ライフサイエンスコーパス: oxidative stress

1 thelial NO pathway and increased endothelial oxidative stress.

2 lso controls cellular defense against ER and oxidative stress.

3 ta activity, possibly useful during cellular oxidative stress.

4 ucing CD36 scavenger receptor expression and oxidative stress.

5 ypoxia, nutrient deprivation, metabolic, and oxidative stress.

6 age the cause of hyperglycemia and resultant oxidative stress.

7 changing environmental conditions and avoid oxidative stress.

8 protein kinase (MAPK) pathway responding to oxidative stress.

9 metastasizing cells depend on MCT1 to manage oxidative stress.

10 gher levels mediates neuronal injury through oxidative stress.

11 lum (ER)-mediated folding inhibition, and 6) oxidative stress.

12 allergen-challenged mice are not mediated by oxidative stress.

13 tion of amino acid metabolism, TCA cycle and oxidative stress.

14 e cycle and nutrient uptake, and lowering in oxidative stress.

15 gy-lysosomal systems), neuroinflammation and oxidative stress.

16 mary melanoma growth and invasion, including oxidative stress.

17 Th2 cytokines production and a modulation of oxidative stress.

18 ctive oxygen species (ROS) leading to higher oxidative stress.

19 icant increase in sensitivity to hypoxic and oxidative stress.

20 n of membrane properties and amelioration of oxidative stress.

21 ility of red blood cells (RBCs) to withstand oxidative stress.

22 facilitated Sir2 accumulation in response to oxidative stress.

23 the loss of melanocyte viability induced by oxidative stress.

24 function due to TEPP-46-induced increases in oxidative stress.

25 assessment of markers of bone metabolism and oxidative stress.

26 cterial processes, including the response to oxidative stress.

27 making them more susceptible to ROS-induced oxidative stress.

28 e activity, which are concurrent to onset of oxidative stress.

29 cumulated from chronic exposure to increased oxidative stress.

30 617F erythrocytes suppressed their effect on oxidative stress.

31 rkers of cardiac function, inflammation, and oxidative stress.

32 improve mitochondrial function and decrease oxidative stress.

33 one may protect against inflammation-induced oxidative stress.

34 ced phenotypes, indicating a causal role for oxidative stress.

35 d from induced pluripotent stem cells during oxidative stress.

36 e level of cytochrome bd-I, which results in oxidative stress.

37 getics, impaired mitochondrial function, and oxidative stress.

38 1 are more resistant to both heat stress and oxidative stress.

39 released from mitochondria during sustained oxidative stress.

40 ceptor type 1 (AT(1) R) axis associated with oxidative stress.

41 enetic factors involved in the regulation of oxidative stress.

42 ioxidant that may protect against As-induced oxidative stress.

43 ed oxidative phosphorylation, causing severe oxidative stress.

44 rotein synthesis during cellular response to oxidative stress.

45 requirement A1 (Htra1), a marker of cellular oxidative stress.

46 tathione synthesis, the Gclm knockout mouse, oxidative stress activated MMP9 (matrix metalloprotease

47 enin are pleiotropic, resulting in decreased oxidative stress, activation of pro-proliferative and pr

48 Together, these findings suggest that oxidative stress actively regulates RGP behavior to ensu

49 d autoxidation (peroxidation) is linked with oxidative stress, age-related diseases, and cancers.

50 Mechanistically, this is due to increasing oxidative stress, alterations to interactions with the m

51 fractions showed bioactive properties, with oxidative stress amelioratory effects, and could be a po

52 resulting in the differentiation of LSCs via oxidative stress and aberrant activation of unfolded pro

53 Psychological distress induces oxidative stress and alters mitochondrial metabolism in

54 rast, induction of cydDC sensitizes cells to oxidative stress and aminoglycosides, which can be suppr

55 er adverse reactions, including weight loss, oxidative stress and angiogenesis, are reported in anima

56 les (Al(2)O(3) and NiO NPs) on plant growth, oxidative stress and antioxidant activities in the hydro

57 Our results confirmed that oxidative stress and antioxidants are a critical contrib

58 treatment reduces CoQ synthesis and promotes oxidative stress and apoptosis in tumors when administer

59 The homeostatic link between oxidative stress and autophagy plays an important role i

60 mide treatment was associated with decreased oxidative stress and axon/myelin loss.

