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1 ective method of biological control of plant parasite infection.
2 t an autonomous response to the challenge of parasite infection.
3 les the immune response to a tissue-invasive parasite infection.
4  with the development of Th2 immunity during parasite infection.
5 -) mice and examined their responsiveness to parasite infection.
6 immunological status of the lungs induced by parasite infection.
7 arvum via upregulation of survivin, favoring parasite infection.
8 ase in C57BL/6 mice with Leishmania mexicana parasite infection.
9 and genotypic differences not related to the parasite infection.
10 ignaling enhances immune responses following parasite infection.
11 sufficient to reproduce the major effects of parasite infection.
12 ective role in the innate immune response to parasite infection.
13 ll subset in the protection against systemic parasite infection.
14 ion, a defense mechanism against pathogen or parasite infection.
15 ation of the inflammatory response following parasite infection.
16 standing the biological processes underlying parasite infection.
17 t cell actin polymerization is necessary for parasite infection.
18  to wheeze in the presence of high-intensity parasite infection.
19  T cells has been shown to be important in a parasite infection.
20 oidan) whereas high concentrations inhibited parasite infection.
21 ad an IgE of 1000 IU/L had fungal allergy or parasite infection.
22 sure a 95% chance of protection from malaria parasite infection.
23 the central repeat region of CSP can prevent parasite infection.
24 which contrasted with enhanced resistance to parasite infection.
25  release without affecting cell viability or parasite infection.
26 haviour, can minimize the risks and costs of parasite infection.
27 r the evolution of migration to minimize the parasite infection.
28 3 in shaping host innate immune responses to parasite infection.
29 y of specific inhibitors designed to prevent parasite infection.
30 tures having the potential to synergise with parasite infection.
31 aRs influence the host's capacity to control parasite infection.
32 expressing mice were much more vulnerable to parasite infection.
33  thereby rendering populations refractory to parasite infection.
34 s coding for proteins that are important for parasite infection.
35 echanism of regulating PV development during parasite infection.
36 l initiator of the type 2 immune response to parasite infection.
37 kinetes and sporozoites (CelTOS) can inhibit parasite infection.
38  received a diagnosis of blood-stage malaria parasite infection.
39 tive immune responses that depend on ongoing parasite infection.
40 n essential early step in successful malaria parasite infection.
41 nfected host cells during the early hours of parasite infection.
42 esponding effector T cells (Teff) in chronic parasite infection.
43  features of nematodes are recognized during parasite infection.
44 sure a 95% chance of protection from malaria parasite infection.
45 way, altering macrophage polarization during parasite infection.
46 e host immune system to sense and respond to parasite infection.
47  these flies were thus highly susceptible to parasite infection.
48 n 1) that were associated significantly with parasite infection.
49 l active today and can be exploited to treat parasite infection.
50 lood stage parasite growth and clear malaria parasite infections.
51 tions, Viral Syndromes, and Blood and Tissue Parasite Infections.
52 issues of allergic conditions and helminthic parasite infections.
53 trol their overall inflammatory responses to parasite infections.
54 n important protective role in bacterial and parasite infections.
55 hway during immune induction by pathogen and parasite infections.
56 ibitors are emerging to manage microbial and parasite infections.
57  at levels theoretically sufficient to clear parasite infections.
58 ologic changes associated with hemoprotozoan parasite infections.
59 nt strategies to compensate for the costs of parasite infections.
60 tions, Viral Syndromes, and Blood and Tissue Parasite Infections.
61 allergic reactions, and host defense against parasite infections.
62 nity, and resistance to concurrent or future parasite infections.
63 use, are now under investigation for various parasite infections.
64 atory method for diagnosing gastrointestinal parasite infections.
65  the doses, will efficiently clear resistant parasite infections.
66 ty, and have wide applicability for studying parasite infections.
67 ular functions and are involved in viral and parasite infections.
68 squitoes as individuals with patent (asexual parasite) infection.
69 ssociated fatty liver disease, and viral and parasite infections(2,3).
70  been shown to confer mosquito resistance to parasite infection, a major challenge for field implemen
71 ed significantly increased susceptibility to parasite infection accompanied by increased numbers of p
72 as p38 MAPK activating kinases revealed that parasite infection activates only MKK3.
