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1 dothelial mitochondrial dysfunction (reduced oxygen consumption rate).
2 P/ADP ratio, CO2 and lactate production, and oxygen consumption rate.
3 xidative phosphorylation on the basis of the oxygen consumption rate.
4 yzed using Fick's law to obtain the per-cell oxygen consumption rate.
5 se, surgery, or topical drugs on the corneal oxygen consumption rate.
6 relationship between oxygen supply rate and oxygen consumption rate.
7 and restores PDH activity and mitochondrial oxygen consumption rate.
8 acid (TCA) cycle intermediates and increased oxygen consumption rate.
9 e stress levels and a significant decline in oxygen consumption rate.
10 eased mitochondrial size and a greater basal oxygen consumption rate.
11 ial size and cristae shape and increased the oxygen consumption rate.
12 ellular acidification rate and decreased the oxygen consumption rate.
13 ATP production in astrocytes, as assessed by oxygen consumption rate.
14 nsport chain (ETC) to direct measurements of oxygen consumption rate.
15 ation efficiency, MYH2 expression, and basal oxygen consumption rate.
16 ion of respiratory chain subunits and normal oxygen consumption rate.
17 ex I of the respiratory chain, decreased the oxygen consumption rate.
18 sufficient to decrease the maximal cellular oxygen consumption rate.
19 ting of the wines studied according to their oxygen consumption rate.
20 cose metabolism, without impacting the basal oxygen consumption rate.
21 E(2) decreased the activity of complex I and oxygen consumption rate.
22 ased basal, spare, and maximal mitochondrial oxygen consumption rates.
23 , with a complimentary reduction in cellular oxygen consumption rates.
24 on-contraction coupling, [Ca(2+)](rest), and oxygen consumption rates.
25 tilage thickness, cell density, and cellular oxygen consumption rates.
26 VEGF+ and Vec cells had equivalent oxygen consumption rates.
27 ited before it affects maximal mitochondrial oxygen consumption rates.
28 cardiac efficiency by decreasing myocardial oxygen consumption rates.
29 t mBEC mitochondrial function via increasing oxygen consumption rates.
30 tion of RBCs resulted in 4- to 6-fold higher oxygen consumption rates.
31 ses in cytosolic and mitochondrial Ca(2+) on oxygen consumption rates.
32 l cortex are mitochondrially dense with high oxygen consumption rates.
33 arly in relation to the body mass scaling of oxygen consumption rates.
34 owed reduced lean mass (-12%), reduced total oxygen consumption rate (-8%), improved glucose toleranc
35 irst characterized in vitro by measuring the oxygen consumption rate, a marker of mitochondrial trica
36 e metabolic disturbances as shown by reduced oxygen consumption rates after application of topical an
37 O-induced basal leak respiration and overall oxygen consumption rate, along with increased triglyceri
39 Compared to M-MO, GM-MO displayed higher oxygen consumption rate and aerobic glycolysis (extracel
40 red mitochondrial respiration with increased oxygen consumption rate and ATP, which was associated wi
42 drial function, as indicated by elevation in oxygen consumption rate and extracellular acidification
43 ed in TSC2(SA), with increased mitochondrial oxygen consumption rate and glycolytic capacity (stresse
44 sporter repertoire, and improving both basal oxygen consumption rate and glycolytic function, thereby
45 ced cellular glucose uptake, higher cellular oxygen consumption rate and greater tolerance to glucose
46 individuals, together with markedly reduced oxygen consumption rate and hyperfragmentation of the mi
47 NRDC knockdown led to a decrease in cellular oxygen consumption rate and hypoxia-induced HIF1alpha ex
48 cells (E10) induced a drop in mitochondrial oxygen consumption rate and impairment of cellular bioen
49 e presence of oxygen, profoundly reduced the oxygen consumption rate and increased the extracellular
53 observed that TSPO deficiency decreased the oxygen consumption rate and mitochondrial membrane poten
55 tes mitochondrial Ca(2+) load, mitochondrial oxygen consumption rate and oxidative phosphorylation.
