ライフサイエンスコーパス: paralysis

1 rve finding that was unilateral or ascending paralysis).

2 nally deleted in astrocytes display episodic paralysis.

3 CP assembly factors to the TZ, and flagellum paralysis.

4 l prostheses being developed for people with paralysis.

5 channels (NaV) of nerve and muscle, causing paralysis.

6 o restore function to people with upper-limb paralysis.

7 d during mechanical ventilation after muscle paralysis.

8 lted in CNS alphaS pathology associated with paralysis.

9 ys receiving 10(8) or 10(9) TCID50 developed paralysis.

10 generalized weakness was described more than paralysis.

11 as hyperphagia, increased weight, and leaden paralysis.

12 an ex vivo assay for BoNT/A-mediated muscle paralysis.

13 ve emotions trigger these episodes of muscle paralysis.

14 as a consequence, instigates ZIKV-associated paralysis.

15 st-developmental expression caused age-onset paralysis.

16 infected, and many of the monkeys developed paralysis.

17 motor function and accelerated the onset of paralysis.

18 onnections, resulting in movement defects or paralysis.

19 ed by three clinical trial participants with paralysis.

20 and an almost complete elimination of muscle paralysis.

21 r restoration of reaching and grasping after paralysis.

22 S fibril muscle injected mice that developed paralysis.

23 ust alphaS pathology and in some cases cause paralysis.

24 edies of millions of children with permanent paralysis.

25 everity, resulting in recovery from hindlimb paralysis.

26 inently featuring motor neuron (MN) loss and paralysis.

27 tore complex movements in people living with paralysis.

28 f full-length tau isoforms did not result in paralysis.

29 uncertain relation to hypokalaemic periodic paralysis.

30 ucilia cuprina) induced rapid but reversible paralysis.

31 people worldwide living with the effects of paralysis.

32 show delayed onset and a shorter duration of paralysis.

33 thus restored grasping abilities after hand paralysis.

34 l-established cause of myotonia and periodic paralysis.

35 ce showed decreased neural invasion and less paralysis.

36 H2O airway pressure under heavy sedation or paralysis.

37 this recruitment translates into more severe paralysis.

38 cause susceptibility to myotonia or periodic paralysis.

39 sure resulted in temporary functional T-cell paralysis.

40 mulus relation, without evidence of periodic paralysis.

41 the neuroinflammatory process that result in paralysis.

42 , Caenorhabditis elegans exhibits reversible paralysis.

43 ons in the spinal cord and subsequent muscle paralysis.

44 ession, even when started after the onset of paralysis.

45 igated during surveillance for acute flaccid paralysis.

46 etylcholine binding to its receptor, causing paralysis.

47 cal symptoms including tremors and hind-limb paralysis.

48 ly, we noted no events of vaccine-associated paralysis.

49 perexpression promotes immune exhaustion and paralysis.

50 otor neuron degeneration, muscle wasting and paralysis.

51 SOD1-G93A mice, modestly delaying the age to paralysis.

52 om ischemia-induced neuron loss and hindlimb paralysis.

53 e selective loss of motor neurons leading to paralysis.

54 r neuron loss that ultimately leads to fatal paralysis.

55 hout histological changes while RFA produced paralysis.

56 ly contributes to the amelioration of muscle paralysis.

57 cation within the brain leading to increased paralysis.

58 o enable hind limb motor functions following paralysis.

59 , a condition characterized by acute flaccid paralysis.

60 r neuron loss correlates with the pattern of paralysis.

61 iminishing T cell function through metabolic paralysis.

62 disease of the CNS that causes blindness and paralysis.

63 als 1 month following C2 hemisection induced paralysis.

64 nerves, which prevented muscle fibrosis and paralysis.

65 eir ability to cause neurological damage and paralysis.

66 1/50), infections 18% (9/50), post-operative paralysis 14% (7/50), neurological diseases 8% (4/50), t

67 Most had no fever (99%), descending paralysis (93%), no mental status change (91%), at least

68 communication in people with complete motor paralysis-a condition called complete locked-in state (C

69 s long-lasting and potentially fatal flaccid paralysis-a major feature of botulism.

