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1 ents with 39 MDCT masses (22 thrombus and 17 pannus).
2 ute, organizing, and organized thrombus (ie, pannus).
3 ion tissue with early fibrous encapsulation (pannus).
4 le patient showed a subepithelial, avascular pannus.
5 ns the aggressive phenotype of the advancing pannus.
6 he rheumatoid arthritis (RA) synovial tissue pannus.
7 l blood and accumulated in inflamed synovial pannus.
8 ected as tissue overgrowth was classified as pannus.
9 e suppression of the angiogenic component of pannus.
10 ght play a role in neovascularization of the pannus.
11  while FLIP remained highly expressed in the pannus.
12 ue and to the growth and proliferation of RA pannus.
13 ation found in the rheumatoid arthritic (RA) pannus.
14 r the chondrocytic cells found in rheumatoid pannus.
15 res of vascularity, villous hypertrophy, and pannus.
16 expanding inflammatory tissue referred to as pannus.
17 sses was significantly lower than that in 17 pannus (87+/-59 versus 322+/-122; P<0.001).
18 tified on cells at the invasive front of the pannus and at sites of cartilage erosion.
19 tis, especially at the interface of synovial pannus and eroding bone.
20  clinical evidence of trachomatous scarring, pannus and Herbert's pits (HPs) or limbal follilcles in
21  Although immune cells, which infiltrate the pannus and promote inflammation, play a prominent role i
22 l and subclinical inflammation, formation of pannus and synovial hyperplasia, and the erosion of cart
23  synovitis characterized by the formation of pannus and the destruction of cartilage and bone in the
24                               Distinguishing pannus and thrombus in patients with prosthetic valve dy
25  tomography (MDCT) in distinguishing between pannus and thrombus, the latter amenable to thrombolysis
26    35/492 (7%) of children had limbal signs (pannus and/or HPs) plus any conjunctival scarring.
27 l, 35/492 (7%) of children had limbal signs (pannus and/or HPs) plus any conjunctival scarring.
28 y, resulting in a dense vascularized corneal pannus, and eventually leading to visual impairment.
29  clinical evidence of trachomatous scarring, pannus, and Herbert's pits (HPs) or limbal follicles in
30 ich contributes to the formation of invasive pannus, and in neutrophil survival, which affects inflam
31 ation; failed: recurrent epithelial defects, pannus, and inflammation), phenotype of cells covering t
32 gnificant reduction in synovial hyperplasia, pannus, angiogenesis, inflammatory infiltration, bone an
33           Histology scores for inflammation, pannus, bone damage, and cartilage damage decreased in p
34 larization, which supports the growth of the pannus by supplying it with nutrients and oxygen.
35 e consistent across groups, while scores for pannus, calcification, and structural change increased o
36 nts showed that C3 deposition, inflammation, pannus, cartilage, and bone damage scores were all signi
37          MT1-MMP was highly expressed at the pannus-cartilage junction in RA joints.
38 number of apoptotic cells was present at the pannus-cartilage junction.
39 canning and in 46%-85% of cases thrombus and pannus coexist, complicating the diagnosis.
40 ectively reduced paw inflammation, inhibited pannus development and mitigated bone degradation compar
41 splanted on the patient's affected eye after pannus excision.
42 g cells and fibroblast-like cells within the pannus expressed both PTHrP and the PTH/PTHrP receptor,
43 nts in TSG-6-treated animals revealed little pannus formation and cartilage erosion, features which w
44 nts showed that DMH-11C treatment attenuated pannus formation and joint tissue injury.
45  parameters that differentiate thrombus from pannus formation as the etiology of obstructed mechanica
46               In the CLAU group, a localized pannus formation at the donor site of the limbal graft w
47  in the rampant fibroblast proliferation and pannus formation characteristic of rheumatoid arthritis.
48 ical prosthetic valve can help differentiate pannus formation from thrombus and may therefore be of v
49 its receptor in fibroblast proliferation and pannus formation in RA.
50                                              Pannus formation was more common in the aortic position
51 rophy and hyperplasia, and highly aggressive pannus formation with erosion of the articular cartilage
52      The changes included synovial invasion (pannus formation) of the enthesis.
53  reduced the inflammatory cell infiltration, pannus formation, and bone and cartilage degradation.
54 lammatory cell infiltrate, cartilage damage, pannus formation, and bone damage.
55 arthritis, characterized by tissue swelling, pannus formation, and bone deformities.
56 e, inflammatory cell infiltration, fibrosis, pannus formation, and bone erosion in joints of BLT1/BLT
57 mmatory cell infiltration, cartilage damage, pannus formation, and bone erosion in the joints of CIA
58 enesis and reduced synovial cell infiltrate, pannus formation, and cartilage erosions.
59 ease characterized by synovial inflammation, pannus formation, and progressive joint destruction.
60 and bone erosions, synovial hyperplasia, and pannus formation, and reduced numbers of vessels (angiog
61                                 Inflammatory pannus formation, bone erosion, and bone marrow inflamma
62 ptosis, proinflammatory cytokine expression, pannus formation, bone erosion, joint swelling, and pain
63 eases in the inflammatory cell infiltration, pannus formation, cartilage and bone destruction, and th
64                                This leads to pannus formation, cartilage breakdown, and eventual bone
65                     Scores for inflammation, pannus formation, cartilage damage, and bone resorption
66 -induced arthritis (CIA) is characterized by pannus formation, cell infiltration, and cartilage erosi
67    The most frequent causes are thrombus and pannus formation, in the absence of infectious data.
68 eficient mice revealed synovial hyperplasia, pannus formation, mononuclear cell infiltration, bone er
69      Fourteen valves had thrombus and 10 had pannus formation.
70 peration of <1 month separated thrombus from pannus formation.