61 The relationship between oxidative stress and cardiac stiffness is thought to inv

62 level in the heart, as well as ameliorating oxidative stress and cell apoptosis activities through p

63 1-specific siRNA mitigated radiation-induced oxidative stress and cellular injury.

64 of intermediate metabolites consistent with oxidative stress and damage.

65 cellular antioxidants under H(2)O(2) induced oxidative stress and disturbances caused to mitochondria

66 ochondrial superoxide scavenger that reduced oxidative stress and DNA damage seen in oxygen-exposed m

67 that melanoma cells in lymph experience less oxidative stress and form more metastases than melanoma

68 compromising membranes, T7SS defects induce oxidative stress and hamper their response to LA challen

69 blockade, with a corresponding reduction in oxidative stress and histologic damage compared to untre

70 These changes correlate with oxidative stress and impaired mitochondrial function in

71 ured with apigenin had significantly reduced oxidative stress and improved antioxidant defense respon

72 indicate that amelioration of mitochondrial oxidative stress and inflammation are key mechanisms und

73 r-kappaB (NF-kappaB) cell signaling, hepatic oxidative stress and inflammation in macrophages during

74 xygen species are linked to the induction of oxidative stress and inflammation in the liver.

75 ponses to many types of insults by enhancing oxidative stress and inflammation.

76 sis and TCA cycle, a milieu which can hasten oxidative stress and inflammation.

77 ated by mitigation of vascular mitochondrial oxidative stress and inflammation.

78 Oxidative stress and inflammatory cytokines affect the h

79 In addition, critical angiogenic oxidative stress and inflammatory factors reported to be

80 tion; (iii) Cellular signaling changes; (iv) Oxidative stress and inflammatory responses.

81 JunD downregulation was associated with oxidative stress and left ventricular dysfunction assess

82 g physiological responses (plasma TH levels, oxidative stress and mitochondrial density).

83 Our results implicated oxidative stress and mitochondrial dysfunction, hormone

84 metabolism, mitochondrial hyperpolarization, oxidative stress and mTOR signalling underwrite the know

85 ffects of the reciprocal interaction between oxidative stress and neuroinflammation, impacting on PVI

86 icacy, and significantly reduced hippocampal oxidative stress and neuroinflammation.

87 rmore, Sspo defects are sufficient to induce oxidative stress and neuroinflammatory responses implica

88 Disruption of these microdomains promotes oxidative stress and Nox isoform-specific redox signalli

89 art mitochondria is unchanged in the face of oxidative stress and point to a critical role of obesity

90 nd proposes a mechanism for exercise-induced oxidative stress and redox-mediated pathophysiological p

91 tored visual pigment formation and decreased oxidative stress and retinal degeneration, which resulte

92 beta1 signaling is a target for iron-induced oxidative stress and suggest that baseline PI-PLCbeta1 q

93 diabetes mellitus, which leads to increased oxidative stress and the activation of multiple inflamma

94 this life extension is due to a reduction of oxidative stress and the activation of the transcription

95 ncreased ULBP2/5 expression was dependent on oxidative stress and the antioxidants N-acetylcysteine a

96 on the vulnerability of the male germline to oxidative stress and the induction of oxidative DNA dama

97 to the media and procedures that may reduce oxidative stress and, as a result, establish a system fo

98 mic reticulum (ER) stress, calcium overload, oxidative stress, and Abeta 1-42 oligomers toxicity.

99 ltration, tissue repair enzymes, pathways of oxidative stress, and altered intestinal barrier functio

100 hin the aerobic respiratory chain, mitigates oxidative stress, and contributes to gene expression in

101 antibacterial activities may be mediated by oxidative stress, and do not necessarily require bacteri

102 tects from endothelial dysfunction, vascular oxidative stress, and hypertrophy and attenuates Ang II

103 e protected against endothelial dysfunction, oxidative stress, and increased Nox2, all of which were

104 asmic reticulum (ER) stress response, causes oxidative stress, and induces apoptosis.

