ライフサイエンスコーパス: oxygen-free

1 and a sulfonated phenanthroline ligand in an oxygen-free 7:3:1 ethanol/water/toluene mixture at 70 de

2 ut oxidative hydrolysis of six samples in an oxygen-free atmosphere at 150 degrees C for 90 min.

3 37.2% conversion and a 78.9% selectivity to oxygen free benzene and the remainder 21.1% as diphenyl

4 ransformation of the oxide precursor into an oxygen-free chalcogenide reagent.

5 Oxygen-free chemical vapour deposition (OF-CVD) synthesi

6 We find no oxygen-free composition that fits the seismological data

7 itated enzyme stabilization and operation in oxygen free conditions.

8 vely, they can be stored for over 20 h under oxygen-free conditions after 365 nm UV illumination for

9 reated with (57)Fe-enriched Fe(II)(aq) under oxygen-free conditions for 30 days at neutral pH and roo

10 Here, we show that under oxygen-free conditions high concentrations of H(2)O(2) c

11 Epitaxial graphene grown in oxygen-free conditions is contamination-free and shows n

12 rformance and possibilities for operation in oxygen-free conditions of an oxidase enzyme biosensor us

13 mental evaluation, constrained by prolonged, oxygen-free conditions, struggles to keep pace with emer

14 O is unable to react with nucleophiles under oxygen-free conditions, suggesting that its higher oxide

15 I) at neutral and acidic pH, under reducing, oxygen-free conditions.

16 modulates Hox hydrogen oxidation rates under oxygen-free conditions.

17 lic substrates, without the need for dry and oxygen-free conditions.

18 idize biomolecules by type I processes under oxygen-free conditions.

19 The primary outcome was supplemental oxygen-free days (SOFDs), defined as the number of days

20 Supplemental oxygen-free days as a clinical endpoint may allow smalle

21 fostamatinib did not increase the number of oxygen-free days compared with placebo.

22 e LRTI had a median of 16 fewer supplemental oxygen-free days than those presenting with URTI (P < .0

23 ulation (TXA-127 or TRV-027) did not improve oxygen-free days vs placebo.

24 The mean (SD) number of oxygen-free days was 13.4 (12.4) in the fostamatinib gro

25 Supplemental oxygen-free days were defined as any day while alive aft

26 The primary outcome was oxygen-free days, an ordinal outcome classifying a parti

27 The primary outcome was oxygen-free days, an ordinal outcome that classifies a p

28 0.48 to 1.13]) resulted in no difference in oxygen-free days.

29 tate is produced upon photoexcitation of the oxygen-free derivatives NPI-PTZ1 and NPI-PTZ4, with the

30 ent conditions a significant fraction of the oxygen-free edge sites are neither H-terminated nor unad

31 conversion under ambient conditions as in an oxygen-free environment in several unsaturated polyphosp

32 However, the requirement for an oxygen-free environment when classic radical polymerizat

33 ng of various ligands to OAM crystals (in an oxygen-free environment) leads to transimination in the

34 there is a 22x decrease in dissolution in an oxygen-free environment, proposing a route to decrease C

35 ure to low-intensity laser irradiation in an oxygen-free environment.

36 paration and manipulation of reactants in an oxygen-free environment; trace quantities of O2 lead to

37 ormation of methane by microbes that live in oxygen-free environments.

38 e functionality of oxidoreductase enzymes in oxygen-free environments.

39 Here we present oxygen-free flow lithography via inert fluid-lubrication

40 erived electrodes at high temperatures in an oxygen-free furnace.

41 The oxygen-free gas was routed through stainless steel tubin

42 g the fraction of substrate that reacts with oxygen free in solution.

43 -gas shift activity of platinum supported on oxygen-free multiwalled carbon nanotubes.

44 Given its origin in oxygen-free photochemistry, mass-independent sulfur isot

45 In contrast, the oxygen-free photoreaction activates previously inaccessi

46 evolved hydrogen facilitates preparation of oxygen-free powders.

47 lity to sequester iron from participation in oxygen free radical formation is consistent with a cytop

48 se results directly demonstrate an increased oxygen free radical generation during hypoxia and sugges

49 The direct demonstration of oxygen free radical generation during hypoxia is the cri

50 The role of oxygen free radical generation during reversible focal c

51 the hypothesis that maternal hypoxia induces oxygen free radical generation in the fetal guinea pig b

52 ly decreased the hypoxia-induced increase in oxygen free radical generation in the term fetal guinea

53 reserved liver cells is not mediated by: (1) oxygen free radical generation or improved by antioxidan

54 Allantoin, a marker of oxygen free radical generation, decreased by 20% after a

55 f neutrophil phagocytosis and a reduction in oxygen free radical generation, which may contribute to

56 are deficient in myeloid cell activation and oxygen free radical generation.

57 TP metabolism are the primary candidates for oxygen free radical generation: (a) MPTP oxidation to MP

58 physiologic outcome following treatment with oxygen free radical inhibitors and scavengers.

