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1 her fled from the situation or experienced a panic attack.
2 all, and one who received placebo suffered a panic attack.
3 icipation of the next uncued (unpredictable) panic attack.
4 f internal and external cues predictive of a panic attack.
5  in both twins, with one twin experiencing a panic attack.
6 d periods of increased anxiety or tension or panic attack.
7 esponse to a conditioned fear stimulus and a panic attack.
8 ptoms and least correlated with reduction in panic attacks.
9 rs to be a genuine risk factor for secondary panic attacks.
10 est for the families of the probands without panic attacks.
11 asing the number of full and limited-symptom panic attacks.
12  the pattern observed during the 34 reported panic attacks.
13  development of neurological symptoms during panic attacks.
14 son subjects, and this increase preceded the panic attacks.
15 cur in different patients from m-CPP-induced panic attacks.
16                                  AS predicts panic attacks.
17 vestibular symptoms between, but not during, panic attacks.
18  substance abuse or substance dependence, or panic attacks.
19 nxiety disorder with recurrent, debilitating panic attacks.
20 ically to suppress excessive arousal such as panic attacks.
21 condition caused by the experience of uncued panic attacks.
22 mpared with young persons with no history of panic attacks.
23 with a significantly greater risk of current panic attacks.
24 n implicated in the pathophysiology of acute panic attacks.
25 ence respiration-related difficulties during panic attacks.
26 r the co-occurrence of cigarette smoking and panic attacks.
27 f the co-occurrence of cigarette smoking and panic attacks.
28 inent depersonalization/derealization during panic attacks.
29 icide attempts than were adolescents without panic attacks.
30  be one of the mechanisms linking smoking to panic attacks.
31 , 27 patients with anxiety but no history of panic attacks, 13 patients with depressive disorders but
32 higher rate than seen among subjects without panic attacks (69.4%).
33 ic ethnicity, two or more prior stressors, a panic attack, a low level of social support, the death o
34                                   During the panic attack, a strong association with the surrounding
35 two were characterized by past or concurrent panic attacks, a rate that was not significantly differe
36                                     Although panic attacks account for only a portion of the morbidit
37 , neutral conditioned stimuli present during panic attacks acquire panicogenic properties.
38      Patients with panic disorder experience panic attacks after intravenous sodium lactate infusions
39 isorder, post-traumatic stress disorder, and panic attack among n=10,206 US Army soldiers and veteran
40                     The associations between panic attack and depression were attenuated in models th
41 ed an increased risk for first occurrence of panic attack and disorder; the risk was higher in active
42 order or for the severity of lactate-induced panic attack and the quantified PET abnormality.
43   Of the 115 patients, 41% (N = 47) reported panic attacks and 17% (N = 20) met screening criteria fo
44      Measures of anxiety included history of panic attacks and a composite variable reflecting curren
45 re used to determine the association between panic attacks and cigarette smoking and to determine whe
46 ents with PTSD experienced yohimbine-induced panic attacks and had significantly greater increases co
47 ts (31%) with PTSD experienced m-CPP-induced panic attacks and had significantly greater increases co
48 ed anxiety and sympathetic drive seen during panic attacks and in hypercapnic states such as COPD.
49 provocation on the subsequent development of panic attacks and panic disorder in nonclinical subjects
50   Evidence from a family study suggests that panic attacks and panic disorder may be related genetica
51  (N = 115) were screened for the presence of panic attacks and panic disorder with a self-report ques
52  sought to determine the association between panic attacks and psychoticism among young adults in the
53             Much of the relationship between panic attacks and psychoticism appears to be explained b
54                              The symptoms of panic attacks and pulmonary disease overlap, so that pan
55 rs is the sudden shift in affect observed in panic attacks and some rapid cycling states.
56 s were used to estimate associations between panic attacks and suicidal ideation and suicide attempts
57 ds to the evidence of an association between panic attacks and suicide attempts during the middle yea
58  study was to investigate the association of panic attacks and suicide attempts in a community-based
59 direction (i.e., from prior smoking to first panic attack) and the possibility of a higher risk in ac
60 ysthymia, generalized anxiety disorders, and panic attacks) and drug use during the previous 12 month
61 ionnaire-2 [PHQ-2], GAD-2, and an item about panic attacks), and a diagnostic evaluation using PHQ-9
62 ssive disorders but no history of anxiety or panic attacks, and 45 normal comparison subjects.
