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1 an be explained by the presence of nonfiring pacemaker cells.
2 ed in other mammalian cells and in amphibian pacemaker cells.
3 ler (7-15 microm in diameter) than relay and pacemaker cells.
4 activation of a variety of ionic currents in pacemaker cells.
5 MP agonists were similar to the responses of pacemaker cells.
6 ells form chemical synapses on the relay and pacemaker cells.
7 anscriptional or translational events in SCN pacemaker cells.
8 ut not GABA(B), receptors are located on SCN pacemaker cells.
9 l types of ion channels expressed in cardiac pacemaker cells.
10 the pacemaker nucleus, i.e., relay cells and pacemaker cells.
11 determine the host response of hESC-SAN-like pacemaker cells.
12 l and functional processes characteristic of pacemaker cells.
13 ontrols automaticity in cardiac and neuronal pacemaker cells.
14  convert chamber cardiomyocytes into induced pacemaker cells.
15  vivo studies to be controlled by intestinal pacemaker cells.
16 of rhythmic activity in cardiac and neuronal pacemaker cells.
17 ides a new perspective on the concept of SCN pacemaker cells.
18 rrent fluctuations on the IBIs of sinoatrial pacemaker cells.
19 cells, hematopoietic cells, and interstitial pacemaker cells.
20 a small structure containing <10,000 genuine pacemaker cells.
21 and physiological automaticity of native SAN pacemaker cells.
22 n program may be especially important in fly pacemaker cells.
23 re initiated by a small number of sinus node pacemaker cells.
24 ncluding thalamocortical neurons and cardiac pacemaker cells.
25 intercellular communications and the role of pacemaker cells.
26 l rhythmic, spontaneous action potentials by pacemaker cells.
27 r potassium current (I(K)) in guinea pig SAN pacemaker cells.
28  of the SR alter pacemaking in these primary pacemaker cells.
29 ends upon input, including PDF, from central pacemaker cells.
30 ate the firing rates of neuronal and cardiac pacemaker cells.
31 is abundant expression of endogenous Pak1 in pacemaker cells; (2) expression of constitutively active
32 cytes, as well as specialized conduction and pacemaker cells, agonist binding to muscarinic acetylcho
33 lts from electrical remodeling of individual pacemaker cells along with structural remodeling and a b
34              Recent findings discovered that pacemaker cells also express the tyrosine kinase recepto
35                                      Primary pacemaker cells also have a SR-dependence of cardiac pac
36                     In spontaneously beating pacemaker cells, an increase in subsarcolemmal intracell
37 s generated by endosomal Ca2+ release in the pacemaker cell and was propagated by gap junctional comm
38 generating electrical rhythmicity in cardiac pacemaker cells and diverse types of brain neurons, key
39 f the regulatory landscape of human SAN-like pacemaker cells and functional assessment of SAN-specifi
40 emonstrate for chemosensing the existence of pacemaker cells and how the presence of gap junctions im
41 fection causes dysfunction of human SAN-like pacemaker cells and induces ferroptosis.
42 K channels control spike frequency in atrial pacemaker cells and inhibitory potentials in neurons.
43 ated chloride channel, ANO1, is expressed by pacemaker cells and may contribute to spontaneous depola
44     Interstitial cells of Cajal (ICC) act as pacemaker cells and possess unique ionic conductances th
45 ontains two well-characterized neuron types: pacemaker cells and relay cells.
46  rapidly increasing mitochondrial calcium in pacemaker cells and that MCU-enhanced oxidative phoshory
47  are histochemically distinct from relay and pacemaker cells and that they receive electrotonic input
48 t a Pak1 signaling pathway exists in cardiac pacemaker cells and that this novel pathway plays a role
49 ndent differentiation of upper urinary tract pacemaker cells and the efficient flow of urine from the
50  to lead to a sustained hyperpolarization of pacemaker cells and thereby reduces heart rate.
