ライフサイエンスコーパス: parenchymal

1 ative compartments: ventricular, midline and parenchymal.

2 d treatment is confounded by the presence of parenchymal Abeta deposition.

3 measure, count, and describe the airway and parenchymal abnormalities in end-stage CF lungs.

4 tomographic imaging of the lungs, a range of parenchymal abnormalities were observed in the five pati

5 d may often result from initially unapparent parenchymal abnormalities.

6 CT findings were analysed based on parenchymal, airway, pleural, mediastinal, and vascular

7 cular emphysema, is characterized by loss of parenchymal alveolar tissue and impaired tissue repair.

8 ted with cortical tissue loss independent of parenchymal Alzheimer's disease pathology.

9 r amyloid, in association with low levels of parenchymal amyloid deposits.

10 APPDutch mice develop CAA in the absence of parenchymal amyloid, mimicking hereditary cerebral hemor

11 ase (AD) is characterized by the presence of parenchymal amyloid-beta (Abeta) plaques, cerebral amylo

12 ebral soluble amyloid-beta1-42, vascular and parenchymal amyloid-beta deposits, and astrocytosis (31%

13 oints largely prevents CAA in the absence of parenchymal amyloid.

14 oking and occupational endotoxin exposure to parenchymal and airway remodeling as defined by quantita

15 C (100 ug/kg/d i.p.) prevents development of parenchymal and cerebrovascular amyloid-beta (Abeta) dep

16 minepentaacetate gadolinium imaging depicted parenchymal and intraventricular inflammation.

17 mune cells, diverse haematopoietic, stromal, parenchymal and neuronal cell types can store inflammato

18 rs are specified, to E10.5 liver, when liver parenchymal and non-parenchymal cell lineages emerge.

19 ALTCs were engineered with human parenchymal and non-parenchymal liver cell lines (HepG2

20 Both parenchymal and non-parenchymal liver cells grown in ALT

21 Purpose To evaluate lung parenchymal and tracheal CT morphology before and 6 mont

22 CT scan findings were compared for cavitary, parenchymal, and non-parenchymal disorders.

23 those of patients with MDR-TB for cavitary, parenchymal, and non-parenchymal lung characteristics.

24 tomography, and use this to investigate the parenchymal architecture of unstained lung tissue from p

25 he other hand, flow/pressure decreases evoke parenchymal arteriole dilation and increased resting pyr

26 On the other hand, decreasing parenchymal arteriole tone increased resting cortical py

27 l-GCaMP3 mice, we demonstrate that increased parenchymal arteriole tone significantly increased intra

28 We showed that, in response to increases in parenchymal arteriole tone, astrocyte intracellular Ca(2

29 During increases in parenchymal arteriole tone, the pyramidal neuron respons

30 ow/pressure-evoked increases or decreases in parenchymal arteriole vascular tone, which result in art

31 responses, increases in flow/pressure within parenchymal arterioles increased the firing activity of

32 nduce rapid and robust dilations of upstream parenchymal arterioles, suggesting a key role of cECs in

33 We conclude that parenchymal astrocytes are latent neural stem cells and

34 in aged mice, compared to a 13% decrease in parenchymal blood flow, itself a leading candidate bioma

35 Encephalitis is parenchymal brain inflammation, commonly due to herpes s

36 These parenchymal brain lesions are considered key contributor

37 No parenchymal brain lesions were evident on imaging, but t

38 d microscopy, we reveal the diversity of non-parenchymal brain macrophages.

39 as taken up by brain regions and entered the parenchymal brain space.

40 y patients with brain hypoxia, as defined by parenchymal brain tissue oxygen tension less than 20 mm

41 SHV infected neurons and oligodendrocytes in parenchymal brain tissues.

42 ever, patients with XDR-TB tend to have more parenchymal calcification and left-sided plural effusion

43 Parenchymal calcification was more common in the XDR gro

44 ntially enhanced the frequency and number of parenchymal CD4+ T cells as well as both CD69 expression

45 9 and CD73 ectonucleotidases was detected on parenchymal CD4+ T cells, we investigated whether CD4+ T

46 nosine triphosphate degradation, impairs the parenchymal CD4+ T-cell response and contributes to the

47 We studied mice with liver parenchymal cell (LPC)-specific disruption of the cylind

48 two different co-culture MT systems with non-parenchymal cell (NPC) fraction sourced from different d

49 ccumulation is an inescapable consequence of parenchymal cell death has not been explored.