105 yr) is produced in cells under conditions of oxidative stress, and m-Tyr has been shown to be toxic t

106 pathogens in circulation, kill them through oxidative stress, and present them to the antigen-presen

107 Also, they reduced weight loss, oxidative stress, and the anthracnose (Colletotrichum gl

108 ects of canagliflozin in ISO-induced cardiac oxidative stress, and their underlying molecular mechani

109 and analyzed for biomarkers of inflammation, oxidative stress, anti-inflammatory lipid mediators, tis

110 porter, and further elucidate a link between oxidative stress, antibiotic resistance, and sulfur meta

111 sregulation of placental Htra1 and placental oxidative stress are features of preeclamptic placentas

112 sensitivity to microbial toxin, osmotic and oxidative stress are seen in both mutants albeit to diff

113 Testing in an in vitro oxidative stress assay and a mouse model of blast-induce

114 Inflammation and oxidative stress associate with such injury, but what dr

115 drial oxidative capacity may protect against oxidative stress associated with birth while ensuring en

116 diac pathological remodeling, apoptosis, and oxidative stress associated with both onset and advancem

117 To avoid iron-mediated oxidative stress, bacteria utilize iron-dependent global

118 etiology and progression of inflammatory and oxidative stress-based diseases.

119 nism for kidney injury is likely mediated by oxidative stress, because treatment with Tempol, an supe

120 d with levels of NT-proBNP, hs-cTnT, CRP, or oxidative stress biomarkers.

121 ere, we assess ribosomal integrity following oxidative stress both in vitro and in cells to elucidate

122 liar application of melatonin alleviated the oxidative stress by increasing GT, CAT, POD, SOD and GR

123 r 2 (Nrf2) plays a critical role in reducing oxidative stress by promoting the expression of antioxid

124 In addition to targeting DNA, oxidative stress can affect proteins like OGG1 itself, s

125 To reduce cellular oxidative stress caused by overproduction of superoxide,

126 We conclude that oxidative stress caused by plumbagin and atovaquone degr

127 etabolic pathways to alleviate the increased oxidative stress caused by statin treatment, and targeti

128 l hypoxia, immune dysfunction, angiogenesis, oxidative stress) causes various disruptions in offsprin

129 and render cells vulnerable to nutrient and oxidative stress conditions.

130 ide radicals and protects the bacterium from oxidative stress conditions.

131 lay in the maintenance of the ribosome under oxidative stress conditions.

132 responsive to inflammatory mediators and to oxidative stress, consistent with a key role in CF lung

133 Similar to oxidative stress, cyclin C is destroyed by the UPS follo

134 tes the cardiomyocyte cell cycle and reduces oxidative stress damage through anabolic pathways and be

135 t OsmC family peroxiredoxin, an indicator of oxidative stress derived from mixing reactive metals and

136 8.5+/-4.1, P=0.014) accompanied by increased oxidative stress (dihydroethidium fluorescence: sham, 1.

137 Inadequate nutrient intake leads to oxidative stress disrupting homeostasis, activating sign

138 electrofusion of rabbit SCNT embryos induced oxidative stress, disturbed the epigenetic state, and ca

139 ty through multiple mechanisms including the oxidative stress, DNA damage, lysosomal dysfunction, inf

140 Mutations indicative of oxidative stress do not increase with increasing latency

141 f HIV proteins and methamphetamine increases oxidative stress, DRP1-mediated mitochondrial fragmentat

142 Conversely, prevention of mitochondrial oxidative stress during chronic T cell stimulation allow

143 enesis will worsen metabolic dysfunction and oxidative stress during NAFLD.

144 e cells and proline metabolism to counteract oxidative stress, during growth at 4 degrees C compared

145 Oxidative stress, endogenous enzyme activities, and othe

146 lation of pathways involved in inflammation, oxidative stress, epigenetics, mitochondrial dysfunction

147 linked to reactive oxygen species (ROS) and oxidative stress exhibit drastic changes in RGPs.

148 se INSTIs induced adipogenesis, lipogenesis, oxidative stress, fibrosis, and insulin resistance.

149 anoradicals, as a yet undiscovered source of oxidative stress, finally convert into hydrogen peroxide

150 Our results provide evidence that reducing oxidative stress following allotransplantation of PVPON/

151 We examined adaptability and survival to oxidative stress following nutrient deprivation in three

152 ination of mitochondrial stresses, including oxidative stress from low levels of purine metabolites a

153 r, we accomplished both light- and energetic/oxidative stress-gated control of this interaction.

