ライフサイエンスコーパス: ozone

1 and -0.9 [95% CI, -1.3 to -0.4; P<0.001] for ozone).

2 ame day in 2-pollutant models (adjusting for ozone).

3 Air Quality Standard (NAAQS) obligations for ozone.

4 ups (teenagers/young adults/seniors) without ozone.

5 ean, prior to the formation of stratospheric ozone.

6 to [Formula: see text], carbon monoxide, and ozone.

7 y related to attack by hydroxyl radicals and ozone.

8 er at the surface shields the inner BaP from ozone.

9 yde, acetaldehyde, peroxyacetyl nitrate, and ozone.

10 nfection with influenza virus or exposure to ozone.

11 o the ocean freeze-up and a seasonal rise in ozone.

12 to and more pronounced than those induced by ozone.

13 was formed, which can be further degraded by ozone.

14 s on the removal of NO (x) by oxidation with ozone.

15 hat facilitate the formation of tropospheric ozone.

16 hylpiperidine-1-oxyl, hydrogen peroxide, and ozone.

17 n SS content was observed at higher doses of ozone.

18 direct radiative forcing due to aerosols and ozone.

19 we did not observe a measurable influence of ozone (0 vs 40 ppb) on human FAP emissions, there was a

20 ancements in PM(2.5) (0.5-1.4 mug m(-3)) and ozone (0.4-0.7 ppbv) occur around the densely populated

21 e exposed to filtered air (FA) or repetitive ozone (0.5 ppm O3, 4 h/d, for 13 consecutive weekdays).

22 and on both days of exposure (air or 0.8ppm ozone, 4 hr/day x 2-days).

23 The effect of gaseous ozone (500-1000 ppm) treatment on the protein, amino aci

24 eaths related to fine particulate matter and ozone (95CI: 25 000-120 000), heart attacks (900-9400),

25 Ozone, a major contributor to poor air quality, has an a

26 NG emissions are predicted to affect surface ozone across a large geographical scale in the United St

27 ductions in maximum daily 8 h average (MDA8) ozone across the four-state domain of 3.5 and 7.1 ppb, r

28 When combined with chloramination, ozone addition after chloramines maintained a higher RO

29 Other conditions including human occupancy, ozone addition, and cleaning with terpene, natural produ

30 nds for the monodentate attack leading to an ozone adduct.

31 ome to quantum chemical characterizations of ozone adducts.

32 n and the greater intracellular diffusion of ozone after the membrane disruption induced by LEEFT.

33 ns allow us to evaluate strategies to attain ozone air quality standards at minimum cost or to maximi

34 Drought conditions affect ozone air quality, potentially altering multiple terms i

35 age) and environmental parameters (level of ozone, air temperature, and relative humidity) on partic

36 nds, conducted below room temperature, using ozone, an iron salt, and a hydrogen atom donor.

37 It drives ozone and aerosol production, modulates atmospheric oxid

38 s well as OH oxidation), as well as aging by ozone and atmospheric dilution can transform the OP(mass

39 Ozone-based treatment trains comprising ozone and biologically activated carbon potentially offe

40 l responses in floral pigmentation linked to ozone and climate change.

41 erated from the reaction of alpha-pinene and ozone and compared and discussed the results in relation

42 oal to surface concentrations of PM(2.5) and ozone and direct radiative forcing due to aerosols and o

43 Observed ozone and elemental mercury depletion rates are quantita

44 pheric reactive halogen chemistry, depleting ozone and elemental mercury, thereby enhancing depositio

45 ntribution to harmful air pollutants such as ozone and fine particulate matter (PM(2.5))(4).

46 ations of health-damaging pollutants such as ozone and fine particulate matter (PM(2.5)).

47 ozone and hydrogen peroxides were found to be responsibl

48 y the simultaneous reduction of tropospheric ozone and methane.

49 Concentrations of gaseous ozone and nitrogen oxides were determined with Fourier-t

50 u the impact of heterogeneous reactions with ozone and other trace gases on ice nucleation.

51 g daily respiratory ED visits with estimated ozone and PM(2.5) concentrations during the week before

52 We assessed the association of exposure to ozone and PM(2.5) with 1-year health status and mortalit

53 the impact of emissions on ambient levels of ozone and PM(2.5), and a health impact assessment tool i

54 n symptoms, formed from the reaction between ozone and squalene.

