ライフサイエンスコーパス: papule

1 d with asymptomatic red to brown macules and papules.

2 the pathogenesis and resistance of Gottron's papules.

3 so greatly increased and similar to those in papules.

4 hough levels were not equivalent to those in papules.

5 areas were significantly smaller than parent papules.

6 condition characterized by widespread warty papules.

7 rnally, causing linear pruritic erythematous papules.

8 er had begun as an asymptomatic erythematous papule 1 week previously.

9 the ciprofloxacin-treated subjects developed papules 1 week after the initial treatment, and the nint

10 patients with only patches and/or follicular papules (10-year OS, 71% vs 80%), while the survival rat

11 he 9 azithromycin-treated subjects developed papules 4-10 weeks (mean, 6.8) after the initial treatme

12 biopsies were obtained from an inflammatory papule and from normal skin in six patients with acne.

13 Overall, the papule and pustule formation rates for 35000HP and FX533

14 aABCD deletion mutant at multiple sites; the papule and pustule formation rates of the mutant and par

15 The papule and pustule formation rates were similar to those

16 5000HP and 35000HP-sodC-cat and observed for papule and pustule formation.

17 fected with 35000 and FX517 and observed for papule and pustule formation.

18 and among those who started treatment in the papule and vesicle lesion stages.

19 , characterized by hyperkeratotic follicular papules and erythematous desquamative plaques.

20 lice bites cause pruritic excoriated macules/papules and hyperpigmentation in areas where clothing se

21 Classical features include beaded eyelid papules and laryngeal infiltration leading to hoarseness

22 Follicular hyperkeratotic papules and marked pruritus were also prominent clinical

23 the epidermis consisting of verrucoid scaly papules and plaques following Blaschko's lines.

24 On examination, there were scattered pink papules and plaques on the trunk, thighs, and buttocks a

25 ifestations include recurrent hyperkeratotic papules and plaques that occur mainly in seborrheic area

26 ic pink, violaceous, or brown hyperkeratotic papules and plaques throughout the body, was conducted a

27 4, he was seen at our clinic with new dermal papules and plaques.

28 Sites inoculated with the mutant produced papules and pustules at rates similar to the rates obser

29 culation experiments, the neuA mutant formed papules and pustules at rates that were no different tha

30 Papules and pustules contained a predominant T cell infi

31 Both papules and pustules contained mixed or T helper 1 type

32 Skin histology of papules and pustules from 5 men having sex with men with

33 that is impaired in its ability to form both papules and pustules in humans.

34 Subsequently, there were numerous papules and pustules--similar to the rash seen in patien

35 /=10 mixed-type lesions such as inflammatory papules and ulcers, located in >/=2 body parts.

36 expression was increased to levels found in papules and vascular adhesion molecule 1 levels were upr

37 All subjects developed papules and were randomized to treatment with a single d

38 pic MF-like scalp plaques with alopecia, (2) papules and/or plaques, (3) photoaccentuated dermatitis,

39 8% progressed to papillomas, 9% persisted as papules, and 22% completely regressed.

40 ions were characterized by purpuric macules, papules, and confluent plaques predominantly on the lowe

41 y finger pad inflammation, palmar violaceous papules, and digital ulcerations.

42 y finger pad inflammation, palmar violaceous papules, and digital ulcerations.

43 aracterized clinically by multiple keratotic papules, and histologically by focal loss of adhesion be

44 of acne vulgaris lesions such as comedones, papules, and pustules.

45 Gottron's papules are a particularly resistant manifestation of DM

46 Such papules are histologically analogous to angiofibromas; t

47 ulation experiments, 35000HPDeltacpxA formed papules at a rate and size that were significantly less

48 om histological sections and 7 of 7 of whole papules by mutation-specific PCR analysis.

49 eated and untreated skin for changes in mean papule counts (-4.2 vs -2.2; P =.08), mean pustule count

50 ons including the heliotrope rash, Gottron's papules, cuticular changes including periungual telangie

51 Gottron's papule dermis contained more C4S and CD44v7 than non-Got

52 Papules developed at 92.8% (95% confidence interval [CI]

53 Papules developed at 95% of 40 sites infected with live

54 Papules developed at similar rates at sites inoculated w

55 Papules developed at similar rates at sites inoculated w

56 Papules developed at similar rates at sites inoculated w

57 When papules developed, the subjects were treated with antibi

58 Sixty-nine percent (95% CI, 36.8%-90.9%) of papules evolved into pustules in the reinfection group,

59 and extended over the whole body, with some papules evolving into vesiculobullous lesions.

60 tules but did not detect bacteria in the one papule examined.

