ライフサイエンスコーパス: oxygen demand

1 otal and volatile suspended solids, chemical oxygen demand).

2 posive sample selected according to greatest oxygen demands).

3 myocardial perfusion and reducing myocardial oxygen demand.

4 t worsening major determinants of myocardial oxygen demand.

5 ontractility to balance oxygen delivery with oxygen demand.

6 rictor nerve activity and reductions in limb oxygen demand.

7 is the major contributor to the increase in oxygen demand.

8 e root tip, likely due to enhanced microbial oxygen demand.

9 well-established regulatory methods based on oxygen demand.

10 nd is prone to dysregulation because of high oxygen demand.

11 ion in which O2 supply is inadequate to meet oxygen demand.

12 ar tone to match local tissue perfusion with oxygen demand.

13 ally at the expense of increasing myocardial oxygen demand.

14 rcise-induced increases in muscle energy and oxygen demand.

15 ssure product, dP/dt, -dP/dt, and myocardial oxygen demand.

16 ncreased right ventricular (RV) workload and oxygen demand.

17 ell with high removal efficiency of chemical oxygen demand.

18 lature to match oxygen delivery to increased oxygen demand.

19 F response is mediated by factors other than oxygen demand.

20 n of large predatory fish, animals with high oxygen demand.

21 ft ventricular (LV) afterload and myocardial oxygen demand.

22 , including motile skeletal benthos, had low oxygen demands.

23 increases red blood cell mass to meet tissue oxygen demands.

24 like flying insects that have high metabolic oxygen demands.

25 rized by elevated concentrations of chemical oxygen demand (13.1-59.6 mg/L), biochemical oxygen deman

26 oxygen demand (13.1-59.6 mg/L), biochemical oxygen demand (7.67-36.5 mg/L), total suspended solids (

27 s as well as the maximum removal of chemical oxygen demand (94.8%), linear alkylbenzenesulfonates (LA

28 with removals of 97% of the soluble chemical oxygen demand, 97% NH3-N, and 91% of total bacteria (bas

29 IPB system removed more than 92% of chemical oxygen demand, 98% of ammonium nitrogen, and 82% of phos

30 However, chemical oxygen demand, a common measure of gross residual organi

31 more likely to result from preventing excess oxygen demand after surgery rather than from deciding wh

32 n agricultural pollution emissions (Chemical Oxygen Demand, ammonia nitrogen, and agricultural carbon

33 n concentrated centrate, 95% of the chemical oxygen demand and 93% of total PFAS were removed after 2

34 h the alternation of the influent biological oxygen demand and ammonium nitrogen load to the aerated

35 when added to leachate; five-day biochemical oxygen demand and biochemical methane potential results

36 ogical diseases characterized by extremes of oxygen demand and carbon dioxide production.

37 leads to increased inflammation, myocardial oxygen demand and dehydration, predisposing people to my

38 approaches that address the balance between oxygen demand and delivery; the manipulation of cardiopu

39 d the potential for mismatch between tubular oxygen demand and delivery; the self-sustained oscillati

40 Chemical oxygen demand and dissolved organic carbon concentration

41 changes decrease LV afterload and myocardial oxygen demand and reduce the number of angina episodes,

42 abolites signalling a mismatch between blood/oxygen demand and supply in exercising muscles.

43 contributes to a mismatch between myocardial oxygen demand and supply, a mechanistic basis for disrup

44 s occur in the setting of a mismatch between oxygen demand and supply, as with severe hypotension.

45 al oxygen delivery to active skeletal muscle oxygen demand and therefore inadvertently impair exercis

46 ignificant correlation of HAAs with chemical oxygen demand and THMs with FC was observed.

47 irculation that combine to reduce myocardial oxygen demand and to increase supply, thereby attenuatin

48 ion (MI) can occur from increased myocardial oxygen demand and/or reduced supply in the absence of ac

49 D (chemical oxygen demand), BOD (biochemical oxygen demand), and COLR (color) from AQEF.

50 wastewater temperature, influent biological oxygen demand, and ammonium nitrogen load, was investiga

51 ndex, global longitudinal strain, myocardial oxygen demand, and left atrial emptying fraction were im

52 (NTG) improves myocardial perfusion, reduces oxygen demand, and may enhance low-dose dobutamine to im

53 Chemical oxygen demand, biological oxygen demand, and total alkalinity removal rate varied

54 ocities than did still-water fish, had lower oxygen demands, and responded less vigorously to small c

55 cting the Ediacaran expansion of macroscopic oxygen-demanding animals.