55 ral pigmentation was associated with altered ozone and temperature in 42 species spanning three conti

56 cate that the cycloaddition reaction between ozone and trans-isoprene follows a stepwise mechanism, w

57 lutants (fine particulate matter [PM2.5] and ozone) and cause-specific risk of hospital admission in

58 ts of different combinations of chloramines, ozone, and biological activated carbon (BAC), applied as

59 and PM2.5, respectively), nitrogen dioxide, ozone, and black carbon.

60 We found that long- and short-term PM2.5, ozone, and nitrogen dioxide exposures were all associate

61 meter less than or equal to 2.5 mum (PM2.5), ozone, and nitrogen dioxide with all-cause mortality on

62 ave combined FACE with temperature, drought, ozone, and nitrogen treatments.

63 , with key impacts on atmospheric oxidation, ozone, and organic aerosols.

64 e conditions (10 min of ultrasound, 5 min of ozone, and pulsed light at 10 V) showed pronounced effec

65 s reveals a formal oxidation state of -2 for ozone anions in CaO(3).

66 as recorded between ethylene antagonists and ozone application in cold storage on the ethylene produc

67 Using ozone as a model gas-phase pollutant, we show that titan

68 ssivate the interfacial defects by employing ozone as a secondary oxidant.

69 The application of ozone as an adjunctive treatment represents a new approa

70 onation of WWTP effluents confirmed that the ozone attack of STG was incomplete even at high ozone do

71 Ozone attacks the primary amine moiety of STG, leading t

72 An ozone-based chemiluminescence approach was used to inves

73 resonance spectroscopy, HPLC), nitrosative (ozone-based chemiluminescence) and inflammatory (fluores

74 d [Hb], blood viscosity, and NO metabolites (ozone-based chemiluminescence) were measured before and

75 sed treatment, we identify opportunities for ozone-based or hybrid treatment trains to reduce treatme

76 states effectively limit the extent to which ozone-based treatment alone can produce recycled water f

77 Ozone-based treatment trains comprising ozone and biolog

78 ment, and hybrid treatment trains comprising ozone-based treatment with a membrane sidestream, and (2

79 cycled water using membrane-based treatment, ozone-based treatment, and hybrid treatment trains compr

80 a preoxidant before membranes or as part of ozone/biological activated carbon (O(3)/BAC) systems.

81 annually combining the effects of NMVOCs and ozone, but could be reduced by nearly 40% by closing the

82 The quantification of ozone by SIFT-MS was investigated in conditions suitable

83 s the Cost And Benefit Optimization Tool for Ozone (CABOT-O(3)), which extends the previous model by

84 Inhalation of the ambient air pollutant ozone causes lung inflammation and can suppress host def

85 how that including interactive stratospheric ozone chemistry in atmospheric model calculations leads

86 een NMVOCs and nitrogen oxides and hence the ozone chemistry in the east and south coast.

87 This suggests that stratospheric ozone chemistry is important for the understanding of se

88 able about the entrance channel and isoprene-ozone complexes thought to define the long-range portion

89 were strongly correlated with the estimated ozone concentration in infiltrating air.

90 Raspberry fruit was ozonated with an ozone concentration of 8-10 ppm for 30 min, every 12 h,

91 For a high ozone concentration range, only NO(2)(-) and O(2)(-) hav

92 et benefits results in greater emissions and ozone concentration reductions in some parts of the coun

93 Temperature, humidity, UV radiation and ozone concentration were recorded and hair swatches of d

94 tribution of emissions reductions to ambient ozone concentrations across the contiguous United States

95 the surface layers but, at sufficiently high ozone concentrations, occurred throughout the film.

96 Yields (ppb product emitted/ppb ozone consumed) for 40 products were quantified.

97 Reconstructions of the UVI from total ozone data show evidence of increasing UVI levels in the

98 rature; second, taxa that experienced larger ozone declines will display larger increases in pigmenta

99 Ozone decreased circulating lymphocytes, increased FFA,

100 Ozone decreased from 2011 to the 2030 baseline, with med

101 Ozone decreased monohydroxy fatty acids and acyl carniti

102 Rapid ozone degradation during the 20(th) century resulted in

103 r water disinfection is hindered by its high ozone demand and the resulting high cost.

104 le with an industrial emission context (high ozone demand, dry air/oxygen as the manufacturing gas of

105 ide (N(2)O) is a powerful greenhouse gas and ozone depleting substance, but its natural sources, espe

106 As a potent greenhouse gas and an ozone-depleting agent, nitrous oxide (N(2)O) plays a cri

107 decline in the atmospheric concentrations of ozone-depleting gases such as chlorofluorocarbons(1).

