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1 ng component of this receptor complex is the p75 neurotrophin receptor.
2 Trk receptor tyrosine kinases and the shared p75 neurotrophin receptor.
3 sociate with the intracellular domain of the p75 neurotrophin receptor.
4 he iota PKC.IRAK complex is recruited to the p75 neurotrophin receptor.
5 very similar gamma-secretase cleavage is the p75 neurotrophin receptor.
6 mechanism, supported by up-regulation of the p75 neurotrophin receptor.
7 the actions of BDNF are mediated through the p75 neurotrophin receptor.
8 so be induced by selective activation of the p75 neurotrophin receptor.
9 uce Schwann cell death via engagement of the p75 neurotrophin receptor.
10 ival or cell death decisions mediated by the p75 neurotrophin receptor.
11 ia a yet undefined mechanism mediated by the p75 neurotrophin receptor.
12 ransducer for the effects of NGF through the p75 neurotrophin receptor.
13 ergic neurons and terminals that express the p75 neurotrophin receptor.
14 the Fas receptor, the p75 TNF receptor, and p75 neurotrophin receptor.
15 f NGF or by antibodies to either trkB or the p75 neurotrophin receptor.
16 dependent signalling mechanism involving the p75 neurotrophin receptor.
17 e extracellular neurotrophin binding site of p75 neurotrophin receptor.
18 n of the kinase-active Trk receptors and the p75 neurotrophin receptor.
19 l-like receptor superfamilies, including the p75 neurotrophin receptor.
20 ARMS can physically associate with TrkA and p75 neurotrophin receptors.
21 GF binds to the TrkA tyrosine kinase and the p75 neurotrophin receptor, a member of the tumor necrosi
25 e studies provide a new reagent for altering p75 neurotrophin receptor actions after injury and sugge
26 t regulated intramembrane proteolysis of the p75 neurotrophin receptor (also known as p75 cleavage).
27 ls via the tyrosine receptor kinase B (TrkB)/p75 neurotrophin receptor and Fyn kinase in transfected
29 eurotrophin receptor types: the multifaceted p75 neurotrophin receptor and the tropomyosin receptor k
31 Thirty years ago, it was discovered that p75 neurotrophin receptor and tropomyosin receptor kinas
32 an interact through its TRAF domain with the p75 neurotrophin receptor and weakly with the lymphotoxi
33 factor induced apoptosis in cells expressing p75 neurotrophin receptor, and enhances neurite outgrowt
35 educes binding of pro-nerve growth factor to p75 neurotrophin receptor, blocks pro-nerve growth facto
36 erve growth factor (NGF) in complex with the p75 neurotrophin receptor but is distinct from that of m
37 t dependence, these neurons express trkB and p75 neurotrophin receptor but not trkA or trkC mRNAs.
38 ng to mature oligodendrocytes expressing the p75 neurotrophin receptor, but not trkA, resulted in a s
39 ta, osteoprotegerin, FOXC2 and FOXF2, BMP-2, p75 neurotrophin receptor, caspase-11, guanylate-binding
41 are closely related to NRAGE, which mediates p75 neurotrophin receptor-dependent apoptosis, and necdi
42 This consolidates a potential role of the p75 neurotrophin receptor during stress and inflammatory
43 e-bolus propofol applications at the peak of p75 neurotrophin receptor expression after experimental
44 cause is the lack of NO, which up-regulated p75 neurotrophin receptor expression, a receptor require
45 ate developmental-like programs and increase p75 neurotrophin receptor expression, probably to foster
46 DNF-tyrosine-related kinase B (TrkB) p75NTR (p75 neurotrophin receptor) facilitates stabilization of
47 ed a cDNA for the LacZ reporter or the human p75 neurotrophin receptor, for which species-specific an
48 combines the 192 monoclonal antibody to the p75 neurotrophin receptor found on terminals and cell bo
50 n contrast, optical density for low-affinity p75 neurotrophin receptor immunoreactivity in the VLDB d
51 terferes with oligomerization of full-length p75 neurotrophin receptor in a dose-dependent manner.
