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1 requiring venovenous extracorporeal membrane oxygenation.
2 3.352; p = 0.18) for extracorporeal membrane oxygenation.
3 tilation (HFOV), and extracorporeal membrane oxygenation.
4 ury in patients with extracorporeal membrane oxygenation.
5 t using venoarterial extracorporeal membrane oxygenation.
6 erfusion (HMPO(2)), the other to HMP without oxygenation.
7 rior mitochondrial preservation with initial oxygenation.
8 cessfully weaned-off extracorporeal membrane oxygenation.
9 nsatory mechanisms to ensure adequate tissue oxygenation.
10 generates new erythrocytes to restore tissue oxygenation.
11 ) had surgery before extracorporeal membrane oxygenation.
12 llowing cessation of extracorporeal membrane oxygenation.
13 ith 28% treated with extracorporeal membrane oxygenation.
14 ents on venoarterial extracorporeal membrane oxygenation.
15 ared with venovenous extracorporeal membrane oxygenation.
16 apeutic range during extracorporeal membrane oxygenation.
17 nd those who died on extracorporeal membrane oxygenation.
18 n piecing together the events around Earth's oxygenation.
19 ane oxygenator function and does not improve oxygenation.
20 quiring venoarterial extracorporeal membrane oxygenation.
21 cute brain injury in extracorporeal membrane oxygenation.
22 ciated with death on extracorporeal membrane oxygenation.
23 lation target during extracorporeal membrane oxygenation.
24 requiring venovenous extracorporeal membrane oxygenation.
25 urs (70-1,008 hr) on extracorporeal membrane oxygenation.
26 ose not supported by extracorporeal membrane oxygenation.
27 penia in patients on extracorporeal membrane oxygenation.
28 erial and venovenous extracorporeal membrane oxygenation.
29 * provides information about relative tissue oxygenation.
30 in monitoring during extracorporeal membrane oxygenation.
31 cal disability after extracorporeal membrane oxygenation.
32 cessfully weaned-off extracorporeal membrane oxygenation.
33 assay in patients on extracorporeal membrane oxygenation.
34 ury in patients with extracorporeal membrane oxygenation.
35 m patients receiving extracorporeal membrane oxygenation.
36 tion that such oxidation enabled atmospheric oxygenation.
37 od cell integrity were maintained throughout oxygenation.
38 ses in listings with extracorporeal membrane oxygenation (+1.2%), intra-aortic balloon pumps (+ 4 %),
39 ation and venovenous extracorporeal membrane oxygenation (13% vs 10%; p = 0.4).
40 ared with venovenous extracorporeal membrane oxygenation (19% vs 10%; p = 0.002).
41 rried out in the gain-of-function fatty acid oxygenation 2 (fou2) mutant that, even when undamaged, s
42 l versus mixed group extracorporeal membrane oxygenation (23.9 vs 34.4 vs 29.4%; p = 0.2) or between
43 10%), HFOV (9%), and extracorporeal membrane oxygenation (3%).
44  1) general condition 2) local perfusion and oxygenation, 3) contamination, and 4) surgery related fa
45 tion of venoarterial extracorporeal membrane oxygenation, 4,918 of these patients had arterial carbon
46 nical ventilation or extracorporeal membrane oxygenation), 433 (90.4%) completed the primary outcome
47 cation of venovenous extracorporeal membrane oxygenation (46.7%).
48 ower in venoarterial extracorporeal membrane oxygenation (48%) than venovenous extracorporeal membran
49 48%) than venovenous extracorporeal membrane oxygenation (64%) (p < 0.001).
50  22 had venoarterial extracorporeal membrane oxygenation (88%) (nine cardiac arrest; 13 cardiogenic s
51 nical ventilation or extracorporeal membrane oxygenation, a 5-day course of remdesivir may provide si
52 requiring venovenous extracorporeal membrane oxygenation according to the pathogen.
53  shear stress inside extracorporeal membrane oxygenation additionally contributed to coagulation and
54 y strategies using respiratory hyperoxia and oxygenation agents in cancer treatment.
55 of barrels with different and known rates of oxygenation allows the effect of different oxygenation c
56          Patients on extracorporeal membrane oxygenation also followed a similar pattern of platelet
57  challenging to depict the potentially small oxygenation alterations with current noninvasive cardiac
58                                   The higher oxygenation altered methionine, lipid, and purine metabo
59 s 30% for venovenous extracorporeal membrane oxygenation and 37.5% for venoarterial extracorporeal me
60 er molecular masses, an increasing degree of oxygenation and a higher fraction of formulas containing
61                                              Oxygenation and acid-base homeostasis are maintained by
62 ty in the cVN related to changes in systemic oxygenation and blood pressure.