59 ogue 21 which were effective in reducing the oxygen free radical level.

60 ated metabolic changes leading to changes in oxygen free radical levels, which in turn lead to the in

61 asic pattern consisting of both acute phase (oxygen free radical mediated) and subacute phase (neutro

62 Oxygen free radical production also increased MCP-1 mRNA

63 ) isolated from piglet cortex to measure CEC oxygen free radical production and determine its role in

64 n is increased xanthine oxidase (XO)-derived oxygen free radical production and endothelial dysfuncti

65 tion in nNOS-/- mice may relate to decreased oxygen free radical production and related NO reaction p

66 plug capillary-sized pores and show enhanced oxygen free radical production may account for the exces

67 -/-) embryonic fibroblasts demonstrated high oxygen free radical production when exposed to hemin, hy

68 Blockade of oxygen free radical production with superoxide dismutase

69 inergic PC12 cell cultures, does not involve oxygen free radical production, but rather may be caused

70 n were increased in response to cytokines or oxygen free radical production, but the magnitude and du

71 A 55% increase in oxygen free radical production, determined by fluorescen

72 f energy metabolism, oxygen consumption, and oxygen free radical production, it becomes imperative to

73 (NO radical) exhibit changes in the rate of oxygen free radical production.

74 (+)/H(+) exchange blocker (amiloride), or an oxygen free radical scavenger (vitamin E).

75 treatment with N-acetyl-l-cysteine (NAC), an oxygen free radical scavenger, led to a reduction in the

76 Significantly, treatment with an oxygen free radical scavenger, N-acetyl-l-cysteine (NAC)

77 se results support the concept of developing oxygen free radical scavengers for both AD and PD and fu

78 ve stability and high generation of reactive oxygen free radical species.

79 ectron oxidation of hydrogen peroxide to the oxygen free radical, superoxide.

80 ly of UV radiation-induced, but also of some oxygen free radical-induced, DNA lesions.

81 d to damage dopamine-secreting neurons by an oxygen free radical-mediated mechanism.

82 Thus, reductions in oxygen free radical-mediated vascular injury may contrib

83 In order to promote oxygen-free radical generation, hypoxanthine (n=9) or xa

84 Targeting uremic toxins, oxygen-free radical production and the mitophagy process

85 ive phosphorylation were also decreased, and oxygen-free radical production increased.

86 in piglets have shown that the generation of oxygen free radicals (O(-)(2)) following traumatic brain

87 The generation of oxygen free radicals (OFR) and tissue infiltration by ac

88 t hypoxia induces an increased production of oxygen free radicals (OFR) in the brain of the guinea pi

89 Oxygen free radicals (OFRs) have been implicated in the

90 rophils by contact, leading to production of oxygen free radicals accompanied by release of granule p

91 (2) Oxygen free radicals also appear to mediate the hypoxic-

92 The UVA-dependent generation of oxygen free radicals also oxidized ASA at a 10(3) faster

93 y to alloxan, a generator of highly reactive oxygen free radicals and a potent beta-cell toxin.

94 ve stress; increased protein modification by oxygen free radicals and an elevated concentration of th

95 Because NFkappaB is a target of oxygen free radicals and Bcl-2 is an antioxidant gene, w

96 This injury is attributed in part to oxygen free radicals and has been partially ameliorated

97 ic newborn piglets through the generation of oxygen free radicals and induction of lipid peroxidation

98 Exercise increases the generation of oxygen free radicals and lipid peroxidation.

99 as previously believed to be accomplished by oxygen free radicals and other reactive oxygen species g

100 n-superoxide dismutase (SOD1) would diminish oxygen free radicals and reduce alcohol-induced liver in

101 elated cytokines; b) increased production of oxygen free radicals associated with ischemia/reperfusio

102 Generation of oxygen free radicals by treatment of telencephalic mitoc

103 The production of oxygen free radicals catalysed by non-haem iron was inve

104 ate that CECs produce significant amounts of oxygen free radicals following ischemia, primarily from

105 3'-blocking groups formed from the action of oxygen free radicals generated during normal cellular me

106 These results suggest that a burst of oxygen free radicals generated during the initial period

107 The involvement of oxygen free radicals has been suggested in c-fos activat

108 Oxygen free radicals have been implicated in beta-cell d

109 Elevated levels of oxygen free radicals have been implicated in the pathway

110 Oxygen free radicals have been invoked as mediators of t

111 Oxygen free radicals have been proposed to mediate amylo

112 These results not only support a role for oxygen free radicals in beta-AP toxicity but also highli

113 data, for the first time, suggest a role of oxygen free radicals in causing abnormality of female re

114 These data support a role of oxygen free radicals in promoting post-anoxic mitochondr

115 e, from the production and detoxification of oxygen free radicals in the mitochondrion to the efficac

116 f the Bad pathway after tFCI and the role of oxygen free radicals in the regulation of apoptosis rema

