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1 nfection and localizes to the surface of the parasite egg.
2 because host eggs hatch predictably ahead of parasite eggs.
3 d immune response against highly immunogenic parasite eggs.
4 ed and Th2-polarized immune response against parasite egg Ags.
5 ss II-restricted CD4+ Th cells sensitized to parasite egg Ags.
6     Polyacrylamide beads coated with soluble parasite egg antigens could induce ICAM-1 expression on
7                                         When parasite eggs are injected into the livers of naive anim
8 ough the granulomas that they develop around parasite eggs are small and devoid of collagen.
9 ology and cytokine production in response to parasite eggs are uneven and strain dependent.
10 d hepatic granulomatous inflammation against parasite eggs associated with a robust Th17 cell respons
11  type 2 (Th2) responses has been ascribed to parasite eggs, because exposure of the host to this life
12  decreased mast cells correlated with higher parasite egg burden and delayed clearance in vivo, T cel
13    In the intestine, Retnla(-/-)mice had low parasite egg burdens compared to those of WT mice, while
14 soni develop small hepatic granulomas around parasite eggs, but concomitant immunization with soluble
15                              Upon infection, parasite eggs can lodge inside of host organs like the l
16                  In schistosomiasis mansoni, parasite eggs cause hepatointestinal granulomatous infla
17 gh versus low parasite burdens (measured via parasite egg counts in faecal samples) were associated w
18    We used data from over two decades of GIN parasite egg counts, host space use, and spatial vegetat
19 rrelation, plant functional traits predicted parasite egg counts.
20 sing the murine host reaction to schistosome parasite eggs deposited in the liver as a model.
21 mRNA expression correlated with the onset of parasite egg deposition and granuloma formation.
22                 Current diagnostics based on parasite egg detection in stool detect infection only at
23 eas signaling in BM-derived cells suppresses parasite egg-driven inflammation within the liver and in
24 ction with the helminth Schistosoma mansoni, parasite eggs elicit a Th cell-mediated hepatic granulom
25  3,000 field-of-view (FOV) images containing parasite eggs, extracted from more than 300 fecal smear
26                                              Parasite eggs identified by microscopy included helminth
27 istosomiasis, either due to the detection of parasite eggs in stool and/or the presence of a concorda
28 uction increases following the deposition of parasite eggs in the liver.
29  and intestines resulting from deposition of parasite eggs in these organs.
30                  In schistosomiasis, chronic parasite egg-induced granuloma formation can lead to tis
31 onicum) infection in humans is attributed to parasite egg-induced granulomatous inflammation and fibr
32 de helminth Schistosoma mansoni results in a parasite egg-induced, CD4 T-cell-mediated, hepatointesti
33 patic granulomatous inflammation surrounding parasite eggs is mediated by CD4(+) T helper (Th) cells
34 ost parents, and in turn leads to mimicry in parasite eggs or chicks [1-7].
35       In infection with Schistosoma mansoni, parasite eggs precipitate an intrahepatic granulomatous
36                  In schistosomiasis mansoni, parasite eggs precipitate an intrahepatic granulomatous
37                In the low-pathology setting, parasite egg-stimulated dendritic cells (DCs) induce rob
38  cell-mediated inflammatory reaction against parasite eggs that varies greatly in magnitude both in h
39  and recruited to build a capsule around the parasite egg to block its development.
40 granulomatous inflammation that forms around parasite eggs trapped in host tissues.
41 ulomatous and fibrosing inflammation against parasite eggs varies considerably in humans and among mo