ライフサイエンスコーパス: oxygen consumption

1 and exertional symptoms (NYHA II-IV and peak oxygen consumption).

2 eflected in decreased membrane potential and oxygen consumption.

3 ism, largely characterized by an increase in oxygen consumption.

4 e phosphorylation genes, increasing cellular oxygen consumption.

5 decreased glucose uptake, and mitochondrial oxygen consumption.

6 ngly compensates to maintain skeletal muscle oxygen consumption.

7 responding increase in maximal mitochondrial oxygen consumption.

8 , but not pravastatin, reduced mitochondrial oxygen consumption.

9 the platelet's activation or overall cell's oxygen consumption.

10 ved in the respective aldehyde formation and oxygen consumption.

11 with decreases in anaplerosis, CAC flux, and oxygen consumption.

12 surements of extracellular acidification and oxygen consumption.

13 siveness, including invasion, migration, and oxygen consumption.

14 simultaneous expired gas analysis to measure oxygen consumption.

15 ased beta-oxidation of fatty acids (FAO) and oxygen consumption.

16 dehydrogenase activity and slightly elevated oxygen consumption.

17 ent ROS formation is linked to mitochondrial oxygen consumption.

18 esses beta-agonist-induced thermogenesis and oxygen consumption.

19 ly must have exceeded the expected increased oxygen consumption.

20 that the ubx4Delta strain displays decreased oxygen consumption.

21 through enhanced DUOX expression and rate of oxygen consumption.

22 ntial resting blood-based biomarkers of peak oxygen consumption.

23 in oxidative phosphorylation and by impaired oxygen consumption.

24 HO protein levels and promotes mitochondrial oxygen consumption.

25 and microrespirometry to estimate changes in oxygen consumption.

26 transport (AET) measured as light-dependent oxygen consumption.

27 id not improve the 4HNE-mediated decrease in oxygen consumption.

28 esulted in specific defects in mitochondrial oxygen consumption.

29 f Cu in wine and their impact on the rate of oxygen consumption.

30 thropoietin and erythrocytosis) and decrease oxygen consumption.

31 fatty acid oxidation, and diminished cardiac oxygen consumption.

32 Mito(16)-ATO inhibit only complex I-induced oxygen consumption.

33 ity when oxygen becomes limiting by reducing oxygen consumption.

34 Milrinone reduced myocardial oxygen consumption (0.15+/-0.06 versus 0.16+/-0.07 mL/be

35 There was increased myocardial oxygen consumption (0.16+/-0.07 versus 0.14+/-0.06 mL O(

36 limitation on exercise testing (reduced peak oxygen consumption, 24+/-1.3 versus 31+/-1.3 mL/kg/min,

37 e, METs (metabolic equivalents of task), and oxygen consumption, (3) methods based on heart rate or (

38 [-22.3 to -4.81]; P=0.009) per 1 unit; peak oxygen consumption (4.05 [1.97-6.59] per 1% change, P=0.

39 ; and lower glomerular filtration rate, peak oxygen consumption, 6-minute walk distance, and active h

40 80% (IQR, 70-88%) of predicted, median peak oxygen consumption 62% (IQR, 45-77%) of predicted, and m

41 athletes (mean +/- SEM age: 29 +/- 2 y; peak oxygen consumption: 66.8 +/- 1.3 mL . min(-1)) were stud

42 FpEF displayed worse exercise capacity (peak oxygen consumption, 7.7+/-2.3 versus 10.0+/-3.4 and12.9+

43 d mitochondria to immediately increase their oxygen consumption after the addition of the exogenous n

44 sion was associated with an increase in mean oxygen consumption after transfusion, especially in pati

45 us (AVPO; an important site for Tb control), oxygen consumption analysis, cardiovascular recordings,

46 developed a proliferative defect, increased oxygen consumption and accumulated reactive oxygen speci

47 as demonstrated in embryos by an ablation of oxygen consumption and an increase in lactate production

48 We recorded oxygen consumption and assessed blood lactate after each

49 Here, using oxygen consumption and ATP assays, RT-qPCR and ChIP-qPCR

50 heme synthesis and uptake causes intensified oxygen consumption and ATP generation, promoting tumorig

51 RK-33 antagonized the increase in oxygen consumption and ATP production observed after exp

52 bolism leads to an increase in mitochondrial oxygen consumption and ATP production.