61 There was a relationship between papule formation and estimated delivered dose.

62 nfected at three sites with each strain, the papule formation rate was 95.8% for 35000HP versus 62.5%

63 The papule formation rates were 88% (95% confidence interval

64 tory portions were dilated, and blisters and papules formed on the skin surface in the knockout mice.

65 He presented to care for a tender, red papule in his right axilla with increasing induration an

66 atments in 2 patients and a reduction of the papules in 2 patients.

67 Here we present a case of atrichia with papules in a patient with a normal HAIRLESS gene but wit

68 he leg and foot in case 1 and small perioral papules in case 2.

69 of the Molluscipoxvirus genus, causes benign papules in healthy people but disfiguring lesions in imm

70 us (MCV) is a poxvirus that causes localized papules in healthy persons.

71 disease characterized by multiple keratotic papules in the seborrheic regions of the body.

72 Generalized atrichia with papules is a rare disorder characterized by loss of hair

73 On May 29, their infant daughter developed a papule on her philtrum.

74 tiple skin lesions, including a 6-mm crusted papule on the left eyebrow, which was resected and, on p

75 led numerous small dome-shaped flesh-colored papules on the head and neck, as well as multiple palpab

76 in his 60s presented with chronic verrucous papules on the lips, tongue, and buccal mucosa refractor

77 currence or pathergy, inflammatory edematous papules on the neck and trunk (sometimes umbilicated), a

78 white man with widespread, crusted, pruritic papules on the scalp, ears, and face and a purpuric and

79 terized by pruritic, flat-topped, violaceous papules on the skin.

80 thema lesion stage) and when initiated late (papule or vesicle stage).

81 ted with multiple erythematous nonfollicular papules partially confluent to plaques on his breast and

82 bies is characterized by excoriated pruritic papules, plaques, and pathognomonic burrows on finger/to

83 , indurated, nonscarring, pink to violaceous papules, plaques, or nodules, devoid of surface changes

84 al manifestations included: piezogenic pedal papules (PPP), joint hyperextensibility, early onset ost

85 Pearly penile papules (PPPs) are benign, dome-shaped lesions found aro

86 Stability was achieved for papule, pustule, and abscess.

87 remove." Eight HS primary lesions, including papule, pustule, nodule, plaque, ulcer, abscess, comedo,

88 om open and closed comedones to inflammatory papules, pustules, cysts, and nodules, and scarring may

89 %), pain/tenderness in 118 of 481 (25%), and papules/pustules in 117 of 481 (24%).

90 Rubbery papules reflected an intense cellular immune reaction do

91 n 24 sites followed to end point, 27% of the papules resolved, 69% (95% CI, 47.1%-86.6%) evolved into

92 on of serial sections of 69 DMBA/TPA-induced papules revealed that they were focal hyperplastic lesio

93 mined non-Gottron's DM lesions and Gottron's papule skin biopsies for C4S, CD44 variant 7 (CD44v7), a

94 where papillomas subsequently developed were papules, slightly raised areas of skin ranging in diamet

95 lesions, including tender red or violaceous papules, sometimes edematous, without fever, arthralgia,

96 rom biopsies of 12 of 15 pustules and 1 of 7 papules, suggesting that H. ducreyi replicates between t

97 However, mutant papule surface areas were significantly smaller than par

98 hree molecules uniquely present in Gottron's papules that contribute to inflammation individually and

99 and myalgia and a characteristic rash, with papules that evolve to vesicles, pustules, and crusts in

100 Although both strains caused papules to form at similar rates, the pustule formation

101 Of 78 DMBA/TPA-induced papules tracked during 15 weeks of TPA treatments, 68% p

102 ress through 4 well-defined stages (macules, papules, vesicles, and pustules) over 2 to 4 weeks.

103 On biopsy, this papule was an invasive acantholytic squamous cell carcin

104 In each patient, the appearance of the papules was either completely diminished or significantl

105 Complete clearance of the papules was noted after 2 to 3 treatments in 2 patients

106 Papules were first detected in DMBA-initiated mice 21 da

107 Sandy patches and rubbery papules were found in 41 of 118 women (35%).

108 However, papules were significantly smaller at mutant sites (mean

109 Papules were significantly smaller at mutant sites than

110 stinguishable from generalized atrichia with papules, which was previously thought to be caused only

111 ficant decrease in the prevalence of rubbery papules with age, even after adjustment for urinary ova

112 thy that appear as atrophic, porcelain-white papules with red, telangiectatic borders.

113 inical morphology consisting of erythematous papules with scale in a variety of distributions.

114 sponsive to vismodegib but with an enlarging papule within the tumor bed.