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1 d by an inhibitor of aldehyde dehydrogenase (pargyline).
2 n vitro in the presence of the MAO inhibitor pargyline.
3 inhibitors, tranylcypromine, clorgyline, and pargyline.
4  monoamine oxidase B inhibitors deprenyl and pargyline.
5 t the enzymatic level with the MAO inhibitor pargyline.
6                            Local infusion of pargyline (100 or 300 microm) into the mPFC or striatum
7   The brains of four adult cats treated with pargyline (a nonhydrazide monoaminoxidase inhibitor) wer
8                       Pragmatic insertion of pargyline, a LSD1 inhibitor, as a surface recognition pa
9                                Combined with pargyline, a monoamine oxidase inhibitor, reserpine incr
10                             However, neither pargyline, a specific monoamine oxidase inhibitor, nor s
11                       Three TEMPO-conjugated pargyline analogues (ParSL-1, ParSL-2, and ParSL-3) have
12               Coincubation of the cells with pargyline and 5-hydroxytryptamine further reduced the re
13      Accordingly, monoamine oxidase blockers pargyline and l-deprenyl had no effect on DAcyt levels i
14 tes of interaction for the enzyme inhibitors pargyline and lazabemide (Ro 19-6327).
15 take inhibitor imipramine, the MAO inhibitor pargyline and the MEK inhibitor PD 98059.
16 aperitoneal injection of the LSD1 inhibitors pargyline and tranylcypromine led to dramatically reduce
17 xpression abolished the inhibitory effect of pargyline and tranylcypromine on neural progenitor proli
18                             The injection of pargyline and tryptophan increased 5-HT immunostaining c
19  reboxetine, the monoamine oxidase inhibitor pargyline, and the atypical antidepressant bupropion, ev
20 nged following a dose of the MAO B inhibitor pargyline at 2mg/kg.
21                                            A pargyline based nitroxide spin labeled irreversible inhi
22 drugs, diazepam, haloperidol, phenobarbital, pargyline, D-amphetamine, imipramine, piracetam or N-met
23 ntidepressant MAO acetylenic inhibitors like pargyline do not inhibit LSD1.
24 ized NEM in clorgyline-inactivated MAO A and pargyline-inactivated MAO B, respectively.
25 " than that of MAO B and that clorgyline and pargyline inactivation of MAO A and B, respectively, inc
26                  We found that 9a, bearing a pargyline moiety, is also able to selectively inhibit MA
27       Inhibition of MAO activity with either pargyline or di-deuterium substitution did not significa
28 ntral nervous system is the direct result of pargyline or if it occurs indirectly as a result of inhi
29 ne (MAO) inhibitors, l-deprenyl, clorgyline, pargyline, or in vivo nialamide produced 30-50% inhibiti
30 linkers to the para or meta positions of the pargyline phenyl ring, respectively.
31 he linkers connecting the TEMPO group to the pargyline phenyl ring.