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1 13; 95% CI, 0.05 to 0.21; P<0.001), and left parietal (1.12 vs. 1.01; adjusted mean difference, 0.12;
2 l and parietal regions, only medial superior parietal activity encoded past and current sensory infor
3 found that although increases in frontal and parietal activity were associated with increases in both
4 analyses of late learning stages showed left parietal activity within a broad bilateral dorsal fronto
5 tudies suggest a causal relationship between parietal alpha and suppression of the representation of
6           However, there is no evidence that parietal alpha controls auditory spatial attention.
7 pose that combined with established roles of parietal alpha in the encoding of sensory information fr
8 vation of the default mode network (DMN) and parietal alpha networks.
9  roles for prefrontal theta oscillations and parietal alpha oscillations in the control of internally
10 sual spotlight of attention." By contrast, a parietal alpha source was modulated by attentional effor
11 ic Helicobacter pylori (H pylori) infection, parietal and chief cell atrophy in the gastric corpus, a
12 ar changes in integration between the fronto-parietal and default mode systems, and integration with
13                        The network formed by parietal and frontal areas lies at the core of cognitive
14 el of dynamics, akin to a physics engine, in parietal and frontal cortex.
15 alami, corpus callosum, occipital, temporal, parietal and frontal lobes, and right hippocampus (p < 0
16 ereas in subjects with higher baseline SUVr, parietal and frontal regions were increasingly affected.
17 lection network" (CRN) centered on posterior parietal and medial temporal lobe regions, but the tempo
18 nsory (predominantly from area 2), posterior parietal and motor cortex, which could provide the subst
19 ine in two clusters involving left temporal, parietal and occipital regions (32, and 18 voxels, CCLAV
20 ne the brain activity in the frontal, motor, parietal and occipital regions, aiming to better underst
21 r functional integration, between the fronto-parietal and other functional systems.
22 aintained through representations in visual, parietal and posterior frontal brain regions, whereas de
23 ent activity is distributed across temporal, parietal and prefrontal cortices, which sequentially gen
24 ty-defined 3D shape includes, in addition to parietal and premotor areas, three clearly distinct regi
25 ttention-related frontoparietal areas (i.e., parietal and premotor cortex).
26 l attention networks, and between the fronto-parietal and somatomotor networks with cingulo-opercular
27 (DMN); specifically, between the DMN, fronto-parietal and somatomotor networks, the DMN and visual, l
28 ments showed confounding effects in frontal, parietal and striatal areas.
29 ordering the Sylvian fissure of the frontal, parietal and temporal lobes.
30 stinct neural substrates: a dorsal (occipito-parietal) and a ventral (occipito-temporal) route, respe
31 cator of mitochondrial function, in frontal, parietal, and cerebellar regions of post-mortem human br
32 thicknesses in the lateral temporal, lateral parietal, and frontal regions were inversely correlated
33 nk multimodal areas of the temporal, lateral parietal, and inferior frontal cortices, including tract
34 functional alterations of striatal, frontal, parietal, and limbic regions.
35 igh-order associative areas of the temporal, parietal, and occipital lobes.
36 cific MD patches distributed across frontal, parietal, and occipitotemporal cortex.
37  prefrontal, dorsomedial prefrontal, lateral parietal, and posterior temporal cortices).
38 e left and right temporo-occipital, inferior parietal, and right insular cortex that were distinctive
39 , the ACC is interconnected with prefrontal, parietal, and subcortical regions involved in valuation
40           We defined left and right frontal, parietal, and temporal areas as seeds (or regions of int
41 s, in addition to scattered lateral frontal, parietal, and temporal areas.
42 t the white/gray matter junction in frontal, parietal, and temporal brain regions, a typical localiza
43  of projections from frontal motor, inferior parietal, and ventrolateral prefrontal hand-related area
44 [V4], lateral intraparietal [LIP], posterior parietal area 7A, frontal eye field [FEF], and prefronta
45 ortex on the medial wall, and from posterior parietal areas 5L and 7b.