154 ng CNS innate immune cells identified a core oxidative stress gene signature coupled to coagulation a

155 t how peripheral changes in a key marker for oxidative stress, glutathione (GSH), may associate with

156 Reactive oxygen species (ROS)-induced oxidative stress has been associated with diseases such

157 or 15 (GDF-15), a marker of inflammation and oxidative stress, has emerged as a biomarker for arteria

158 bolites upon exposure to PCB-153, leading to oxidative stress, hemolytic anemia, and tumor developmen

159 eral pathological conditions associated with oxidative stress; however, its role in periodontal disea

160 ng sequences to act as long-range sensors of oxidative stress, impacting gene expression via the DNA

161 ization experienced by Salmonella undergoing oxidative stress impairs folding of periplasmic proteins

162 Oxidative stress in adipocyte plays a central role in th

163 pment of chronic, low-grade inflammation and oxidative stress in age-related dry eye.

164 To test whether increased oxidative stress in Atg7 (Delta/Delta) mice was responsi

165 PGRPs induce oxidative stress in bacteria through a block in the resp

166 he inflammatory processes and the effects of oxidative stress in Caco-2 cells, and preserved the inte

167 increases mitochondrial activity and reduces oxidative stress in children with SCD/VOE.

168 ere we demonstrate, using multiple models of oxidative stress in conjunction with mechanical loading,

169 The retina experiences increased oxidative stress in diabetes, and the transcriptional ac

170 ne oxidase for dopamine metabolism, triggers oxidative stress in dopaminergic neurons and alpha-Syn a

171 astrocyte activation, neuroinflammation, and oxidative stress in FBN-ARO-KO mice.

172 bin might be a potential biomarker to assess oxidative stress in oral cancer patients.

173 ential therapeutic target for regulating the oxidative stress in PD.

174 atus, providing a mechanism for the enhanced oxidative stress in pre-eclampsia.

175 ized cells provide insights into the role of oxidative stress in senescence bypass and immortalizatio

176 ness of lipofuscin as a marker of cumulative oxidative stress in the brain.

177 kinase (p38) activated by neutrophil-derived oxidative stress in the pathogenesis of NASH.

178 of A. baumannii to form biofilms and resist oxidative stress in the respiratory tract facilitates sy

179 of GSK3 enhanced Nrf2 activity and prevented oxidative stress in the retina of diabetic mice.

180 (REDD1) is necessary for the development of oxidative stress in the retina of streptozotocin-induced

181 f Setx accumulate DNA damage when exposed to oxidative stress in vitro and during aging in vivo, afte

182 to constitutive NRF2 activity and decreased oxidative stress in wild Neoaves tissues and cells.

183 role of lipid peroxidation, a potent form of oxidative stress, in mediating RV hypertrophy and failur

184 , we observed specific markers reflective of oxidative stress, including irreversible oxidation of be

185 sol and mitochondrion to cope with exogenous oxidative stresses, indicating a direct link between bot

186 Our results suggest that the oxidative stress induced high mutation frequency on mtDN

187 ed a mitophagy induction paradigm where mild oxidative stress induced low levels of mitochondrial dam

188 Recently, we reported that Hb-dependent oxidative stress induced post-translational modification

189 on by protecting multiple myeloma cells from oxidative stress-induced apoptosis.

190 ion of the redox-sensitive S70pBcl2 prevents oxidative stress-induced DNA damage and cell death by su

191 To investigate the role of oxidative stress-induced DNA damage and mutagenesis in c

192 al studies found noise to be associated with oxidative stress-induced vascular and brain damage, medi

193 Oxidative stress induces coccoid formation and is associ

194 he mitochondrial compartment, in addition to oxidative stress, inflammation, myocardial cell death pa

195 can modulate the cellular processes, such as oxidative stress, inflammatory responses, and apoptosis,

196 Oxidative stress inhibits E2F1 transcriptional activity,

197 Oxidative stress inhibits the interaction between E2F1 a

198 Here, we provide the oxidative stress innate immune cell atlas in neuroinflam

199 Oxidative stress is a central part of innate immune-indu

200 We show that oxidative stress is a key factor in the mode of action o

201 elevance for human amyloid diseases in which oxidative stress is often an associated hallmark.

202 source of reactive oxygen species (ROS), and oxidative stress is thought to contribute to pathology i

203 "oxidative stress." Today, we recognize that oxidative stress is two-sided.