56 ived, and from this the relationship between oxygen consumption rate and oxygen partial pressure is d
57 Furthermore, the effects of mitochondria on oxygen consumption rate and reactive oxygen species prod
58 betaInsr(KO) islets have reduced ATP-coupled oxygen consumption rate and reduced expression of genes
59 ated from mesenchymal stromal cells restored oxygen consumption rate and reduced total reactive oxyge
61 hat mdx-Mss51 KO mice had increased myofiber oxygen consumption rates and an amelioration of muscle h
62 inoma cells overexpressing TIGAR have higher oxygen consumption rates and ATP levels when exposed to
63 on measured by basal, ATP-linked and maximal oxygen consumption rates and by spare respiratory capaci
64 evels showed that exposure to SSRI increased oxygen consumption rates and decreased carbohydrate leve
65 I assembly, higher complex I-linked state 3 oxygen consumption rates and decreased superoxide produc
66 ial membrane potential, cellular metabolism (oxygen consumption rates and fatty acid oxidation), and
68 cy or high glutamine levels result in higher oxygen consumption rates and metabolic profiles unfavora
69 hese hypoxic "non-Warburg" cells had highest oxygen consumption rates and mitochondrial capacity cons
70 hondrial levels of RMRP, in turn suppressing oxygen consumption rates and modestly reducing mitochond
74 instrument can also be used to measure cell oxygen consumption rates and thereby calculate glycolyti
75 iffusion, have been linked to differences in oxygen consumption rates and/or aerobic activity levels
76 sal oxygen consumption rates, higher maximal oxygen consumption rate, and augmented spare respiratory
78 n show altered metabolism, including reduced oxygen consumption rate, and reduced in vitro regulatory
79 d found that aging is associated with higher oxygen consumption rates, and especially higher extracel
80 succinate dehydrogenase activity, succinate oxygen consumption rates, and heart adenosine triphospha
81 tion rate as a measure of glycolysis and the oxygen consumption rate as a measure of mitochondrial re
87 We measured extracellular acidification and oxygen consumption rates, assessed mitochondrial DNA cop
88 ted in significantly increased mitochondrial oxygen consumption rates, ATP production rates, and enha
89 production consumed, and (iii) bottom-water oxygen consumption rates become less dependent on relati
91 lts showed no significant differences in the oxygen consumption rate between white and red wines, and
92 thermogenic activation of EPA by increasing oxygen consumption rate, brown-specific marker genes, an
93 radual but significant reduction in cellular oxygen consumption rate by 6 hours, corresponding with u
94 reases of brown-specific signature genes and oxygen consumption rate by EPA were concurrent with up-r
97 ne and KCl were all reduced without altering oxygen consumption rate compared with scramble control.
98 MX and KCl were all reduced without altering oxygen consumption rate compared with scramble control.
99 from a large database of routine metabolic (oxygen consumption) rates composed of a range of species
100 sensitive, and using a population of cells, oxygen consumption rates could be calculated down to a s
101 emainder of the model is parameterised using oxygen consumption rate data, indicative of hydroquinone
102 health was compromised, evidenced by reduced oxygen consumption rate, declined adenosine triphosphate
104 imum force of contraction, increased maximum oxygen consumption rate, decreased peak rise time, and i
106 nction, through measurement of mitochondrial oxygen consumption rate, demonstrated that overexpressio
107 the vertical profile of the microbial-driven oxygen consumption rate demonstrating a close linkage be
108 d the measurement volume biases the measured oxygen consumption rate depending on the actual [O(2)] g
109 on of mitochondrial OXPHOS proteins, reduced oxygen consumption rate, dissipated mitochondrial membra
111 edicted from biomechanical modeling, and the oxygen consumption rate during dives decreased with dept
113 glycolysis, or glucose oxidation revealed by oxygen consumption rate experiments and (13)C(6) glucose
114 que imaging system for GSCs, we assessed the oxygen consumption rate, extracellular acidification rat
115 oenergetics through enhanced basal and total oxygen consumption rate, extracellular acidification rat
116 lia, TDF caused a dose-dependent increase in oxygen consumption rate, extracellular acidification rat
118 Ascorbic acid increased the first-order oxygen consumption rate from 0.030 +/- 0.001 to 0.040 +/
120 AURKA inhibition, we note an increase in the oxygen consumption rate fueled by enhanced fatty acid ox
121 cretion, partly due to reduced mitochondrial oxygen consumption rate, glucose-stimulated Ca(2+) flux,
122 cretion, partly due to reduced mitochondrial oxygen consumption rate, glucose-stimulated Ca(2+) flux,
124 ired glucose uptake, mitochondrial function, oxygen consumption rates, glycolysis, lactic acid, and A
125 tochondrial respiration with increased basal oxygen consumption rates, higher maximal oxygen consumpt
126 drial dysfunction, as evidenced by decreased oxygen consumption rate in cardiomyocytes, increased lev
127 ondrial bioenergetic parameters, such as the oxygen consumption rate in cell cultures, enzyme activit
129 lated and phosphomimetic Cytc showed a lower oxygen consumption rate in reaction with isolated Cytc o
131 ivity in C. albicans and drastically reduced oxygen consumption rate in the fungus, similar to that s
132 d with veratridine, despite a lower baseline oxygen consumption rate in the presence of glucose.
