ライフサイエンスコーパス: oxidase

1 nicotinamide adenine dinucleotide phosphate oxidases).

2 he progression of EP and the levels of NADPH oxidase.

3 ophils by the electrogenic activity of NADPH oxidase.

4 coacervate micro-droplets containing glucose oxidase.

5 ctivities of citrate synthase and cytochrome oxidase.

6 , mixed lineage kinase-like (MLKL) and NADPH oxidase.

7 zed in other enzymes, including cytochrome c oxidase.

8 by the copper-containing bovine serum amine oxidase.

9 regulates the abundance of a conserved NADPH oxidase.

10 species generation by neutrophil NOX2 NADPH oxidase.

11 g the fallacy of DET to FAD-dependent native oxidases.

12 with high sequence similarity to known GA 3-oxidases.

13 nction mutations in the peroxisomal acyl-CoA oxidase 1 (ACOX1) gene cause neurodegeneration via disti

14 Acyl-CoA oxidase 1 (Acox1), the enzyme that catalyzes the first s

15 absent in mice that lacked epithelial NADPH oxidase 1 (NOX1) activity.

16 n of TLR4, dual oxidase 2 (DUOX2), and NADPH oxidase 1 (NOX1).

17 of inducible nitric oxide synthase and NADPH oxidase 1 enzymes at myoendothelial projections in obese

18 ACOX1 (acyl-CoA oxidase 1) encodes the first and rate-limiting enzyme of

19 M (apolipoprotein M), PCYOX1 (prenylcysteine oxidase 1), PON1, and APOE correlate with stroke recover

20 arget oxygen free radicals produced by NADPH oxidase-1 (Nox1)(3,6) that otherwise elicited ER stress

21 s were analyzed for expression of TLR4, dual oxidase 2 (DUOX2), and NADPH oxidase 1 (NOX1).

22 acing model of AF, we demonstrate that NADPH oxidase 2 (NOX2) generated oxidative injury causes upreg

23 nal intrinsic signaling axis PKC-STAT3-NADPH oxidase 2 (NOX2), enhancing redox signaling as shown by

24 nks diastolic stretch to generation of NADPH oxidase 2 (NOX2)-dependent reactive oxygen species (ROS)

25 otubule-dependent mechanoactivation of NADPH oxidase 2 (NOX2)-generated reactive oxygen species (ROS)

26 ular Ca2+ signaling and a reduction of NADPH oxidase 2 (NOX2)/ROS production.

27 gene, which encodes a component of the NADPH oxidase 2 complex that mediates neutrophil oxidative bur

28 altered calcium release dynamics, how NADPH oxidase 2 is activated by and responds to stretch, and f

29 RPHOGENIC DWARF, DWF4, and BRASSINOSTEROID-6-OXIDASE 2 Phenotypic characterization of knockout and kn

30 ure, glucose, F(2)-isoprostanes, NOX2 (NADPH oxidase 2), and PKC (protein kinase C) were measured in

31 ction) and rs269868-C/Ser1067 in DUOX2 (dual oxidase 2, related to thyroid function and innate immuni

32 n pathway linking diastolic stretch to NADPH oxidase 2-derived reactive oxygen species signals that r

33 gulated pro-oxidative enzymes, such as amine oxidase 3/vascular adhesion protein 1 (AOC3/VAP1) as wel

34 stabilizes 1-aminocyclopropane-1-carboxylate oxidase 4 (ACO4), which may in part explain increased et

35 uggests a role for stromally expressed NADPH oxidase 4 (NOX4) as a modulator of reactive oxygen speci

36 NADPH oxidase 4 (NOX4) is the most abundant NOX isoform in the

37 sion of FOXO transcription factors and NADPH oxidase 4 (Nox4), a key regulator of reactive oxygen spe

38 (NOX)4 promoter and induction of NOX4 (NADPH oxidase 4) expression.

39 enic, and pro-oxidant activity via the NADPH oxidase 4.

40 oxygen species (ROS)-producing enzyme NADPH oxidase-4 (Nox4) in the pathogenesis of DPN.

41 ive oxygen species (ROS)-forming Nox5 (NADPH oxidase 5).

42 RNA), acting as a regulator of the monoamine oxidase A (MAOA) gene in the brain, and named it MAOA-as

43 tify for the first time a role for monoamine oxidase A (MAOA) in NPC.