46 CS reduced EEG interhemispheric coherence in parietal areas and affected the phasic EMG correlation b
47 o control of action and inhibition, and meso-parietal areas associated with minimally conscious and c
48 interconnected network, receiving input from parietal areas implicated in 3D-structure processing.SIG
49  inferotemporal cortex, receiving input from parietal areas in the dorsal stream.
50  number of cortical areas including anterior parietal areas, from primary motor cortex (M1), premotor
51 tantly, no increased activation was found in parietal areas, nor in PFC, whereas microstimulation in
52 ned than untrained sequences in premotor and parietal areas, without any evidence of learning-related
53 ll as subcentral and more posterior temporal-parietal areas.
54 ssociation of these remains with exceptional parietal art.
55 , the ventral posterior midline, and lateral parietal association cortex.
56 , while VWFA connectivity with dorsal fronto-parietal attention network predicted visuo-spatial atten
57 enting is associated with activity in fronto-parietal brain areas that play a pivotal role in oculomo
58 r and white matter integrity within a fronto-parietal brain network underlying executive function.
59 h targets could be decoded from premotor and parietal but not motor cortical activity during movement
60 of B cells stimulates the production of anti-parietal cell antibodies, the serological hallmark of AI
61     The prevalence of positive serum gastric parietal cell antibody (PCA) was 61.8%.
62 r proliferation and increases acid-secreting parietal cells (PCs) in mice and organoids.
63                    The findings suggest that parietal cells involved in oculomotor decisions show unc
64  characterized by the destruction of gastric parietal cells, leading to the loss of intrinsic factor
65 ic mucus layer, and increased vacuolation of parietal cells.
66 f the hydrogen/potassium pump in the gastric parietal cells.
67  left temporo-occipital and bilateral medial parietal cluster, the inhibit-generate component was mai
68 ons are implemented by a distributed, fronto-parietal cognitive control network in the brain.
69 no significant impact on the dynamics of the parietal community, which already exhibited increased fl
70  in amygdala-frontal connectivity and insula-parietal connectivity were associated with larger PTSD s
71  V1 and V2, and long-range feedback occipito-parietal connectivity.
72 ynamic coupling or de-coupling of the fronto-parietal control network adjusted to cognitive effort.
73 om other somatosensory areas of the anterior parietal cortex (areas 1, 3b, and 3a), the second somato
74        We recorded the activity of posterior parietal cortex (including lateral intraparietal area LI
75 cohort of patients (n = 50), from the medial parietal cortex (MPC) and the medial temporal lobe (MTL)
76                                 Human medial parietal cortex (MPC) is implicated in multiple cognitiv
77                                    Posterior parietal cortex (PPC) activity correlates with monkeys'
78 om fMRI studies emerged, including posterior parietal cortex (PPC) and hippocampus.
79                         Neurons in posterior parietal cortex (PPC) control several effectors (e.g., e
80 s (RMS) on the connectivity of the posterior parietal cortex (PPC) in adolescent male mice.
81 y evaluated the influence of right posterior parietal cortex (PPC) on a direct measure of visual proc
82                                The posterior parietal cortex (PPC) performs many functions, including
83 nterfering with left but not right posterior parietal cortex (PPC) using high-definition cathodal tra
84 l connections between PIVC and the posterior parietal cortex (PPC), a major brain region of the corti
85 3PNs from macaque monkey DLPFC and posterior parietal cortex (PPC), two key nodes in the cortical wor
86 er-order sensory areas such as the posterior parietal cortex (PPC).
87 al experiment, we hypothesized that inferior parietal cortex (specifically supramarginal gyrus [SMG])
88                          The ventral lateral parietal cortex (VLPC) shows robust activation during ep
89 een linked to alpha oscillations in occipito-parietal cortex [3,11].
90                                     Although parietal cortex activity is thought to represent accumul
91 citatory projections from auditory cortex to parietal cortex and found this was sufficient to increas
92 f visual salience, we reversibly inactivated parietal cortex and simultaneously recorded salience sig
93 howed increased connectivity in the superior parietal cortex and striatum in their global network.