73 velopment of T helper type 2 immunity during parasite infection and allergic inflammation.
74 localization of QTL conferring resistance to parasite infection and beetle fitness may result from th
75 abnormal amphibians are associated with both parasite infection and chemical contaminants, but that t
76 gets because of their indispensable roles in parasite infection and development, especially in the pr
77            The lack of miR-155 caused robust parasite infection and diminished survival of infected m
78 ve trait loci (QTL) mapping of resistance to parasite infection and fitness traits using the red flou
79 from suppression of collateral damage during parasite infection and from reduced allergic, autoimmune
80  the hypothesis that 'pace-of-life' predicts parasite infection and host pathology.
81  consequences of these changes for recurrent parasite infection and infection-associated pathologies
82 hat type I IFN can be induced in response to parasite infection and influence the outcome of infectio
83 at TLR9 mediates the innate response to oral parasite infection and is involved in the development of
84 onal light microscopy in detecting low-grade parasite infection and offers an exceptional advantage f
85 tion but were overtly normal with respect to parasite infection and pathological responses.
86 section of human immune responses to malaria parasite infection and the evaluation of therapeutics an
87 bodies against P. vivax CSP strongly inhibit parasite infection and thus support the notion that thes
88  They were first described in the context of parasite infections and allergic processes.
89 e roles of miRNAs in hepatic fibrogenesis by parasite infections and discusses the strategies using m
90 contaminants, (ii) land use practices, (iii) parasite infection, and (iv) targeted interactions betwe
91 ppressor cells have been described in tumor, parasite infection, and severe trauma models.
92 TPLEELYPT211, was observed after the initial parasite infection, and the anti-iB-1 antibodies were no
93 e models with acute virus infection, chronic parasite infection, and tumor growth.
94 ors, limited power to detect effects on rare parasite infections, and that it was not feasible to bli
95 is granulocyte, including allergic diseases, parasite infections, and tumorigenesis.
96 ortality tolerance-higher survival following parasite infection-and in parasites adapting to microbia
97 prove child health outcomes (diarrhea, HCGI, parasite infection, anemia, growth).
98 .20; 2.29-7.11), and a history of intestinal parasite infection (AOR = 1.17; 0.55-2.49) were found to
99                                     Helminth parasite infections are associated with a battery of imm
100                                        Mixed parasite infections are common in many parts of the worl
101                                 Pathogen and parasite infections are increasingly recognized as power
102 e findings call for further investigation of parasite infection as a cause of amphibian deformities i
103  stickleback and three ecologically relevant parasite infections as a "wild" model.
104 l host responses to a non-replicating type I parasite infection associated with development of long-l
105 arbor seals (Phoca vitulina), a weak HFC for parasite infection based on 27 microsatellites strengthe
106  the expected long-term effect of climate on parasite infections but can also shift the seasonal peak
107 evamisole is commonly used to treat nematode parasite infections but therapy is limited by resistance
108 -macroglobulin responded strongly to malaria parasite infection, but displayed little or no response
109 s can reduce the negative fitness effects of parasite infection by avoiding contact with parasites or
110  IIGP is reported to constrain intracellular parasite infection by disrupting the parasitophorous vac
111 1, has been found to constrain intracellular parasite infection by disrupting their vacuole membranes
112 wheeze as an outcome measure and ascertained parasite infection by fecal examination.
113          We assessed intervention effects on parasite infections by measuring Ascaris lumbricoides, T
114            Furthermore, demonstration that a parasite infection can counteract the deleterious effect
115 nally, we find that the transmission rate of parasite infection can either decrease or increase the t
116               Intrapopulation differences in parasite infections can develop from specialist individu
117                However, we also suggest that parasite infections can drive this niche specialisation.
118                       Chronic, low-intensity parasite infections can reduce host fitness through nega
119 roles in diverse chronic diseases, including parasite infections, cancer, and allergic responses.
120  protection against controlled human malaria parasite infection (CHMI) and natural exposure.
121 ssibility by chromatin remodeling during the parasite infection cycle.