63 in a catalyst-controlled fashion between C-H oxygenation and C-H amination.
64    Treatment with nimodipine restores spinal oxygenation and can rapidly improve function.
65 hics, comorbidities, extracorporeal membrane oxygenation and cannulation characteristics, occurrence
66 tress syndrome aims for providing sufficient oxygenation and carbon dioxide clearance, while limiting
67  age (+/- SD) of the extracorporeal membrane oxygenation and cardiopulmonary bypass cohorts was 45.4
68  thrombocytopenia in extracorporeal membrane oxygenation and cardiopulmonary bypass were 6.4% (19/298
69 equired venoarterial extracorporeal membrane oxygenation and for whom sublingual microcirculation mea
70 ions associated with extracorporeal membrane oxygenation and identify prognostic and predictive facto
71 lloon pump in 2, and extracorporeal membrane oxygenation and Impella CP in 1.
72 whom were symptomatic) required supplemental oxygenation and inpatient treatment.
73 ne oxygenation in 2, extracorporeal membrane oxygenation and intra-aortic balloon pump in 2, and extr
74 stress syndrome and high compliance improves oxygenation and lung aeration but may result in alveolar
75       Although alterations in retinal vessel oxygenation and morphology have been reported in DR, the
76 gnition and management of failure to restore oxygenation and reduce the risk of cardiopulmonary arres
77 patients who died on extracorporeal membrane oxygenation and those successfully weaned.
78 cessfully weaned-off extracorporeal membrane oxygenation and those who died on extracorporeal membran
79 r between venovenous extracorporeal membrane oxygenation and venoarterial extracorporeal membrane oxy
80 between venoarterial extracorporeal membrane oxygenation and venovenous extracorporeal membrane oxyge
81                                              Oxygenation and ventilation inhomogeneity improved but a
82 t: 67%) were achievable in both cohorts when oxygenation and ventilation were allowed to vary within
83 sed by pulmonary embolism, postresuscitation oxygenation and ventilation, prophylactic antibiotics af
84 l ARDSnet settings to improve lung function, oxygenation and ventilation/perfusion matching, without
85 l metastases, tumor inflammation, perfusion, oxygenation, and acidity.
86 monary hypertension, extracorporeal membrane oxygenation, and cardiac arrest.
87 n 34% of the days on extracorporeal membrane oxygenation, and cryoprecipitate on 14%.
88  as enzymes for aromatic carbon degradation, oxygenation, and decarboxylation, and (ii) releasing low
89 e position sessions, extracorporeal membrane oxygenation, and inhaled nitric oxide; Pao2:Fio2 ratio m
90 erpretations related to changes in energy or oxygenation, and the analysis of reservoir quality given
91  vs 1; p = 0.01), preextracorporeal membrane oxygenation antiplatelet use (7 vs 0; p = 0.03), and a h
92 vival and various preextracorporeal membrane oxygenation as well as extracorporeal membrane oxygenati
93     We identified preextracorporeal membrane oxygenation as well as extracorporeal membrane oxygenati
94 athomechanism of the extracorporeal membrane oxygenation-associated coagulopathy and identify options
95 lying factors of the extracorporeal membrane oxygenation-associated coagulopathy differ between venov
96                  The extracorporeal membrane oxygenation-associated coagulopathy is a multifactorial
97 otocols for treating extracorporeal membrane oxygenation-associated coagulopathy should be further va
98 ian, 0.5; range, 0-2.0) correlated with both oxygenation at low PEEP and the oxygenation response; at
99             The FIO2 needed to obtain viable oxygenation at lower positive end-expiratory pressure wa
100  supported or not by extracorporeal membrane oxygenation at the time of heart transplantation.
101 l mortality, but not extracorporeal membrane oxygenation at the time of heart transplantation.
102  118 (28.4%) were on extracorporeal membrane oxygenation at the time of transplantation (peripheral,
103       Median time on extracorporeal membrane oxygenation before heart transplantation was 9 days (int
104 ate, respiration rate, temperature and blood oxygenation, but also provides a range of important addi
105 al versus venovenous extracorporeal membrane oxygenation, but described a variable correlation with c
106 ction, particularly when combined with micro-oxygenation, but fined wines after long term aging in bo
107 it thrombosis during extracorporeal membrane oxygenation, but no consensus exists on the optimal meth
108 and greater respiratory and locomotor muscle oxygenation, but there were no differences in ventilatio
109 en content that is the main driver of tissue oxygenation by RBCs.