56 Chemical oxygen demand, biological oxygen demand, and total alkal

57 astal DO, rain event sampling of biochemical oxygen demand (BOD) in samples collected from the outfal

58 well as a detector for measuring biochemical oxygen demand (BOD) using Rhodotorula mucilaginosa UICC

59 (Chemical Oxygen Demand (COD) and Biological Oxygen Demand (BOD)) significantly overestimated populat

60 aracterize phosphorus, nitrogen, biochemical oxygen demand (BOD), and fecal coliform pollution from h

61 dissolved oxygen (DO), salinity, biological oxygen demand (BOD), chemical oxygen demand (COD), elect

62 ee critical pollutants including biochemical oxygen demand (BOD), total nitrogen (TN) and total suspe

63 TSS (total suspended solids), COD (chemical oxygen demand), BOD (biochemical oxygen demand), and COL

64 apability to rotate and determine biological oxygen demand, BOD), that uniquely enables automated oxy

65 10(-2) g CH(4) (g influent 5 day biochemical oxygen demand; BOD(5))(-1) [0.6-9.9 x 10(-2) g CH(4) (g

66 mical oxygen demand (COD) 90-95%, biological oxygen demand (BOD5) 94-98%, total nitrogen (TN) 70-80%,

67 ) and a 4-fold increase in 5-day biochemical oxygen demand (BOD5).

68 of dicarboxylates induces not only a higher oxygen demand but also a higher NADH/NAD(+) ratio than s

69 ng with a local hypoxia induced by increased oxygen demands by proliferating cells which supports chr

70 e extent to which apnoea-induced extremes of oxygen demand/carbon dioxide production impact redox reg

71 ly been associated with increased myocardial oxygen demand, cardiac arrhythmias, and mortality in a v

72 DNA degradation rate declined as biochemical oxygen demand, chlorophyll, and total eDNA (i.e., from a

73 g in large values of chemical and biological oxygen demand (COD and BOD) in the aquatic systems into

74 ecedent determinations, theoretical chemical oxygen demand (COD(th)) as well as theoretical and stoic

75 ded solids (TSS) < 30 mg L(-1), and chemical oxygen demand (COD) < 150 mg L(-1).

76 high biological elimination rates (chemical oxygen demand (COD) 90-95%, biological oxygen demand (BO

77 er ratio (R(2) = 0.87), and between chemical oxygen demand (COD) and AA concentrations (R(2) = 0.87).

78 Treatment efficiency (chemical oxygen demand (COD) and ammonia removal), Ag dissolution

79 Greater than 95% reductions in chemical oxygen demand (COD) and ammonium ion were achieved withi

80 Traditional load-based indicators (Chemical Oxygen Demand (COD) and Biological Oxygen Demand (BOD))

81 d the 24-h and 7-day Pb toxicity to chemical oxygen demand (COD) and NH3-N removal, bacterial viabili

82 ystem oxidized only 10% of influent chemical oxygen demand (COD) and recovered up to 55% of incoming

83 Both MFCs removed 65-70% chemical oxygen demand (COD) at a hydraulic retention time (HRT)

84 average removal rate of 94.23% for Chemical Oxygen Demand (COD) compared to 88.76% for standard syst

85 For nearly a century, chemical oxygen demand (COD) has been widely used for assessment

86 method for the determination of the chemical oxygen demand (COD) in heterogeneous solid or semisolid

87 used and mainly contribute to high chemical oxygen demand (COD) in textile effluents.

88 rs applied to the rapid analysis of chemical oxygen demand (COD) in urban waste waters.

89 te, the SCMFC acted as a sensor for chemical oxygen demand (COD) in water.

90 g rate (OLR) from 2.5 to 15 g total chemical oxygen demand (COD) L.d(-1).

91 Condensate pH was 6.2 and its chemical oxygen demand (COD) level was 36,000 ppm.

92 retention time (SRT), matching the chemical oxygen demand (COD) loading rate to the removal rate in

93 weeks, with a mass balance based on chemical oxygen demand (COD) of 100 +/- 2% (COD(out)/COD(in)).

94 synthetic acetate solution having a chemical oxygen demand (COD) of 320 mg/L.