122                                              Parasite infection decreased 57 (inclusive of IL-12 and
123 lead to a simple blood test for sub-clinical parasite infection detection, reducing anthelmintic use
124 etermine whether protective immunity against parasite infection develops following repeat CHMI and th
125                                              Parasite infections do not in general protect against as
126                                              Parasite infection does not prevent allergen sensitisati
127                                      Because parasite infections elicit a similar immunologic environ
128 rmation, the exploration of the effects that parasite infections exert on populations of commensal gu
129 ch was reflected by increased trypanosomatid parasite infections following heatwave exposure.
130                   Data on confirmed malarial parasite infections from health facilities in interventi
131 est known correlation with susceptibility to parasite infection (>15 fold effect).
132                                We found that parasite infection had no impact on age-associated decli
133                                              Parasite infection has been shown to decrease grazing an
134                               Trypanosomatid parasite infections have a devastating impact on human h
135 stigated in treatments of cancer, virus, and parasite infections (i.e., malaria) as well as in crop s
136 to hemocytes to differentiate and respond to parasite infection, implicating hemocytes as critical mo
137 en-related protein 1 (FREP1) is critical for parasite infection in Anopheles gambiae and facilitates
138                 Second, the low incidence of parasite infection in industrial nations is cited as a f
139 monstrated that Wnt5a-mediated inhibition of parasite infection in macrophages is Rac1/Rho dependent.
140                           Progression of the parasite infection in the fly depends on factors inheren
141 ing 20E signaling pathways to reduce malaria parasite infection in the mosquito vector and provide ne
142 8 cells and observed consistent reduction of parasite infection in these knockdown cells.
143 _P reduces host-seeking behavior and malaria parasite infection in vector mosquitoes in ways that fur
144 demonstrated that 1294 significantly reduces parasite infection in vitro, with a half maximal effecti
145 damage during development and by controlling parasite infections in adults that can otherwise reduce
146 ombined field-derived estimates of trematode parasite infections in aquatic snails with measurements
147 Some nectar metabolites, for example, reduce parasite infections in bees [4-7].
148     The mechanisms of protective immunity to parasite infections in humans are still elusive.
149                                     However, parasite infections in insect mass-production systems ca
150                  CD8(+) CTLs protect against parasite infections in mice primarily by secreting inter
151                                      Chronic parasite infections in the liver pose a global threat to
152  reduction in the risk of mortality in mixed parasite infections, indicating that T. ovis confers het
153 ne whether total or species-specific current parasite infection is associated with a reduced risk of
154      To determine whether host resistance to parasite infection is associated with fitness costs, we
155 s of their physiology that may influence the parasite infection is essential to better understanding
156 rst, obligatory replication phase of malaria parasite infections is characterized by rapid expansion
157 n, the role of endogenous Gal-1 during acute parasite infections is uncertain.
158                       Susceptibility to many parasite infections is, to a greater or lesser extent, c
159  antibodies were not readily produced during parasite infection, it may be desirable to direct antibo
160 periodic relapses of symptomatic blood stage parasite infections likely initiated by activation of do
161 urs (through anaesthesia exposure) increased parasite infection loads in isolated hosts by 62-102% re
162 e, two such proteins that antagonize malaria parasite infections, LRIM1 and APL1C, circulate in the h
163  dataset of 934 collections (without data on parasite infection), malformation frequency was best pre
164 urce availability, and seasonal variation in parasite infections may further alter ruminant body cond
165 bin mediated) and secondary changes (Babesia parasite infection mediated) to the RBC membrane using w
166                                       During parasite infection, mice with epithelial-specific deleti
167                              High degrees of parasite infection might prevent asthma symptoms in atop
168 man PBMCs from HVL, similar to the wild type parasite infection, mimicking a naturally acquired prote
169          Among collections also examined for parasite infection (n = 154), average parasite load and
170 development of effective vaccines that block parasite infection of erythrocytes is identifying the pl
171  P. berghei we are able to robustly quantify parasite infection of hepatocyte cell lines by flow cyto
172 c cleavage of the CSP, a key requirement for parasite infection of hepatocytes.
173 tive caseinolytic protease, as important for parasite infection of host macrophages and a potential t
174 mes play distinctive biological roles during parasite infection of mammalian cells.
175 ns most of the genetic variation for malaria parasite infection of mosquitoes in nature.