94  the layer IV and V pyramidal neurons of the parietal cortex and the CA1 basilar tree of the hippocam
95 al cortex in carnivores, where the posterior parietal cortex and the central temporal region (PMLS) p
96 is linked to altered activity in the lateral parietal cortex and the connectivity between the hippoca
97          Evolutionary adaptations of temporo-parietal cortex are considered to be a critical speciali
98                                              Parietal cortex compensates for this by updating reach g
99 n PFC, whereas microstimulation in posterior parietal cortex did activate the ITC.
100                             We recorded from parietal cortex during flexible switching between catego
101 nto oscillatory bursts in bilateral inferior parietal cortex during multiple-object processing.
102 plicating PMLS as a potential gateway to the parietal cortex for dorsal stream processing.
103 rrences of alpha-oscillatory burst events in parietal cortex for processing objects versus ensembles
104    These results support the general role of parietal cortex for the integration of visuospatial pert
105 sly rewarded stimuli in posterior visual and parietal cortex from ~260 ms after stimulus onset.
106                               Stimulation to parietal cortex had no significant impact on the dynamic
107 icipants' peak of activation within the left parietal cortex impaired their ability to generalize lea
108 rm calcium imaging recordings from posterior parietal cortex in mice (Mus musculus), we show that dri
109 right dorsolateral prefrontal cortex and the parietal cortex in non-relapsers.
110 se observations demonstrate a causal role of parietal cortex in regulating salience signals within th
111       Direct pharmacological inactivation of parietal cortex increased minimum integration times, sug
112                   Here, we asked whether the parietal cortex integrates acoustic features from audito
113                          The right posterior parietal cortex is part of a broad cortical network invo
114                              Inactivation of parietal cortex not only caused pronounced and selective
115 tory and inhibitory neurons in the posterior parietal cortex of mice judging multisensory stimuli.
116 r, our findings provide causal evidence that parietal cortex plays a role in the network that integra
117 ere focused on regions of the prefrontal and parietal cortex potentially implicated in delusion proce
118 s were revealed within the control posterior parietal cortex region.
119 t activity was followed by similar posterior parietal cortex spectral power increase that decreased i
120            The study of the evolution of the parietal cortex supplies an interesting case-study in wh
121 onnectivity between regions in occipital and parietal cortex supported enhanced decoding of the curre
122       The neural data suggest that posterior parietal cortex supports serial learning by representing
123 y suggesting enhanced sensitivity of temporo-parietal cortex to positive emotional stimuli at this st
124  interrupted the gait cycle, while posterior parietal cortex tracked obstacle location for planning f
125 networks, whereby prefrontal regions inhibit parietal cortex under internal implicit control.SIGNIFIC
126 suppression) is triggered and in the lateral parietal cortex when control is exerted, with the latter
127            Our work focuses on the posterior parietal cortex, a brain region supporting short-term me
128  bundles innervating posterior cingulate and parietal cortex, basal ganglia, and temporal cortex.
129   The dorsal circuit, that includes inferior parietal cortex, dorsal lateral prefrontal cortex, and t
130 ns of several white matter tracts in temporo-parietal cortex, including the middle and superior longi
131 ith FOG had less activation of the posterior parietal cortex, less deactivation of the dorsolateral p
132           By contrast, carbachol delivery to parietal cortex, or noradrenaline delivery to either pre
133  prefrontal cortex and alpha oscillations in parietal cortex, respectively.
134 ems to elicit more activation in the temporo-parietal cortex, thereby suggesting enhanced sensitivity
135                          In vivo, in the rat parietal cortex, these electrodes could detect brain NO
136                                       Within parietal cortex, this decreased branching was most evide
137 movement signals were strongest in posterior parietal cortex, where gradients of single-feature repre
138 ontal cortex, precentral gyrus, and inferior parietal cortex, whereas activation was higher in alcoho
139 ted how delay-period activity in frontal and parietal cortex, which is known to correlate with the de
140 io of alpha power over the left versus right parietal cortex.
141 error magnitude and past errors in posterior parietal cortex.
142 ated activity, and motor-related activity in parietal cortex.
143 ported by parahippocampal cortex and lateral parietal cortex.