117 We tested the hypothesis that oxygen free radicals induce an increase in ODC activity

118 ment of S phase cells with agents that cause oxygen free radicals induces the dephosphorylation of DN

119 hat in tumor cells, endogenous production of oxygen free radicals may be a major factor in promoting

120 nhanced muscle fatigue, whereas formation of oxygen free radicals may be attenuated by endogenous pro

121 Oxygen free radicals may play a pivotal role in the mito

122 Oxygen free radicals oxidize arachidonic acid to a compl

123 However, the role of oxygen free radicals produced after ischemia/reperfusion

124 anistically, spermine neutralized off-target oxygen free radicals produced by NADPH oxidase-1 (Nox1)(

125 Short-lived oxygen free radicals react with the spin trap and produc

126 d coronary endothelial dysfunction caused by oxygen free radicals released during reperfusion.

127 ine and xanthine, xanthine oxidase generates oxygen free radicals that cause postischemic injury.

128 es are associated with the overproduction of oxygen free radicals that inflict cell damage.

129 Activation and oxygen free radicals were assessed using gp91(phox-/-) m

130 Oxygen free radicals were measured by ESR spectroscopy i

131 ble of generating significantly greater ROS (oxygen free radicals) than nondiabetic blood (P < 0.05).

132 such mucosal injury is initially mediated by oxygen free radicals, and because mitogen-activated prot

133 se is especially vulnerable to inhibition by oxygen free radicals, and the upstream metabolites, pyru

134 MPP(+), unlike rotenone, did not produce oxygen free radicals, but rather blocked ATP production

135 Oxygen free radicals, generated by cerebral ischemia, ha

136 Anticancer modalities based on oxygen free radicals, including photodynamic therapy and

137 cally accompanied by increased production of oxygen free radicals, is a cause and a consequence of ce

138 (1) Oxygen free radicals, particularly hydroxyl radical (OH.

139 Variable factors, including accumulation of oxygen free radicals, protein conformational changes, de

140 t the idea that MPTP toxicity is mediated by oxygen free radicals, we assessed lipid peroxidation and

141 pathophysiologic production of cytokines and oxygen free radicals, which potentiate organ injury in s

142 ave been proposed as a significant source of oxygen free radicals.

143 n thiols and inhibits cellular damage due to oxygen free radicals.

144 are related to its role in the generation of oxygen free radicals.

145 s in PD involves the accumulation of harmful oxygen free radicals.

146 stnatal exposure to environmental sources of oxygen free radicals.

147 tients may be related to oxidative damage by oxygen free radicals.

148 smutases (SODs) protect cells from damage by oxygen free radicals.

149 of MCP-1 may be stimulated by cytokines and oxygen free radicals.

150 disorders linked to excessive generation of oxygen free radicals.

151 philic response, which causes the release of oxygen free radicals.

152 are mediated via the formation of NE-derived oxygen free radicals.

153 nt in tumor tissue prevents the formation of oxygen free radicals.

154 cytochrome oxidase, and (5) the formation of oxygen free radicals.

155 Both STGP and LTGP gels produced oxygen free radicals.

156 , at least in part through the generation of oxygen free radicals.

157 nhance phagocytic activity and generation of oxygen free radicals.

158 he affected tissues produce large amounts of oxygen-free radicals and NO.

159 hypoxic-ischemic brain injury by generating oxygen-free radicals during reperfusion.

160 Oxygen-free radicals formed during normal aerobic cellul

161 Superoxide anion and other oxygen-free radicals have been implicated in the pathoge

162 ): Each does indeed bind copper and scavenge oxygen-free radicals in vivo.

163 e hypoxia results in increased generation of oxygen-free radicals including nitric oxide (NO), expres

164 We examined the role of XO in generating oxygen-free radicals that cause brain injury, hypothesiz

165 These results suggest that oxygen-free radicals, especially superoxide anions, are

166 and which are formed in DNA by the action of oxygen-free radicals.

167 In this study, the oxygen-free reaction of U(VI) with varying quantities of

168 in inorganic oxides and hydroxides using an oxygen-free solution containing a biradical polarization

169 escent (Phi = 0.2-0.3) in a room-temperature oxygen-free solution.

170 enzymatic systems were designed to maintain oxygen-free solutions in open, small volume electrochemi

171 esponse over prolonged (6-7 h) operations in oxygen-free solutions, indicating no depletion of the in

172 provide convenient biosensing of glucose in oxygen-free solutions.

173 um tungsten nitride (LaWN(3)) in the form of oxygen-free sputtered thin films, according to spectrosc

174 h Eubacteria and Archeabacteria in the early oxygen-free stages of the earth's evolution.

175 reover, requires the availability and use of oxygen-free starting materials.

176 ion in organic light-emitting diode devices, oxygen-free systems show very efficient photoinduced ICT

177 uboxic (0.2% oxygen), and anoxic conditions (oxygen-free with nitrate).