44 Here, we show that upregulation of monoamine oxidase A (MAOA), a mitochondrial enzyme that degrades m

45 GABA suppressed polyamine oxidase, a gene related to polyamine catabolism, when pl

46 In this setting, NADPH oxidase, a source of free radicals, decreased in the myo

47 we report that high expression of monoamine oxidase-A (MAO-A) is associated with positive ARv7 detec

48 ation is mediated in part by microglia NADPH oxidase activation, and this is alleviated by the CNS-pe

49 se subunit p47(phox), which results in NADPH oxidase activation.

50 PK activation but proximal to PI3K and NADPH oxidase activation.

51 soluble quinones, peroxidase and polyphenol oxidase activities in contrast to control.

52 te kinase, creatine kinase, and cytochrome c oxidase activities, and increased myoglobin content.

53 be readily adapted for measurements of other oxidase activities.

54 Ox A96L/N212K mutant, which is minimized its oxidase activity and b-type cytochrome protein was const

55 ot detected, pDCs regulated neutrophil NADPH oxidase activity and conidial killing.

56 lectrochemical method to investigate cuprous oxidase activity and to understand the physiological rol

57 llows us to attribute this wave to a cuprous oxidase activity displayed by the laccase and induced by

58 tylcholine in aortic rings and reduced NADPH oxidase activity in DOCA-salt animals.

59 leviated, suggesting that dysregulated NADPH oxidase activity is a key factor promoting autoinflammat

60 , and Thr(202), respectively, conferred GA 3-oxidase activity to CsGA1ox/ds and thereby augmented its

61 m resulted in lower ethylene production, ACC oxidase activity, membrane permeability, <beta>-galactos

62 g phenotypes, including enhancement of NADPH oxidase activity, shedding of l-selectin, or mobilizatio

63 iated dehydrogenase activity with negligible oxidase activity, showing quasi-DET properties after PES

64 CblC suppressed the NO(2)Cbl-dependent thiol oxidase activity, whereas the disease-associated R161G v

65 f these three conserved amino acids for GA 3-oxidase activity.

66 onally, AtGA2ox9 and AtGA2ox10 exhibit GA 20-oxidase activity.

67 ata suggested she has <2% residual glycolate oxidase activity.

68 coa improved mitochondrial COX (cytochrome c oxidase) activity (P=0.013), increased capillary density

69 variant in which these three canonical GA 3-oxidase amino acids were changed to the ones present in

70 measured the oxidation state of cytochrome c oxidase, an intracellular measure of metabolism, in huma

71 working electrode was modified with glucose oxidase and dihexadecyl phosphate film in order to obtai

72 zymatic reactions catalyzed by AChE, choline oxidase and horseradish peroxidase, leading to productio

73 tments suppressed the activity of polyphenol oxidase and increased activity of superoxide dismutase e

74 The device comprises a lactate oxidase and osmium-polymer -based anode connected to a c

75 mes including papain, alpha-amylase, glucose oxidase and phytase stabilized dough structure to facili

76 cGMP, which prompts the stimulation of NADPH oxidase and protein kinase C (PKC).

77 ally, we demonstrate that anaerobic arsenite oxidase and respiratory arsenate reductase catalytic sub

78 the thylakoid-localized respiratory terminal oxidases and CytM (DeltaCox/Cyd/CytM) displayed a phenot

79 cleotide (FAD) dependent family of monoamine oxidases and is vital in regulation of mammalian biology

80 s previously trapped in ferritin-like diiron oxidases and oxygenases (FDOs) and, more recently, the H

81 f the emerging superfamily of HO-like diiron oxidases and oxygenases (HDOs).

82 than 40 enzymes, prominently including NADPH oxidases and the mitochondrial electron transport chain.

83 e hits, TraesCS4D02G352200 (TaNox8; an NADPH oxidase) and TraesCS4D02G350300 (a rhomboid-like protein

84 FIS1 encodes a GA2-oxidase, and its mutation leads to increased bioactive g

85 mination through the action of MPO and NADPH oxidase, and restricts fungal growth through NET release

86 ioenergetics predominantly via cytochrome bd oxidase, and that H(2)S reverses *NO-mediated inhibition

87 zymes such as myeloperoxidase (MPO) or NADPH oxidase, and the release of neutrophil extracellular tra

88 proach for electrochemical tuning of alcohol oxidase (AOx) and alcohol dehydrogenase (ADH) biocatalys

89 cold conditioning, activation of alternative oxidase (AOX) occurs pre-climacterically.