133 nd two-photon excited fluorescence data, the oxygen consumption rate in the stratum basale is estimat
135 ortholog-encoding gene SNF1 can restore the oxygen consumption rate in ubx4Delta strain, thereby ree
137 ombine to match oxygen diffusion capacity to oxygen consumption rates in both air- and water-breathin
138 nadione sodium bisulfite (MSB) increased the oxygen consumption rates in both cell lines, whereas CCC
139 PCMB (p-chloromercurobenzoate) inhibited the oxygen consumption rates in both WT and rho(0) cells, wh
140 tiproliferative activities and inhibition of oxygen consumption rates in cells were distinctly differ
141 GS, -11%)] levels and improved mitochondrial oxygen consumption rates in comparison to vehicle-4L;C*
142 k, and decreased ATP-coupling efficiency and oxygen consumption rates in comparison with their wild-t
143 dative phosphorylation than TFM by measuring oxygen consumption rates in isolated liver mitochondria
145 m patients displayed significantly decreased oxygen consumption rates in patients, suggesting mitocho
146 chondrial function was assessed by measuring oxygen consumption rates in permeabilized muscle fibers.
148 We also observed elevated basal and maximal oxygen consumption rates in the fibroblasts from the pro
151 timulation conditions induced increased OCR (oxygen consumption rate) in the presence of Ca2+, which
152 the mitochondria, results in enhanced muscle oxygen consumption rate, increased endurance capacity, a
153 tochondrial functional capacity, measured as oxygen consumption rate, increased primarily after birth
155 errucosus continuously showed 15-36% reduced oxygen consumption rates indicating metabolic depression
156 Mechanistically, SCH772984 increased the oxygen consumption rate, indicating that these cells rel
157 rial respiratory chain enzyme activities and oxygen consumption rates indistinguishable from controls
158 eases glucose uptake, lactate secretion, and oxygen consumption rates; inhibition of glucose consumpt
161 scavenging DPPH, and, even without modifying oxygen consumption rate, it protected quite well wine co
162 active oxygen species (mtROS) production and oxygen consumption rates (JO(2) ) in a manner that was d
163 ving mitochondrial creatine kinase activity, oxygen consumption rate, LV energetics (by (31)phosphoro
164 ho developed BPD or died had a lower maximal oxygen consumption rate (mean +/- SEM, 107 +/- 8 vs. 235
166 ue by performing high-throughput single-cell oxygen-consumption-rate measurements of cultured cells a
167 Similarly, C2C12 myoblasts show a reduced oxygen consumption rate mediated by Pi transport-depende
168 the mutant did not exhibit major changes in oxygen consumption rate, mitochondrial membrane potentia
169 sm, we found that S100A4 depletion decreases oxygen consumption rates, mitochondrial activity, and AT
171 roduces cells with gene expression profiles, oxygen consumption rates, nitric oxide production levels
172 depletion from serum blunts the induction of oxygen consumption rate observed in tubule cells treated
173 st, loss of Piezo1 reduced the mitochondrial oxygen consumption rate (OCR) and adenosine triphosphate
174 Results indicate that MPP(+)-induced loss in oxygen consumption rate (OCR) and ATP production by mito
176 nd macrophages, omega-3 increased ATP-linked oxygen consumption rate (OCR) and omega-3 with carnitine
178 n particular, the biological implications of oxygen consumption rate (OCR) and substrate level phosph
179 lecting an increased production of NADH, and oxygen consumption rate (OCR) by 0.32 nmol/min/100 islet
182 wer peripheral blood mononuclear cell (PBMC) oxygen consumption rate (OCR) is strongly associated wit