53 e endurance that is associated with enhanced oxygen consumption and carbon dioxide production.

54 a complex and dynamic oxygen cycle in which oxygen consumption and corresponding carbon oxidation ar

55 identify parameters of a numerical model for oxygen consumption and diffusion.

56 present a technology platform for performing oxygen consumption and extracellular acidification measu

57 The oxygen consumption and extracellular acidification rate,

58 Mitochondrial oxygen consumption and fatty-acid oxidation were unalter

59 lly, 3-HIB treatment decreases mitochondrial oxygen consumption and generation of reactive oxygen spe

60 The p32 (+/-) mice show increased oxygen consumption and heat production, indicating that

61 relevant events that guard against wasteful oxygen consumption and inappropriate cell growth during

62 a3-AR stimulation, associated with decreased oxygen consumption and increased lactate production in a

63 ia exposure reduced mitochondrial ATP-linked oxygen consumption and increased state 4 respiration lin

64 Whereas higher doses may increase myocardial oxygen consumption and induce arrhythmias, diastolic hyp

65 ing of peroxynitrite increased mitochondrial oxygen consumption and membrane potential, mediated by t

66 ATPHI islets have an increased basal rate of oxygen consumption and mitochondrial mass.

67 Quantifying oxygen consumption and mitochondrial stability, we demon

68 Oxygen consumption and myocardial external efficiency (M

69 We show that CDNs decrease oxygen consumption and oxidative phosphorylation, cause

70 rowth phase human melanoma cells show higher oxygen consumption and preferential utilization of gluta

71 culating formats had a significant impact on oxygen consumption and pressure increase rate in the bot

72 ere found to be substrates for COX, based on oxygen consumption and product formation.

73 eukaryotic cells by Shigella flexneri boosts oxygen consumption and promotes the synthesis of phospha

74 abundance and cell metabolism with enhanced oxygen consumption and proton leak.

75 An increase in mitochondrial oxygen consumption and reserve capacity was observed in

76 hondrial function, attributable to increased oxygen consumption and slightly increased mitochondrial

77 arameters nu (e) and K (trans), representing oxygen consumption and supply, respectively.

78 arization maintains viability through slower oxygen consumption and/or a shift to a more reduced meta

79 ression of mitochondrial electron transport, oxygen consumption, and ATP generation.

80 Glucose oxidation, oxygen consumption, and ATP production correlated well w

81 t chain, oxidative phosphorylation, elevated oxygen consumption, and ATP synthesis.

82 mitochondrial membrane potential, increased oxygen consumption, and attenuated the Warburg effect at

83 In response to CL 316, 243 treatments, oxygen consumption, and BAT thermogenesis were diminishe

84 chondrial ATP output), mitochondrial fusion, oxygen consumption, and Ca(2+) uptake increased in wild-

85 with dynamic changes such as thermal power, oxygen consumption, and extracellular acidification rate

86 decreased cellular ATP levels, mitochondrial oxygen consumption, and extracellular acidification rate

87 n localization, wound healing, mitochondrial oxygen consumption, and glycolysis extracellular acidifi

88 e differentiation, myoblast fusion, cellular oxygen consumption, and glycolysis in skeletal muscle ce

89 lower valvuloarterial impedance, myocardial oxygen consumption, and improved myocardial efficiency d

90 gher levels of muscle AMPK, greater rates of oxygen consumption, and increased oxidative phosphorylat

91 Atpenin A5 partially inhibits oxygen consumption, and neither hypoxia nor atpenin A5 i

92 ient, New York Heart Association class, peak oxygen consumption, and patient-reported outcomes assess

93 alities in glucose metabolism, mitochondrial oxygen consumption, and production of reactive oxygen sp

94 cluded changes in LV systolic function, peak oxygen consumption, and quality of life.