90 expression of Ciona intestinalis alternative oxidase (AOX)(12), which also oxidizes ubiquinol to ubiq

91 ytochrome-mediated pathway to an alternative oxidase (AOX), an increase in proline consumption, eleva

92 ciated with the loss of function of aldehyde oxidase (AOX1).

93 Aldehyde oxidases (AOXs) are a small group of enzymes belonging t

94 Milk oxidases are an integral part of milk immune system, and

95 Current assay methods for milk oxidases are either insensitive, tedious or not cost-eff

96 e bimetallic active sites in the heme-copper oxidases are reviewed.

97 Heme-copper oxidases are transmembrane enzymes involved in aerobic a

98 a family of cell wall-localized multicopper oxidases, are involved in lignin biosynthesis.

99 Using glucose oxidase as a functional protein core, we show activity-b

100 An oxalate oxidase assay measured fecal and P(ox) concentrations.

101 competitive antagonist AMG-21629, the NADPH oxidase assembly inhibitor apocynin, and the reactive ox

102 ssembly proteins)1/2 and COX10 (cytochrome c oxidase assembly protein-10), indicating endothelial imp

103 siae) and human (Homo sapiens) mitochondria, Oxidase assembly protein1 (Oxa1) is the general insertas

104 e into the inner membrane while Cytochrome c oxidase assembly protein18 (Cox18/Oxa2) is specifically

105 ed direct electron transfer (DET) to glucose oxidase at carbon nanotubes (CNT).

106 Previously unknown for GA 2-oxidases, AtGA2ox9 performs 2alpha-hydroxylation of C(19

107 th an active proton-translocating cytochrome oxidase, ATP synthesis readily occurred at the measured,

108 Illudinine was found to inhibit monoamine oxidase B (MAO-B) with an IC(50) of 18 +/- 7.1 muM in pr

109 eroxide (H(2)O(2)) originated from monoamine oxidase B in severe reactive astrocytes causes glial act

110 Monoamine oxidase-B inhibitors and dopamine agonists can be reserv

111 oring by bioaffinity of biotinylated glucose oxidase (biot-GOx) as proof-of-concept of the analytical

112 Bilirubine oxidase (BOx) immobilized on MWCNT coated GCE (GCE|BOx)

113 that the RIPK3-MLKL pathway activates NADPH oxidase but requires, in addition to p38 MAPK and PI3K,

114 e lowering of enzymatic activity of all four oxidases by CNT.

115 neage of DMSORs compared to aerobic arsenite oxidase catalytic subunits, which evolved from the assim

116 represents the first crystal structure of an oxidase catalyzing direct, intermolecular C-H amination.

117 and by inhibiting mitochondrial cytochrome c oxidase (CcO), thereby decreasing oxygen delivery and in

118 onsume, reducing it to water by cytochrome c oxidase (CcO).

119 n the activity of mitochondrial Cytochrome-C Oxidase (CCO).

120 ccinate dehydrogenase (SDH) and cytochrome c oxidase (CCO).

121 e largest subgroup contains the cytochrome c oxidases (CcO), which reduce molecular oxygen to water.

122 alcohol (AOx), pyranose (PyOx), and choline oxidase (ChOx).

123 In Saccharomyces cerevisiae, cytochrome c oxidase (CIV) forms SCs of varying stoichiometry with cy

124 ochondria and histochemistry of cytochrome c oxidase (CO) activity were examined in pre-BotC neurons

125 r jaw skin surface was developed, cytochrome oxidase (CO) was used to label flattened-cut LJBSF secti

126 The NADPH oxidase complex (NOX) produces reactive oxygen species (

127 be human COX6B2, a component of cytochrome c oxidase (complex IV).