50 E10.5 liver, when liver parenchymal and non-parenchymal cell lineages emerge.

51 rtance of inter-donor variability of the non-parenchymal cell population in the overall governance of

52 Whereas the initial step of parenchymal cell proliferation was not affected by acute

53 Further, parenchymal cell release may have applications in other

54 Adipocytes are the primary parenchymal cell type in adipose tissue, yet these cells

55 ells, yielding molecular definitions for non-parenchymal cell types that are found in healthy and cir

56 To do so, we created mice harboring liver parenchymal cell-specific deletion of HOIP (Hoip(Deltahe

57 epithelial cells acquire a cancer-associated parenchymal-cell-like phenotype when co-cultured with ca

58 While ablating either RIPK1 or RelA in liver parenchymal cells (LPCs) did not cause spontaneous liver

59 nhibition of catalytic IKK activity in liver parenchymal cells (LPCs; IKKalpha/beta(LPC-KO) ) were in

60 stem can uniquely address the ability of CNS parenchymal cells (neurons, astrocytes, and microglia) t

61 nd cues from surrounding environment and non-parenchymal cells (NPCs).

62 ous HGD promoter was localised to only liver parenchymal cells and kidney proximal tubules in adultho

63 Our findings show for the first time that parenchymal cells are released from organs under non-pro

64 sms of cellular crosstalk between immune and parenchymal cells are still elusive.

65 ults in the generation of significantly more parenchymal cells by P28, composed mostly of ventromedia

66 n recognition receptors (PRRs) on immune and parenchymal cells can detect danger-associated molecular

67 local lipid release and a mechanism by which parenchymal cells can modulate tissue macrophage differe

68 the crosstalk between liver lymphocytes and parenchymal cells during liver regeneration after partia

69 lk between organotypic endothelial cells and parenchymal cells for identification of determinants of

70 important not only in leukocytes but also in parenchymal cells for the progression of inflammation.

71 xpressed by sinusoidal endothelial cells and parenchymal cells in the liver, respectively.

72 argeted sampling of the xylem sap and single parenchymal cells in the pith, thereby differentiating t

73 we perform single-cell RNA sequencing on non-parenchymal cells isolated from healthy and NASH mouse l

74 hat inflammatory mediators produced by renal parenchymal cells may influence the function of remote o

75 Hepatocytes, the highly metabolic parenchymal cells of the liver, are efficient at differe

76 ibutes are all performed by hepatocytes, the parenchymal cells of the liver.

77 Fusion of donor mesenchymal stem cells with parenchymal cells of the recipient can occur in the brai

78 Astrocytes are neural parenchymal cells that ubiquitously tile the central ner

79 hiPSCs) with various types of supporting non-parenchymal cells to attain a higher differentiation yie

80 lation, in vivo mRNA paracrine transfer from parenchymal cells to ECs, or cell-autonomous expression

81 range of stromal cells that co-develop with parenchymal cells to form tissues.

82 demonstrate coordination between T cells and parenchymal cells to regulate sympathetic innervation.

83 HOIP deficiency in liver parenchymal cells triggered tumorigenesis at 18 months o

84 ermore, normally beta-tanycytes give rise to parenchymal cells via an intermediate population of alph

85 art by driving the expression of TGFbeta1 in parenchymal cells via the IL-17 receptor C (IL-17RC).

86 ntrifugation into two portions, hepatocytes (parenchymal cells) and LSEC (non-parenchymal cells).

87 epatocytes (parenchymal cells) and LSEC (non-parenchymal cells).

88 se data demonstrate an unanticipated role of parenchymal cells, as shown here for hepatocytes, in tis

89 Fv was found to associate with postvascular, parenchymal cells, indicating its successful receptor-me

90 that form a barrier between blood and liver parenchymal cells, NS2(H126R) activates RNase L, which l

91 rating and resident immune cells, as well as parenchymal cells, present in nephritic kidneys.

92 linear ubiquitination specifically in liver parenchymal cells, we investigated the physiological rol

93 tained within endosomes of hematopoietic and parenchymal cells, whereupon IgG is diverted from degrad

94 We report the presence of cancer-associated parenchymal cells, which exhibit stem-cell-like features

95 e generation of lipid nutrients for adjacent parenchymal cells.

96 ciated with proteinuria and injury by kidney parenchymal cells.

97 in innate and adaptive immunity, as well as parenchymal cells.

98 to malignant cells by disadvantaging normal parenchymal cells.

