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1 s the airways of patients and leads to blood oxygen desaturation.
2 uration of respiratory events, and degree of oxygen desaturation.
3 were accompanied by moaning, tachypnea, and oxygen desaturation.
4 tively correlated with severity of nocturnal oxygen desaturation.
5 s and none of the 10 controls had sleep-time oxygen desaturation.
6 in during intermittent hypoxia to accentuate oxygen desaturation.
7 and had severe acute respiratory syndrome or oxygen desaturation.
9 rface displacements (32% vs. 56%; P = 0.01), oxygen desaturations (40% vs. 75%; P < 0.001), required
12 after completion of the vascular anatomoses oxygen desaturation and increased airway pressure was no
14 airway obstruction during sleep resulting in oxygen desaturation and sleep fragmentation, and associa
16 obesity, the frequency of obstructive apnea, oxygen desaturation, and arousal contributes to abnormal
17 n of the amygdala co-occurred with apnea and oxygen desaturation, and electrical stimulation of the a
19 or the presence of respiratory disturbances, oxygen desaturations, and cortical arousals within a 90-
20 y insufficient unless clinical findings like oxygen desaturation, apnea, and bradycardia are part of
22 ratory events >9.4 b.p.m. (third tertile) or oxygen desaturation area under baseline (hypoxic burden)
24 Six children (0.9%) had brief periods of oxygen desaturation below 95%, none of which required ai
28 cheal intubation-associated events or severe oxygen desaturation compared to primary tracheal intubat
29 , nocturnal REM sleep latency, and extent of oxygen desaturation-could reflect neurophysiological mec
30 ose tolerance was related to the severity of oxygen desaturation (DeltaSa(O(2))) associated with slee
35 raperitoneal, leptin decreased the number of oxygen desaturation events in REM sleep, and increased v
36 fic tracheal intubation-associated events or oxygen desaturation events occurred in 40.2% of patients
37 m notifications were for clinically relevant oxygen desaturation, facilitating timely nursing respons
38 on with adverse procedure-related events and oxygen desaturation, future study is warranted to improv
41 idence of postreinfusion nausea, emesis, and oxygen desaturation in comparison to unselected PBSC rei
44 hough the duration and magnitude of cerebral oxygen desaturation increased in the low-trigger group (
47 fference: -0.47, 95% CI -0.74 to -0.20), and oxygen desaturation index (mean difference: -0.99, 95% C
49 udies of obstructive sleep apnea, namely the oxygen desaturation index (ODI) and oxygen desaturation
50 y effectiveness end points comprised AHI and oxygen desaturation index (ODI) responder rates (RRs).
53 indicating moderate-to-severe apnea) and the oxygen desaturation index (ODI; the number of times per
56 thickness was only associated with the mean oxygen desaturation index among the polysomnography para
58 mass index, smoking, drinking habits, and 3% oxygen desaturation index in the middle and worse sleep
59 sleep apnoea (Epworth score > 9, and a > 4% oxygen desaturation index of > 10 per h) who were assign
60 lycated hemoglobin level of 6.5-8.5%, and an oxygen desaturation index of 15 or more events per hour
61 saturation < 90%, apnea-hypopnea index, and oxygen desaturation index-did not show any significant a
62 e concordance between overall and positional oxygen desaturation indices (ODI) and apnea-hypopnea ind
63 relationship between intradialytic cerebral oxygen desaturation, intradialytic BP, and CVR in differ
64 ETCO2, supporting the assumption that ictal oxygen desaturation is a consequence of hypoventilation.
69 pneumothorax, pneumomediastinum) and severe oxygen desaturation (< 70%) were recorded prospectively.
71 ned as lung aspiration of digestive content, oxygen desaturation, major hemodynamic instability, sust
74 ical rating scale), interface displacements, oxygen desaturations, need for ventilator support, and r
75 p efficiency without changing apnoea length, oxygen desaturation, next-day perceived sleepiness and a
78 Adverse respiratory event was defined as oxygen desaturation of at least 5%, pulmonary aspiration
80 itical vulnerability, resulting in saw-tooth oxygen desaturation possibly representing the earliest m
82 n adenosine that occur during jugular venous oxygen desaturations suggest that adenosine may play an
83 uat exposure with ventilatory equivalent and oxygen desaturation suggests that paraquat may be associ
84 f bronchial hemorrhage of > 30 mL; transient oxygen desaturation to < 90% in seven (8.4%) patients; h
85 with adverse respiratory events: 58 cases of oxygen desaturation, two pulmonary aspirations, 10 cases
92 ere coded as clinically relevant (i.e., true oxygen desaturation with SpO2<89 for >15s) or irrelevant
93 tients with COPD who have nocturnal arterial oxygen desaturation without qualifying for long-term oxy