110 cterization of intratumoral angiogenesis and oxygenation by using dynamic susceptibility contrast age
111 three had venovenous extracorporeal membrane oxygenation cannulation (12%).
112 outcomes and whether extracorporeal membrane oxygenation causes secondary brain injury.
113  were more common in extracorporeal membrane oxygenation compared with cardiopulmonary bypass through
114 us- and venoarterial-extracorporeal membrane oxygenation compared with cardiopulmonary bypass.
115 itation venoarterial extracorporeal membrane oxygenation compared with venovenous extracorporeal memb
116 ndrome on venovenous extracorporeal membrane oxygenation, compared with current ventilation practice
117 f oxygenation allows the effect of different oxygenation conditions throughout the process in barrels
118  care of patients on extracorporeal membrane oxygenation continues to evolve and grow, especially whe
119 ed on ELISA studies, indicating that singlet oxygenation could be an important natural detoxification
120 ontext, venoarterial extracorporeal membrane oxygenation could rapidly restore hemodynamics and tissu
121 ning criteria for an extracorporeal membrane oxygenation course as a vehicle for delivering the curri
122 (control) during the extracorporeal membrane oxygenation course.
123 ediatric acute respiratory distress syndrome oxygenation criteria for greater than or equal to 6 hour
124 e hypothesis that pediatric patients meeting oxygenation criteria for pediatric acute respiratory dis
125 ediatric acute respiratory distress syndrome oxygenation criteria with bilateral infiltrates on chest
126 implement, in vivo-like perfusion and stable oxygenation culture conditions in vitro semi-independent
127 on of a standardized extracorporeal membrane oxygenation curriculum; 2) defining criteria for an extr
128  circuit changes per extracorporeal membrane oxygenation day compared with less than 0.15 U/mL.
129 transfused on 68% of extracorporeal membrane oxygenation days, plasma on 34% of the days on extracorp
130 ventilation and post-extracorporeal membrane oxygenation decannulation.
131  during venoarterial extracorporeal membrane oxygenation decreased from 10% to 6% in 5 years.
132 longer time on the ventilator independent of oxygenation defect severity.
133 ric Risk of Mortality score, and severity of oxygenation defect, presence of bilateral infiltrates wa
134 death, initiation of extracorporeal membrane oxygenation, denial of valve replacement in patients wit
135  proposal for therapeutic use of antihypoxic oxygenation described here was motivated by the need to
136 tients on venovenous extracorporeal membrane oxygenation despite the delivery of volume- and pressure
137 third of patients on extracorporeal membrane oxygenation develop vascular complications; elderly male
138 16-2.22; p = 0.004), extracorporeal membrane oxygenation duration (adjusted odds ratio, 1.01; 95% CI,
139 dy was to evaluate the optimal start time of oxygenation during continuous hypothermic machine perfus
140 idneys were randomized to receive 2 hours of oxygenation during HMP either at the start (n = 6), or e
141 vival advantage over extracorporeal membrane oxygenation (ECMO) as a bridge to transplant.
142 ccur when initiating extracorporeal membrane oxygenation (ECMO) in patients with respiratory failure
143 od coagulation in an extracorporeal membrane oxygenation (ECMO) setting in rabbits, all without incre
144 recommend the use of extracorporeal membrane oxygenation (ECMO) support for COVID-19-related acute hy
145 who had a history of extracorporeal membrane oxygenation (ECMO) underwent PT (11% vs 2%, p=0.049).
146 use of pretransplant extracorporeal membrane oxygenation (ECMO), and on index hospitalization length
147  trial in the USA of extracorporeal membrane oxygenation (ECMO)-facilitated resuscitation versus stan
148 tomy or venoarterial extracorporeal membrane oxygenation [ECMO]) is safe and effective.
149 imitations in global extracorporeal membrane oxygenation education exist.
