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1 s, at least for a time, for the treatment of palmar and axillary hyperhidrosis.
2                                          The palmar and dorsal ulnotriquetral and ulnolunate ligament
3           Immunostaining of Cx26 in lesional palmar and knuckle skin was weak or absent, although its
4 c signaling pathways in fibroblasts from the palmar and nonpalmar dermis of Dupuytren's patients and
5 3000), which each entail painful calluses on palmar and plantar skin.
6 ling arthritic changes, marked osteoporosis, palmar and plantar subcutaneous nodules and distinctive
7 alignant melanoma typically occurring on the palmar and plantar surfaces.
8 rome include multiple basal cell carcinomas, palmar and/or plantar pits, odontogenic keratocysts, ske
9 iated with incompleteness of the superficial palmar arch ( P=0.13).
10            Incompleteness of the superficial palmar arch (SPA) was present in 46%, the deep palmar ar
11 measure the pressure-derived function of the palmar arch and forearm arterial collateral circulation
12 irect invasive hemodynamic assessment of the palmar arch and forearm arterial function reveals collat
13                                              Palmar arch and forearm collateral function was determin
14   Before invasive CFI measurements, arterial palmar arch and forearm function was tested noninvasivel
15                                  The rate of palmar arch incompleteness and the clinical consequences
16 lmar arch (SPA) was present in 46%, the deep palmar arch was complete in all patients.
17 upply is always preserved by a complete deep palmar arch.
18                                          The palmar arches serve as the most important conduits for d
19 romoted differentiation into specifically of palmar dermal fibroblasts from Dupuytren's patients in t
20 ith the peak systolic velocity of the second palmar digital artery (Pearson coefficient: 0.621; p < 0
21 CR) analysis and immunohistochemistry of the palmar epidermis demonstrated significantly increased ex
22 ratum granulosum of both normal and affected palmar epidermis, indicating that the altered AQP5 prote
23                                        Human palmar fascia fibroblasts and human fetal lung fibroblas
24 ive fibroproliferative disease affecting the palmar fascia of the hands, causing fingers to irreversi
25 disabling fibroproliferative disorder of the palmar fascia, which leads to flexion contractures of th
26 nonpalmar dermis of Dupuytren's patients and palmar fibroblasts from non-Dupuytren's patients.
27                                              Palmar finger skin reacts to extension under mechanical
28                     Facial, axillary, and/or palmar hyperhidrosis (excessive sweating of the face, ar
29  altered thumb print due to other causes and palmar hyperhidrosis were excluded.
30            A prospective randomized study on palmar hyperhidrosis, May 2016-September 2019.
31 bioenergetics in 13 patients with idiopathic palmar hyperhidrosis.
32 cise performance in patients with idiopathic palmar hyperhidrosis.
33 mediate phenotype most readily identified by palmar hyperlinearity and in some cases fine-scale and/o
34 mon skin disorder characterised by dry skin, palmar hyperlinearity and keratosis pilaris.
35        Trained observers were able to define palmar hyperlinearity in the majority (3191/3656, 87%) o
36                                              Palmar hyperlinearity is a feature of ichthyosis vulgari
37 s of IV are fine-scale on the arms and legs, palmar hyperlinearity, and keratosis pilaris.
38 ong adduction of the thenar, hypothenar, and palmar interosseous muscles offer powerful rigidity to t
39 il thickening in PC-K6a and PC-K17; (3) more palmar keratoderma in PC-K16; (4) cysts primarily in PC-
40                       Our findings show that palmar keratoderma is a clinical indicator of recessive
41 pe of EPP which is characterized by seasonal palmar keratoderma, relatively low erythrocyte protoporp
42 ited Kingdom identified six individuals with palmar keratoderma.
43 he new species exhibits the oldest record of palmar (or true) opposition of the pollex, which is unpr
44 at can progress to parasagittal fractures or palmar osteochondral disease (POD).
45                                The digit and palmar pads are also represented by barrelettes located
46        Dose-limiting toxicities were grade 3 palmar plantar erythrodysesthesia (PPE), mucositis, and
47 nt but required early dose reductions due to palmar plantar erythrodysesthesia, and liver decompensat
48 stinal toxicity, hypothyroidism, fatigue and palmar plantar erythrodysesthesia.
49 a subtype of cutaneous melanoma arising from palmar, plantar, or subungual skin.
50 ns affecting nails, glands, oral mucosa, and palmar-plantar epidermis.
51  worse treatment-related adverse events were palmar-plantar erythrodysaesthesia (1 [<1%] of 367 patie
52 ing placebo, the most frequent of which were palmar-plantar erythrodysaesthesia (13 [10%] vs 0), hype
53 , fatigue (24 [8%]), dyspnoea (21 [7%]), and palmar-plantar erythrodysaesthesia (18 [6%]) in the sora
54  vs 39 [12%] of 320 in the sunitinib group), palmar-plantar erythrodysaesthesia (25 [8%] vs 26 [8%]),
55 ase (37 [9%] vs eight [4%] vs 18 [10%]), and palmar-plantar erythrodysaesthesia (35 [8%] vs 17 [8%] v
56 n (90 [17%] of 535), fatigue (51 [10%]), and palmar-plantar erythrodysaesthesia (49 [9%]).