99 l between the sinusoidal lumen and the liver parenchymal cells.

100 myofibroblasts (MFBs) in place of functional parenchymal cells.

101 rves distinct functions in immune and tissue parenchymal cells.

102 mitting diffuser, with no intervening viable parenchymal cells.

103 Parenchymal changes were also seen, mostly in a patchy a

104 und to be valuable parameters in determining parenchymal changes.

105 ion Radiomic phenotypes capture mammographic parenchymal complexity beyond conventional breast densit

106 rpose To identify phenotypes of mammographic parenchymal complexity by using radiomic features and to

107 lied to identify and reproduce phenotypes of parenchymal complexity in separate training (n = 1339) a

108 e identified four phenotypes with increasing parenchymal complexity that were reproducible between tr

109 Stroma is a poorly defined non-parenchymal component of virtually every organ with key

110 cterized by 2,8-dihydroxyadenine (DHA) renal parenchymal crystal deposition.

111 characterized by 2,8-dihydroxyadenine renal parenchymal crystal deposition.

112 Approximately 38% of parenchymal cysts calcify after antiparasitic treatment.

113 with calcification and focal gliosis, renal parenchymal damage and liver lobular inflammation depend

114 The parenchymal damage seems to be induced by placing suture

115 ure to reduce ischemic cerebral vascular and parenchymal damages.

116 The t-test was used to compare parenchymal densities and renal pelvic diameter differen

117 to investigate the diagnostic value of renal parenchymal density differences in distinguishing betwee

118 Parenchymal density values (HU) and renal pelvic anterio

119 PP colocalized with amyloid plaques in brain parenchymal deposits, suggesting that these peptides may

120 ation of airways disease (bronchiolitis) and parenchymal destruction (emphysema), whose relative prop

121 iability in the severity and distribution of parenchymal destruction throughout the lungs.

122 mphysema with severe hyperinflation and less parenchymal destruction.

123 tional small airways disease (PRM(fSAD)) and parenchymal disease (PRM(PD)) were compared between bila

124 diopathic pulmonary fibrosis (IPF) is a lung parenchymal disease of unknown cause usually occurring i

125 component of the pathophysiology of diffuse parenchymal diseases including idiopathic pulmonary fibr

126 ed to treat a broad range of airway and lung parenchymal diseases, such as asthma, emphysema, and chr

127 compared for cavitary, parenchymal, and non-parenchymal disorders.

128 ng and ex vivo autoradiography revealed more parenchymal distribution of Bapi-TXB2 compared with Bapi

129 t grade per patient), the mean difference in parenchymal dose (Gy) per step increase in CTCAE grade c

130 tumor-absorbed dose of more than 90 Gy and a parenchymal dose of less than 55 Gy.

131 a positive association between toxicity and parenchymal dose was found.

132 ese mice also showed significantly decreased parenchymal EB levels.

133 ial fibrosis are associated with lower renal parenchymal elasticity.

134 ells within skeletal muscle, that regenerate parenchymal elements following damage.

135 Conclusion The pattern of parenchymal emphysema at baseline CT was an independent

136 = 80, 18.3%), moderate or marked background parenchymal enhancement (BPE) (n = 91, 20.9%), posttreat

137 BackgroundThe higher level of background parenchymal enhancement (BPE) at breast MRI has the pote

138 Purpose To assess the extent of background parenchymal enhancement (BPE) at contrast material-enhan

139 utomated quantitative measures of background parenchymal enhancement (BPE) derived from an early vers

140 Purpose To evaluate the effect of background parenchymal enhancement (BPE) on breast magnetic resonan

141 from diffusion-weighted imaging), background parenchymal enhancement (BPE), and amount of fibroglandu

142 tion) and normal tissue features (background parenchymal enhancement [BPE] volume, mean BPE) were qua

143 I revealed immediate and sizable hippocampal parenchymal enhancement indicating BBB opening, followed

144 risk.ConclusionA higher level of background parenchymal enhancement measured at breast MRI is associ

145 ositivity, fibroglandular volume, background parenchymal enhancement, and being mass or non-mass were

146 on deciphering regenerative cells within the parenchymal epithelium, cell lineages in the stroma that

147 On human lung parenchymal explants, chloroquine concentration clinical

148 transport; (ii) transport of dextrans in the parenchymal extracellular space, measured by 2-photon fl

149 sue samples corresponding to eight different parenchymal feature classes from 208 CT scans.