51 human pluripotent stem cell-derived SAN-like pacemaker cells and ventricle-like cells and identified
52  myenteric ICC (ICC-MY) in human jejunum are pacemaker cells and whether these cells actively propaga
53 iption factors could be utilized to generate pacemaker cells, and suggest ISL1 mutations may underlie
54 to convert quiescent heart-muscle cells into pacemaker cells, and the successful generation of sponta
55 connexin expression phenotypes in sinus node pacemaker cells, and to define the spatial distribution
56 acent to the inner nerve ring where swimming pacemaker cells are located.
57          These findings suggest that cardiac pacemaker cells are physically segregated and molecularl
58                                              Pacemaker cells are specialized cell types that drive bi
59 erlying muscles fail to propagate beyond the pacemaker cell, are slow, initiate in abnormal locations
60  secondary heart fields, we found that chick pacemaker cells arise from a discrete region of mesoderm
61  to a model of the evolutionary emergence of pacemaker cells as neurons using components of innate im
62                         Rhythmic activity in pacemaker cells, as in the sino-atrial node in the heart
63 e anatomical and neurochemical properties of pacemaker cells, as well as in the mechanisms of clock-g
64 at Isl1 is a specific marker for a subset of pacemaker cells at developmental stages examined, and su
65                       Electrical coupling of pacemaker cells at gap junctions appears to play an impo
66 pled-clock system that drives normal cardiac pacemaker cell automaticity.
67                                      Cardiac pacemaker cells autonomously generate electrical impulse
68  medial septum/diagonal band of Broca (MSDB) pacemaker cells, because, in addition to previously desc
69 ten terminated at the periphery of relay and pacemaker cell bodies.
70 d in both sino-atrial node and latent atrial pacemaker cells but not in working atrial myocytes.
71 tomaticity in isolated sinoatrial node (SAN) pacemaker cells, but the potential role of NCX1 in deter
72 noatrial node myocytes (SAMs) act as cardiac pacemaker cells by firing spontaneous action potentials
73      Sinoatrial node myocytes act as cardiac pacemaker cells by generating spontaneous action potenti
74 nally, it has been shown that bio-engineered pacemaker cells can be generated from non-rhythmic ventr
75 lines) were selectively expressed in cardiac pacemaker cells, cardiomyocytes, vascular endothelial an
76 t interstitial cells of Cajal (ICC) serve as pacemaker cells, conduits for active transmission of ele
77 eveal that they consist of three subtypes of pacemaker cells: Core Pacemaker, Sinus Venosus, and Tran
78                                      Cardiac pacemaker cells (CPCs) rhythmically initiate the electri
79                                      Cardiac pacemaker cells create rhythmic pulses that control hear
80  subunit also affects the reliability of the pacemaker cell; cycle timing is often irregular.
81 nfection of hESC-derived functional SAN-like pacemaker cells demonstrates ferroptosis as a potential
82 ng molecular mechanisms, especially of human pacemaker cell development, are poorly understood.
83 c pacemaker program, including activation of pacemaker cell differentiation transcription factors Isl
84                 These results link defective pacemaker cell differentiation with hydronephrosis and p
85 to the molecular processes involved in human pacemaker cell differentiation, opening new avenues for
86 e preparations revealed three populations of pacemaker cells distinguished on the basis of connexin i
87       These results indicate that subsets of pacemaker cells express distinct connexin phenotypes.
88 al node is compartmentalized, with a core of pacemaker cells, fibroblasts and glial cells supporting
89  of the hypothalamus are described as master pacemaker cells for biological rhythms.
90 Cs) can be a source to derive human SAN-like pacemaker cells for disease modeling.
91 erstandings for generating stable and robust pacemaker cells from non-pacemaking VMs by the interplay
92 sarcoplasmic reticulum (SR) regulate cardiac pacemaker cell function by activation of electrogenic Na
93 lar Ca(2+) cycling dynamics regulate cardiac pacemaker cell function on a beat-to-beat basis remains
94  of SAN-specific REs potentially involved in pacemaker cell gene regulation.
95 ar from these studies that ICC serve as: (i) pacemaker cells, generating the spontaneous electrical r
96 rker to identify and isolate sinoatrial node pacemaker cells has been reported.
97 ontrolling target gene expression in the SAN pacemaker cells have remained undefined.