95 1.4 g dL(-1) respectively) are used to model oxygen consumption as a fraction of delivery at rest ( V

96 rometric detection was carried out following oxygen consumption at -0.7V vs. the Ag reference electro

97 ociated haemoglobin concentration influences oxygen consumption at rest and during exercise via alter

98 hich predicts low haemoglobin concentration, oxygen consumption at rest can be sustained with the ass

99 in understanding the determinants of muscle oxygen consumption at the microvascular level.

100 ondrial heme, oxygen-utilizing hemoproteins, oxygen consumption, ATP generation, and key mitochondria

101 oblasts derived from the patient reveal that oxygen consumption, ATP output and reactive oxygen speci

102 y a few attempts to describe the kinetics of oxygen consumption based on the chemical composition of

103 declined from 8.0 to 7.5, calcification and oxygen consumption both decreased, suggesting a reduced

104 se from ATTM reduces metabolism (measured as oxygen consumption) both in vivo in awake rats and ex vi

105 creased glucose and palmitate metabolism and oxygen consumption, but maintained power and function.

106 e micelles, have been determined in terms of oxygen consumption by a Clark electrode in an oxygen-tig

107 The kinetics of oxygen consumption by different oenological tannins were

108 es in pulmonary function, mucus plugging and oxygen consumption by host neutrophils gives rise to reg

109 4HNE decreases mitochondrial oxygen consumption by inhibiting electron transport chai

110 lobin, the model shows that normal levels of oxygen consumption can be achieved at rest and during ex

111 The study of wines oxygen consumption capacity has been tied to colour modi

112 ts of both siblings showed glucose-repressed oxygen consumption compared to their mother, whereas gal

113 d insulin sensitivity and have lower maximal oxygen consumption compared with the exercised wild-type

114 Up to 38% of equine COC oxygen consumption could be attributed to non-mitochondr

115 With increasing energy state, oxygen consumption decreases rapidly until a threshold i

116 Silencing MICU1 in vitro increases oxygen consumption, decreases lactate production, inhibi

117 As the temperature dependence of oxygen consumption depends on activity levels, these fin

118 by elevated mitochondrial mass and increased oxygen consumption during activation.

119 ident with a large decrement in peak rate of oxygen consumption during aerobic exercise, respectively

120 , vascular conductance, oxygen delivery, and oxygen consumption during exercise; interestingly, these

121 However, oxygen consumption during PDT can result in an inadequat

122 vates central carbon metabolism activity and oxygen consumption, enhancing the killing effects of ant

123 Integrated chemical oxidation and oxygen consumption experiments, coupled with electron pa

124 Levels of oxygen consumption, extraction, creatinine fall and frac

125 in Ad-FLD mice was associated with increased oxygen consumption, fat utilization, and the expression

126 The regression equations showed that oxygen consumption followed a 2nd polynomial equation wh

127 chondrial respiratory function, and uncouple oxygen consumption from ATP production.

128 e primary outcome measure was change in peak oxygen consumption from baseline to 16 weeks.

129 ux, as occurs with exercise, exhibit reduced oxygen consumption from fatty acids, with higher oxygen

130 en consumption from fatty acids, with higher oxygen consumption from glucose.