144 mplars in inferior frontal gyrus and lateral parietal cortex.
145 ar to recent observations in hippocampus and parietal cortex.
146 out the dorsal visual pathway into posterior parietal cortex.
147 e tract strength between parahippocampus and parietal cortex.
148 four cell classes across primate frontal and parietal cortex.
149  also found in frontal eye fields and dorsal parietal cortex.
150 oss-regional dynamics in the hippocampus and parietal cortex.
151 isinhibition of the homotopic left posterior parietal cortex.
152 al selection throughout posterior visual and parietal cortex.
153 ralized alpha (8-14 Hz) oscillatory power in parietal cortex: alpha increases in the hemisphere ipsil
154 nt girls was associated with altered frontal/parietal cortical morphology.
155 cluding code, depend on a distinctive fronto-parietal cortical network.
156                              Lower bilateral parietal cortical thickness, greater left ventrolateral
157  affect action-goal encoding in premotor and parietal cortices and if they bias subsequent free choic
158 nhanced neural activity over dorsal occipito-parietal cortices for pseudowords, when compared to irre
159 rk includes both cortical (e.g., frontal and parietal cortices) and subcortical (e.g., the superior c
160  is female biased in prefrontal and superior parietal cortices, and male biased in ventral occipitote
161       The parahippocampal, retrosplenial and parietal cortices, as well as the hippocampal formation
162 r cingulate/paracingulate gyri, and inferior parietal cortices, as well as the left middle temporal g
163 radrenaline delivery to either prefrontal or parietal cortices, failed to restore wakefulness.
164 substantially overlapped in the premotor and parietal cortices, whereas individual movements were uni
165 f the cortex, most pronounced in frontal and parietal cortices; and (c) a significant disruption of t
166 of the more complex symptoms associated with parietal damage, such as constructional ataxia.
167 thin-subject correlation between frontal and parietal delay-period activity and whole-trial estimates
168 n explained the most variance in frontal and parietal delay-period activity.
169           These findings suggest that fronto-parietal disconnection might be particularly relevant fo
170 n cognitively salient brain networks (fronto-parietal; dorsal attention, DAN; ventral attention; and
171 relative to the stimulated hand over central-parietal electrodes but relative to its external locatio
172 in the context of abundant (and spectacular) parietal engravings.
173 1/2, and CD44, but not with synaptopodin, in parietal epithelial cells (PECs) infiltrating cFSGS glom
174 lomerular tuft and marked hyperplasia of the parietal epithelial cells (PECs).
175 d mTOR signalling and proliferation in human parietal epithelial cells after rapamycin treatment.
176 odocytes, activation of proximal tubule-like parietal epithelial cells identified by ultrastructural
177 lls of the afferent and efferent arterioles, parietal epithelial cells, and three types of endothelia
178 tion and with higher dementia risk scores in parietal, frontal and medial occipital cortices, (3) wit
179  We found that a large set of regions in the parietal, frontal, and insular cortices shows increases
180                        Right SMG and several parietal grasp regions, namely, left anterior intraparie
181 rus, and right insular, lingual and superior parietal gyri were significantly smaller, on average, in
182 y/attention network was altered in AD due to parietal inactivation.
183  parietal (supramarginal gyrus) and superior parietal (intraparietal and superior parietal) regions t
184 creased synchronization of the right temporo-parietal junction (R TPJ) during the collaborative phase
185 e posterior cingulate cortex (PCC), temporal parietal junction (TPJ), and medial prefrontal cortex (M
186 ve middle temporal complex (MT+) and temporo-parietal junction (TPJ).
187 mentalizing-related (e.g. precuneus, temporo-parietal junction) and reward-related regions (e.g. puta
188 ecifically, selectivity of the right temporo-parietal junction) in these children resembles responses
189 white matter tracts deep to the left temporo-parietal junction.
190 brain data across a bilateral fronto-temporo-parietal language network.