128 aerobes and one confirmed obligate anaerobe, oxidase complexes (fox, sox, dox and a new putative cyto

129 the latest data on the pathobiology of each oxidase component, the complex crosstalk between differe

130 ent, the complex crosstalk between different oxidase components and the consequences of this crosstal

131 enes, predicted to encode a cytochrome bd-II oxidase, conferred a fitness advantage for E. coli in ch

132 y mitochondrial genes including cytochrome C oxidase copper chaperone (COX17) and ATP Synthase, H(+)

133 (3) compared to analogous steps in the aa(3) oxidases could reflect evolutionary adaptation of the en

134 was associated with the gain of cytochrome c oxidase (COX) function, and the COX subunit, COX4, was l

135 encoding an assembly factor of cytochrome c oxidase (COX) specifically in mouse ECs, providing a mod

136 aquo-cob(III)alamin or enter a futile thiol oxidase cycle forming GSH disulfide.

137 ied the time course of changes of cytochrome oxidase (CytOx) blob spatial density and blob cross-sect

138 glucose consumption while inhibition of NOX2 oxidase decreased glucose consumption.

139 ized with neurodegeneration and cytochrome c oxidase deficiency.

140 Using different NADPH oxidase-deficient mice, we show that TSPO is a key regul

141 In the absence of high-affinity cytochrome c oxidases, deletion of mhr no longer caused a fitness dis

142 e presence of glucose and FcMeOH(+), glucose oxidase delivers electrons to FcMeOH(+), and the potenti

143 usion enzyme between an engineered l-lactate oxidase derived from Aerococcus viridans, AvLOx A96L/N21

144 d was mediated via their production of NADPH oxidase-derived reactive oxygen species and MMP-9.

145 ese experiments provide direct evidence that oxidase-derived ROS promotes the growth of leukemia cell

146 mutant defective in two peroxisomal acyl-CoA oxidases does not metabolize ascr#18 and does not repel

147 id rapidly attracted leukocytes through dual oxidase (Duox) and 5-lipoxygenase (Alox5a).

148 red energy generation caused by cytochrome c oxidase dysfunction in the mitochondria.

149 During our genetic functional analysis of oxidase-encoding genes in the everninomicin producer Mic

150 We also report a novel role for the NADPH oxidase enzymes (NOXs), namely NOX1, and NOX-derived rea

151 The case study of four FAD-dependent oxidase enzymes is presented in the context of the often

152 th a focus on laccases which are exocellular oxidase enzymes produced by this fungus during fruit con

153 DUOX1, an NADPH oxidase family member, catalyzes the production of hydro

154 The flavin-dependent amine oxidase (FAO) superfamily consists of over 9000 nonredun

155 MAOB, a crucial monoamine oxidase for dopamine metabolism, triggers oxidative stre

156 an regenerate NAD+ by expression of the NADH oxidase from Lactobacillus brevis (LbNOX)(13) targeted t

157 The quinoprotein glycine oxidase from the marine bacterium Pseudoalteromonas lute

158 and a flavin mononucleotide (FMN)-dependent oxidase (Fub9) in synthesizing the picolinic acid scaffo

159 Galactose oxidase (GAO) contains a Cu(II)-ligand radical cofactor.

160 Activated NADPH oxidase generates reactive oxygen species and elevated o

161 he Anr-controlled high-affinity cytochrome c oxidase genes.

162 probe by covalently immobilizing the glucose oxidase (GOD) enzyme onto an ultramicro electrode (UME)

163 In-channel immobilization of glucose oxidase (GOx) at the DLC surface was realized through ca

164 n-microcapsule system is loaded with glucose oxidase (GOx) in the inner aqueous compartment and insul

165 uction of H(2)O(2) in honey requires glucose oxidase (GOx) that oxidizes glucose to gluconolactone an

166 Glucose oxidase (GOx) was chosen as a model bioreceptor to valid

167 ne hemoglobin, immunoglobulin G, and glucose oxidase (GOx)), which have different shapes, charges, an

168 etection of pathogens coupled with a glucose oxidase (GOx)-based electrochemical readout is reported.

169 gene encoding the catalytic subunit of NADPH oxidase gp91phox.

170 zymes to program a spatially coupled glucose oxidase/haemoglobin reaction cascade, which in the prese

171 ochrome P450 3A4 (CYP3A4) and human aldehyde oxidase (hAOX) for more in-depth data interpretation, an

172 The catalytic mechanism of the organic oxidases has been elucidated, and the high TOF is achiev

173 roduced by tissue-specific respiratory burst oxidase homolog (RBOH) enzymes to drive development.