131 ne defined as either (1) an increase in peak oxygen consumption >=1.5 mL/kg/min and reduction of at l

132 ar body mass index, resting heart rate, peak oxygen consumption (H = 40 +/- 13 vs. C = 42 +/- 7 ml/kg

133 During EVKP lower levels of oxygen consumption, higher oxygen extraction, a lower de

134 for understanding the determinants of muscle oxygen consumption; however, no investigation has direct

135 owever, these agents can increase myocardial oxygen consumption, impair tissue perfusion, and are fre

136 ogenitor cell (NPC) lines revealed increased oxygen consumption in CDD mutant lines, which is associa

137 IS1 suppression with siRNA increased maximal oxygen consumption in fetal cells but not in postnatal c

138 Oxygen consumption in freshly isolated mitochondria from

139 Gboxin rapidly and irreversibly compromises oxygen consumption in glioblastoma cells.

140 Oxygen consumption in its deep waters leads to the build

141 oped pressure (LVDP), coronary flow rate and oxygen consumption in LANG and LWHs.

142 ndent and -independent energy biogenesis and oxygen consumption in mice without a concomitant increas

143 ease in the ratio of l-lactate production to oxygen consumption in primary hippocampal cultures.

144 nd degradation in NSCLC cells and suppresses oxygen consumption in purified mitochondria.

145 uffer from a severe deficit in mitochondrial oxygen consumption in response to the respiratory comple

146 anges in LV mass, LV ejection fraction, peak oxygen consumption in the cardiopulmonary exercise test,

147 portantly, idebenone supported mitochondrial oxygen consumption in the presence of a Complex I inhibi

148 eart, endothelial nitric oxide (NO) controls oxygen consumption in the working heart through paracrin

149 pret this negative impact of antioxidants on oxygen consumption in vitro and adipose tissue browning

150 energy expenditure and reduced mitochondrial oxygen consumption in white adipose tissue, brown adipos

151 s significantly increased, despite unaltered oxygen consumption, in isolated mitochondria from the sy

152 thelial cells results in decreased uncoupled oxygen consumption, increased fission, decreased membran

153 The estimated mean difference for oxygen consumption index in the patients with sepsis was

154 Oxygen consumption index increased significantly during

155 The oxygen delivery index, oxygen consumption index, central venous oxygen saturati

156 9 mL/min/m (0.9-9.0 mL/min/m) (p = 0.02) for oxygen consumption index.

157 lactate production; however, high concurrent oxygen consumption indicated a comparatively increased r

158 ly mitigated the reductions in mitochondrial oxygen consumption induced by these stresses.

159 ng circuits is directly linked to changes in oxygen consumption mediated by NNT.

160 Peak oxygen consumption (milliliters per kilogram of body wei

161 iogenesis as measured by increased uncoupled oxygen consumption, mitochondrial DNA content, and volta

162 Tissue level oxygen consumption (MRO2) was determined and used to cal

163 cardial external energy efficiency (MEE) and oxygen consumption (MVO(2)); and (4) whether the cardiov

164 Rates of cellular oxygen consumption (OCR) and extracellular acidification

165 sustained increases in the rate of cellular oxygen consumption (OCR) and reactive oxygen species (RO

166 Diabetes increased mitochondrial oxygen consumption (OCR) at complex II-IV.

167 ce-trained males, with a mean +/- SD maximal oxygen consumption of 58.2 +/- 5.3 mL . min(-1), followe

168 he little difference of the time of complete oxygen consumption on concentration of different antioxi

169 fficiency (VE/VCO2) (P < 0.001) but not peak oxygen consumption or anerobic threshold (P > 0.2).

170 Empagliflozin did not change myocardial oxygen consumption or MEE.

171 and SV response to exercise, or the maximal oxygen consumption or peak power output.

172 hemical and genetic perturbations that alter oxygen consumption or redox state support a model in whi

173 , whereas loss of NLRX1 results in increased oxygen consumption, oxidative stress, and subsequently a

174 [measured by the predicted maximal volume of oxygen consumption (p$\dot{V}$O2 max), n = 47] and 24-h

175 The oxygen consumption parameters were positively correlated

176 ction of at least 40%, impaired peak rate of oxygen consumption (peak Vo2), and at least 2 conditions

177 us) ferric carboxymaltose (FCM) affects peak oxygen consumption [peak VO2], an objective measure of e

178 as absolute value and as percentage of peak oxygen consumption (peakVO(2)), ventilation efficiency (

179 d, lower blood pressure response, lower peak oxygen consumption predicted, and higher minute ventilat

180 above the heart, resulting in similar muscle oxygen consumption profiles throughout exercise.