150 et, and skewness and power law beta from the parenchymal feature set-were selected more than 50% of t

151 nd behavioral impairment, but never develops parenchymal fibrillar amyloid deposits.

152 Histologic airway and parenchymal fibrosis were semiquantitatively graded in a

153 Parenchymal findings of the lung also included fewer and

154 ry endpoint, PC, was defined by new abnormal parenchymal findings on chest imaging in the setting of

155 intestinal or neurologic) symptoms, had lung parenchymal findings suspicious for COVID-19 at non-ches

156 strated increasing heterogeneity of regional parenchymal flow with increasing lung size, with decreas

157 ells ameliorated fibrosis and restored renal parenchymal function and metabolic homeostasis.

158 ide eliciting dilation and preserving normal parenchymal function by inhibiting inflammation and prol

159 od-brain barrier compromise was suggested by parenchymal gadolinium enhancement, leukocyte recruitmen

160 damage and scarring of intraventricular and parenchymal (glia-lymphatic) CSF pathways.

161 g of POMC neurons was reversed ex vivo or by parenchymal glucose administration.

162 the systemic circulation, resolution of the parenchymal "ground glass" opacity and absence of furthe

163 s accumulate lipids before the major wave of parenchymal growth.

164 Mortality at 90 days and risk of parenchymal haematoma and symptomatic intracranial haemo

165 cluded patients aged 18 years and older with parenchymal haemorrhage on the first CT scan.

166 cteplase, 7; alteplase, 2; P=0.018) and less parenchymal hematoma (2 of 75 versus 10 of 71; P=0.02).

167 Parenchymal hematoma (PH) following intravenous thrombol

168 67-1.97; RD, 0.00; 95% CI, -0.02 to 0.03) or parenchymal hematoma (RR, 1.18; 95% CI, 0.71-1.94; RD, 0

169 ause mortality, intracranial hemorrhage, and parenchymal hematoma at 90 days were also assessed.

170 Parenchymal hematoma triggers a series of reactions lead

171 %) and 2 treatment patients (7%) developed a parenchymal hematoma type 2 (P > .99).

172 t growth at 24 hours and occurrence of large parenchymal hematoma.

173 No sICH or parenchymal hematomas occurred.

174 Mild chronic parenchymal hemorrhage was noted in 3 left superior PV l

175 tients had hemorrhagic infarction and 15 had parenchymal hemorrhage.

176 matic intracranial hemorrhage (1% vs 4%) and parenchymal hemorrhages type 1 (1% vs 3%) or type 2 (1%

177 We discovered high SYK expression in the parenchymal hepatocyte, hepatic stellate cell (HSC), and

178 ments were evaluated visually before PEA for parenchymal hypoperfused segments.

179 : six ambulatory neurosurgical patients with parenchymal ICP-sensors and four former cancer patients

180 (-/) (-) mice, marked by airway and adjacent parenchymal immune cell infiltration and mucus productio

181 y allergic immune response, whereas the lung parenchymal immune system has been largely neglected.

182 perivascular predominance as well as diffuse parenchymal infiltration (14/14), present in meninges, w

183 de lung involvement in the form of airway or parenchymal inflammation and fibrosis.

184 ve oxygen species mediates both dilation and parenchymal inflammation leading to cellular dysfunction

185 trauma research has historically focused on parenchymal injuries and the risk of bleeding.

186 ytic anaemia, even when subclinical, lead to parenchymal injury and chronic organ damage, causing sub

187 develop persistent pathological responses to parenchymal injury or stress.

188 R3(injury) displayed increased expression of parenchymal injury transcripts (eg, hypoxia-inducible fa

189 SIRT(oe) mice showed exacerbated parenchymal injury whereas SIRT(hep-/-) mice evidenced a

190 nal model reproduced this behavior only when parenchymal interdependence between neighboring acini wa

191 reviewed to identify the frequency of liver parenchymal invasion (LPI) from perihepatic peritoneal m

192 assign a CT severity score for the degree of parenchymal involvement per lobe.

193 nd poor oxygenation despite relatively minor parenchymal involvement.

194 transporter ferroportin (Fpn), resulting in parenchymal iron overload.

195 group of genetic disorders characterized by parenchymal iron overload.