174 ly bind to the promoter of RESPIRATORY BURST OXIDASE HOMOLOG 1 (RBOH1), and that RBOH1-mediated ROS p

175 ies further identified the RESPIRATORY BURST OXIDASE HOMOLOG D (RBOHD) protein as a key player in med

176 ic stress, the key role of respiratory burst oxidase homologs in the integration of ROS and hormone s

177 ansferase, 1-aminocyclopropane-1-carboxylate oxidase homologs, receptor-like protein kinases were exp

178 n sequencing of the mitochondrial cytochrome oxidase I (COI) 'barcode' gene, which remains costly for

179 Cytochrome oxidase I (COI) mitochondrial DNA sequences were generat

180 NA and sequenced mitochondrial (cytochrome c oxidase I [mtCOI]) and nuclear (small subunit 18S rRNA [

181 We sequenced a portion of the cytochrome oxidase I gene for 432 gnathiids, collected from six loc

182 troformation approach to reconstitute bo (3) oxidase in giant unilamellar vesicles made of PDMS-g-PEO

183 d to elevated oxidative stress through NADPH oxidase in lineage-traced microglia.

184 Inactivation of NOX2 NADPH oxidase in Pstpip2(cmo) mice did not affect IL-1beta lev

185 unction that directly activates cytochrome c oxidase in the mitochondria and functions in the nucleus

186 Succinate alone primed the PMN oxidase in vitro at physiologically relevant levels.

187 nt to prevent confounding bovine serum amine oxidase-induced cytotoxicity in mechanistic studies of t

188 NADPH oxidase inhibition did not prevent cross-talk inhibition

189 A xanthine oxidase inhibitor or IL-1 receptor antagonist was admini

190 stration of beta-aminopropionitrile, a lysyl oxidase inhibitor.

191 Monoamine oxidase inhibitors (MAOIs) exert therapeutic actions by

192 atypical antidepressants [AA], and monoamine oxidase inhibitors [MAOI]), age, sex, smoking, mild syst

193 al related synthetic analogues are monoamine oxidase inhibitors, which is the first reported indicati

194 We also found that cytochrome P450 oxidases involved in cutin and suberin production are ab

195 Cytochrome bo (3) ubiquinol oxidase is a transmembrane protein, which oxidizes ubiqu

196 The phagocyte NADPH oxidase is responsible for the neutrophil's great capaci

197 (PPO), also known as tyrosinase and catechol oxidase, is the enzyme responsible for enzymatic brownin

198 Nicotinamide adenine dinucleotide phosphate oxidase isoform 2 is an enzyme complex, which generates

199 We found NADPH oxidase isoform 4 (NOX4) to be the main producer of cyto

200 ), Akt and eNOS, and inhibits iNOS and NADPH oxidase isoform 4 (NOX4), all of which are associated wi

201 forming growth factor beta (TGF-beta), NADPH oxidase isoform 4 (Nox4), caspase-3 and Bax.

202 the mitochondria and increased cytochrome c oxidase levels in the brain.

203 nine dinucleotide phosphatase oxidase (NADPH oxidase) levels, in comparison to DM+INS and DM+RSV+INS

204 nipulating stromal matrix with an anti-lysyl oxidase like-2 (anti-LOXL2) antibody in syngeneic orthot

205 ic predisposition due to variations in lysyl oxidase-like 1 (LOXL1) function, leading to altered elas

206 slinking genes/enzymes (lysyl oxidase, lysyl oxidase-like 2-4, tissue transglutaminase-2), and ECM tu

207 The oxidase-like activity and exogenous ROS generation have

208 oluble Pd(12) nanocage 1 has shown excellent oxidase-like activity upon irradiation with white light.

209 Lysyl oxidase-like-1 (LOXL1), a vital crosslinking enzyme in e

210 neered LOXCAT, a fusion of bacterial lactate oxidase (LOX) and catalase (CAT), which irreversibly con

211 entify hypoxia-induced ECM re-modeler, lysyl oxidase (LOX) as a key inducer of chemoresistance by dev

212 The l-lactate oxidase (LOx) based lactate sensors are widely used for

213 The lysyl oxidase (LOX) family of extracellular proteins plays a v

214 hat Atox1, ATP7A, and the proenzyme of lysyl oxidase (LOX; copper-loaded via ATP7A) are all in close

215 cysteine), crosslinking genes/enzymes (lysyl oxidase, lysyl oxidase-like 2-4, tissue transglutaminase