70 er rate of reporting non-polio acute flaccid paralysis (AFP) (OR = 1.13, 95% CI 1.02-1.26 for a 1-uni

71 ool specimens of patients with acute flaccid paralysis (AFP) and sewage samples collected from 60 env

72 Surveillance for acute flaccid paralysis (AFP) is a fundamental cornerstone of the glob

73 old identified with non-polio acute flaccid paralysis (AFP) reported through polio surveillance, inf

74 ess indicators associated with acute flaccid paralysis (AFP) surveillance, routine immunization, and

75 n activity that can complement acute flaccid paralysis (AFP) surveillance.

76 Loss of LSD1 leads to paralysis, along with widespread hippocampus and cortex

77 ared with mechanical ventilation with muscle paralysis and absence of diaphragmatic activity.

78 ic mice that overexpress mutant SOD1 develop paralysis and accumulate misfolded SOD1 onto the cytopla

79 er metabolite for MDSCs that mediates T cell paralysis and can serve as a target to improve cancer im

80 regated alphaS in M83(+/-) mice also induced paralysis and CNS alphaS pathology, although less effici

81 om patients with MS and reversed established paralysis and CNS inflammation in four different EAE mod

82 din-3-yl)phenol (PHTPP) reversed established paralysis and CNS inflammation, characterized by a drama

83 ess a cluster of children with acute flaccid paralysis and cranial nerve dysfunction geographically a

84 We report that sPIF reduces paralysis and de-myelination of the brain in a clinicall

85 rized by motor neuron loss and that leads to paralysis and death 2-5 years after disease onset.

86 omyelitis mouse model with complete hindlimb paralysis and death by 30 d after induction of QKI delet

87 uired for neurotransmission, causing flaccid paralysis and death by asphyxiation.

88 brain inflammation, and protected mice from paralysis and death during treatment.

89 erative motor neuron disease, causing muscle paralysis and death from respiratory failure.

90 It reduced paralysis and death in an acute pulmonary thrombosis mod

91 ic killing assay, whereas worms succumbed to paralysis and death in its absence.

92 hibitor of ferroptosis, delayed the onset of paralysis and death induced by Gpx4 ablation.

93 rons in the brain and spinal cord leading to paralysis and death.

94 tor neuron dysfunction disease that leads to paralysis and death.

95 beginning at 16 days, followed by hind-limb paralysis and death.

96 m the time of infection delayed the onset of paralysis and death.

97 mitant progressive muscle weakness ending in paralysis and death.

98 sive motor neuron disease that culminates in paralysis and death.

99 urred, virus was cleared, and mice developed paralysis and died.

100 l cord syndrome that can result in permanent paralysis and disability.

101 wake symptoms, such as hallucinations, sleep paralysis and disturbed sleep.

102 or neuron loss, resulting in muscle wasting, paralysis and eventual death.

103 used by the loss of motor neurons leading to paralysis and eventually death.

104 autoimmune encephalomyelitis (EAE), reducing paralysis and inflammation, while inducing several pathw

105 recovery in humans with different degrees of paralysis and levels of spinal cord injury.

106 indness/retinal detachment, temporary facial paralysis and maternal death, were adopted.

107 It can result in paralysis and may be fatal.

108 Both paralysis and muscle damage could be rescued with collag

109 ic dy(2j)/dy(2j) mice with apparent hindlimb paralysis and muscle fibrosis.

110 nce that loss of LSD1 in adult mice leads to paralysis and neurodegeneration in the hippocampus and c

111 variants exhibiting polio-like acute flaccid paralysis and other central nervous system manifestation

112 th soluble, nonaggregated alphaS resulted in paralysis and pathology in only a subset of mice, wherea

113 severity ranging from progressive infantile paralysis and premature death (type I) to limited motor

114 ed by extensive motor neuron loss leading to paralysis and premature death.

115 e expression of TDP-43 in motoneurons led to paralysis and premature lethality.

116 on diseases associated with muscle weakness, paralysis and respiratory failure.

117 Using acute flaccid paralysis and routine, monthly countrywide environmental

118 and can be associated with cataplexy, sleep paralysis and sleep-related hallucinations.

119 oped severe encephalomyelitis with hind-limb paralysis and succumbed within 7 days.