57 n with sorafenib than with axitinib included palmar-plantar erythrodysaesthesia (PPE; 37 [39%] of 96
58 %]), diarrhoea (109 [27%] vs 105 [54%]), and palmar-plantar erythrodysaesthesia (seven [2%] vs 100 [5
59 d to erdafitinib were stomatitis (25 [12%]), palmar-plantar erythrodysaesthesia syndrome (12 [6%]), a
60 ] vs 7 [2%]), fatigue (36 [11%] vs 24 [7%]), palmar-plantar erythrodysaesthesia syndrome (27 [8%] vs
61 5%] of 269 patients in the sorafenib group), palmar-plantar erythrodysaesthesia syndrome (33 [12%] vs
62 5%] of 269 patients in the sorafenib group), palmar-plantar erythrodysaesthesia syndrome (33 [12%] vs
63 hoea (103 [21%] of 488 patients) followed by palmar-plantar erythrodysaesthesia syndrome (87 [18%]),
64  fatigue in the axitinib arm, and diarrhoea, palmar-plantar erythrodysaesthesia, and alopecia in the
65        PLD-treated patients experienced more palmar-plantar erythrodysesthesia (37%; 18% grade 3, 1 p
66 atigue (6% v 15%), hypertension (28% v 22%), palmar-plantar erythrodysesthesia (8% v 4%), and hematol
67 a (n = 69 [48.6%]), nausea (n = 65 [45.8%]), palmar-plantar erythrodysesthesia (n = 62 [43.7%]), cons
68                                              Palmar-plantar erythrodysesthesia (PPE) became evident a
69 cause of adverse events related to the drug (palmar-plantar erythrodysesthesia [PPE], n = 3; asthenia
70 ful neuropathy occurred in 7.5%; and grade 3 palmar-plantar erythrodysesthesia occurred in 2.5%.
71  worse treatment-related adverse events were palmar-plantar erythrodysesthesia syndrome (18 [10%] in
72          The most common adverse events were palmar-plantar erythrodysesthesia syndrome (38.1%), alop
73  fatigue, hypertension, febrile neutropenia, palmar-plantar erythrodysesthesia syndrome, and stomatit
74 ts in the tucatinib group included diarrhea, palmar-plantar erythrodysesthesia syndrome, nausea, fati
75 ncidences of diarrhea, nausea, vomiting, and palmar-plantar erythrodysesthesia were higher with lapat
76  events were fatigue, weight loss, diarrhea, palmar-plantar erythrodysesthesia, and hypertension.
77 mg twice per day; n = 1); grade 3 mucositis, palmar-plantar erythrodysesthesia, and hypokalemia (400
78                                   Skin rash, palmar-plantar erythrodysesthesia, and thrombocytopenia
79  of whom had three dose-limiting toxicities: palmar-plantar erythrodysesthesia, cerebral ischaemia, a
80 associated adverse events included diarrhea, palmar-plantar erythrodysesthesia, decreased weight and
81 5% of patients) were diarrhea, nausea, rash, palmar-plantar erythrodysesthesia, mucositis, vomiting,
82 as 1,657 mg/m2/d with limiting toxicities of palmar-plantar erythrodysesthesia, nausea, vomiting, ver
83 ction, elevated thyroid stimulating hormone, palmar-plantar erythrodysesthesia, weight loss, and head
84  grade 3 events were skin toxicity (rash and palmar-plantar erythrodysesthesia; five [4%]) and hypert
85                                      Painful palmar-plantar keratoderma (PPK) severely impairs mobili
86 on in a family with diffuse nonepidermolytic palmar-plantar keratoderma was shown to be the loss in o
87 Ewing sarcoma, three [7%] for osteosarcoma), palmar-plantar syndrome (three [7%] for Ewing sarcoma, t
88 cognized, but important, normal functions in palmar-plantar tissues.
89          The remarkable occurrence of severe palmar--plantar hyperkeratosis in both patients suggests
90 ressed in ectoderm-derived appendages and in palmar/plantar epidermis and is robustly induced when th
91 d protein electrophoretic features closer to palmar/plantar or mucosal-like epithelia.
92 erse metacarpal ligament (DTML) (n = 5), and palmar plate (n = 10) were analyzed.
93 ittal MR images were best for evaluating the palmar plate and the capsule.
94 n of lesions of the extensor hood, DTML, and palmar plate structures.
95  many medical conditions such as plantar and palmar psoriasis, dyshidrosis, and cholestasis.
96 , radioscapholunate, dorsal radiotriquetral, palmar scaphotriquetral, and dorsal scaphotriquetral lig
97    Results were compared to tape strips from palmar skin of age/race/sex-matched healthy controls (HC
98   By simulating skin deformations across the palmar surface of the hand and tiling it with receptors
99   Injection of capsaicin into the plantar or palmar surface of the paws produced a depression of brad
100                              We examined the palmar surfaces of the dominant and nondominant hands of
101 gueta (GUA), Roseira (ROS), Santa Vitoria do Palmar (SVP), and Taubate (TBT).
102                   Despite limited sensation, palmar tactile stimulation delivered 4 months post-trans
103                    The capitate head is more palmar than in all other known hominoids, permitting ext
104 rgone AHSCT, namely finger pad inflammation, palmar violaceous papules, and digital ulcerations.
105 rgone AHSCT, namely finger pad inflammation, palmar violaceous papules, and digital ulcerations.

 
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