216 Monoamine oxidase (MAO) metabolizes cytosolic dopamine (DA), there

217 Tryptophan hydroxylase (TPH1) and monoamine oxidase (MAO-A) are the key enzymes for the synthesis an

218 The resurgence of interest in monoamine oxidases (MAOs) has been fueled by recent correlations o

219 Multicopper oxidases (MCOs) catalyze the oxidation of a variety of s

220 The multicopper oxidases (MCOs) couple four 1e(-) oxidations of substrat

221 The MGCN exhibited glucose oxidase-mimicking activity and chitin-AcOH mirrored pero

222 esults are consistent with the hypothesis of oxidase molecules displaying a spectrum of enzymatic act

223 pper enzyme Myrothecium verrucaria bilirubin oxidase (MvBOD) for direct electron transfer (DET) in th

224 icotinamide adenine dinucleotide phosphatase oxidase (NADPH oxidase) levels, in comparison to DM+INS

225 ed two further members of this class of GA 2-oxidases, namely AtGA2ox9 and AtGA2ox10, in the Arabidop

226 mtDNA depletion and cytochrome c oxidase-negative cells were found ex vivo in biopsies of

227 uction of Igs, or by the activation of NADPH oxidase (NOX) complexes.

228 derived from mitochondria and from the NADPH oxidase (NOX) enzymes of innate immune cells are known t

229 Indeed, ROS forming NADPH oxidase (Nox) genes associate with hypertension, yet tar

230 sing on the central role of particular NADPH oxidase (NOX) isoforms that are activated in specific ca

231 iotensin II type 1 receptor (AT(1) R), NADPH oxidase (NOX) subunits, D(5) R, and NaCl cotransporter.

232 of SpNOX, the Streptococcus pneumoniae NADPH oxidase (NOX), a prokaryotic model system for exploring

233 nicotinamide adenine dinucleotide phosphate oxidase (NOX)-dependent ROS production might be a driver

234 oxidative stress via direct binding on NADPH oxidase (NOX)4 promoter and induction of NOX4 (NADPH oxi

235 The epithelial NADPH oxidase NOX1 is the primary source of luminal H(2)O(2) e

236 , and twofold higher gut expression of NADPH oxidase (NOX2) and translocator protein (TSPO).

237 y understood, and establishing how the NADPH oxidase (NOX2) kills microbes has proven elusive.

238 The NOX2 NADPH oxidase (NOX2) produces reactive oxygen species to kill

239 that RAC2(E62K) retains binding to an NADPH oxidase (NOX2) subunit, p67(phox), and to the RAC-bindin

240 Here, we identified type 5 NADPH oxidase (NOX5), a calcium-activated, ROS-forming enzyme,

241 ng evidence supports a central role of NADPH oxidases (NOXs) in the regulation of platelets, which ar

242 adenine dinucleotide phosphate, reduced form oxidases (NOXs) were studied in young/old mice on fast f

243 arising from constitutive activation of NOX2 oxidase, occurs in >60% of patients with AML and that RO

244 tion based on covalently immobilized glucose oxidase on PCB-integrated electrodes.

245 Interactions between different oxidases or oxidase systems have been intensively invest

246 n measuring oxidation states of cytochrome c oxidase (oxCCO), an intracellular marker of cellular met

247 ation of xanthine oxidase (XO) and polyamine oxidase (PAO) activities in milk samples was developed.

248 the action of oxygen-sensing plant cysteine oxidases (PCOs).

249 ould be eliminated by overexpressing PNP/PMP oxidase (PdxH) to decrease the PNP levels.

250 ucose-responsive moieties, including glucose oxidase, phenylboronic acid, and glucose-binding molecul

251 Leukocyte reduced NADP (NADPH) oxidase plays a key role in host defense and immune regu

252 er neutrophil) and the persistence of 16-46% oxidase-positive neutrophils.

253 ine ammonia-lyase (PAL) and lower polyphenol oxidase (PPO) activities.

254 kinetics of peroxidase (POD) and polyphenol oxidase (PPO) were investigated under conventional and m

255 Polyphenol oxidase (PPO), also known as tyrosinase and catechol oxi

256 tions for the inactivation of two polyphenol oxidases (PPOs): mushroom tyrosinase in buffer and the P

257 process in the Mn/Fe R2-like ligand-binding oxidase (R2lox) protein is investigated using a suite of

258 The plant NADPH oxidase RBOHD is a primary player in ROS production duri

259 cess reminiscent of flavoprotein nitroalkane oxidase reactivity, which is achieved by relatively stab

260 two natural enzymes (peroxidase and glucose oxidase) realized through real sample analysis.