53 rescence for either ChAT or the low-affinity p75 neurotrophin receptor in the nucleus basalis Meynert
54 arget-derived NGF promotes expression of the p75 neurotrophin receptor, in turn causing a reduction i
58 a valine residue at position 264 in the rat p75 neurotrophin receptor is necessary for the ability o
61 itory signals through the same Nogo receptor/p75 neurotrophin receptor/LINGO-1 (NgR1/p75/LINGO-1) com
65 enesis and recent evidence suggests that the p75 neurotrophin receptor (NTR) contributes significantl
66 sal forebrain neurons immunoreactive for the P75 neurotrophin receptor (NTR) in male and female Sprag
68 tors while down-regulating the expression of p75 neurotrophin receptor (NTR), phospho-JNK, and Bcl-2-
69 -hybrid library with the death domain of the p75 neurotrophin receptor (NTR), we identified the Sall2
73 ived compound, EVT901, which interferes with p75 neurotrophin receptor oligomerization through direct
75 that TAp73 is a transcriptional activator of p75 neurotrophin receptor (p75(NTR)) and that p75(NTR) m
78 eurotrophic factor (BDNF)-activated TrkB and p75 neurotrophin receptor (p75(NTR)) by disrupting the e
80 al role in Alzheimer's disease (AD), and the p75 neurotrophin receptor (p75(NTR)) has been implicated
89 f nerve growth factor (NGF) signaling by the p75 neurotrophin receptor (p75(NTR)) is critical for neu
90 e of distinct structural determinants in the p75 neurotrophin receptor (p75(NTR)) is crucial for the
96 can be initiated by activation of either the p75 neurotrophin receptor (p75(NTR)) or the more selecti
99 cellular signaling networks regulated by the p75 neurotrophin receptor (p75(NTR)) substantially overl
101 neuron loss, distal axonal degeneration and p75 neurotrophin receptor (p75(NTR)) upregulation in the
102 Strikingly, mutant male mice lacking the p75 neurotrophin receptor (p75(NTR)) were resistant to t
103 erestingly, mutant male mice that lacked the p75 neurotrophin receptor (p75(NTR)) were seen to be res
104 structures of complexes formed by the DD of p75 neurotrophin receptor (p75(NTR)) with RhoGDI, for ac
105 s the de novo expression of the low-affinity p75 neurotrophin receptor (p75(NTR)), a gene that plays
108 examined the expression of the low affinity p75 neurotrophin receptor (p75(NTR)), an excellent marke
109 c efferents, identified by the low-affinity, p75 neurotrophin receptor (p75(NTR)), with interneurons
110 This produced a substantial loss of both p75 neurotrophin receptor (p75(NTR))-positive and cholin
111 proNGF, is a potent apoptotic ligand for the p75 neurotrophin receptor (p75(NTR))-sortilin complex.
116 tropomyosin kinase receptor B (TrkB) and the p75 neurotrophin receptor (p75) are the primary regulato
118 pomyosin-related kinase receptors (Trks) and p75 neurotrophin receptors (p75) compete to regulate sur
121 Proneurotrophins bind with high affinity to p75 neurotrophin receptor (p75NTR) and lack the capacity
123 atory ligand B7-1 (hB7-1) interacts with the p75 neurotrophin receptor (p75NTR) and that the B7-1:p75
124 tment with antisense oligonucleotides to the p75 neurotrophin receptor (p75ntr) decreased basal survi
125 pment of the sympathetic nervous system, the p75 neurotrophin receptor (p75NTR) has a dual function:
126 tate gyrus, the distribution of low-affinity p75 neurotrophin receptor (p75NTR) immunoreactivity (IR)
127 septum, choline acetyltransferase (ChAT) and p75 neurotrophin receptor (p75NTR) immunoreactivity, and
128 Here, we investigate the involvement of p75 neurotrophin receptor (p75NTR) in Abeta-treated huma
129 , we identify an anti-migratory role for the p75 neurotrophin receptor (p75NTR) in cerebellar develop
132 Our goal was to determine the role of the p75 neurotrophin receptor (p75NTR) in the loss of islet
133 ad, myelin-associated glycoprotein recruited p75 neurotrophin receptor (p75NTR) into a complex with L
142 nizes trkA, trkB, and trkC) and low-affinity p75 neurotrophin receptor (p75NTR) was examined in the h
143 ors, tropomyosin related kinase B (TrkB) and p75 neurotrophin receptor (p75NTR), are increasingly app
145 ohol drinking produces a mobilization of DLS p75 neurotrophin receptor (p75NTR), whose activities opp
152 mesenchymal cells expressing both desmin and p75 neurotrophin receptor (p75NTR): HSCs in the liver pa
153 Here, we demonstrate that mice lacking the p75 neurotrophin receptor, p75NTR, decrease their feedin
155 the same set of transcription factors from a p75 neurotrophin receptor peptide (p75NTRp)-tagged adeno
157 NF appear to be mediated by the low-affinity p75 neurotrophin receptor, rather than a trk receptor.
160 (brain-derived neurotrophic factor, proBDNF, p75 neurotrophin receptor, S100B), neuropsychometric tes
161 that of NRAGE, a MAGE protein that mediates p75 neurotrophin receptor signaling and neuronal apoptos
162 e results revealed new functions for proBDNF-p75 neurotrophin receptor signaling pathway in the contr
164 ermore, we demonstrate that EVT901 abrogates p75 neurotrophin receptor signalling by other ligands, s
166 and gp120 were blocked by inhibitors of the p75 neurotrophin receptor, suggesting that proBDNF and g
167 c inhibition of the cell death domain of the p75 neurotrophin receptor (TAT-Pep5) and in mice lacking
168 urotrophins such as BDNF can bind to p75NTR (p75 neurotrophin receptor), their precursors are the hig
169 rve growth factor (NGF) and the low-affinity p75 neurotrophin receptor, their sensory nerve fibers ex
170 ecule, nicotinic acetylcholine receptor, and p75 neurotrophin receptor), thus demonstrating that pseu
171 t Par-3 directly associates and recruits the p75 neurotrophin receptor to the axon-glial junction, fo
172 s ability to redirect the apically localized P75 neurotrophin receptor to the basolateral membrane do
173 ding assays with cysteine-rich domains-fused p75 neurotrophin receptor, we confirmed that EVT901 inte
174 opomyosin receptor kinase (Trk) A, TrkB, and p75 neurotrophin receptor were all expressed in primary
175 he p75 TNF receptor, the Fas antigen, or the p75 neurotrophin receptor, which are other members of th