181 lls partially reverses the effect of CycT on oxygen consumption, proliferation, and tumorigenic funct

182 ation from scans such as blood flow rate and oxygen consumption provides new perspectives on the dise

183 .5 mL/kg per min or greater increase in peak oxygen consumption (pVO(2)) and at least one NYHA class

184 econdary end points included changes in peak oxygen consumption (pVO2), resting and Valsalva LVOT gra

185 action fraction (OEF), whereas outer retinal oxygen consumption (QO2) relies on oxygen availability b

186 efficiency defined as stroke work/myocardial oxygen consumption (r=0.63-0.65; all P<0.01).

187 ho developed BPD or died had a lower maximal oxygen consumption rate (mean +/- SEM, 107 +/- 8 vs. 235

188 Results indicate that MPP(+)-induced loss in oxygen consumption rate (OCR) and ATP production by mito

189 nd macrophages, omega-3 increased ATP-linked oxygen consumption rate (OCR) and omega-3 with carnitine

190 Oxygen Consumption Rate (OCR) is an established measure

191 Therefore, the oxygen consumption rate (OCR) of the lees of sparkling w

192 The oxygen consumption rate (OCR) was measured in an oxygen

193 ve no detectable AK2 protein, as well as low oxygen consumption rate (OCR), extracellular acidificati

194 Alde-red assays for CSCs, and Seahorse-based oxygen consumption rate (OCR), extracellular acidificati

195 ted hearts showed significant suppression of oxygen consumption rate (OCR).

196 etabolites (glucose, lactate, pyruvate), and oxygen consumption rate (OCR).

197 The circadian rhythmicity of oxygen consumption rate (Vo2) was disrupted in aged obes

198 red mitochondrial respiration with increased oxygen consumption rate and ATP, which was associated wi

199 ced cellular glucose uptake, higher cellular oxygen consumption rate and greater tolerance to glucose

200 tion rate as a measure of glycolysis and the oxygen consumption rate as a measure of mitochondrial re

201 ar metabolic rates in vitro using changes in oxygen consumption rate as a readout.

202 Using oxygen consumption rate as an assay for determining unco

203 lts showed no significant differences in the oxygen consumption rate between white and red wines, and

204 MX and KCl were all reduced without altering oxygen consumption rate compared with scramble control.

205 ne and KCl were all reduced without altering oxygen consumption rate compared with scramble control.

206 emainder of the model is parameterised using oxygen consumption rate data, indicative of hydroquinone

207 but did not decrease coupling efficiency or oxygen consumption rate during beta-oxidation.

208 The results indicate that the oxygen consumption rate follows second-order kinetics de

209 drial dysfunction, as evidenced by decreased oxygen consumption rate in cardiomyocytes, increased lev

210 -coated microRNA-211 partially augmented the oxygen consumption rate in PIG3V cells.

211 lated and phosphomimetic Cytc showed a lower oxygen consumption rate in reaction with isolated Cytc o

212 d with veratridine, despite a lower baseline oxygen consumption rate in the presence of glucose.

213 ortholog-encoding gene SNF1 can restore the oxygen consumption rate in ubx4Delta strain, thereby ree

214 They also confirm that the oxygen consumption rate is influenced by temperature in

215 Similarly, C2C12 myoblasts show a reduced oxygen consumption rate mediated by Pi transport-depende

216 depletion from serum blunts the induction of oxygen consumption rate observed in tubule cells treated

217 oxic conditions, decreased the mitochondrial oxygen consumption rate of cultured cells and mice.

218 ortance of the light-activation step and the oxygen consumption rate on the drug activity.