98                                   In cardiac pacemaker cells, hetero-tetramer GIRK1/4 channels and ho
99                                     Putative pacemaker cells, identified by immunoreactivity for hype
100                vri is expressed in circadian pacemaker cells in larval and adult brains.
101 otypical ANO/SCN/TRPM ion channel-expressing pacemaker cells in the basal metazoan Hydra by using a c
102 ells of Cajal (ICCs) have been identified as pacemaker cells in the gastrointestinal (GI) tracts of v
103 sing a hamster model, we showed that primary pacemaker cells in the heart can be infected by SARS-CoV
104 cillations (6-9 Hz), which are controlled by pacemaker cells in the medial septal area.
105 acemaker potentials from isolated sinoatrial pacemaker cells in the presence of endogenous cAMP conce
106 terstitial cells of Cajal (ICC) are putative pacemaker cells in the rabbit urethra.
107                                     Resident pacemaker cells in the renal pelvis are critical to this
108                  The spontaneous activity of pacemaker cells in the sinoatrial (SA) node controls hea
109 vity decreases heart rate (HR) by inhibiting pacemaker cells in the sinoatrial node (SAN).
110 or decreasing the spontaneous firing rate of pacemaker cells in the sinoatrial node.
111 een firing and recently discovered nonfiring pacemaker cells in the sinoatrial node.
112        Interstitial cells of Cajal (ICC) are pacemaker cells in the small bowel, and therefore this c
113 ration of oscillations in neural activity of pacemaker cells in the suprachiasmatic nucleus (SCN).
114         Thus, Nkx2-5 defines a population of pacemaker cells in the transitional zone.
115 ived cardiac progenitors produces functional pacemaker cells in vitro, advancing the therapeutic pote
116 19) identify the embryonic origin of cardiac pacemaker cells in zebrafish and implicate Wnt5b in prom
117                  CRY overexpression in brain pacemaker cells increases behavioral photosensitivity, a
118 ditis elegans, a periodic calcium spike in a pacemaker cell initiates a calcium wave in the intestine
119 oneDB.org module, we identify trans-synaptic pacemaker cell interactions with glia.
120 ctivity and neoplastic transformation of gut pacemaker cells (interstitial cells of Cajal).
121 by gene transfer or by delivering engineered pacemaker cells into normally quiescent myocardium.
122 frequency, but the identity of the lymphatic pacemaker cell is still debated.
123 yperpolarization-activated current (I(f)) in pacemaker cells is passed by hyperpolarization-activated
124 ntracellular Ca2+ release to automaticity of pacemaker cells isolated from cat right atrium.
125  heat-triggered rate acceleration in cardiac pacemaker cells, isolated hearts and in vivo.
126 strointestinal (GI) muscles are generated by pacemaker cells, known as interstitial cells of Cajal (I
127                                              Pacemaker cells, known as interstitial cells of Cajal (I
128 us, the biological clock of sinoatrial nodal pacemaker cells, like that of many other rhythmic functi
129 in physiology and behavior are controlled by pacemaker cells located in the suprachiasmatic nucleus (
130 right atrium coexpressed the sinoatrial node pacemaker cell marker HCN4.
131 titial cells of Cajal, suggesting that these pacemaker cells may also be involved in neural reflexes.
132 n concert with the autonomic nervous system, pacemaker cells, myogenic mechanisms, and/or electrotoni
133                                     Putative pacemaker cells near the plexus showed immunoreactivity
134                                           In pacemaker cells of the brain, PER and TIM proteins rise
135 ulated rhythmically in nuclei of eyes and in pacemaker cells of the brain.
136 e 5-HT(2B) receptor is also expressed on the pacemaker cells of the gastrointestinal tract, the inter
137                                          The pacemaker cells of the heart initiate the heartbeat, sus
138 tivated potassium current (IKACh) channel in pacemaker cells of the heart.
139  coronavirus 2 (SARS-CoV-2) infection on the pacemaker cells of the heart.
140 d chloride channel anoctamin 1 (ANO1) to the pacemaker cells of the human and mouse urethra, the inte
141 e sequential activation of ionic currents in pacemaker cells of the sinoatrial node.
