ライフサイエンスコーパス: papillary carcinoma

1 nomas); and group C (medullary, mucinous and papillary carcinomas).

2 ies that included follicular hyperplasia and papillary carcinoma.

3 s in the alemtuzumab group developed thyroid papillary carcinoma.

4 n reported in patients with hereditary renal papillary carcinomas.

5 in the development of post-Chernobyl thyroid papillary carcinomas.

6 Papillary carcinoma (67.2%) and follicular carcinoma (21

7 Most cancers were papillary carcinoma (68 [88.3%] of 77).

8 5-22.3) years after HCT and included 13 with papillary carcinoma and 5 with benign adenomas.

9 oidectomy was done in 85.3% of patients with papillary carcinoma and 71.3% of patients with follicula

10 ents with high-risk thyroid cancer (303 with papillary carcinoma and 82 with follicular carcinoma).

11 ations of c-Met have been described in renal papillary carcinoma and gastrointestinal cancers includi

12 thyroid hyperplasia, solid tumor variants of papillary carcinoma and metastatic cancer.

13 and spontaneously progressed to both cystic papillary carcinoma and metastatic PDAC.

14 It is possible to distinguish between papillary carcinomas and other lesions with the thyroid

15 linkage between chronic pancreatitis, cystic papillary carcinoma, and PDAC.

16 e VHL gene and clear cell carcinoma, MET and papillary carcinoma, and the families of genes that they

17 as, 1 of 30 Hurthle cell carcinomas, 1 of 90 papillary carcinomas, and 0 of 10 nodular goiters had 3p

18 inomas, grade I and II from grade III serous papillary carcinomas, and ovarian from breast carcinomas

19 NA was overexpressed in three of six thyroid papillary carcinomas as compared with paired normal thyr

20 ally positive p53 staining was found in four papillary carcinomas being primarily confined to solid a

21 ype p53-overexpressing derivative of the NPA papillary carcinoma cell line was obtained, and these ce

22 ase 8 construct effectively killed the SW579 papillary carcinoma cell line, arguing that the action o

23 0 thyroid tumor cell lines, including 2 of 2 papillary carcinoma cell lines, 4 of 5 anaplastic carcin

24 as preferentially found in radiation-induced papillary carcinomas developing after a short latency, w

25 errant expression in mice is associated with papillary carcinoma development.

26 high prevalence of ret/PTC3, whereas typical papillary carcinomas do not, suggesting that the differe

27 by well-differentiated carcinomas, including papillary carcinoma, follicular carcinoma, and medullary

28 ry carcinoma) or papillary (any component of papillary carcinoma); for papillary tumors, the proporti

29 Papillary carcinomas formed a tightly clustered group, a

30 characteristics of 38 post-Chernobyl thyroid papillary carcinomas from Belarussian children 5-18 year

31 rangements are highly prevalent in pediatric papillary carcinomas from children exposed to radiation

32 ren 5-18 years old with those of 23 sporadic papillary carcinomas from the same age children without

33 vating mutations in cancers other than renal papillary carcinoma has not been well defined.

34 Osteosarcomas in juvenile Wwox(-/-) and lung papillary carcinoma in adult Wwox(+/-) mice occurred spo

35 lial tumours of the bladder are diagnosed as papillary carcinomas in accordance with the WHO classifi

36 The presence of a component of papillary carcinoma is more common than previous reports

37 In thyroid papillary carcinomas, mutations of genes coding for effe

38 situ (n=2), ductal carcinoma in situ (n=7), papillary carcinoma (n=2), and invasive ductal carcinoma

39 was effective in helping distinguish between papillary carcinomas (n = 10) and other lesions (n = 43)

40 re as follows: clear cell carcinomas (n=23), papillary carcinomas (n=6), and chromophobe carcinomas (

41 that produced false-positive PET scans were papillary carcinoma of the thyroid (1), bronchogenic car

42 radiation-induced thyroid tumorigenesis, 33 papillary carcinomas, one follicular carcinoma and 22 be

43 ified as nodular-sclerosing (no component of papillary carcinoma) or papillary (any component of papi

44 inomas but not in 20 follicular adenomas, 10 papillary carcinomas, or 10 multinodular hyperplasias.

45 onal profiles of four normal thyroids and 51 papillary carcinomas (PCs) were generated using DNA micr

46 and malignant follicular carcinoma (FTC) and papillary carcinoma (PTC) thyroid tissues.

47 Five of the N-terminal genes found in papillary carcinoma rearrangements are also involved in

48 The morphological variants of papillary carcinoma showed different prevalence of the s

49 Thirty-two clear cell carcinomas, 10 papillary carcinomas, six chromophobe carcinomas, four o

50 ts with hyperplasia and Group B of rats with papillary carcinoma (stages Ta and T1).

51 expressed in radiation-induced and childhood papillary carcinomas than other members of the fusion on

52 lary lesions suspicious at SCNB proved to be papillary carcinomas; the fourth had no residual carcino

53 Higher CXCR6 expression in serous papillary carcinoma tissues suggests its association wit

54 ase types that range from indolent localised papillary carcinomas to the fulminant and lethal anaplas

55 s (WDCs; two [6.7%] of 30 of the tumors were papillary carcinomas, two [10.5%] of 19 of them were fol

56 g radiation-induced tumors, solid variant of papillary carcinoma was found in 37%, follicular in 29%,

57 (n = 10) and other lesions (n = 43) because papillary carcinomas were stiffer than other lesions (P

58 alphavbeta3-positive vessels in the group of papillary carcinomas whereas it correlated with integrin

59 Solid papillary carcinoma with reverse polarity (SPCRP) is a r

60 nes, severe cystic pathology, and ultimately papillary carcinomas with hepatic metastases.

61 and RET genes in 12 post-Chernobyl pediatric papillary carcinomas with known RET/PTC3 rearrangement.

62 the cases were histologically classified as papillary carcinomas, with 2 of them exhibiting follicul