191 s +/- 0.22; right, 1.99 years +/- 0.22), and parietal (left, 1.92 years +/- 0.30; right, 2.03 years +
192 G) in post-stroke patients with left temporo-parietal lesions prior to functional neuroimaging.
193 .001), temporal lobe (g = -0.84; p < 0.001), parietal lobe (g = -0.73; p = 0.053), cerebellum (g = -1
194            Our understanding of the inferior parietal lobe (IPL) remains challenged by inconsistencie
195 erately intense stimulation was found in the parietal lobe (P2, P4, and P6 electrodes).
196 vity (mediated by the beta band) to inferior parietal lobe and right middle temporal gyrus (MTG).
197  n = 117; temporal lobe, n = 244 vs n = 137; parietal lobe n = 240 vs n = 93; and occipital lobe, n =
198 l association with surface area in the right parietal lobe, a region related to nonverbal cognitive f
199 e cortex, and increases in activation in the parietal lobe, posterior cingulate cortex, and inferior
200 10.85, 95% CI -17.91, -3.79, p < 0.0125) and parietal lobes (B = -12.75, 95% CI -21.58, -3.91, p < 0.
201 ations of FC between left and right inferior parietal lobes and right insular cortex.
202 r white matter hyperintensities (frontal and parietal lobes).
203 y matter volume of the frontal, temporal and parietal lobes, insula and whole brain.
204 symmetric uptake was evident in temporal and parietal lobes, precuneus, and posterior cingulate corte
205 ant WM abnormalities within the temporal and parietal lobes.
206  with higher MT in the bilateral frontal and parietal lobes.
207 l (PMv)-motor cortex (M1), anterior inferior parietal lobule (aIPL)-M1, and dorsal inferior parietal
208 ) and anterior intraparietal sulcus/superior parietal lobule (consistent with sensorimotor output).
209 rietal lobule (aIPL)-M1, and dorsal inferior parietal lobule (dIPL)-M1 before and after inducing a lo
210  reveal the contribution of rostral inferior parietal lobule (IPL) regions, in particular PFt, and th
211 x (AIC), premotor cortex (PMd), and inferior parietal lobule (IPL) were modulated by prior belief on
212 , ventral premotor cortex (PMv) and inferior parietal lobule (IPL), presumably consisting of motor-re
213 ed with social and moral cognition (inferior parietal lobule [IPL], prefrontal cortex [PFC], and cing
214 (p < 0.0003), and a decrease in NAc-inferior parietal lobule FC relative to controls (p < 0.001).
215 ight middle frontal gyrus and right inferior parietal lobule in ECN, as well as increased RSFC betwee
216 ons, particularly the right and left lateral parietal lobule, and the Language Network, including the
217 ve to HC in both the left and right inferior parietal lobule, including the supramarginal and angular
218 eased GMV in the right insula, left inferior parietal lobule, left dorsolateral prefrontal cortex/sup
219  intraparietal sulcus and bilateral superior parietal lobule, met our criteria for transsaccadic orie
220 , the inferior parietal sulcus, and superior parietal lobule.
221 uperior temporal gyrus and the left inferior parietal lobule.
222 dle ripples are tightly coupled to posterior parietal locations activated by conscious recollection.
223 theta coherence, and rs116445911 with centro-parietal low theta coherence.
224 in the PMC that matches the known multifocal parietal maps of grasping representations.
225 raction and GMD across the whole frontal and parietal medial cortex reflecting the consequence of HF
226              These findings suggest that the parietal multimodal sensory association region could hav
227  spread, from occipital visual areas through parietal multisensory areas to frontal action planning a
228 ks [e.g., default mode network (DMN), fronto-parietal network (FPN), and sensory-motor network (SMN)]
229  forms of attention engaging a common fronto-parietal network at different time lags.
230                    A left-lateralized fronto-parietal network was recruited for code comprehension.
231 structural integrity of a brain-scale fronto-parietal network, including prefrontal regions related t
232 comprehension of code could depend on fronto-parietal networks shared with other culturally-invented
233               Hippocampal and fronto-temporo-parietal networks that respectively support episodic mem
234 related with the dorsal attention and fronto-parietal networks; and third subdivision does not have a
235  We simultaneously recorded from frontal and parietal nodes of the attention network while macaques p
236 ssion showed confounding effects in frontal, parietal, occipital, accumbal and thalamic regions.