71 and synovial inflammation but also mitigated pannus formation.
72 edema, fibrosis, epithelial downgrowths, and pannus formation.
73 re performed on a corneal button and corneal pannus from 2 EEC patients.
74 %) and specificity (95.5%) in discriminating pannus from thrombus.
75  Features of vascularity, villous formation, pannus, granularity, and capillary hyperemia were record
76 on the valve in 92% of cases, whereas 29% of pannuses had a soft echo density (p= 0.007).
77 Cia5d) rats preserved a normal joint without pannus, hyperplasia, or erosions.
78 thrombosis in 3 (7%), mismatch in 2 (5%) and pannus in 1 (2%).
79  The cause of mechanical AVR obstruction was pannus in 26 cases (53%), mismatch (P-PM) in 19 (39%) an
80 ve cellular infiltration and fully developed pannus in arthritic joints of non-GTP-fed mice.
81 4 main phenomena: 1) thrombosis; 2) fibrotic pannus ingrowth; 3) degeneration; and 4) endocarditis.
82                Tissue sections from the bone-pannus interface at sites of bone erosion were examined
83         Focal resorption of bone at the bone-pannus interface is common in rheumatoid arthritis (RA)
84 d arthritis (RA) is invasion of the synovial pannus into cartilage, and this process requires degrada
85 gressive growth and invasion of the synovial pannus into the surrounding cartilage and bone.
86 invasion and its associated joint damage and pannus invasion and destruction in RA.
87 ike synoviocyte (FLS), has a central role in pannus invasion and destruction of cartilage and bone in
88 tilage may be one of the factors that impede pannus invasion following an inflammatory insult to the
89 sential collagen-degrading proteinase during pannus invasion in human RA.
90 d in bone marrow in and adjacent to areas of pannus invasion in RA erosions.
91 ified in bone resorption lacunae in areas of pannus invasion into bone in RA patients.
92 t precursor cells are identified in areas of pannus invasion into bone in RA.
93  damage at sites adjacent to and distal from pannus invasion, and tartrate-resistant acid phosphatase
94 rophils, destruction of articular cartilage, pannus invasion, bone resorption, extra-articular fibrop
95 , membrane type 1 MMP (MT1-MMP), in synovial pannus invasiveness.
96                             The expansion of pannus is supported by extensive formation of new blood
97            A major cellular component of the pannus is the fibroblast-like synoviocyte (FLS), whose m
98 undantly expressed in cells at the cartilage-pannus junction in rheumatoid synovitis.
99 ive to new blood vessel formation, and hence pannus mass, adding to other therapeutic effects of anti
100 ll-thickness expression of K19 in the entire pannus of all eight specimens.
101 ogic verification of LSCD, the fibrovascular pannus of each cornea was removed.
102 n the pathogenic processes that arise in the pannus of rheumatoid arthritis and also interfere with c
103 ntation, pump surfaces become covered with a pannus of smooth muscle-like cells (myofibroblasts).
104                 Distinction of thrombus from pannus on obstructed prosthetic valves is essential beca
105 ical AVR obstruction, TEE differentiation of pannus or thrombus from mismatch is challenging.
106 iography in detecting the obstruction cause (pannus or thrombus), bioprosthesis calcifications, and e
107 rosion, bone erosion, and fibroproliferative pannus) or frozen, cryosectioned, and assayed for enzyme
108            Here, we show that osteoclasts in pannus originate exclusively from circulating bone marro
109       A high (HU>/=145) attenuation suggests pannus overgrowth, whereas a lower value is associated w
110 ases with thrombus formation but in 60% with pannus (p=0.0198).
111 function, Prosthetic Heart Valve Thrombosis, Pannus, Paravalvular Leak, CT Angiography, Cardiac, Valv
112 MP activity over TIMP action in the invading pannus, periarticular tissue, or SF.
113 age of origin of the entire conjunctivalized pannus removed from eight corneas with a diagnosis of to
114  were transplanted on the affected eye after pannus resection.
115 er of neutrophils in the synovial lining and pannus significantly decreased from day 28 to day 35, su
116                         In culture, all five pannus specimens generated a compact, small epithelial c
117 thelial outgrowth from segments of five such pannus specimens were analyzed by Western blot and rever
118 mbal epithelial explant, but not in all five pannus specimens.
119 ssed in the control specimen and in all five pannus specimens.
120 at sites of erosion and was localized to the pannus starting on day 21.
121 f extracellular matrix (ECM) proteins in the pannus suggest that intracellular signals generated thro
122 f an aggressive, tumor-like structure called pannus that erodes the joint.
123 ce studies showed that, aside from the joint pannus, the subchondral bone tissue constitutes an essen
124                               The mechanism (pannus/thrombus vs. mismatch) was identified in 10% by T
125 s in vitro and also promote joint erosion by pannus tissue in vivo.
126    The resultant epithelial phenotype of the pannus tissue was not corneal, as evidenced by the negat
127 mity to MNCs, and in occasional cells within pannus tissue, but not in the MNCs in bone resorption la
128                     Thrombi were larger than pannuses (total length 2.8+/-2.47 cm vs. 1.17+/-0.43 cm;
129 e 6 historical controls that developed LSCD, pannus was noted in 1 (13%) and pseudopterygium extendin
130 sion and localization of MT1-MMP in human RA pannus were investigated by Western blot analysis of pri
131 is characterized by an inflammatory synovial pannus which mediates tissue destruction.
132 ke expansion of inflamed synovial tissue, or pannus, which causes much of the joint damage in this di
133  for example, underlies the formation of the pannus, while proliferation of endothelial cells results
134 ignificant findings included the presence of pannus without inflammatory changes in the regions in wh

 
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