204 em (CNS) innate immune cells contributing to oxidative stress is unknown, and therapies to target the

205 homeostasis after SCI, mediated primarily by oxidative stress, is considered to play a crucial role i

206 efore, when evaluating compounds that induce oxidative stress, it is important to consider the contri

207 Lipofuscin is a nondegradable end-product of oxidative stress; its cerebral presence reflects the cum

208 ironments can expose tumor cells to hypoxia, oxidative stress, lack of growth signals, inadequate ami

209 eased levels of stress hormones and vascular oxidative stress, leading to endothelial dysfunction and

210 Furthermore, we show that acute oxidative stress leads to increased RECQL4 acetylation a

211 cocoa polyphenols, that induced a decreased oxidative stress levels.

212 substrate for the heart and kidneys, reduced oxidative stress, lowered serum uric acid level, reduced

213 x + placebo had a greater improvement in the oxidative stress marker protein carbonyls (P < 0.01).

214 -treated rats not only prevents elevation of oxidative stress markers but also rescues levels of depl

215 genous antioxidants but also reduces cardiac oxidative stress markers, fibrosis and apoptosis.

216 ammatory cytokines (TNF-alpha and IL-1beta), oxidative stress (MDA and OSI), and proteases (MMP-8, MM

217 Here, we investigate the role of oxidative stress-mediated by plumbagin and atovaquone in

218 ns as a redox sensor to prevent drug-induced oxidative stress-mediated DNA damage and execution with

219 ugh NOD2 activation upon an otherwise lethal oxidative stress-mediated signal.

220 ted mitochondrial dysfunction and associated oxidative stress might induce senescence in joint tissue

221 ription factors involved in inflammation and oxidative stress, mitigate mitochondrial dysfunction, ac

222 s, which identified consistent enrichment in oxidative stress, mitochondrial dysfunction, and transcr

223 With oxidative stress, MSH3 with this deletion (Delta27bp MSH

224 ss glucose is present; and (c) mitochondrial oxidative stress must precede the insulin stimulus to ca

225 uction in the heart contributes to increased oxidative stress, myocyte hypertrophy, ECM remodeling, a

226 ing microbial metabolism, hepatic steatosis, oxidative stress, nitric oxide modulation, and collagen

227 a in the altered iron handling and increased oxidative stress observed in AD pathogenesis.

228 ure GNNQQNY fibers did not exert significant oxidative stress or cytotoxicity upon incubating with di

229 Inducing oxidative stress or exogenous DNA damage leads to an ear

230 ng proteinopathy of AD, possibly by inducing oxidative stress or ferroptotic cell death, or may be re

231 upon transition to latency or by decreasing oxidative stress or iron content.

232 turnover of rRNA during ribophagy induced by oxidative stress or mTOR inhibition to show that 28S and

233 n throughout a lifetime and may cause higher oxidative stress (OS).

234 emical (malonyl-aldehyde [MDA], glutathione, oxidative stress [OSI], tumor necrosis factor [TNF]-alph

235 ures of kidney function, tubular injury, and oxidative stress over time in a cohort of children with

236 tent pattern of increased tubular injury and oxidative stress over time, which have been shown to aff

237 utritional value, in vitro cytotoxicity, and oxidative stress parameters in cells of selected insect

238 nction, suppression of neuroinflammatory and oxidative stress pathways, and a reduction in persistent

239 Oxidative stress plays a key role in the activation of a

240 l mitochondrial hyper-function and increased oxidative stress, possibly resulting in neurodegeneratio

241 Transcriptional profiling of oxidative stress-producing CNS innate immune cells ident

242 ra extracts to decrease the damage caused by oxidative stress promoted by H(2)O(2) in Saccharomyces c

243 Upregulation of TRIM25 ameliorates oxidative stress, promotes ER-associated degradation (ER

244 ve oxygen species and increased abundance of oxidative stress proteins.

245 During cardiac response to acute oxidative stress, proteome changes are consistent with a

246 iated non-amyloidogenic APP processing), and oxidative stress (reduced cortical oxidized proteins and

247 feri during mammalian infection of borrelial oxidative stress regulator (bosR) and decorin binding pr

248 chondrial reactive oxygen species-associated oxidative stress, remains elusive, partially due to the

249 2 in a manner that decreased and scaled with oxidative stress, respectively.

250 icative of the aryl hydrocarbon receptor and oxidative stress response (AREc32).