261 trophil LTB4 generation as a target of NADPH oxidase regulation, which could potentially be exploited

262 chondrial electron transport chain and NADPH oxidase, respectively.

263 Genetic defects in NADPH oxidase result in chronic granulomatous disease (CGD), c

264 mus thermophilus ba(3) (Tt ba(3)) cytochrome oxidase revealed that O(2) binding was slowed down by a

265 HAO1 (glycolate oxidase) silencing is the mechanism of lumasiran, an inv

266 ost response to H. pylori, and that spermine oxidase (SMOX), which metabolizes the polyamine spermine

267 The pyruvate oxidase (SpxB)-dependent production of H(2)O(2) is widel

268 providing a suitable environment for glucose oxidase stability.

269 embled individual mitochondrial cytochrome c oxidase subunit 1 (COI) sequences (n = 866) and found hi

270 cotinamide adenine dinucleotide phosphatase] oxidase subunit 2) and NOX4 (NADPH [nicotinamide adenine

271 cotinamide adenine dinucleotide phosphatase] oxidase subunit 4) were upregulated.

272 of the mitochondrial (mt) genes cytochrome c oxidase subunit I (COI) and Cytochrome b as well as in t

273 3 is due to the phosphorylation of the NADPH oxidase subunit p47(phox), which results in NADPH oxidas

274 e nuclear-encoded mitochondrial cytochrome c oxidase subunit, COX4I2, in glomus cell sensitivity to h

275 which codes for an isoform of a cytochrome c oxidase subunit.

276 to chromatin regulatory regions of the NADPH oxidase subunits increases in the mdx muscle and JQ1 adm

277 ding for NADH dehydrogenase and cytochrome c oxidase subunits.

278 of novel therapeutic agents targeting these oxidase systems and their interactions, which could be e

279 Interactions between different oxidases or oxidase systems have been intensively investigated for t

280 ralization, and Loxl4, which encodes a lysyl oxidase that catalyzes collagen crosslinking.

281 This degradation is mediated by NADPH oxidase that produces highly oxidizing reactive oxygen s

282 A2ox7 and AtGA2ox8, which are class III GA 2-oxidases that 2beta-hydroxylate C(20)-GAs.

283 GA catabolic enzymes include GA 2-oxidases that are classified into three classes.

284 li the component of cytochrome bd-I terminal oxidase, the CydDC complex, shuttles excessive l-cystein

285 naling domain, which activates SYK and NADPH oxidase to cause phagosomal damage even when spliced int

286 Activated neutrophils assemble NADPH oxidase to convert substantial amounts of molecular oxyg

287 s (Lb)NOX(1), a bacterial water-forming NADH oxidase, to assess the metabolic consequences of directl

288 , generated by semicarbazide-sensitive amine oxidase, to cause the metabolic phenotype of MDSCs and M

289 hydroxymandelic acid (DHMA) by the monoamine oxidase TynA and the aromatic aldehyde dehydrogenase Fea

290 h organic cation transporter 3 and monoamine oxidase type B, two key proteins for DA uptake and metab

291 Members of class III GA 2-oxidases typically act on GA precursors containing a C(2

292 ), with those typically conserved among GA 3-oxidases, Tyr(93), Met(106), and Thr(202), respectively,

293 damage, mediated by activation of the NADPH oxidase, uncoupling of endothelial and neuronal nitric o

294 ) and, more recently, the HO-like fatty acid oxidase UndA.

295 LR4, MD2, and subunits of the vascular NADPH oxidases under diabetes and hypertension.

296 Glucose oxidase was studied as a test-case, but the method is ge

297 ouse liver, and isolated bovine cytochrome c oxidase were directly inhibited by the drug, while activ

298 nicotinamide adenine dinucleotide phosphate oxidase, which generates microbicidal reactive oxidants,

299 In the hypoxic mitochondrion, cytochrome c oxidase, which is a major source of NO, also is inhibite

300 e assay method for determination of xanthine oxidase (XO) and polyamine oxidase (PAO) activities in m