150 escribe the state of extracorporeal membrane oxygenation education worldwide, noting current limitati
151                                        Renal oxygenation, eGFR, and SCr improved after MT + PTRA.
152 corporated into enzymes only after the Great Oxygenation Event, are either absent or present at conce
153                     Ultra-slow variations in oxygenation follow a cascade.
154 ed with venoarterial extracorporeal membrane oxygenation for refractory postcardiotomy cardiogenic sh
155 ported by venovenous extracorporeal membrane oxygenation for severe acute respiratory syndrome corona
156 e of all patients on extracorporeal membrane oxygenation from 2012 to 2018 at a tertiary referral cen
157 re subregion analysis revealed steep spatial oxygenation gradients in growing tumors that were reduce
158 t transplantation on extracorporeal membrane oxygenation had similar survival compared with those not
159 tion or venoarterial extracorporeal membrane oxygenation) had surgery before extracorporeal membrane
160 rted by venoarterial extracorporeal membrane oxygenation has been associated with poor prognosis.
161               During extracorporeal membrane oxygenation, hemodilution and contact of blood component
162 uration, serious complications, apneic time, oxygenation, ICU length of stay, or overall survival whe
163                        Background Myocardial oxygenation imaging could help determine the presence of
164 ted the venoarterial extracorporeal membrane oxygenation impact on macrocirculatory hemodynamics and
165 hether the timing of extracorporeal membrane oxygenation implantation influences the renal prognosis
166  before venoarterial extracorporeal membrane oxygenation implantation regressed within 12 hours after
167 s after venoarterial extracorporeal membrane oxygenation implantation were excluded.
168 %), Impella CP in 2, extracorporeal membrane oxygenation in 2, extracorporeal membrane oxygenation an
169  long been known, the role of extracorporeal oxygenation in dictating changes in the respiratory quot
170  and it selectively increased cerebral blood oxygenation in fetuses with single ventricle or aortic o
171 es of maternal hyperoxia increased placental oxygenation in healthy fetuses and fetuses with congenit
172 e results support the notion that reduced AT oxygenation in individuals with obesity contributes to i
173 teria for the use of extracorporeal membrane oxygenation in poisoning.
174  We have measured slow net flow and high net oxygenation in the placenta in vivo, which are consisten
175 culties with laryngoscopy and provide rescue oxygenation in the setting of failed attempts.
176                              Due to improved oxygenation in the spinner flasks, organoids rapidly pro
177 ents and as a proxy measure of perfusion and oxygenation in tumor tissue undergoing antiangiogenic tr
178 erapy; corticosteroids started at the lowest oxygenation index (13.0; interquartile range, 7.6-22.0)
179 Variability was noted in the median starting oxygenation index of each therapy; corticosteroids start
180  five topic areas of extracorporeal membrane oxygenation initiation and management.
181 , and 48 hours after extracorporeal membrane oxygenation initiation, respectively.
182 rior to venoarterial extracorporeal membrane oxygenation initiation, with 18% cannulated during it.
183 is already common at extracorporeal membrane oxygenation initiation.
184 more candidates with extracorporeal membrane oxygenation, intra-aortic balloon pumps, and exception r
185 sis after venovenous extracorporeal membrane oxygenation is a frequent complication.
186           Venovenous extracorporeal membrane oxygenation is an effective intervention to improve gas
187             However, extracorporeal membrane oxygenation is associated with a coagulopathy characteri
188 e recommendations is extracorporeal membrane oxygenation is best performed by a multidisciplinary tea
189 ave shown that decreased adipose tissue (AT) oxygenation is involved in the pathogenesis of obesity-i
190  patients undergoing extracorporeal membrane oxygenation, it is unclear which cannulation method carr
191 etween local neuronal activity and the blood oxygenation level dependent (BOLD) signal can be describ
192        Ultra-slow, ~0.1-Hz variations in the oxygenation level of brain blood are widely used as an f
193 in the anesthetized rat, we identified blood oxygenation level-dependent (BOLD) responses directly re
194                           However, the blood oxygenation level-dependent (BOLD) signal provides an in
195 aging in our healthy controls and with blood oxygenation level-dependent functional imaging in 35 hea
196 de conclusive evidence that changes in blood oxygenation level-dependent signal amplitude and frequen
197   Here, we simultaneously measured the blood-oxygenation level-dependent signal from 12 triads (n = 3
198  luminance contrast and abnormal rapid blood oxygenation level-dependent signal saturation to high lu
199 ng Proton Imaging of Siloxanes to map Tissue Oxygenation Levels (PISTOL) magnetic resonance imaging.
200             As research proceeds appropriate oxygenation levels should be carefully considered.
201 umab treatment was followed by a recovery in oxygenation levels.