120 Loss of either of these kinases results in paralysis and worm death in a mammalian host.

121 scular disease involving motor neuron death, paralysis and, ultimately, respiratory failure.

122 eight gain, hypersomnia, fatigue, and leaden paralysis) and may moderate the antidepressant effects o

123 Neuroinvasion, paralysis, and death occurred for all mice infected with

124 s, resulting in progressive muscle weakness, paralysis, and death within 5 years of diagnosis.

125 e associated with myositis, atrophy, paresis/paralysis, and death.

126 is, dilated cardiomyopathy, type I diabetes, paralysis, and even death.

127 ons degenerate, resulting in muscle atrophy, paralysis, and fatality.

128 it problems, muscle denervation and atrophy, paralysis, and have diminished life expectancy.

129 roencephalogram and complete skeletal muscle paralysis, and is associated with vivid dreams.

130 n, muscle and cartilage dysmorphogenesis and paralysis, and lethality by 2-5 wk, which indicates an e

131 <10 or >29, flail chest, hemo/pneumothorax, paralysis, and multisystem trauma.

132 accine (OPV), surveillance for acute flaccid paralysis, and OPV "mop-up" campaigns.

133 ing myocarditis, pancreatitis, acute flaccid paralysis, and poliomyelitis.

134 ike lipid pathology, MN death, astrogliosis, paralysis, and reduced survival.

135 and found that these larvae exhibit episodic paralysis, and their astrocytes poorly infiltrate the CN

136 s transient ischemic attacks, migraine, Todd paralysis, and Uhthoff phenomenon.

137 ucing symptoms such as loss of coordination, paralysis, and violent spasms.

138 sponse, arguing against a generalized T cell paralysis as a major cause of protracted immune suppress

139 een 2007 and 2017, demonstrating chronic bee paralysis as an emergent disease.

140 itochondrial electron transport chain causes paralysis as well as muscle structural damage in the nem

141 e used to assess early disease onset, before paralysis, as well as disease progression in diverse mou

142 set and protects from the development of the paralysis associated with a murine model of multiple scl

143 res of the increasing cases of acute flaccid paralysis associated with anterior myelitis noted in the

144 Ps, and drugs for the treatment of diaphragm paralysis associated with high cervical spinal cord inju

145 e results in progressive ataxia and hindlimb paralysis associated with motor neuron degeneration, sev

146 therapies for SMA, it is unclear whether the paralysis associated with the disease derives solely fro

147 behavior and heterogeneous perceived risk of paralysis based on the individual's comprehension of the

148 primary spinal cord injury (SCI) results in paralysis below the level of injury and is associated wi

149 le for persons suffering from complete motor paralysis but intact cognitive and emotional processing,

150 LeTx is one of the toxins causing immune paralysis by cleaving and inactivating the mitogen-activ

151 rove the quality of life of individuals with paralysis by directly mapping neural activity to limb- a

152 ighly potent neurotoxin that elicits flaccid paralysis by enzymatic cleavage of the exocytic machiner

153 extensive nerve cell dysfunction and severe paralysis by the age of 3 weeks.

154 The number of chronic bee paralysis cases increased exponentially between 2007 and

155 th received several reports of acute flaccid paralysis cases of unknown etiology.

156 Cases of paralysis caused by poliovirus have decreased by >99% si

157 rts should be prioritised to protect against paralysis caused by type 2 poliovirus; however, this inc

158 ted the mechanism of temporary acute flaccid paralysis caused by Zika virus infection in aged interfe

159 We demonstrate that hemidiaphragm paralysis causes muscle fibre hypertrophy, maintaining g

160 h genetically confirmed hypokalemic periodic paralysis (Cav1.1-R1239H mutation, n = 5; Cav1.1-R528H m

161 ream mentation combined with skeletal muscle paralysis characterizes rapid eye movement sleep.

162 extending both safety range and duration of paralysis compared with the control BoNT/B.

163 shock syndrome, encephalitis, acute flaccid paralysis, congenital abnormalities and fetal death.

164 tween 2009 and 2014, using the acute flaccid paralysis database at the World Health Organization Nige

165 contractile force and complete pectoral fin paralysis, demonstrating that mylpf impairment most seve

166 of toxic organophosphates that induce human paralysis due to severe axonopathy of large neurons.