176 ity have decreased substantially, malaria, a parasite infection of red blood cells, still kills rough
177 lished on mosquito midguts, greatly limiting parasite infection of salivary glands and transmission t
178  for gametocyte-expressed genes required for parasite infection of the mosquito Anopheles coluzzii.
179                                 Experimental parasite infections of advanced mosquito intercrosses de
180 onsidered for virotherapy to control certain parasite infections of man and animals.
181                                              Parasite infections often lead to dramatically different
182 e to confirm previously described effects of parasite infection on host dopamine either in vitro or i
183 the effects of rainfall and gastrointestinal parasite infections on springbok (Antidorcas marsupialis
184 ittle is known of the effects of concomitant parasite infections on the immune response or severity o
185 circulation that could predict the extent of parasite infection outside of circulation.
186 e mice were shown to protect against mucosal parasite infection (P < 0.05), demonstrating that mucosa
187                                  Elucidating parasite infection patterns of endangered species with l
188 udies and case reports suggest that internal parasite infections (PI) can cause CSU.
189 isease include microbial exposures including parasite infection, pollution, diet and obesity.
190                                Prior malaria parasite infection primed the production of anti-native
191 l and plant species.(5)(,)(6) By suppressing parasite infection, protective microbiota could reduce t
192 city of macrophages is evident in helminthic parasite infections, providing protection from inflammat
193 ell derived neurons show an ~50% decrease in parasite infection rate when compared to unstimulated cu
194  recombinant fragments of EphA2 enhanced the parasite infection rate, thus establishing its role in P
195                  Current therapies for human parasite infections rely on a few drugs, most of which h
196 ction of autophagic events during Leishmania parasite infection remain unknown.
197                   However, their role during parasite infection remains to be clarified.
198 ally, each pair of matched source/nosocomial parasite infections showed <1% of different STRs and <6.
199                                              Parasite infection significantly decreased host shoot bi
200 cing fibrinogen-related protein 30 increased parasite infection significantly, whereas ablation of fi
201 on of electron-dense band at the base of the parasite infection site.
202 er level of ITGA2 protein was present in the parasite infection sites.
203 pose an improved indicator that incorporates parasite infection status (as assessed by a rapid diagno
204 mucosal immunity to intestinal intracellular parasite infections such as Eimeria infection.
205 may not have other means of diagnosing blood parasite infections such as parasitology thick and thin
206 issions were paired with data from community parasite infection surveys.
207                     Women with >/=2 malarial parasite infections tended to have lower z scores than u
208 d be more sustainable control strategies for parasite infections than mass drug administration, while
209                                      Malaria parasite infections that are only detectable by molecula
210  available to organisms for dealing with the parasite infection, that is they migrate to a different
211  of 20-50 mg/kg given after establishment of parasite infection, the compounds reduced parasitemia in
212 an important and preferential target cell of parasite infection, the injection of ROP16 has multiple
213 es, daily fluctuations in temperature affect parasite infection, the rate of parasite development, an
214 ate their functions in mosquito survival and parasite infection, these genes were knocked down by RNA
215 odified course of infection, controlling its parasite infection to levels below detection by thick bl
216                      The ability of helminth parasite infections to manipulate the immune system of t
217 en colitis was induced in AKR/J mice using a parasite infection, Trichuris muris.
218                            Gene induction by parasite infection was associated with trimethylation of
219 sults from previous studies at local scales; parasite infection was more influential in the West and
220                                        Blood parasite infection was significantly different between p
221                                      Malaria parasite infection weakens colonization resistance again
222                         Asymptomatic malaria parasite infections were detected in 1361 of 14732 sampl
223 evalence, as well as the intensity of midgut parasite infections were found to be significantly highe
224 hrough active screening algorithms, or blood-parasite infection, which is likely to be diagnosed if t
225 er progress to either asymptomatic skin-only parasite infection, which would not be diagnosed through
226  choriomeningitis virus and Leishmania major parasite infections, which were rescued with diet supple
227 FREP1(Q) allele has been reported to prevent parasite infection, while supporting essential physiolog
228 t has a complex life cycle; however, asexual parasite infection within the blood stream is responsibl

 
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