108 Chlorofluorocarbons (CFCs) are ozone-depleting substances previously thought to be pers

109 e Antarctic stratosphere due to emissions of ozone-depleting substances(9-11).

110 from these banks, and associated impacts on ozone depletion and climate change.

111 established that these trends were driven by ozone depletion in the Antarctic stratosphere due to emi

112 Yet, although our main concern about ozone depletion is the subsequent increase in harmful so

113 Reduced ozone depletion potential for VRFB and LFP can be achiev

114 , especially in Antarctica, where effects of ozone depletion were larger.

115 entrations have contributed to stratospheric ozone depletion(1) and climate change(2), with the curre

116 with regard to human health, air pollution, ozone depletion, acidification, and land transformation.

117 Following complete ozone depletion, elevated bromine concentrations are sus

118 he stratosphere would result in catastrophic ozone depletion, extending the surface cooling caused by

119 er stratospheric cooling-primarily caused by ozone depletion-yields [Formula: see text] values betwee

120 so been speculated to have led to wide-scale ozone depletion.

121 l) significantly contribute to stratospheric ozone depletion.

122 Adduction of these receptors with ozone-derived oxysterols impaired ligand binding and cor

123 g macrophage proteins that form adducts with ozone-derived oxysterols.

124 osed anthers experiencing larger declines in ozone displayed more dramatic pigmentation increases.

125 reatment process is beneficial to reduce the ozone dosage and disinfection by-product formation with

126 OM isolates were studied by varying specific ozone doses (0.1-1.3 mg-O(3)/mg-C) at pH 7.

127 R) was treated by O(3)-BAF at three specific ozone doses (0.5, 0.7, and 1.0 mg O(3)/mg DOC) and diffe

128 ne attack of STG was incomplete even at high ozone doses of 1.7 and 0.9 mg O(3)/mg DOC, respectively.

129 ver, previous research has demonstrated that ozone drastically increases the formation potential of g

130 lternative possible cause for the postulated ozone drop: a nearby supernova explosion that could infl

131 The results showed that the ozone effect depends on the profile and content of antho

132 Since ozone effects are mediated through the activation of AR

133 winter, reduced nitrogen oxides resulted in ozone enhancement in urban areas, further increasing the

134 Ozone-enriched atmosphere appears to counteract postharv

135 ate that long-range dynamics in the isoprene-ozone entrance channel can impact the overall reaction i

136 ents factors (epsilon(C)) for reactions with ozone (epsilon(C) = -3.6 to -4.6 per mille) and hydroxyl

137 Ozone exposure (37 ppb) was found to have little influen

138 he dynamic increase in emission rates during ozone exposure also varied among participants, possibly

139 The association between short-term ozone exposure and respiratory diseases was robust.

140 Periodic ozone exposure during gate oxide ALD on SiGe is shown to

141 ortality associated with long-term PM2.5 and ozone exposure increased substantially at low levels.

142 n diabetic animal strains repetitive ambient ozone exposure led to early and exaggerated pulmonary in

143 Ozone exposure of vehicle-treated rats increased broncho

144 , HCAR1, and LINC00336 DNA methylation after ozone exposure relative to clean air.

145 ly insulin-resistant KKAy mice were used for ozone exposure studies.

146 ression of macrophage phagocytosis following ozone exposure through the generation of oxysterols and

147 icroRNA expression obtained before and after ozone exposure was also used to identify changes associa

148 llion (ppb) increase in long- and short-term ozone exposure was associated with 2.35 (95% CI: 1.08, 3

149 mpletely adjusted models, higher PM(2.5) and ozone exposure were independently associated with poorer

150 e relationship between long-term PM(2.5) and ozone exposure with health status outcomes 1 year after

151 tions of incomplete PAH decay upon prolonged ozone exposure.

152 utrophil counts in the sputum in response to ozone exposure.

153 ne whether pulmonary responses to repetitive ozone exposures are exacerbated in murine strains that a

154 feedstock olefins with beta-nitrostyrenes by ozone/Fe(II) -mediated radical substitutions, are report

155 esidence-based daily nitrogen dioxide (NO2), ozone, fine particulate, and black carbon concentrations

156 10/group/sex) were exposed to air or 0.8 ppm ozone for 5 h.