251 e of DNA-binding protein associated with the oxidative stress response and that this molecular functi

252 aracterized proteins involved in prokaryotic oxidative stress response are rare, we sought to learn m

253 ed factor 2 (NRF2), a major regulator of the oxidative stress response implicated in cell survival af

254 on of C. albicans drug-efflux, regulation of oxidative stress response, and maintenance of cell membr

255 factor erythroid 2-related factor 2-mediated oxidative stress response, which collectively contribute

256 aquat, suggesting they may have functions in oxidative stress response.

257 RNA-regulated growth-suppressive pathway for oxidative stress response.

258 for scavenging free radicals in wolfberries oxidative stress response.

259 dicated activation of lipid biosynthetic and oxidative-stress response pathways, including the antife

260 s and regulates the transcription of various oxidative stress-response genes.

261 essential for SpxA1-dependent activation of oxidative stress responses but dispensable for SpxA2-med

262 nd data set here could provide insights into oxidative stress responses in the heart and avail the se

263 ctivity, mHAMs induced greater Nrf2-mediated oxidative stress responses, demonstrating their distinct

264 elevates ROS level and induces expression of oxidative stress-responsive genes.

265 itochondrial membrane potential and elevated oxidative stress, results in apoptosis.

266 lbs of affected human scalp remains unclear, oxidative stress sensing appears to be a key factor invo

267 Oxidative stress serves as a hallmark in developing pros

268 reactive oxygen species (ROS) and consequent oxidative stress, specifically in the gut.

269 with increases (decreases) of resistance to oxidative stress, starvation stress and sleep indices.

270 ects mitochondrial redox balance, increasing oxidative stress status, which in turn is proposed as a

271 atally exposed to PCB-153 displayed elevated oxidative stress, symptoms of hemolytic anemia, and prem

272 glycerides in liver of young mice, caused by oxidative stress that activated FOXO1 to promote express

273 l presence reflects the cumulative amount of oxidative stress the brain has endured.

274 only MutT enzyme required for resistance to oxidative stress, this effect is not due to OG processin

275 xonal and myelin loss attributed to elevated oxidative stress through NADPH oxidase in lineage-traced

276 There was evidence for oxidative stress, tissue injury and microscopic intersti

277 oitation of the plant defense system against oxidative stress to engineer tolerant plants in the clim

278 hannels, while hydrogen peroxide distributes oxidative stress to sensitize the network to mitochondri

279 s) and molecules that exacerbate LIP-induced oxidative stress to trigger ferroptosis.

280 arotenoids, flavonoids), and the concept of "oxidative stress." Today, we recognize that oxidative st

281 Our results point to an oxidative stress tolerance network that is important for

282 Thus, oxidative stress transcriptomics identified neurotoxic C

283 inorganic As(V) and As(III) and relieved the oxidative stress undergone by cells exposed to either ox

284 ac dysfunction, myocardial inflammation, and oxidative stress, underlining the importance of inflamma

285 yses confirmed target engagement and reduced oxidative stress, unfolded protein response and fibrogen

286 intained basal Sir2 levels in the absence of oxidative stress; Upl3 facilitated Sir2 accumulation in

287 nalyses showed that Sir2 levels responded to oxidative stress via a mechanism involving ubiquitinatio

288 es mitochondrial fission leading to elevated oxidative stress via DRP1.

289 Oxidative stress was analyzed by measuring protein thiol

290 RVLM)] cytokine surges were blunted, whereas oxidative stress was higher in SHR.

291 nt roles in iron homeostasis and controlling oxidative stress, we examined whether chelating copper l

292 ory cytokine profile and multiple markers of oxidative stress were not substantially different betwee

293 here is a disconnect between respiration and oxidative stress, whereby mitochondrial oxidant levels d

294 anslation quality control breaks down during oxidative stress, wherein ThrRS is rendered inactive.

295 CIN can be induced by tissue oxidative stress, which is determined by the cellular ba

296 viding a model of translation control during oxidative stress, which supports elongation halt at pret

297 ty for providing cellular protection against oxidative stress while serving as a reactive oxygen spec

298 or arsenite, but not for sorbitol, quenching oxidative stress with N-acetylcysteine did suppress both

299 ways, which were costabilized by spontaneous oxidative stress within 3D cultures.

300 f normal mitochondrial function and elevated oxidative stress within the lymphatic muscle.