167 for improving motor function in humans with paralysis due to spinal cord injury.

168 ioning, and a cohort with complete hind limb paralysis due to T8 spinal cord transection.

169 immunometabolic conditions, including immune paralysis during septic shock.

170 n uncontrollable onset of muscle weakness or paralysis during wakefulness.

171 tory disease and is associated with cases of paralysis, especially among children.

172 diagnostic criteria for other acute flaccid paralysis etiologies were excluded.

173 eurological decline resulting in generalized paralysis, extreme emaciation and death.

174 and chemical characterization of Schistosome Paralysis Factor (SPF), a novel tetracyclic alkaloid pro

175 ted varying efficacy in transiently reducing paralysis following BoNT poisoning, the precise mechanis

176 nces, and REM-sleep-related phenomena (sleep paralysis, hallucinations) that intrude into wakefulness

177 However, bilateral vocal cord paralysis has rarely been described.

178 aiming to restore standing and walking after paralysis have been extensively studied in animal models

179 ents with myotonia or hyperkalaemic periodic paralysis (HyperPP).

180 ent through the VSD and hypokalemic periodic paralysis (HypoPP), but these have hitherto not been ass

181 ed attacks can quickly lead to blindness and paralysis if undiagnosed and untreated.

182 Joining the Miami Project to Cure Paralysis in 1989 brought the opportunity to use this kn

183 35%), encephalitis in 6 (11%), acute flaccid paralysis in 4 (7%), and autonomic dysregulation with pu

184 d malformation of astrocytes and episodes of paralysis in a Drosophila model.

185 es for the ability to reduce the severity of paralysis in a mouse model of EV-D68 infection: (1) huma

186 ive in vivo where they inhibit Abeta-induced paralysis in a transgenic Abeta Caenorhabditis elegans m

187 t strain of Sindbis virus (NSV) causes fatal paralysis in adult C57BL/6 mice during clearance of infe

188 studying and eventually treating progressive paralysis in ALS patients.

189 death of motor neurons leads to progressive paralysis in amyotrophic lateral sclerosis (ALS).

190 s a bilateral, symmetric, descending flaccid paralysis in an afebrile and alert patient without senso

191 fests with cranial nerve palsies and flaccid paralysis in children and adults.

192 n, a process contributing to immunometabolic paralysis in human and mouse sepsis monocytes, can be re

193 d neutralizing antibodies to EV-D68, reduced paralysis in infected mice and decreased spinal cord vir

194 read to the central nervous system and cause paralysis in infected patients, especially young childre

195 an 2014 EV-D68 isolate that reliably induces paralysis in mice due to infection and loss of spinal co

196 We observed modestly decreased time to paralysis in mice transgenic for human A53T alphaS (line

197 VAMP4 in cultured neurons and causes flaccid paralysis in mice.

198 ha2-Na/K ATPase that triggers episodic motor paralysis in mice.

199 minin-alpha1, ameliorates muscle wasting and paralysis in mouse models of MDC1A, demonstrating its im

200 indicate that the cytopathology and episodic paralysis in our Drosophila EA6 model stem from a gain-o

201 Flexible bronchoscopy revealed vocal cord paralysis in paramedian position, potentially due to ext

202 ant from AQP4(-/-), but not WT, mice induced paralysis in recipient WT and B-cell-deficient mice.

203 t PMSCs have the potential to cure hind limb paralysis in the fetal lamb model of SB via a paracrine

204 Muscle paralysis increased the number of Hh-responsive cells in

205 , the dramatic motor neuron degeneration and paralysis induced by Gpx4 ablation suggest that ferropto

206 Bilateral vocal cord paralysis is a rare but potentially fatal complication o

207 Chronic bee paralysis is a well-defined viral disease of honey bees

208 ngland and Wales to test whether chronic bee paralysis is an emerging infectious disease and investig

209 ion during mechanical ventilation and muscle paralysis may be a contributing factor to unsuccessful r

210 Injury, resulting in VF paralysis, may contribute to subsequent swallowing, voic

211 nding and clinical treatment of facial nerve paralysis, mitigating facial asymmetry, aberrant reinner

212 orf72 ALS/FTD that shows decreased survival, paralysis, muscle denervation, motor neuron loss, anxiet

213 entified in patients with myotonia, periodic paralysis, myasthenia, or congenital myopathy.