157 separated occasions to clean air or 0.3-ppm ozone for two hours.

158 ing may affect atmospheric OH reactivity and ozone formation rates downwind of urban centers through

159 In chemical reactions such as ozone formation, a delta(17) O/delta(18) O=1 is observed

160 ozone precursors and its impacts in shaping ozone formation.

161 g hazardous air pollutant concentrations and ozone formation.

162 Long-term ozone ([Formula: see text]) exposure is associated with

163 nitrogen dioxide ([Formula: see text]), and ozone ([Formula: see text]) from satellite-derived data,

164 e to nitrogen dioxide ([Formula: see text]), ozone ([Formula: see text]), fine particulate matter [Fo

165 d to increased human exposure to PM(2.5) and ozone from seven emission sectors for 2005 to 2018.

166 articles <=2.5 um in diameter (PM(2.5))) and ozone gas and, therefore, these species have been the ma

167 Further exposure of PAH-SOA to ozone generally increased the concentration ratio of PAH

168 Ozone generation is observed under all conditions, with

169 e of dry air as the manufacturing gas of the ozone generator) affected the ozone quantification by SI

170 y air/oxygen as the manufacturing gas of the ozone generator, and high humidity levels beyond saturat

171 ate matter <2.5 um in diameter (PM(2.5)) and ozone has been associated with the development and progr

172 Gaseous ozone has been recently proposed as sanitizing agent to

173 Anthropogenic (pollution) ozone has increased iodine emissions since preindustrial

174 cleaner air for decades, unhealthy levels of ozone have decreased but remain stubbornly frequent even

175 se temporally and be correlated with reduced ozone (higher UV) when accounting for effects of tempera

176 eal Protocol has begun to heal the Antarctic ozone hole and avoided more global warming than any othe

177 The Antarctic ozone hole is decreasing in size but this recovery will

178 vered, these CFC banks could delay Antarctic ozone hole recovery by about six years and contribute 9

179 me for policymakers to plug the holes in the ozone hole treaty.

180 oduction over 10 years, disappearance of the ozone hole will be delayed by a few years, although ther

181 e, using model simulations, we show that the ozone hole will largely cease to occur by 2065 given com

182 unusual meteorology of 2002 is repeated, an ozone-hole-free-year could occur as soon as the early 20

183 sotope effects (AKIEs) for the reaction with ozone, however, was nontrivial due to challenges in assi

184 n and photo-oxidation caused by ground level ozone impacts hair properties such as melanin oxidation,

185 NO (400 ppm) and a variable concentration of ozone in a mixture of N(2) and O(2) was directed through

186 Application of ozone in cold storage maintained higher levels of sugars

187 anisms of chemical transformations of BaP by ozone in indoor and outdoor environments are still not f

188 ly responsible for the continued decrease of ozone in the lower stratosphere.

189 mission reductions on the occurrence of high ozone in the region.

190 m(3).s(-1), and the diffusion coefficient of ozone in the thin film is 9 x 10(-10) cm(2).s(-1) These

191 component, and the reacto-diffusive depth of ozone in the triolein coating is estimated to be between

192 linear production chemistry and titration of ozone in winter, reduced nitrogen oxides resulted in ozo

193 (0.6-1.2 V) LEEFT significantly enhances the ozone inactivation.

194 Ozone increased various amino acids, polyamines, and met

195 gonist (dexamethasone; DEX) would exacerbate ozone-induced pulmonary and systemic changes.

196 and consequent desorption from the SiGe with ozone insertion during the ALD growth process is confirm

197 Because ozone is a common atmospheric oxidant, such compounds ma

198 Ozone is an important oxidant in the environment.

199 Whether exposure to PM(2.5) and ozone is associated with patients' health status and qua

200 So far the impact on ozone is small, but if these emissions indicate producti

201 ed over dense cities, whereas warming due to ozone is widespread, peaking at 4.2 mW m(-2) over the At

202 le evidence that air pollution, specifically ozone, is associated with declines in bird abundance in

203 The recovery of the stratospheric ozone layer relies on the continued decline in the atmos

204 a significant threat to the recovery of the ozone layer.

205 ulations Case" results in a maximum peak 8 h ozone level of 162 ppb and 24 h PM(2.5) of 42.7 mug/m(3)

206 ncluding some carcinogenic species) and peak ozone levels by 20-30% and 6-15% respectively, in the co

207 of premature deaths using modeled changes in ozone levels resulting from the application of emission

208 of the increased emissions on summer surface ozone levels.

