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1 ad less ecchymosis compared with controls in paranasal and eyelid ecchymosis at postoperative day (PO
8 and cervical vasculature, and the sellar and paranasal regions is recommended in every patient presen
9 al runoff CT angiography (-48.6%), CT of the paranasal sinus (-39.6%), cerebral or carotid CT angiogr
12 ultidetector computed tomography (CT) of the paranasal sinus by using computer simulation of the effe
16 iple orbital abscesses, and lack of adjacent paranasal sinus disease may be predictive factors that s
21 flammatory condition affecting the nasal and paranasal sinus mucosa, often accompanied by olfactory d
22 is an infection of the nasal epithelium and paranasal sinus mucosa, usually caused in children by St
23 We identified studies of nasal cavity and paranasal sinus tumours through searches of databases in
29 y invasion of adjacent structures, including paranasal sinuses (n = 10), anterior fossa (n = 7), orbi
30 ), tracheobronchial tree (n=4), colon (n=3), paranasal sinuses (n=1), and shoulder joint (n=1) were g
31 ed-release Microsphere-based-delivery to the Paranasal Sinuses (TEMPS) is developed with the corticos
32 usitis (AFS) is a devastating disease of the paranasal sinuses afflicting immunocompromised individua
33 or the presence of fluid and sediment in the paranasal sinuses and airways, mastoid air cell fluid, f
34 or correlations between anatomic variants of paranasal sinuses and chronic or recurrent sinusitis.
35 (CT) showed soft tissue lesions in the left paranasal sinuses and destruction of the left lamina pap
37 gested that the upper airways (including the paranasal sinuses and nasopharynx) play an important rol
38 ion between the degree of development of the paranasal sinuses and the growth of the cranium, standar
39 d in 252 patients (30.1%), with nasal cavity/paranasal sinuses and trachea/larynx subsites as the mos
40 ent locoregional mucosal inflammation of the paranasal sinuses and upper airway that has substantial
41 tumours arising within the nasal cavity and paranasal sinuses are rare and composed of several histo
42 he assessment of inflammatory changes in the paranasal sinuses due to different parameters of width (
43 ent underwent unenhanced CT of the brain and paranasal sinuses followed by contrast-enhanced MRI of t
44 ent underwent unenhanced CT of the brain and paranasal sinuses followed by contrast-enhanced MRI of t
46 examinations of pathological changes in the paranasal sinuses may be due to selection of unsuitable
47 flammatory condition affecting the nasal and paranasal sinuses of approximately 11.5% of the United S
52 red in 290/4155 (7.0%) patients (orbita, 31; paranasal sinuses, 93; main nasal cavity, 38; tongue, 27
54 inflammatory process in the nasal cavity and paranasal sinuses, and bacteria have been considered to
55 is inaccurate in complex regions such as the paranasal sinuses, but brain region activities in 48 reg
56 performed to look for mucosal disease of the paranasal sinuses, drainage pathways, and presence of an
57 mation and tissue remodeling of the nose and paranasal sinuses, frequently occurring with nasal polyp
58 cavity, oropharynx, larynx, hypopharynx, and paranasal sinuses, is the seventh most common cancer wor
59 to avoid missing inflammatory lesions in the paranasal sinuses, it is reasonable to use CT windows de
60 tracts, leading to chronic infections of the paranasal sinuses, middle ear, and bronchi that begin du
62 nflammatory condition affecting the nose and paranasal sinuses, posing a significant socio-economic i
63 e, with or without axial planes, through the paranasal sinuses, reconstructed in a sharp algorithm an
64 ned to demonstrate abnormal processes in the paranasal sinuses, temporal bones, base of the skull, an
65 neck region, especially in the nasal cavity, paranasal sinuses, tonsillar fossa, and oral cavity.
66 neck region, especially in the nasal cavity, paranasal sinuses, tonsillar fossa, and oral cavity.
78 m processing of bacteria in the lumen of the paranasal spaces rather than through invasion or samplin