214 ns ranges from sub-clinical to acute flaccid paralysis, myocarditis and meningitis.

215 hen present, is not directly responsible for paralysis nor learning disabilities induced in the worm,

216 Paralysis occurring in amyotrophic lateral sclerosis (AL

217 PZQ causes Ca(2+) influx and spastic paralysis of adult worms and rapid vacuolization of the

218 tabolic phenotype of MDSCs and MDSC-mediated paralysis of CD8(+) T cells.

219 ervation, and consequent partial-to-complete paralysis of hemidiaphragm.

220 ce the course of DSB repair, indicating that paralysis of meiotic chromosome mobility in a genotoxic

221 botulism, a fatal illness caused by flaccid paralysis of muscles due to botulinum neurotoxin (BoNT)

222 These events resulted in complete paralysis of primary CD4(+) T cell activation, affecting

223 I are respiratory complications secondary to paralysis of respiratory muscles.

224 ting motor neurons that leads to progressive paralysis of skeletal muscle.

225 urological disorder that is characterized by paralysis of the facial nerves and variable other congen

226 , we find immediate, thorough, but temporary paralysis of the worms.

227 Reversion to neurovirulence, assessed as paralysis of transgenic mice, was low in isolates from t

228 ome, 50% were found to have bilateral facial paralysis on examination.

229 ents (5.8%): one had transient diaphragmatic paralysis, one vascular access complication, and one had

230 Clusters of acute flaccid paralysis or cranial nerve dysfunction in children are u

231 No gross signs of paralysis or paresis were also observed.

232 Patients initially seen with acute flaccid paralysis or pulmonary edema had significantly greater f

233 oth the dominant clinical presentation, e.g. paralysis or tremor and additional symptomatology such a

234 salivation (OR = 34.6, 95% CI 11.3-106.5) or paralysis (OR = 19.0, 95% CI 4.8-74.8) and when the dog

235 Removal of loading via microgravity, paralysis, or bed rest leads to rapid loss of muscle mas

236 e behaviour, facial dysmorphism, left facial paralysis, post-axial polydactyly, and for the first tim

237 drome including lung protective ventilation, paralysis, prone positioning, and inhaled nitric oxide.

238 mTORC1 paralysis protects multiple myeloma cells against DEPTOR

239 OPV after the switch would risk outbreaks of paralysis related to type 2-circulating vaccine-derived

240 came resistant to P. ostreatus We found that paralysis-resistant mutants all harbored loss-of-functio

241 rapy for the treatment of respiratory muscle paralysis resulted from cervical SCI.

242 bital-induced sleep and zoxazolamine-induced paralysis, secondary to decreased expression and activit

243 Now we know that patients with gaze paralysis show conventional benefits of exogenous (invol

244 V103Z SOD1 moderately accelerated the age to paralysis, similar in magnitude to WT/L126Z mice.

245 omuscular disorders with features of ataxia, paralysis, skeletal muscle wasting, and degeneration.

246 Besides obvious motor and sensory paralysis, spinal cord injury also induces a functional

247 y also induces a functional SCI-IDS ('immune paralysis'), sufficient to propagate clinically relevant

248 KccB also caused astroglial malformation and paralysis, supporting the idea that the EAAT1(P>R) mutat

249 cted a retrospective review of acute flaccid paralysis surveillance in the security-compromised state

250 ntary immunization activities, acute flaccid paralysis surveillance, and routine immunization with th

251 human resources and strengthen acute flaccid paralysis surveillance.

252 gans based on the phenotype swimming-induced paralysis (Swip), a paralytic behavior observed in herma

253 erature-based screening method for synthetic paralysis that can be used to rapidly identify genetic p

254 that RLN transection created ipsilateral VF paralysis that did not recover by 13 weeks postsurgery.

255 exhibit an increased incidence of hind limb paralysis that is linked to productive HSV-2 infection i

256 loped motor deficits including acute flaccid paralysis that peaked 8-10 days after viral challenge.

