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1 (larynx, trachea and bronchus) and the lung parenchymal tissue.
2 pes, without substantial delivery into brain parenchymal tissue.
3 f antigen-responsive T cells across all lung parenchymal tissues.
4 matory cytokines systemically and within the parenchymal tissues.
5 d found predominantly in the bone marrow; in parenchymal tissues.
6 ion leading to migration of lymphocytes into parenchymal tissues.
7 nsported across the capillary endothelium to parenchymal tissues.
8 red mainly by their counterparts residing in parenchymal tissues.
9 atched, paired NSCLC tumor and non-malignant parenchymal tissues.
10 ed a marked accumulation of iron in affected parenchymal tissues, a finding consistent with early wor
12 pillary beds, efferent vein, and surrounding parenchymal tissue are explanted and maintained for 24 h
13 FN-gamma expression in cognate Ag-expressing parenchymal tissues as well as death via a mechanism tha
14 ve been implicated in the repair of inflamed parenchymal tissue, but the signals that regulate their
16 Clara cell of the airway protect surrounding parenchymal tissue by inducing apoptosis of Fas(+) immun
20 reased apoptosis of lymphoid organs and some parenchymal tissues contributes to this immune suppressi
24 apid response mechanism that is operative in parenchymal tissues for effective defense against tissue
26 regarding the cellular origin of human liver parenchymal tissue generation during embryonic developme
27 etabolic rate of the vessel wall relative to parenchymal tissue in the rat mesentery suggests that in
28 or restore expression of B7-H1 expression by parenchymal tissues in the recipient could prevent or am
29 lobase message was present in multiple human parenchymal tissues including heart, skeletal muscle, pl
30 way hyperresponsiveness to methacholine, and parenchymal tissue inflammation were also dramatically r
34 In chronic disease, failure to regenerate parenchymal tissue leads to the replacement of lost cell
37 ant tumors could "reawaken" after changes in parenchymal tissue mechanical properties, as may arise d
38 ng enzyme (ACE) in pulmonary artery and lung parenchymal tissues (obtained at the time of resection f
39 ome fibroblasts in various organs, including parenchymal tissue of the gut, lung, skin, and liver.
40 ted KT inhibitor associated with the gut and parenchymal tissues of the infective juvenile stage of F
41 the lung, unable to populate either the lung parenchymal tissue or the airway under homeostatic condi
42 lorative single-center clinical trial, renal parenchymal tissue perfusion of 32 stable kidney allogra
43 8(+) effector cells, and PD-L1 expression on parenchymal tissues protected against effector OT-I T ce
44 histologic evidence of ongoing vascular and parenchymal tissue remodeling, including interstitial fi
46 (TNF) superfamily is a conserved response of parenchymal tissues to injury and inflammation that comm
47 ve immunity, dendritic cells (DCs) move from parenchymal tissues to lymphoid organs by migrating alon
48 ater (H(2)(17)O), which has direct access to parenchymal tissues via aquaporin-4 water channels, exhi
49 recipient vs donor) and localization (eg, in parenchymal tissue vs lymphoid organs) of these response
50 origin was from the vasculature rather than parenchymal tissue, while interindividual variability in
51 s represents the uncontrolled replacement of parenchymal tissue with extracellular matrix (ECM) produ