219 The highest oxygen consumption rate values were detected in three st

220 Oxygen consumption rate was assessed with the Seahorse m

221 eases in pyruvate dehydrogenase activity and oxygen consumption rate were reversed by dichloroacetate

222 nGLT-1 KO synaptosomes exhibited an elevated oxygen consumption rate when stimulated with veratridine

223 the haemoglobin oxygen change (that is, the oxygen consumption rate) in the microwells.

224 Net flux (oxygen consumption rate) is determined by demand for ATP

225 O-induced basal leak respiration and overall oxygen consumption rate, along with increased triglyceri

226 The lower oxygen consumption rate, changes in lipid and metabolite

227 Mitochondrial oxygen consumption rate, citrate synthase activity, and

228 imum force of contraction, increased maximum oxygen consumption rate, decreased peak rise time, and i

229 lia, TDF caused a dose-dependent increase in oxygen consumption rate, extracellular acidification rat

230 cretion, partly due to reduced mitochondrial oxygen consumption rate, glucose-stimulated Ca(2+) flux,

231 cretion, partly due to reduced mitochondrial oxygen consumption rate, glucose-stimulated Ca(2+) flux,

232 tochondrial functional capacity, measured as oxygen consumption rate, increased primarily after birth

233 Mechanistically, SCH772984 increased the oxygen consumption rate, indicating that these cells rel

234 ion of respiratory chain subunits and normal oxygen consumption rate.

235 ex I of the respiratory chain, decreased the oxygen consumption rate.

236 sufficient to decrease the maximal cellular oxygen consumption rate.

237 ting of the wines studied according to their oxygen consumption rate.

238 E(2) decreased the activity of complex I and oxygen consumption rate.

239 acid (TCA) cycle intermediates and increased oxygen consumption rate.

240 ue by performing high-throughput single-cell oxygen-consumption-rate measurements of cultured cells a

241 active oxygen species (mtROS) production and oxygen consumption rates (JO(2) ) in a manner that was d

242 HSP2-treated tumors exhibited reduced oxygen consumption rates (OCR) and ATP levels.

243 Oxygen Consumption Rates (OCRs) are faster with higher c

244 d mitochondrial dysfunction evidenced by low oxygen consumption rates (OCRs), complex activities, ATP

245 As a consequence, oxygen consumption rates and intracellular ATP concentra

246 The Mcl-1 transgene increased oxygen consumption rates and mROS expression in mock-inf

247 GS, -11%)] levels and improved mitochondrial oxygen consumption rates in comparison to vehicle-4L;C*

248 at match the reduced hindlimb blood flow and oxygen consumption rates in IUGR fetal sheep.

249 chondrial function was assessed by measuring oxygen consumption rates in permeabilized muscle fibers.

250 PDH-KO cells had increased oxygen consumption rates in response to glutamate, but n

251 rates match reduced hindlimb blood flow and oxygen consumption rates in the IUGR fetus.

252 errucosus continuously showed 15-36% reduced oxygen consumption rates indicating metabolic depression

253 hput single-cell photoacoustic microscopy of oxygen consumption rates should enable the faster charac

254 Oxygen consumption rates were determined in red, white a

255 accurately predicting metabolic rates (i.e., oxygen consumption rates) of aquatic organisms and restr

256 ired glucose uptake, mitochondrial function, oxygen consumption rates, glycolysis, lactic acid, and A

257 sm, we found that S100A4 depletion decreases oxygen consumption rates, mitochondrial activity, and AT

258 roduces cells with gene expression profiles, oxygen consumption rates, nitric oxide production levels

259 ions as a metabolic regulator by controlling oxygen consumption rates, suppressing hypoxic glycogen l

260 from a large database of routine metabolic (oxygen consumption) rates composed of a range of species

261 riate data techniques identify six different Oxygen-Consumption-Rates (OCRs) as required to completel

262 d redox state, increased maximum and reserve oxygen consumption ratio (OCR) and higher VDAC protein l

263 educed anaerobic glycolysis and an increased oxygen consumption ratio.