202 n patients receiving extracorporeal membrane oxygenation, little is known regarding the mechanism and
203  0.025), duration of extracorporeal membrane oxygenation (&lt; 66 hr: odds ratio, 1; 66-128 hr: odds rat
204                  Pre-extracorporeal membrane oxygenation macrocirculation, echocardiography, arterial
205 pecific protocol for extracorporeal membrane oxygenation management encompassing patient selection, i
206 arly implantation of extracorporeal membrane oxygenation may help prevent acute kidney injury.
207 sure basic cardiovascular physiology, spinal oxygenation, mitochondrial function, and tissue perfusio
208 c evaluation, 68% of extracorporeal membrane oxygenation nonsurvivors developed acute brain injury.
209 nal complications on extracorporeal membrane oxygenation (odds ratio, 2.346; 95% CI, 1.203-4.572; p =
210 hod can be applied to the chemoselective C-H oxygenation of benzylic, allylic, and propargylic C(sp(3
211                     This report presents the oxygenation of C-H bonds via the merger of photocatalysi
212 n of hydrocarbons (e.g., propane) by surface oxygenation of platinum (Pt)-alloyed multicomponent nano
213  peroxodiiron(III) intermediate (P) from the oxygenation of the diiron(II) enzyme and its subsequent
214 deoxyhaemoglobin, we observed an increase in oxygenation of the entire tumour volume immediately afte
215 cate that this pathway primarily affects the oxygenation of the inner retina involved in signal proce
216 e system designed to mimic the physiological oxygenation of the pancreas, we demonstrate high viabili
217                      Earlier attempts to use oxygenation of tumors as a monotherapy or to improve rad
218 , we evaluated the potential influence of AT oxygenation on AT biology and insulin sensitivity in peo
219 ithin 12 hours after extracorporeal membrane oxygenation onset.
220  early Cambrian, such as episodic changes in oxygenation or productivity.
221 er group (venovenous extracorporeal membrane oxygenation or venoarterial extracorporeal membrane oxyg
222 cular assist system, extracorporeal membrane oxygenation, or a combination of MCS device use), or med
223 onse; at PEEP 15, high recruiters had better oxygenation (P = 0.004), whereas low recruiters experien
224 penia in patients on extracorporeal membrane oxygenation (p = 0.79).
225  The administration of hMSCs did not improve oxygenation (Pa(O(2))/Fi(O(2)) mean difference = -146 mm
226  care of patients on extracorporeal membrane oxygenation: patient selection, management, mitigation o
227 95 in 459 venovenous extracorporeal membrane oxygenation patients (odds ratio, 2.35; 95% CI, 1.87-2.9
228 ion and venoarterial extracorporeal membrane oxygenation patients and are best diagnosed by a combina
229 ree of 10 venovenous extracorporeal membrane oxygenation patients and in four of eight venoarterial e
230 n 6,124 venoarterial extracorporeal membrane oxygenation patients compared with 95 in 459 venovenous
231         Venoarterial extracorporeal membrane oxygenation patients had more ischemic stroke (10% vs 1%
232         Venoarterial extracorporeal membrane oxygenation patients had more overall brain injury compa
233 ysical disability in extracorporeal membrane oxygenation patients plays a significant role in psychia
234  10,342 venoarterial extracorporeal membrane oxygenation patients, 401 (3.9%) experienced ischemic st
235 hort of venoarterial extracorporeal membrane oxygenation patients.
236 f eight venoarterial extracorporeal membrane oxygenation patients.
237 .5% for venoarterial extracorporeal membrane oxygenation patients.
238 topenia was lower in extracorporeal membrane oxygenation patients.
239 l of nine complex natural products with rich oxygenation patterns and skeletal diversity in 10 steps
240 to existing blood vessels, thereby affecting oxygenation, perfusion, and systemic dissemination.
241  surgery, providing information about tissue oxygenation, perfusion, hemoglobin concentration and wat
242 third of the days on extracorporeal membrane oxygenation, plasma on one third, and cryoprecipitate on
243          Even before extracorporeal membrane oxygenation, plasmatic coagulation and platelet aggregat
244  We conclude that this mechanism for retinal oxygenation played a vital role in the adaptive evolutio
245 n the development of extracorporeal membrane oxygenation practitioner certification; and 5) promoting
246                                         With oxygenation proposed as a resuscitative measure during h
247  to improve radiotherapy have failed because oxygenation protocols were not combined with immunothera
248 lar complications on extracorporeal membrane oxygenation, published from 1972 to January 31, 2020, wa
249 ed here exhibited a linear relationship with oxygenation (R(1) = A' + B'*pO(2)) at all temperatures a
250 report a visible-light-mediated benzylic C-H oxygenation reaction.