95 The model was able to predict the chemical oxygen demand (COD) removal efficiency and methane produ

96 elized cost and carbon intensity of chemical oxygen demand (COD) removal.

97 ly from wastewater streams with low chemical oxygen demand (COD) to nitrogen (C/N) ratios.

98 of initial pH (pH 4, 5, and 7) and chemical oxygen demand (COD) to nitrogen (COD/N) ratio of 3.6:1,

99 time and EO time, on the removal of chemical oxygen demand (COD), colour, turbidity, and total organi

100 ty, biological oxygen demand (BOD), chemical oxygen demand (COD), electrical conductivity (EC), nitra

101 ir impact on key indicators such as Chemical Oxygen Demand (COD), NH(4)(+)-N, Total Phosphorus (TP),

102 ed as total organic carbon (TOC) or chemical oxygen demand (COD), though these parameters do not prov

103 tified accounted for only 2.1 mg of chemical oxygen demand (COD)/L (16% of total SMP as COD) because

104 the highest solubilization (0.16 mg chemical oxygen demand (COD)/mg volatile solids (VS), at 2.13 mg

105 ter column features arising from respiratory oxygen demand during organic matter degradation in strat

106 We examined heart rate and oxygen demand during squats, exploring variations across

107 Our findings suggest that the oxygen demand during squatting is notably substantial, w

108 Acute increases in myocardial oxygen demand evoke coronary vasodilation and enhance pe

109 t adequately restore tissue oxygenation when oxygen demand exceeds supply.

110 idation result in reduced economy (increased oxygen demand for a given speed) at velocities that tran

111 the filtration barrier, albuminuria, and the oxygen demand for tubular reabsorption.

112 tive to riverine nutrient loads and sediment oxygen demand from settled organic carbon.

113 VFA yield of 0.55 +/- 0.12 g VFA as chemical oxygen demand g volatile solids (VS)(fed)(-1) were obser

114 explained by its ability to reduce cerebral oxygen demand has been replaced by an increasingly docum

115 mperatures, low oxygen supply and increasing oxygen demand, high and rising exposure to ultraviolet r

116 tion extends the ability to sustain elevated oxygen demand, implying a buffering role for myelin agai

117 western basin bloom growth and central basin oxygen demand in distinct ways that merit further invest

118 the diaphragm to augment blood flow to match oxygen demand in response to contractile activity and co

119 The importance of increase in myocardial oxygen demand in the genesis of ischemia in both men and

120 e tropical Pacific, which would have reduced oxygen demand in the subsurface.

121 hat enhanced productivity leading to greater oxygen demand in the thermocline.

122 The comorbidities that increase oxygen demands in these infants, like bronchopulmonary d

123 ater consumption, discharge of COD (chemical oxygen demand) in effluent water, cumulative COD and dil

124 Significant increases in myocardial oxygen demand, including systolic blood pressure, occur

125 oals of treatment are to decrease myocardial oxygen demand, increase coronary blood flow and oxygen s

126 bility declines and cardiac and whole-animal oxygen demand increases.

127 h the influent total phosphorus and chemical oxygen demand instead of geographical factors (e.g. lati

128 -shivering thermogenesis the increase in BAT oxygen demand is met by a local and specific increase in

129 ion fraction is increased in layer IV, where oxygen demand is presumably the highest.

130 forms, E. coli, enterococci, and biochemical oxygen demand (Kendall's tau = 0.348 to 0.605, p < 0.05)

131 ischemia induced by a sustained increase in oxygen demand may not progress to necrosis but may inste

132 n muscle glucose metabolism during increased oxygen demand may promote central fatigue and thereby di

133 logy similar to that of a modern sponge, its oxygen demands may have been met well before the enhance

134 e COD method with an optode-based biological oxygen demand method to accurately and efficiently asses

135 to reductases, we tested the hypothesis that oxygen demand modifies arterial [NAD(H)](i), and that re

136 ncreases in these determinants of myocardial oxygen demand, myocardial perfusion decreased by 30% (10

137 To meet the extreme oxygen demand of insect flight muscle, tracheal (respira

138 ggest that a fine-tuning on CO(2) uptake and oxygen demand of the cells is essential to reach a highe

139 carbon-paste matrix is shown to satisfy the oxygen demand of the enzymatic reaction and to provide c

140 ter so that significant reduction of initial oxygen demand of wastewater by 61% was observed.

