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1 e.g., insomnia, obstructive sleep apnea, and parasomnias).
2 s a genetic predisposition to many childhood parasomnias.
3 ructive sleep apnea syndrome (OSAS) (0.06%), parasomnia (0.7%), restless leg syndrome (0.9%), or psyc
5 tic challenges between nonrapid eye movement parasomnia and nocturnal frontal lobe epilepsy remain.
6 ents during REM sleep, is a male-predominant parasomnia and the strongest prodromal marker of synucle
7 te the strong familial aggregation for the 2 parasomnias and lend support to the notion that sleepwal
8 ly, symptoms of some sleep disorders such as parasomnias and narcolepsy can be confused with those of
9 bility of a common pathogenic background for parasomnias and nocturnal seizures that is summarized in
12 ehavior disorder is distinguished from other parasomnias by clinical features and the demonstration o
15 ith isolated REM sleep behaviour disorder, a parasomnia considered to be a prodromal phenotype of a-s
18 1; anosmia, OR, 2.16; 95% CI, 1.59-2.93; and parasomnias (including RBD), OR, 1.62; 95% CI, 1.42-1.84
19 l study of a male population, probable sleep parasomnias, including both SW and RBD, were associated
22 t (REM) sleep behaviour disorder, which is a parasomnia manifested by vivid dreams associated with dr
23 ts with Parkinson disease (PD) reported that parasomnias other than rapid eye movement (REM) sleep be
24 nto the functional mechanisms of this common parasomnia: sleepwalkers exhibited improved movement aut
27 REM) sleep behaviour disorder (RBD) is a REM parasomnia that often predicts the later occurrence of a
28 azepine receptor agonist hypnotics can cause parasomnias, which in rare cases may lead to suicidal id
29 antibodies identify a unique non-REM and REM parasomnia with sleep breathing dysfunction and patholog
30 ildhood sleepwalking and sleep terrors are 2 parasomnias with a risk of serious injury for which fami