251                                Enzymatic C-H oxygenation reactions are often complementary to small m
252 ediatric patients on extracorporeal membrane oxygenation receiving anticoagulant therapy.
253                             Our bottom-water oxygenation record is compared to other similar high-res
254 and the frequency of extracorporeal membrane oxygenation-related complications.
255 ygenation as well as extracorporeal membrane oxygenation-related factors that are associated with mor
256 ygenation as well as extracorporeal membrane oxygenation-related factors.
257 rted with venovenous extracorporeal membrane oxygenation remains high, and this may be due in part to
258 eath or venoarterial extracorporeal membrane oxygenation removal.
259             Hematologic values pre- and post-oxygenation, respectively were (median +/- IQR): Red blo
260 ivery, export productivity, and bottom-water oxygenation, respectively.
261 ed with both oxygenation at low PEEP and the oxygenation response; at PEEP 15, high recruiters had be
262 d respiratory rate, systolic blood pressure, oxygenation, retractions, capillary refill, atelectasis
263 e issues surrounding extracorporeal membrane oxygenation selection, cannulation, and management are a
264               Before extracorporeal membrane oxygenation start, patients were randomized to either re
265 re direct assessment of tumor glycolysis and oxygenation status quantitatively.
266 vasive imaging surrogate biomarker of tissue oxygenation status.
267 piratory failure, treatment with noninvasive oxygenation strategies compared with standard oxygen the
268                                        First oxygenation strategies included standard oxygen (n = 245
269                   Treatment with noninvasive oxygenation strategies such as noninvasive ventilation a
270  with ARDS, early exposure to a conservative-oxygenation strategy with a Pao(2) between 55 and 70 mm
271        Results showed that outcomes of micro-oxygenation strongly depend on aging treatments.
272 scharge and need for extracorporeal membrane oxygenation support post-transplant.
273           The median extracorporeal membrane oxygenation support time was 96 hours (interquartile ran
274  during venoarterial extracorporeal membrane oxygenation support, before future routine use of this p
275   Among venoarterial extracorporeal membrane oxygenation-supported drug-refractory electrical storm p
276                      Extracorporeal membrane oxygenation survivors' physical quality of life was wors
277 to survive following extracorporeal membrane oxygenation than those with other poisonings (49% vs 72%
278 nd blood are associated with impaired tissue oxygenation, the development of new therapies based on t
279 t decreased tumor volumes, angiogenesis, and oxygenation, thereby reflecting its effectiveness for ex
280 f prevalence, combinations of therapies, and oxygenation threshold for which the therapies are applie
281 of arylboronic acids and benzylic C(sp(3))-H oxygenation through reductive quenching.
282           The use of extracorporeal membrane oxygenation to support critically ill, poisoned patients
283  use of venoarterial extracorporeal membrane oxygenation to treat severe cardiogenic shock patients,
284 rs towards regioselective intramolecular C-H oxygenations to provide cyclic carbonates, hydroxylated
285 reased rate of early extracorporeal membrane oxygenation use (12% vs 7%, P = 0.04).
286 aphical variation in extracorporeal membrane oxygenation use by geospatially mapping the ZIP code ass
287                      Extracorporeal membrane oxygenation use in poisoned patients in the United State
288 the highest rates of extracorporeal membrane oxygenation use.
289 he [4 + 2]-cycloaddition, site-selective C-H oxygenation using a novel tandem acylation/[3,3]-rearran
290 oring heparin during extracorporeal membrane oxygenation using activated clotting times to anti-Xa he
291         Venoarterial extracorporeal membrane oxygenation (VA-ECMO) is increasingly used to treat card
292  use of venoarterial extracorporeal membrane oxygenation (VA-ECMO) may facilitate graft rescue.
293 ssessed venoarterial extracorporeal membrane oxygenation (VA-ECMO) support for sepsis-induced cardiog
294 lity was higher when extracorporeal membrane oxygenation was used for metabolic or hematologic poison
295                      Extracorporeal membrane oxygenation was used in the care of 514 consecutive chil
296 ratory hyperoxia as a means to improve tumor oxygenation, we provide in vivo evidence that hypoxia is
297 om the initiation of extracorporeal membrane oxygenation were associated with neurologic complication
298 y 24% of the days on extracorporeal membrane oxygenation were free of any hemostatic transfusions.
299 atients treated with extracorporeal membrane oxygenation were successfully weaned.
300                       This improves cortical oxygenation, which, together with lesser tubular gluco-t

 
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