戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 tive therapeutic target for this devastating pain disorder.
2 algorithm indicating severity of the chronic pain disorder.
3 s inherited erythromelalgia, a human genetic pain disorder.
4 gional pain syndrome (CRPS), a posttraumatic pain disorder.
5 iple Na(V)1.8 variants associated with human pain disorders.
6 ng chronic urticaria, atopic dermatitis, and pain disorders.
7 ausally related to a broad spectrum of human pain disorders.
8 ritable bowel syndrome and related abdominal pain disorders.
9 on is enhanced in and predictive of clinical pain disorders.
10 unction as a causative factor in neuropathic pain disorders.
11 inform the development of new analgesics for pain disorders.
12 t of better treatments for acute and chronic pain disorders.
13 across multiple aetiologically heterogeneous pain disorders.
14 voltage-gated sodium channel Nav1.9 in human pain disorders.
15 TRESK has been implicated in nociception and pain disorders.
16 ated sodium channel (VGSC) variants to human pain disorders.
17 n approved for treatment of multiple chronic pain disorders.
18 ion, and certain mental health disorders and pain disorders.
19 es but rather associated with behavioral and pain disorders.
20 n the development and maintenance of chronic pain disorders.
21 d a critical contribution of Nav1.7 to human pain disorders.
22 derstanding, and therefore treating, chronic pain disorders.
23 le stimuli and is implicated in a variety of pain disorders.
24 pain pathway is believed to promote clinical pain disorders.
25 ing of nociception in a number of pathologic pain disorders.
26 esent a class of agents for the treatment of pain disorders.
27 been linked to a spectrum of inherited human pain disorders.
28 ysiology of fibromyalgia and related chronic pain disorders.
29 cific hypotheses in a broad range of chronic pain disorders.
30 given to clinical studies related to chronic pain disorders.
31 al substance used to treat neuromuscular and pain disorders.
32 f negative context on symptoms in functional pain disorders.
33 ral sensitisation may contribute to visceral pain disorders.
34 le orgasmic disorder and female genitopelvic pain disorders.
35 gets is required to advance the treatment of pain disorders.
36 e included 1422 participants (median time of pain disorder 10 years [IQR 5-20]; median length of stro
37 mary erythromelalgia; (3) paroxysmal extreme pain disorder; (4) febrile seizures and recently (5) sma
38 e second most common musculoskeletal chronic pain disorder after low back pain, affecting 6-9% of adu
39                                 Many chronic pain disorders alternate between bouts of pain and perio
40  for studying the pathophysiology of chronic pain disorders and as screening tools for new therapies.
41 ic patients, a high risk group for co-morbid pain disorders and increased perception of pain.
42 ls, and are associated with episodic extreme pain disorders and insensitivity to pain, respectively.
43      TRPM8 is also implicated in cold-evoked pain disorders and migraine, highlighting its inhibitors
44 rly depression, are often comorbid with both pain disorders and opioid abuse.
45            These findings may integrate many pain disorders and provide an approach for developing an
46 lation of pain, review specific IgG-mediated pain disorders and summarise recent development in thera
47 nherited erythromelalgia, paroxysmal extreme pain disorder, and small-fibre neuropathy.
48 d to its classification as a chronic primary pain disorder, and subtypes of CRPS have been updated.
49 tified in three families with rare heritable pain disorders, and in patients with painful small-fibre
50 in various diseases, such as psychiatric and pain disorders, and may play important roles in myelinat
51 cated voltage-gated sodium channels in human pain disorders, and targeted and massively parallel geno
52 mall fibre neuropathy and paroxysmal extreme pain disorder are caused by gain-of-function mutations i
53                     Numerous musculoskeletal pain disorders are based in dysfunction of peripheral pe
54 lood flow/oxygenation and a number of muscle pain disorders are based in problems of peripheral perfu
55                         Functional abdominal pain disorders are common disorders with a prevalence of
56                 Although chronic neuropathic pain disorders are more prevalent in the senescent popul
57                                      Chronic pain disorders are often associated with negative emotio
58       Alcohol use disorder (AUD) and chronic pain disorders are pervasive, multifaceted medical condi
59 cause erythromelalgia and paroxysmal extreme pain disorder as a result of hyperexcitability of sensor
60 5c, C1, and C2 regions, plays a role in some pain disorders associated with craniofacial structures (
61 the development of specialized treatment for pain disorders associated with I(NaR).
62 , 1.25; 95% CI, 1.10-1.42), and preoperative pain disorders (back pain: aOR, 1.57; 95% CI, 1.42-1.75;
63 al neuropathic pain is the most debilitating pain disorder but current treatments including opiates a
64 d pain is a major characteristic of clinical pain disorders, but it is difficult to assess in isolati
65 is question for inherited erythromelalgia, a pain disorder caused by gain-of-function mutations in Na
66             Fibromyalgia is a common chronic pain disorder characterized by complex symptomatology an
67        Trigeminal neuralgia (TN) is a facial pain disorder characterized by intense and paroxysmal pa
68        Trigeminal neuralgia (TN) is a unique pain disorder characterized by intense paroxysmal facial
69              Paediatric functional abdominal pain disorders, currently referred to as disorders of gu
70 h chronic pain, but the patients without the pain disorder do not show this neuropathology, indicatin
71 ezing cold injury is a disabling neuropathic pain disorder due to a sensory neuropathy.