257 For people with severe paralysis, the ability to display emotions intentionally

258 worldwide suffer from diseases that lead to paralysis through disruption of signal pathways between

259 b was transected at postnatal day 8 to cause paralysis to that limb.

260 demonstrated that for long-term spastic limb paralysis, transferring the seventh cervical nerve (C7)

261 profiles based on limb-specific paresis and paralysis, tremors and seizures, and other clinical sign

262 ronic sleepiness and cataplexy-sudden muscle paralysis triggered by strong, positive emotions.

263 900S, R1239H) linked to hypokalemic periodic paralysis type 1 and of CaV1.3-R3 (R990H) identified in

264 ein thrombosis, major procedure, spinal cord paralysis, venous injury, lower extremity fracture, pelv

265 nesis, demonstrating that the fungus induced paralysis via the cilia of nematode sensory neurons.

266 The chronic bee paralysis virus (CBPV), extracted from sick or dead bees

267 The cricket paralysis virus (CrPV) uses an internal ribosomal entry

268 The intergenic IRES of Cricket Paralysis Virus (CrPV-IRES) forms a tight complex with 8

269 characterized how the IRES of Israeli acute paralysis virus (IAPV) intergenic region captures and re

270 of the honey bee dicistrovirus Israeli acute paralysis virus (IAPV) IRES PKI domain can uncouple 0 an

271 rus (DWV)] and dicistrovirids [Israeli acute paralysis virus (IAPV), black queen cell virus (BQCV)] i

272 altered by a virus of concern, Israeli acute paralysis virus (IAPV).

273 Many of them, including slow bee paralysis virus (SBPV), cause lethal diseases in honeybe

274 that MV is most closely related to Slow bee paralysis virus (SBPV), which is highly virulent in hone

275 study, we identify the dicistrovirus cricket paralysis virus 1A (CrPV-1A) protein that functions to i

276 gut symbiont) and a decrease in Aphid lethal paralysis virus and Black queen cell virus - particularl

277 The dicistrovirus Cricket Paralysis virus contains a unique dicistronic RNA genome

278 w a 40S subunit was recruited by the cricket paralysis virus intergenic region (CrPV IGR) IRES to for

279 acterized at high resolution how the Cricket Paralysis Virus Internal Ribosomal Entry Site (CrPV-IRES

280 complex formed by Nsp1, 40S, and the cricket paralysis virus internal ribosome entry site (IRES) RNA,

281 Using the cricket paralysis virus internal ribosome entry site element, co

282 ed CrPV-1A, within the dicistrovirus cricket paralysis virus that can inhibit host transcription, mod

283 us, Black queen cell virus, or Israeli acute paralysis virus, however.

284 m the intergenic region (IGR) of the Cricket Paralysis Virus.

285 at hemisection model, diaphragm function and paralysis was assessed and recovered at chronic time poi

286 ugh the effect of pifk-1 mutation on time to paralysis was considerably delayed.

287 Unilateral diaphragm paralysis was identified in 3 infants.

288 lecular mechanisms underlying rapid nematode paralysis, we conducted genetic screens in Caenorhabditi

289 Patients with extraocular muscle fibrosis or paralysis were excluded.

290 rved, and no abnormalities suggesting facial paralysis were noted.

291 onal properties following chronic injury and paralysis, which are normalized following restored muscl

292 Botulism is characterized by flaccid paralysis, which can be caused by intoxication with any

293 displayed tremor that progressed to hindlimb paralysis, which correlated with diminished numbers of m

294 Options for people with severe paralysis who have lost the ability to communicate orall

295 rare but distinct syndrome of acute flaccid paralysis with evidence of spinal motor neuron involveme

296 In addition, group 2 demonstrated paralysis with histopathologic changes of spinal cord is

297 lation using PLP-CreERT resulted in hindlimb paralysis with immobility at approximately 30 d after 4-

298 olorado (Aurora, CO, USA) with acute flaccid paralysis with spinal-cord lesions involving mainly grey

299 median survival by 50% and delayed hindlimb paralysis, with animals remaining ambulatory until the h

300 illain-Barre syndrome which causes temporary paralysis, yet the mechanism is unknown.