63 gonists acting at this receptor can suppress panic attacks, and both inverse agonists and antagonists
64 rder are highly susceptible to CO(2)-induced panic attacks, and depressed patients appear to be insen
65                            Active avoidance, panic attacks, and increased sympathetic arousal are ass
66 rformance fears, unexpected and/or triggered panic attacks, anticipatory anxiety, and avoidance behav
67 xcept PMDD), the physiological features of a panic attack appear similar across groups.
68                                              Panic attacks are a common complication of affective dis
69                                              Panic attacks are a hallmark in panic disorder (PAND).
70 h previous findings, these data suggest that panic attacks are associated with greater risk of cigare
71                                              Panic attacks are relatively common among postmenopausal
72     Individuals with panic disorder perceive panic attacks as unpredictable.
73 ienced significantly more panic symptoms and panic attacks, as well as elevated respiratory rates.
74  were used to estimate the risk for onset of panic attacks associated with prior smoking and vice ver
75      The role of lung disease in the smoking-panic attacks association was explored.
76  scored highly for PTSD-related symptoms and panic attacks at 8 wks (p = .023 and .014, respectively)
77                             No subject had a panic attack before hyperventilation.
78 sorder, with anxiety disorders (particularly panic attacks) being the most common comorbid condition.
79 -occurrence of regular cigarette smoking and panic attacks but did not predict either panic attacks o
80 ession predicted a first onset of subsequent panic attacks but not of panic disorder.
81             That primary depression predicts panic attacks but not panic disorder suggests that secon
82 hors sought to determine the relationship of panic attacks, cigarette smoking, and neuroticism.
83 o Pavlovian conditioning, failure to predict panic attacks could be due to a basic deficit in conditi
84           Specific symptoms occurring during panic attacks differ by gender.
85 cantly lower in those who subsequently had a panic attack during 5% CO(2) breathing than those who di
86 ic ethnicity, two or more prior stressors, a panic attack during or shortly after the events, residen
87                                              Panic attacks during adolescence are associated with sig
88  in minute ventilation at baseline predicted panic attacks during CO2 inhalation.
89 ensitivity to CO(2) and in the threshold for panic attacks during hypoxic and hypercapnic states.
90 e placebo-treated patients were free of full panic attacks during the 2 weeks ending at week 10.
91           Despite a mean of 9.5 to 11.6 full panic attacks during the screening period, 86.0% of the
92              A 6-month history of full-blown panic attacks, endorsed by 10% of postmenopausal women i
93 ty Survey respondents with panic disorder or panic attacks, female respondents were more likely than
94      Primary outcome measures were change in panic attack frequency and clinician-rated Clinical Glob
95 ic disorder have generally used reduction in panic attack frequency as the primary measure of improve
96 rtion of the morbidity of panic disorder and panic attack frequency assessments are unreliable, studi
97                                Reductions in panic attack frequency in subjects given either fluoxeti
98 easures, including global improvement, total panic attack frequency, phobic symptoms, and functional
99 th placebo for the primary outcome variable, panic attack frequency.
100 laxis, undifferentiated somatoform disorder, panic attacks, globus hystericus, vocal cord dysfunction
101             The majority of individuals with panic attacks had been regular smokers during their life
102 th panic disorder who had a low frequency of panic attacks had elevated daytime corticotropin levels
103                  Subjects with CO(2)-induced panic attacks had similarly high ratings on the behavior
104 ' goal was to determine whether treatment of panic attacks has a protective effect on the risk of maj
105                The symptoms of CO(2)-induced panic attacks have a similar intensity regardless of the
106                                     Having a panic attack in the preceding 3 years was associated wit
107 re used to determine the association between panic attacks in adolescence (age 15-21) and psychoticis
108 idence of an independent association between panic attacks in adolescence and psychoticism during you
109 ion between A2AR polymorphisms and phobia or panic attacks in humans and prompts a therapeutic intere
110 oradrenergic agent yohimbine reliably induce panic attacks in humans with panic disorder but not in h
111 fusion has induced flashbacks accompanied by panic attacks in male combat veterans with posttraumatic
112                                              Panic attacks in panic disorder may be explained by inef
113 ough CO(2) breathing causes a higher rate of panic attacks in patients with PD than other groups (exc
114 r in panic disorder, evokes intense fear and panic attacks in susceptible individuals.
