epithelium and induces colonic oxalate secretion, thereby reducing urinary oxalate excr

259 cretion and stimulated (>42%) distal colonic oxalate secretion.

260 dentify the derived factors inducing colonic oxalate secretion.

261 For the first time, we show that oxalate selectively attacks edge surface sites and enhan

262 nsistent 100% recovery, at pH 4, using 0.1 M oxalate solution as an effective stripping agent.

263 The adsorbent is fully regenerable, using oxalate solution.

264 contribute to the increased risk for calcium oxalate stone formation observed in patients with cystic

265 t affects BP and biochemical risk of calcium oxalate stone formation, thus providing a potential mole

266 e relationship of gut microbiota and calcium oxalate stone has been limited investigated, especially

267 rols, patients with occasional renal calcium oxalate stones (OS) and patients with recurrent stones (

268 rovided Sprague Dawley rats of renal calcium oxalate stones with antibiotics and examined the renal c

269 e low urinary citrate and to develop calcium oxalate stones) had a 40% decrease in urinary excretion

270 However, oxalate strongly enhanced biotite dissolution and induce

271 ers, dolphins and ferrets, that form calcium oxalate, struvite, uric acid, cystine and other stone ty

272 colate oxidase, to deplete the substrate for oxalate synthesis.

273 itively correlated with genes involvement in oxalate synthesis.

274 grandiflora had a very high content of total oxalates, tannins and dietary fibers, which reduced calc

275 ding affinity are due to the formation of Ni-oxalate ternary surface complexes.

276 ormed in one-pot reactions containing (89)Zr-oxalate, the photoactive chelate desferrioxamine B (DFO)

277 Upon reaction with DFOB or DFOB + oxalate, the remaining solids became enriched in Cr rela

278 FDA-approved drugs tramadol and escitalopram oxalate, they release or uptake serotonin in a dose- and

279 Oxalate thus largely enhances Ni mobility, thereby incre

280 The molar ratios of phytate or oxalate to minerals (calcium and zinc) in all composite

281 The addition of oxalate to the reactions of Fe(III) with all organics, e

282 so significantly restricted the induction of oxalate transport by CM.

283 rmigenes-derived bioactive factors stimulate oxalate transport in intestinal cells through mechanisms

284 fonic acid completely blocked the CM-induced oxalate transport.

285 Knockdown of the oxalate transporter SLC26A6 also significantly restricte

286 The oxalate transporter Slc26a6 is a candidate for contribut

287 alate back secretion mediated by the SLC26A6 oxalate transporter.

288 etion in the colon in models of CKD, but the oxalate transporters remain unidentified.

289 m, O. formigenes CM significantly stimulated oxalate uptake (>2.4-fold), whereas CM from Lactobacillu

290 ture conditioned medium (CM) on apical (14)C-oxalate uptake by human intestinal Caco-2-BBE cells.

291 Oxalate-vanadium complexes markedly reduced the vanadini

292 contributing to the extrarenal clearance of oxalate via the gut in CKD.

293 Lead oxalate was precipitated by Aspergillus niger during bio

294 In spinach, [(14)C]oxalate was the major product of [(14)C]ascorbate degrad

295 The 3,3-diarylazetidine oxalates were then easily converted into N-alkyl and N-a

296 lated to give the tert-alkyl N-phthalimidoyl oxalate, which in the presence of visible-light, catalyt

297 st in RS patients with high level of urinary oxalate, which was positively correlated with genes invo

298 h is known to contain significant amounts of oxalates, which are toxic and, if consumed regularly, ca

299 dical coupling of tert-alkyl N-phthalimidoyl oxalates with electron-deficient alkenes is terminated b

300 CO2(*-) produced can either dimerize to form oxalate within the nanogap between SECM tip and substrat