196 Retained gadolinium colocalized with parenchymal iron.

197 FD also resulted in immediate enhancement of parenchymal labeling with the fluorescent dye Hoechst 33

198 yndromes (both acute and progressive), brain parenchymal lesions seen on neuroimaging and a set of di

199 re engineered with human parenchymal and non-parenchymal liver cell lines (HepG2 and LX2 cells, respe

200 te hepatic organoids that comprise different parenchymal liver cell types and have structural feature

201 Both parenchymal and non-parenchymal liver cells grown in ALTCs exhibited markedl

202 tive targeting and specific drug delivery to parenchymal liver cells is a promising strategy to treat

203 th MDR-TB for cavitary, parenchymal, and non-parenchymal lung characteristics.

204 O has a complementary effect on vascular and parenchymal lung development.

205 estigated, the mechanisms causing airway and parenchymal lung disease are not well defined.

206 ugal, and the UK) evaluated cases of diffuse parenchymal lung disease in a two-stage process between

207 ic pulmonary fibrosis (IPF) is a progressive parenchymal lung disease of complex cause.

208 Diffuse parenchymal lung disease represents a diverse and challe

209 team agreement for the diagnosis of diffuse parenchymal lung disease.

210 tions, and transbronchial biopsy to diagnose parenchymal lung disorders.

211 Anti-IL-17A modestly decreased airway and parenchymal lung fibrosis, along with a striking reducti

212 nt (HCT) manifests as progressive airway and parenchymal lung fibrosis.

213 Influenza induced persistent parenchymal lung inflammation, alveolar epithelial metap

214 Findings outside the parenchymal lung, including mediastinal lymphadenopathy

215 e in the lungs under the instruction of lung parenchymal lymphocytes.

216 Microglia are parenchymal macrophages of the CNS; as professional phag

217 mal macrophages, known as microglia, and non-parenchymal macrophages, collectively termed border-asso

218 The central nervous system hosts parenchymal macrophages, known as microglia, and non-par

219 luding detailed pathological features of the parenchymal margin) with recurrence after surgical resec

220 ventional noncontrast CT provides airway and parenchymal measurements but cannot be used to directly

221 uted through the lungs according to regional parenchymal mechanics.

222 sly in the same pancreas were actually intra-parenchymal metastases, not independent primary tumors.

223 is characterized by changes in the pial and parenchymal microcirculations.

224 that provide a deeper understanding of both parenchymal microglia and extraparenchymal brain macroph

225 a diverse myeloid compartment that includes parenchymal microglia and perivascular macrophages, as w

226 While the roles of parenchymal microglia in brain homeostasis and disease a

227 ured at treatment day 7 in 118 patients with parenchymal NCC enrolled in a treatment trial.

228 Data from 220 patients with parenchymal NCC from three randomized trials of antipara

229 the proportion of residual calcification in parenchymal NCC, and defined risk factors associated wit

230 For parenchymal NCC, the new criteria had a sensitivity of 8

231 ased antiparasitic efficacy in patients with parenchymal NCC.

232 tic fluid along with a limited area of intra-parenchymal necrosis, indicating necrotizing pancreatiti

233 er demonstrated normal portal tract, with no parenchymal necrosis, inflammation, fibrosis, or other p

234 lanchnic vein thrombosis and pancreatic head parenchymal necrosis.

235 alanine aminotransferase) and areas of liver parenchymal necrosis.

236 cacy of albendazole therapy in patients with parenchymal neurocysticercosis (NCC) is suboptimal.

237 are involved in the still high prevalence of parenchymal neurocysticercosis and ocular cysticercosis

238 Portal fibrosis, bridging, parenchymal nodules, portal inflammation, hepatocellular

239 lung, ground-glass opacification, and dense parenchymal opacification were 23.5 +/- 16.7%, 36.3 +/-

240 is that they consider parasite location (ie, parenchymal or extraparenchymal), which is an important

241 (POPH) may occur as a consequence of hepatic parenchymal or vascular abnormalities.

242 notyping of immune cells circulating between parenchymal organs and draining lymph nodes; injection o

243 f periepithelial infiltrates in exocrine and parenchymal organs or resulting from immunocomplex depos

244 Thus, we identify an immune-parenchymal pair in the murine heart that enables transf

245 lationships among pulmonary ossification and parenchymal patterns, clinical parameters, and multidisc

246 breast parenchyma to assess the mammographic parenchymal patterns.

247 c resonance-guided focused ultrasound allows parenchymal penetration of gadobutrol contrast, creating

248 Catheter navigation times, renal parenchymal perfusion, and renal artery flow rates were

249 phocreatine concentrations, as well as brain parenchymal pH, were normal.