209 s responsible for 32% of the halogen-induced ozone loss (bromine 40%, chlorine 28%), due primarily to

210 aqueous and hydrate phases that can lead to ozone loss.

211 niors), relative humidity (low or high), and ozone (<2 ppb or ~35 ppb).

212 The rate coefficients (k(ozone)) measured for the BCA + ozone (O(3)) reaction at

213 No water interference was identified during ozone measurements by SIFT-MS using NO(2)(-) and O(2)(-)

214 ificantly obscures key biomarker gases (e.g. ozone, methane) in simulated transmission spectra, imply

215 be strongly dependent on the film thickness, ozone mixing ratio, and ozone reactivity of the surface

216 a function of average coating thickness and ozone mixing ratio, we determined that the reactive upta

217 Oceanic emissions of iodine destroy ozone, modify oxidative capacity, and can form new parti

218 Satellite observations of the Ozone Monitoring Instrument (OMI) for tropospheric sulfu

219 nt advancement in a retrieval method for the Ozone Monitoring Instrument (OMI) sensor enabled detecti

220 al predictor variables include data from the Ozone Monitoring Instrument, Copernicus Atmosphere Monit

221 4 mg/h of indoor SOA formation due to indoor ozone-monoterpene chemistry.

222 conomic status, seven contextual covariates, ozone, nitrogen dioxide, and combined oxidative potentia

223 Annual mean community-level ozone, nitrogen dioxide, and particulate matter less tha

224 Exposures to ozone, nitrogen dioxide, sulfur dioxide, carbon monoxide

225 Rising ozone (O(3) ) concentrations, coupled with an increase i

226 TL) and estimated annual average residential ozone (O(3)) and fine particulate matter with a diameter

227 onation of drinking and wastewater relies on ozone (O(3)) and hydroxyl radical ((*)OH) as oxidants.

228 ere aged under visible light with or without ozone (O(3)) at an atmospherically relevant level in an

229 low-tube reactor at atmospherically relevant ozone (O(3)) exposure levels (5-30 ppb h) with pure squa

230 Urban ozone (O(3)) formation can be limited by NO(x), VOCs, or

231 r dioxide (SO(2)), carbon monoxide (CO), and ozone (O(3)) in 2013-17 were averaged from 35 monitoring

232 Tropospheric ozone (O(3)) is a key component of air pollution and an

233 Urban ozone (O(3)) pollution is influenced by the transport of

234 efficients (k(ozone)) measured for the BCA + ozone (O(3)) reaction at 295 +/- 2 K were 4.8 +/- 0.6 x

235 y studies have linked short-term exposure to ozone (O(3)) with morbidity and mortality, but epidemiol

236 gen dioxide (NO(2)), sulfur dioxide (SO(2)), ozone (O(3)), and carbon monoxide (CO)-and daily hospita

237 <=2.5 mum in aerodynamic diameter (PM(2.5)), ozone (O(3)), and nitrogen dioxide (NO(2)) exposures at

238 such as carbon dioxide (CO(2)), tropospheric ozone (O(3)), and particulate matter (PM) can be reduced

239 Total hydroxyl radical (OH), ozone (O(3)), nitrate radical (NO(3)), and chlorine atom

240 ic compounds (VOCs), carbon dioxide (CO(2)), ozone (O(3)), nitric oxide (NO), nitrogen dioxide (NO(2)

241 rvous system activity and more so with added ozone (O(3)).

242 % CI: 17 to 45%), with marginal increases in ozone (O(3): 4%; 95% CI: -2 to 10%) in 34 countries duri

243 h risk associated with time-varying prenatal ozone (O3) exposure over pregnancy.

244 s pulmonary responses to air pollutants like ozone (O3).

245 most cooking oils was oxidized by gas-phase ozone on a surface.

246 amic diameter, oxides of nitrogen (NOx), and ozone on characterized chronic obstructive pulmonary dis

247 ound little evidence of an overall effect of ozone on the DNA methylome but some suggestive changes i

248 of organic pollutants with oxidants such as ozone or hydroxyl radicals by compound-specific stable i

249 idate the contribution of the reactions with ozone or hydroxyl radicals to overall transformation.

250 o statistically significant associations for ozone or PM10.