264 Fractional Na(+) excretion was increased and oxygen consumption reduced in the low Na(+) group after

265 irds (Aves) display high metabolic rates and oxygen consumption relative to mammals, increasing react

266 plementation reduced adiposity and increased oxygen consumption, respiratory exchange ratio, and heat

267 temperature, BAT temperature, and whole-body oxygen consumption) response to acute cold exposure, pro

268 Furthermore, a similar increase in oxygen consumption specific to RTT patient-derived isoge

269 in expression and reduced glucose-stimulated oxygen consumption: targeted metabolomics reveals impair

270 on- and time-dependent, relying on increased oxygen consumption that triggered enhanced production of

271 or hypoxic hearts, beyond decreasing cardiac oxygen consumption to alleviate hypoxia and decrease tra

272 utaneous white adipose tissue (WAT) promotes oxygen consumption, uncoupled respiration, and heat prod

273 s and tissue perfusion and reduce myocardial oxygen consumption, until adequate anti-arrhythmic drug

274 creased mitochondrial content, and increased oxygen consumption upon activation with cAMP analogs.

275 nal pro-B-type natriuretic peptide, and peak oxygen consumption (Vo(2)).

276 s after adjuvant therapy) with impaired peak oxygen consumption (VO(2)peak) to 1 of 2 supervised exer

277 obic exercise capacity, assessed as the peak oxygen consumption (Vo(2)peak).

278 (NO) on skeletal muscle force production and oxygen consumption ( VO2 ).

279 Patients underwent measurement of peak oxygen consumption (Vo2 [mL/kg per minute]) and ventilat

280 sus 11.4 +/- 4.8 min; P = 0.008), lower peak oxygen consumption (VO2) (18.4 +/- 5.4 versus 21.4 +/- 6

281 eart rate (HR), BT, motor activity (MA), and oxygen consumption (Vo2) were measured 24 h/d at normal

282 erence, 20.93 m [CI, 5.91 to 35.95 m]), peak oxygen consumption (Vo2max) (mean difference, 3.17 mL/kg

283 Peak oxygen consumption (VO2peak) in the exercise group rose

284 Average oxygen consumption was 1.1 +/- 0.2 mL/kg per minute.

285 Mean peak oxygen consumption was 33 mL/kg per minute, and 92% of p

286 cortical oxygen tension were lower and renal oxygen consumption was higher in low Na(+) groups.

287 M compared with controls, whereas myocardial oxygen consumption was lower in HOCM.

288 The results for white wines showed that the oxygen consumption was sensitive to the non-sulfide-boun

289 ause of a higher cardiac mass, total cardiac oxygen consumption was significantly higher in HOCM than

290 glycolytic enzymes as well as mitochondrial oxygen consumption were all highly sensitive to CD28 blo

291 edance (ie, global afterload) and myocardial oxygen consumption were reduced by -11% and -12% (P=0.03

292 exhibited an increased rate of mitochondrial oxygen consumption when compared with low MPO-expressing

293 ions in the maximum specific growth rate and oxygen consumption when cultured under conditions promot

294 and duroquinol-dependent complex III-induced oxygen consumption whereas Mito(12)-ATO and Mito(16)-ATO

295 ial membrane and decreases the mitochondrial oxygen consumption, which may result in AMPK activation

296 ing Na(+) intake for 2 weeks increased renal oxygen consumption, which was normalized by mineralocort

297 ls with LKB1 knockdown had a reduced rate of oxygen consumption, which was partially restored by PDK4

298 s increase whole-body energy expenditure and oxygen consumption, while reducing body-weight in recipi

299 ation of coral reefs predicts that microbial oxygen consumption will cause reef deoxygenation.

300 2) an improvement of >=3.0 mL/kg/min in peak oxygen consumption with no worsening of New York Heart A