250 creas, and aorta were recorded on pancreatic parenchymal phase (PPP) dual-energy CT 70-keV, 52-keV, a

251 scular drainage pathways, resulting in fewer parenchymal plaques but more CAA because of loss of CLU

252 ate its ability to penetrate brain and label parenchymal plaques in transgenic mice.

253 humans, and can be distinguished from other parenchymal populations, including mature BECs, by disti

254 interaction with Tregs via other immune and parenchymal populations.

255 producible technique that enables anatomical parenchymal preserving liver resections for selected cen

256 nervous system, tegument, oesophageal gland, parenchymal/primordial gut cells, and stem cells.

257 Nearly all parenchymal progenitors at P4 are Sox2(+)Olig2(+), but b

258 e JCI, Sevillano and authors determined that parenchymal PrPSc plaques of the mouse brain preferentia

259 nning with lissencephalic aspect to moderate parenchymal rarefaction, severe to mild ventriculomegaly

260 ial cells (BECs), playing important roles in parenchymal regeneration.

261 luate the contribution of non-hepatocytes to parenchymal regeneration.

262 inflammation and subsequent airway and lung parenchymal remodelling and fibrosis.

263 as endotoxins and bile acids, might mediate parenchymal renal injury in patients with cirrhosis, sug

264 hnique was applied since 1991 for anatomical parenchymal resections including central hepatectomy (re

265 roduce the S1P egress signal, whereas thymic parenchymal S1P levels are kept low through S1P lyase (S

266 Parenchymal segments supplied by segmental arteries with

267 Parenchymal sequelae included fibrosis with architectura

268 dy-state pulmonary rEos are IL-5-independent parenchymal Siglec-FintCD62L+CD101lo cells with a ring-s

269 Background Brain MRI parenchymal signal abnormalities have been associated wi

270 These results identify parenchymal SIRP-alpha as an independent driver of IR-me

271 use model of AD, plaques fail to form in the parenchymal space following microglial depletion, except

272 Parenchymal stellate cells are the primary contributors

273 These results suggests that non-neurogenic, parenchymal structural plasticity might be more importan

274 NOX4 is necessary to maintain parenchymal structures, increase cell-cell and cell-to-m

275 applied clinically, such as calcifications, parenchymal T1 signal changes, focal or diffuse gland at

276 not readily distinguish between normal (ie, parenchymal tau) and pathological tau species and showed

277 alopathy, but did not label nonpathological, parenchymal tau.

278 used to quantify breast density and extract parenchymal texture features in a cross-sectional sample

279 Previous studies have suggested that breast parenchymal texture features may reflect the biologic ri

280 tperformed that of B-mode parameters such as parenchymal thickness (AUC, 0.64; 95% CI: 0.51, 0.77; P

281 ive B-mode findings such as renal length and parenchymal thickness.

282 nderdevelopment, ranging from major cerebral parenchymal thinning with lissencephalic aspect to moder

283 days before such T cells can enter the lung parenchymal tissue and airway.

284 Although kidney parenchymal tissue can be generated in vitro, reconstruc

285 regarding the cellular origin of human liver parenchymal tissue generation during embryonic developme

286 the lung, unable to populate either the lung parenchymal tissue or the airway under homeostatic condi

287 nsported across the capillary endothelium to parenchymal tissues.

288 f antigen-responsive T cells across all lung parenchymal tissues.

289 is a 2-stage hepatectomy, which incorporates parenchymal transection at stage 1 enabling resection of

290 conferred an ischemic stress response within parenchymal tumor cells, with ROS triggering the evoluti

291 oblastoma is the most common malignant brain parenchymal tumor yet remains challenging to treat.

292 Optimal grading and treatment of pineal parenchymal tumours of intermediate differentiation (PPT

293 Affected individuals present with cerebral parenchymal underdevelopment, ranging from major cerebra

294 he contralateral (tumor-free) breast: breast parenchymal uptake (BPU) (from (18)F-FDG PET), mean appa

295 aptic or spiking responses around individual parenchymal vessels in cats and established that the vas

296 responses for the first time and found that parenchymal vessels in cortical layer 2/3 were orientati

297 mal volume did not improve the equation over parenchymal volume alone.

298 Clinical characteristics combined with parenchymal volume did not improve the equation over par

299 d as the true values for cortical volume and parenchymal volume, respectively.

300 y variables including unenhanced CT-measured parenchymal volume.