183 transport function (dextran/albumin uptake), oxygen consumption rate (OCR) measurements in isolated t
186 urs, we conduct a high-throughput screen for oxygen consumption rate (OCR) reduction and identify a n
187 measures of cellular respiration, including oxygen consumption rate (OCR) to produce ATP, were uncha
189 sults: DM1 induced a multi-state decrease in oxygen consumption rate (OCR) with a corresponding reduc
190 es promotes thermogenesis, showing increased oxygen consumption rate (OCR) with higher thermogenic ge
191 Alde-red assays for CSCs, and Seahorse-based oxygen consumption rate (OCR), extracellular acidificati
192 ve no detectable AK2 protein, as well as low oxygen consumption rate (OCR), extracellular acidificati
193 Alde-red assays for CSCs, and Seahorse-based oxygen consumption rate (OCR), extracellular acidificati
194 rat islets by acetylcholine and response of oxygen consumption rate (OCR), NAD(P)H, cytosolic Ca2+,
201 , HepG2 cells exhibited significantly higher oxygen consumption rates (OCR) and respiratory capacity
202 des to predict chemical composition and wine oxygen consumption rates (OCR) by PLS-modeling of the vo
203 isol-treated macrophages exhibited increased oxygen-consumption rate (OCR) to extracellular-acidifica
204 fn2 gene in myoblasts (Mfn2(MKO) ) increases oxygen-consumption rates (OCR) associated with the maxim
205 iling in LCLs revealed significantly reduced oxygen consumption rates (OCRs) and NAD+/NADH ratios, co
208 ion rates (ECARs) to estimate glycolysis and oxygen consumption rates (OCRs) for oxidative metabolism
209 d mitochondrial dysfunction evidenced by low oxygen consumption rates (OCRs), complex activities, ATP
210 riate data techniques identify six different Oxygen-Consumption-Rates (OCRs) as required to completel
211 oxic conditions, decreased the mitochondrial oxygen consumption rate of cultured cells and mice.
212 analyze the onset of the pathology, maximal oxygen consumption rate of left ventricular permeabilize
213 of the metabolite oxaloacetate decreased the oxygen consumption rate of the aged blastema and increas
214 s allowed us to experimentally determine the oxygen consumption rate of the intestinal epithelial cel
215 oxygen behaviour in upper basement suggests oxygen consumption rates of 1 nmol cm(-3)ROCK d(-1) or l
217 accurately predicting metabolic rates (i.e., oxygen consumption rates) of aquatic organisms and restr
221 viability, ROS levels, oxidative DNA damage, oxygen consumption rates, or extracellular acidification
222 uction was increased in patient cells, while oxygen consumption rate, particularly under stress, alon
227 hput single-cell photoacoustic microscopy of oxygen consumption rates should enable the faster charac
228 CBF, in the presence of virtually unaltered oxygen consumption rates, show concurrent regional age-a
229 ions as a metabolic regulator by controlling oxygen consumption rates, suppressing hypoxic glycogen l
231 The algorithm increases the useful range of oxygen consumption rates, the temporal resolution, and d
232 erently dampened and thus underestimates the oxygen consumption rate; the discrepancy is much larger
241 from capillary networks in muscle at a high oxygen consumption rate was simulated using a computatio
242 srupted mitochondrial morphology and reduced oxygen consumption rate were observed in DMXAA-treated a
243 eases in pyruvate dehydrogenase activity and oxygen consumption rate were reversed by dichloroacetate
244 n, perfusate flow rates, urinary output, and oxygen consumption rates were compared between all group
247 nGLT-1 KO synaptosomes exhibited an elevated oxygen consumption rate when stimulated with veratridine
249 levels of ETC supercomplexes, DeltaPsim and oxygen consumption rates, while isolated mitochondrial m