142 the idea that Ca(2+) regulates CL in cardiac pacemaker cells on a beat-to-beat basis, and suggest a m
143 eretofore focused on intrinsically bursting "pacemaker" cells operating in conjunction with synaptic
144 s and Ca transient decay to insure fail-safe pacemaker cell operation within a wide range of rates.
145 because they either modify the rhythm of the pacemaker cell or are essential for pattern generation i
146    Streptozotocin-treated mice had increased pacemaker cell ox-CaMKII and apoptosis, which were furth
147 lock spontaneous slow waves, suggesting that pacemaker cells populate all regions of the circular mus
148                 A single isolated sinoatrial pacemaker cell presents intrinsic interbeat interval (IB
149 along with smooth myosin heavy chain for the pacemaker cells (previously termed 'atypical' smooth mus
150 s driving heart rate acceleration in cardiac pacemaker cells remain incompletely understood.
151 icantly lower input resistances in relay and pacemaker cells, respectively, exhibiting drastically di
152 es instruct the motor steps and regulate the pacemaker cell's authority and reliability.
153  on the surface membrane of sinoatrial nodal pacemaker cells (SANCs) are the proximal cause of an act
154 polarization (DD) in rabbit sinoatrial nodal pacemaker cells (SANCs) generate an inward current (I(NC
155  Studies in animal models have revealed that pacemaker cells share a common progenitor with the (pro)
156                Their isolated peripheral SAN pacemaker cells showed a reduced Na+ channel expression
157                                 We validated pacemaker cell-specific elements in the SHOX2 and TBX3 l
158           A similar oscillator is present in pacemaker cells such as the interstitial cells of Cajal
159                         This sinoatrial node pacemaker cell surface marker is highly valuable for ste
160  electrical rhythmicity, which originates in pacemaker cells surrounding the myenteric plexus, called
161 ficantly less ox-CaMKII, exhibited increased pacemaker cell survival, maintained normal heart rates,
162  slow waves that are thought to originate in pacemaker cells termed interstitial cells of Cajal (ICC)
163 ity mediates axonal growth of the Drosophila pacemaker cells, termed "small ventrolateral neurons" (s
164                                  Specialized pacemaker cells, termed atypical smooth muscle cells (AS
165 n the interstitial cells of Cajal (ICC), the pacemaker cells that control smooth muscle contraction.
166        Interstitial cells of Cajal (ICC) are pacemaker cells that generate electrical activity to dri
167  In the gastrointestinal (GI) tract, ICC are pacemaker cells that generate spontaneous electrical slo
168  Lipsius demonstrated that in cat subsidiary pacemaker cells the late phase of diastolic depolarizati
169 ER in two significant clusters of behavioral pacemaker cells: the large and the small ventral lateral
170  in survival, proliferation, and function of pacemaker cells throughout development.
171 ural and technological systems, from cardiac pacemaker cells to coupled lasers.
172  model and human ESC (hESC)-derived SAN-like pacemaker cells to explore the impact of severe acute re
173 nditions can hamper the ability of the SAN's pacemaker cells to generate consistent action potentials
174 hrough a polysynaptic circuit extending from pacemaker cells to PI neurons.
175  flow of temporal information from circadian pacemaker cells to selective behaviors.
176 esponsiveness reflects the properties of the pacemaker cell type, not the receptor.
177 ," whose compositions differ between E and M pacemaker cell types.
178 l cells of Cajal (ICC), the gastrointestinal pacemaker cells, underlie a number of gastrointestinal m
179       In the present study, in mouse SA node pacemaker cells, we investigated Na+ currents under phys
180       The s-LNvs are the principal circadian pacemaker cells, whereas recent evidence indicates that
181      A variety of receptors are found on SCN pacemaker cells which permit the clock mechanism to resp
182  derive from hESCs functional human SAN-like pacemaker cells, which express pacemaker markers and dis
183 strategy to derive functional human SAN-like pacemaker cells, which was further characterized by sing
184                                              Pacemaker cells within the hamster suprachiasmatic nucle
185 or circadian rhythms of bioluminescence from pacemaker cells within the head for several days in indi
186 shift activity rhythms by acting directly on pacemaker cells within the SCN.

 
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