72 pain conditions often overlap with nonpelvic pain disorders (eg, fibromyalgia, migraines) and nonpain
73                                The inherited pain disorder erythromelalgia (IEM) has been linked to N
74 ng allergy/anaphylaxis and neuroinflammatory pain disorders, exhibit a sex bias, with females at incr
75 ID) has been changed to functional abdominal pain disorders (FAPD) and we have derived a new term, "f
76                         Functional abdominal pain disorders (FAPDs) are more prevalent in female pati
77                         Functional abdominal pain disorders (FAPDs) can severely affect the life of c
78 tions for children with functional abdominal pain disorders (FAPDs).
79 aine has traditionally been categorized as a pain disorder, focusing on headache as its central featu
80 al nausea and vomiting, functional abdominal pain disorders, functional diarrhea, and functional cons
81 ociation of genetic variants with idiopathic pain disorders has appeared in the literature, and here
82 terestingly, many of the heritable monogenic pain disorders have been mapped to mutations in genes en
83 ndromes, molecular substrates for hereditary pain disorders have been poorly understood.
84            Human hereditary gain- or loss-of-pain disorders have demonstrated an essential role of Na
85      Genetic and functional studies of human pain disorders have identified Na(V)1.7, Na(V)1.8 and Na
86                          Mendelian heritable pain disorders have provided insights into human pain me
87  Other pain disorders, like complex regional pain disorder, have been associated with IgGs against be
88 R 0.97, [0.83-1.13]) or functional abdominal pain disorders (HR 0.90, [0.81-1.00]).
89  syndrome (FMS) is a chronic musculoskeletal pain disorder in children and adolescents for which ther
90       Erythromelalgia is the first inherited pain disorder in which it is possible to link a mutation
91 een identified as promising targets to treat pain disorders in patients.
92 erapies may be more appropriate for treating pain disorders in which hyperalgesia and not allodynia i
93 lth care services for symptoms of functional pain disorders, including irritable bowel syndrome, than
94 sory hyperinnervation, a hallmark of several pain disorders, including vulvodynia.
95 nction SCN9A variant associated with extreme pain disorder induced hypersynchrony.
96 ral Nav isoform) associated with two genetic pain disorders, inherited erythromelalgia (IEM) and paro
97 oved drug for fibromyalgia and other chronic pain disorders, into the CeA or chemogenetic inactivatio
98 the role of dietary fatty acids in promoting pain disorders is not completely understood.
99 inal neuropathic pain, the most debilitating pain disorder, is often triggered and exacerbated by coo
100 surgical pain, fibromyalgia, and neuropathic pain disorders, is highly prevalent but remains poorly t
101                                        Other pain disorders, like complex regional pain disorder, hav
102          Migraine is a complex neurovascular pain disorder linked to the meninges, a border tissue in
103 This study expands the spectrum of heritable pain disorders linked to gain-of-function mutations in N
104                 Understanding rare heritable pain disorders not only improves diagnosis and treatment
105 ivity associated with inherited and acquired pain disorders occurs through increased excitability of
106 drome (IC/BPS) is a chronic and debilitating pain disorder of the bladder and urinary tract with poor
107 ause hyperexcitability and familial episodic pain disorder or painful neuropathy, while mutations evo
108 at alcohol use can predispose individuals to pain disorders or exacerbate existing pain conditions, b
109                           Paroxysmal extreme pain disorder (PEPD) and inherited erythromelalgia (IEM)
110    We demonstrated that a paroxysmal extreme pain disorder (PEPD) mutation in the human peripheral ne
111  nonoverlapping syndrome, paroxysmal extreme pain disorder (PEPD), characterized by rectal, periocula
112 erythromelalgia (IEM) and paroxysmal extreme pain disorder (PEPD), enhance Nav1.7 function via distin
113                           Paroxysmal extreme pain disorder (PEPD), previously known as familial recta
114 erythromelalgia (IEM) and paroxysmal extreme pain disorder (PEPD).
115 erythromelalgia (IEM) and paroxysmal extreme pain disorder (PEPD).
116 nful inherited neuropathy paroxysmal extreme pain disorder (PEPD).
117                Inherited "paroxysmal extreme pain disorder" (PEPD) differs in its clinical picture an
118 uld contribute to the development of certain pain disorders, possibly including those modulated by es
119 tage-gated Na(+) channel NaV1.9 cause severe pain disorders ranging from neuropathic pain to congenit
120 mal models, the role of glial cells in human pain disorders remains unknown.
121                                   In chronic pain disorders, rumination can impede treatment efficacy
122                  Evidence from human genetic pain disorders shows that voltage-gated sodium channel a
123  contribute to etiology of functional pelvic pain disorders such as interstitial cystitis/bladder pai
124                             Chronic visceral pain disorders, such as interstitial cystitis/bladder pa
125                                     Visceral pain disorders, such as irritable bowel syndrome, exhibi
126 Migraine is a highly prevalent and disabling pain disorder that affects >1 billion people worldwide.
127 plex regional pain syndrome (CRPS) is a rare pain disorder that usually occurs in a limb after trauma
128 different neuronal types distributed between pain disorders that display different genetic susceptibi
129 sodium channel (NaV) mutations cause genetic pain disorders that range from severe paroxysmal pain to
130 date molecular mechanisms underlying chronic pain disorders to develop novel therapeutic approaches.
131 netic studies have now linked multiple human pain disorders to voltage-gated sodium channels, includi
132           Small fibre neuropathy is a common pain disorder, which in many cases fails to respond to t
133 75 years who had been diagnosed with chronic pain disorder with psychological and somatic factors (In

 
Page Top