141 ue and specialized trait associated with the oxygen demands of flying, their endothermic metabolism a

142 However, the effects of remote increases in oxygen demand on a circulation with limited ability to r

143 are associated with an increased myocardial oxygen demand on reperfusion.

144 e observed the effect of remote increases in oxygen demand on splanchnic and renal blood flow in hemo

145 ion was related to a reduction in myocardial oxygen demand or preservation of myocardial oxygen suppl

146 d disease-associated hypoxia (where cellular oxygen demand outstrips supply) for immune cell function

147 Additionally, biochemical oxygen demand, partial pressure of CO(2), and organic ca

148 r NAD(P)(H) redox ratios reflecting elevated oxygen demand potentiate native coronary Kv1 activity in

149 ponses were expressed relative to myocardial oxygen demand (rate-pressure product).

150 DO level was attributable to biofilm-induced oxygen demand rather than changes in oxygen diffusivity.

151 between cerebral vascular geometry and local oxygen demand, recent experiments have reported that mic

152 ogens, which led to the decrease in chemical oxygen demand removal.

153 We hypothesized that increased myocardial oxygen demand resulting from hypotension and reflex tach

154 Increased myocardial oxygen demand resulting from hypotension and reflex tach

155 tized, hemorrhaged dog, increased peripheral oxygen demand results in further redistribution of blood

156 ons significantly decreased soluble chemical oxygen demand (S(COD)) removal efficiency (11% to 31%) a

157 nse variables analyzed were soluble chemical oxygen demand (sCOD) and volatile suspended solids (VSS)

158 ch aims to predict effluent soluble chemical oxygen demand (SCOD) in anaerobic digestion (AD) process

159 isioned cyber-physical system with real-time oxygen demand sensing and delivery for improved patient

160 f MFCs were proposed earlier (as biochemical oxygen demand sensing) only lately a myriad of new uses

161 Besides, the use of MFCs as biochemical oxygen demand sensors (perhaps the main analytical appli

162 on rate, renal oxygen consumption, and renal oxygen demand/supply relationship, i.e., renal oxygen ex

163 domestic wastewater having a total chemical oxygen demand (tCOD) of 210 +/- 11 mg/L.

164 Secondary-treated PPM effluent has lower oxygen demand than primary-treated effluent, but ASB acc

165 ations and functions for estimating sediment oxygen demand that are linked to settled organic carbon

166 whole-limb blood flow is related to a lower oxygen demand that is independent of tissue mass.

167 However, the heart has intrinsic oxygen demands that must be met to maintain effective co

168 A2 class of adenosine receptors and reducing oxygen demand through A1 adenosine receptors (A1AR).

169 using equatorial productivity and subsurface oxygen demand to be less concentrated in the east.

170 on the filtration barrier and increases the oxygen demand to drive reabsorption.

171 investigation illustrated that the chemical oxygen demand, total nitrogen, and total phosphorus remo

172 ciencies of total suspended solids, chemical oxygen demand, total phosphorus, and total nitrogen were

173 d minor effect on determinants of myocardial oxygen demand, vasodilator stress myocardial perfusion i

174 ine oxygen extraction fraction and regulated oxygen demand via oxygen extraction fraction changes, wh

175 On exercise, oxygen demand was highest (rate-pressure product: 22,157

176 Statistical analysis indicated that cerebral oxygen demand was maintained to an Hct of 0.14, 0.11, an

177 iency of total suspended solids and chemical oxygen demand was observed for recovered aluminum (85-60

178 ples where water temperature and biochemical oxygen demand were measured.

179 These increases in oxygen demand were met by increases in both oxygen deliv

180 ad (aortic pressure, P=0.030) and myocardial oxygen demand were seen (tension-time index, P=0.024; ra

181 r side, high heart rates increase myocardial oxygen demand, which can be a problem in patients with f

182 ulation was used to increase lower extremity oxygen demand while lower extremity, splanchnic, renal b

183 Balancing oxygen demand with availability is crucial to cardiac fu

184 ormal pigs, there is no change in myocardial oxygen demand with CPAP, whatever the change in cardiac

185 al oxygen supply or unmet need in myocardial oxygen demand, without atherothrombosis, usually in the

186 calized vascular damage and increased tissue oxygen demand, wound healing occurs in a relatively hypo