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1 f the head, neck, or uncinate process of the pancrease.
2 ISM was used to isolate islets from 10 human pancreases.
3 and IL18r on acinar cells in human and mouse pancreases.
4 eaky vessels were apparent in cd93-deficient pancreases.
5 clinical n=42) and 150 chronic pancreatitis pancreases.
6 ution in relation to islets in infused human pancreases.
7 labeling between perfused and syringe-loaded pancreases.
8 signal intensity between normal and diabetic pancreases.
9 31 (65%) of the 48 patients with functioning pancreases.
10 s to improve islet recovery from young donor pancreases.
11 s were not detectable in fetal and postnatal pancreases.
12 n), shorter cold ischemia time for recovered pancreases (355 vs. 630 min), and reduced mean cost per
13 onors and 31 organs (13 kidneys, 6 livers, 3 pancreases, 5 hearts, and 4 lungs) were ultimately trans
15 der-drained pancreas transplants (25 SPK, 14 pancrease after kidney transplants [PAK], amd two pancre
16 c endothelial cells (ASECs) from three human pancreases and corroborate these findings with three pub
17 icensis; Jamaican fruit bat) bat kidneys and pancreases and identify key cell population, gene expres
21 eelike structures in lungs, kidneys, livers, pancreases, and mammary glands as well as digits and bon
22 ion after brain death (DBD) and low-risk DCD pancreases, as the number of DCD pancreas transplants in
23 tively) in human fetal, postnatal, and adult pancreases, as well as in isolated adult islets, and exa
24 yielded more than 200,000 IEQ from 19 of 21 pancreases (averaging 422,893 +/- 181,329 total IEQ and
25 for transplantation from 8 of 10 consecutive pancreases, averaging 6510 +/- 2150 islet equivalent num
27 d bihormonal endocrine cells in 7 of 9 human pancreases between CS16 and CS18, which were no longer d
29 romote regeneration and reduce damage in the pancreases, blood, guts, and nervous systems of mice, bu
30 a-cells were rarely found in human cadaveric pancreases but were in the range of 0.2-0.5% in human is
31 Insulin secretion was observed in all four pancreases, but was lowest in an older donation after ca
32 ication method (SM, standard method) and 132 pancreases by a refined University of Illinois at Chicag
33 isolated islets from 114 human cadaver donor pancreases by the automated Ricordi method, followed by
37 arge collection of human embryonic and fetal pancreases covering the first trimester of pregnancy to
41 pectively analyzed 376 islet isolations from pancreases flushed and transported with IGL-1 (n=95), UW
45 ked for female islet beta cells in four male pancreases from autopsies, one from a T1D patient, emplo
48 PTP1B and TCPTP expression was determined in pancreases from chow and high fat fed mice and the impac
50 brain stem death and DCD hearts, livers, and pancreases from donors who died following ligature asphy
51 in recipients of lungs, hearts, livers' and pancreases from donors who died following ligature asphy
52 a subset of these fatty acids was tested in pancreases from fed animals, the same rank order of effe
54 bserved in the kinetics of CVB4 clearance in pancreases from NOD, NOD IL-4-/-, and NOD IFN-gamma-/- m
55 ber of islet transplants, the suitability of pancreases from organ donors considered inappropriate fo
56 ence among transplant surgeons to transplant pancreases from small pediatric donors despite the optim
57 isolation of younger and fibrotic pediatric pancreases, gave increased islet yield with improved pat
61 ing analyses in hereditary and idiopathic CP pancreases indicate differences in innate and adaptive i
63 suring sufficient islet yield from preserved pancreases is a critical step in clinical islet transpla
65 ucose metabolism in islets isolated from the pancreases of children with KATPHI who required pancreat
66 volved, as human amylin forms amyloid in the pancreases of diabetic patients, and oligomers have been
67 ntly larger beta-cell areas were observed in pancreases of fetuses and neonates from prepregnant HF-f
71 increased, and TCPTP expression decreased in pancreases of mice fed a high fat diet, as well as in MI
72 l three virus doses, but day 3 titers in the pancreases of mice inoculated with 10(4) PFU were reduce
73 ormed on cDNAs from isolated islets or whole pancreases of NOD/Lt females 4 weeks after intrathymic i
85 nt at cell membranes in homozygous p120(f/f) pancreases, potentially providing stability for maintena
87 port ex vivo normothermic perfusion of human pancreases procured but declined for transplantation, wi
89 lying pathology in human as well as rat LADA pancreases provided identical results, allowing the conc
90 on of transplanted organs was less than 10%; pancreases refused due to "trauma", "age", or "resuscita
93 mmunofluorescent staining of fetal and adult pancreases revealed colocalization of GAD65 with alpha-
96 study, we examined 10 human cadaveric donor pancreases (tail and body regions) via integration of st
97 -dose RCEM was efficient in digesting entire pancreases to obtain higher yield (5535 +/- 830 and 2582
98 d by amylase, and exocrine function of human pancreases using EVNP and demonstrates the feasibility o
106 eases were transplanted; among these, 62% of pancreases were of potentially high quality (favorable d
108 ompared between two purification methods; 32 pancreases were processed by conventional Biocoll purifi
110 atic islets when sections from rat and mouse pancreases were reacted with a polyclonal antibody to re
115 ta-cells appeared histologically normal, the pancreases were unresponsive to perfusion with stimulato
116 ch as nitrite, are generated in precancerous pancreases, which induce massive DNA damage, including D
117 r islet yield from deceased and pancreatitis pancreases while retaining islet quality and function.