115          Proportion of subjects remitted (no panic attacks in the past month, minimal anticipatory an
116 ompleted a questionnaire about occurrence of panic attacks in the previous 6 months.
117 tory of panic disorder, panic attacks, or no panic attacks in the probands.
118  of 35% CO(2) evoked not only fear, but also panic attacks, in three rare patients with bilateral amy
119 ent in frequency of full and limited-symptom panic attacks, intensity of full panic attacks, phobic f
120                 Other assessments included a panic attack inventory, clinician-rated and patient-rate
121 -response relationship suggests that primary panic attack is a marker, rather than a causal risk fact
122 , the exact mechanism of lactate eliciting a panic attack is still unknown.
123                                       Once a panic attack is triggered, minute ventilation and respir
124 nts, which were drug hypersensitivity (n=1), panic attack (n=1), pyrexia (n=1), and COVID-19 (n=1).
125 hypersensitivity (n=1), nephritis (n=1), and panic attack (n=1).
126                                              Panic attacks occurred in five patients with generalized
127 of the respiratory dysfunction manifested in panic attacks occurring in panic disorder.
128 set and was less likely to be complicated by panic attacks or alcoholism.
129 and panic attacks but did not predict either panic attacks or cigarette smoking in the absence of the
130 onset of daily smoking in persons with prior panic attacks or disorder.
131 tic anxiety ratings, but not the presence of panic attacks or of any lifetime anxiety disorder, added
132 der if the proband with bipolar disorder had panic attacks or panic disorder was calculated with logi
133 s increased significantly if the proband had panic attacks or panic disorder.
134 any panic, which included panic disorder and panic attacks (OR = 2.1; 95% CI, 1.1, 4.5).
135  any anxiety disorder [OR = 1.3 (1.1, 1.6)], panic attacks [OR = 1.6 (1.1, 2.1)], panic disorder [OR
136 groups based on a history of panic disorder, panic attacks, or no panic attacks in the probands.
137 tion exists about the epidemiology of DSM-IV panic attacks (PAs) and panic disorder (PD).
138                                    Recurrent panic attacks (PAs) are a common feature of panic disord
139  who completed the study, the mean number of panic attacks per week dropped by 88% in the sertraline-
140 ted-symptom panic attacks, intensity of full panic attacks, phobic fear, anxiety, and depressive symp
141 d safety of fluoxetine treatment in reducing panic attacks, phobic symptoms, anxiety, and depressive
142                                   History of panic attacks proved to be a significant correlate of no
143  The findings suggest that the perception of panic attacks reflects central rather than peripheral re
144  suggest a causal hypothesis for the smoking-panic attacks relationship.
145 accompanying a PTSD flashback and those in a panic attack remains unclear.
146 site variable and patients with a history of panic attacks reported more severe medication side effec
147  common in women and are not associated with panic attacks, suggesting a late-life subtype.
148 n other men to evidence major depression and panic attack syndromes.
149                            Yohimbine-induced panic attacks tended to occur in different patients from
150  patients had a higher rate of CO(2)-induced panic attacks than depressed patients and normal subject
151           More patients with PD and PMDD had panic attacks than did controls or patients with major d
152 subsyndromal panic disorder characterized by panic attacks that failed to meet either the criterion o
153 adjustment for confounding factors, having a panic attack was still associated with an increased rate
154  that depersonalization/derealization during panic attacks was associated with childhood trauma.
155                                Occurrence of panic attacks was judged with DSM-IV criteria by a blind
156 met DSM-III-R criteria for panic disorder or panic attacks were analyzed to test for gender differenc
157 uency of certain symptoms between and during panic attacks were obtained.
158                           Panic symptoms and panic attacks were rated with the Acute Panic Inventory
159                       However, patients with panic attacks were significantly more likely to report d
160 ugs, the authors found that adolescents with panic attacks were three times more likely to have expre
161 umed alcohol experienced significantly fewer panic attacks when applying liberal panic criteria; howe
162  major depressive episodes (odds ratios: for panic attacks with depression, 6.2; for panic disorder w
163                           Temporally primary panic attacks, with or without panic disorder and whethe
164 neration, including revisiting alexithymia, 'panic attack without panic', dissociation, insecure atta
165 ncing depersonalization/derealization during panic attacks would be more likely to have a history of

 
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