251 emistry is represented by using an empirical ozone-organics reaction probability that matches the obs

252 e enzymes of allergens, detergents, tobacco, ozone, particulate matter, diesel exhaust, nanoparticles

253 kg of carbon per hectare, 8.1 and 1.42 kg of ozone per hectare, and 8.4 and 8 kg of PM(10) per hectar

254 ncided with a dramatic drop in stratospheric ozone, possibly due to a global temperature rise.

255 States, can regionally offset reductions of ozone precursor emissions made in other sectors, and can

256 ly chain relocates emissions of tropospheric ozone precursors and its impacts in shaping ozone format

257 ow that an air pollution regulation limiting ozone precursors emissions has delivered substantial ben

258 d but remain stubbornly frequent even as the ozone precursors NO(x) (nitrogen oxides = NO(2) + NO) an

259 Ozone premature mortalities away from the leak increased

260 With ozone present, OH reactivity nearly doubled, with the in

261 With ozone present, the total OH reactivity decreased slightl

262 Several compounds were transformed during ozone pretreatment that were poorly removed in the open-

263 water unit-process wetlands with and without ozone pretreatment was studied over a 2-year period.

264 tion of thin solid films of NPM by gas-phase ozone produces unexpected products, the majority of whic

265 The hydroxyl radical (OH) fuels tropospheric ozone production and governs the lifetime of methane and

266 is recognized to be necessary when modelling ozone production).

267 issions include higher rates of tropospheric ozone production, increases in the lifetime of methane,

268 ing gas of the ozone generator) affected the ozone quantification by SIFT-MS.

269 asing iodine emissions have implications for ozone radiative forcing and possibly new particle format

270 Per 20-ppb increase in ozone, rate ratios were 1.017 (1.011-1.023) among childr

271 the film thickness, ozone mixing ratio, and ozone reactivity of the surface substrate.

272 Ozone reacts with cholesterol in the lung to form oxyste

273 Ozone reacts with four negative precursor ions available

274 ssions of 67 Gg yr(-1) would delay Antarctic ozone recovery by well over a decade.

275 al Protocol and the associated stratospheric ozone recovery might therefore manifest, or have already

276 rthermore, we demonstrate that stratospheric ozone recovery, resulting from the Montreal Protocol, is

277 participant-to-participant heterogeneity in ozone responses.

278 Except for ozone, satellite measurements of the troposphere indicat

279 Major products of the primary reaction of ozone-squalene included 6-methyl 5-hepten-2-one (6-MHO)

280 mpared to minimizing costs to meet a uniform ozone standard, maximizing net benefits results in great

281 Upon exposure to ozone, the decay of thin coatings of triolein was observ

282 For ozone, the IRR for asthma was 0.85 (95% CI, 0.71-1.02) f

283 Without ozone, the whole-body OH reactivity was dominated by bre

284 [Formula: see text], cleaved and annealed in ozone to increase the doping all the way to the non-supe

285 oxide anion radical and hydrogen peroxide by ozone-treated fruit was significantly lower than in the

286 m of this work was to evaluate the impact of ozone treatment during winegrapes dehydration (10 and 20

287 ast, lactic acid bacteria, enzyme, peroxide, ozone, UV light and cold plasma.

288 creased number of days when the averaged 8-h ozone values exceed 70 ppb, with the highest sensitivity

289 alculations predict multiple stable isoprene-ozone van der Waals complexes for trans-isoprene in the

290 rmed global and regional tests examining the ozone versus clean air effect on the DNA methylome and c

291 ssover experiment to estimate the effects of ozone (vs.

292 n our study, greater exposure to PM(2.5) and ozone was associated with poorer 1-year health status fo

293 ), a major component of cannabis smoke, with ozone was examined as a pure compound and within cannabi

294 high-fat diet (HFD) on metabolic response to ozone was examined in Long-Evans rat offspring.

295 With O(2)(-) precursor ion, ozone was successfully measured in the presence of nitro

296 Moreover, higher PM(2.5) exposure, but not ozone, was independently associated with greater mortali

297 in regulating tropospheric and stratospheric ozone, while some hHNPs bioaccumulate and have toxic pro

298 ic pollutants and precursors of tropospheric ozone, while the Middle East is a global emission hotspo

299 nment of reactive positions for reactions of ozone with aromatic carbons in ortho-, meta-, or para-po

300 izes the lasting environmental damage to the ozone, with R134a (1,1,1,2-tetrafluoroethane) being used