237 ephalography findings showed asymmetric left parietal-occipital high-amplitude spike-and-wave dischar
238 s reflecting sensory inference and memory in parietal-occipital regions, while the cumulative exposur
239 oefficient observed in scalp electrodes over parietal-occipital regions.
240 le (IPL) regions, in particular PFt, and the parietal opercular regions in decision processing and de
241          We previously demonstrated that the parietal operculum (parts OP1/OP4) is activated with CMR
242 chronic form (OR 3.29, 95% CI 1.70 to 6.49), parietal (OR 14.82, 95% CI 6.32 to 37.39) or temporal (O
243 opercularis (p = 0.03), ipsilateral inferior parietal (p = 0.04) and contralateral frontal pole (p =
244 rol (frontal theta inter-trial coherence and parietal P3b latency), as measured by electroencephalogr
245 sting EEG and TEPs from prefrontal (PFC) and parietal (PAR) cortex were measured before and after adm
246 ity of the ipsilateral white matter inferior parietal parcel showed a marginally significant increase
247 eritoneal pouch was formed by dissecting the parietal peritoneum from the transversalis fascia of mic
248       In a novel preparation of the isolated parietal peritoneum PD fluid or 3,4-DGE alone, at concen
249 national cohort of 1280 patients to localize parietal pleura/lung parenchyma followed by classificati
250 late event-related EEG potential (the Centro-Parietal Positivity, CPP) to be a correlate of the accum
251 verlapping brain circuits including frontal, parietal, posterior, and cingulate regions with the resu
252 sions also tended to have relatively reduced parietal-prefrontal feedforward effective connectivity d
253  observations support a role for feedforward parietal-prefrontal information processing deficits in d
254 ddress individual variability in feedforward parietal-prefrontal information updating in patients wit
255 = 0.001), temporal (r = 0.74; P = 0.002) and parietal (r = 0.89; P < 0.001) cortex correlated with gl
256                                 Premotor and parietal regions also exhibited changes in the fine-grai
257  level occur along a network where posterior parietal regions are connected to homologous premotor re
258 and 11C-PK11195 were derived from 15 temporo-parietal regions characteristically affected by Alzheime
259 ly, lateralization of alpha-band activity in parietal regions during attentional orienting in expecta
260 r theta or alpha frequency to prefrontal and parietal regions identified using functional MRI.
261 mapping demonstrated that damage to temporal-parietal regions impacted the ability to retrieve words
262          Our results posit a central role of parietal regions in shaping multisensory spatial recalib
263                        Our data suggest that parietal regions involved in multisensory and spatial me
264  resting state connectivity with frontal and parietal regions of the dorsal attention network, encode
265  the remembered stimulus, as did frontal and parietal regions of the dorsal attention network.
266 t, TFCE) and in FH +group (left temporal and parietal regions p<0.01, TFCE).
267 roup (left cingulate and lateral frontal and parietal regions p<0.01, threshold-free cluster enhancem
268 re seen across the cortex, the occipital and parietal regions undergo the greatest rate of cortical a
269 ortical activation in frontal, temporal, and parietal regions using multiple regression models, adjus
270 EM delta power especially in the frontal and parietal regions, and (iii) progressive increases in ind
271 , ventral and dorsal system in the posterior parietal regions, but no systematic causal description o
272 multisensory integration within temporal and parietal regions, only medial superior parietal activity
273 back layer-to-layer connectivity in occipito-parietal regions, suggesting that sensory plasticity gat
274 ong temporal, inferior frontal, and inferior parietal regions, underpinning the verb's modification o
275 ors have different impacts on prefrontal and parietal regions, we designed a dual route decision-maki
276 ed with activation in frontal, temporal, and parietal regions, whereas both implicit regulation and e
277 ning, goal-directed attention-involving left parietal regions-integrates "what" and "when" stimulus i
278 ions were observed predominantly in temporal-parietal regions.
279 e, and another weighted to posterior temporo-parietal regions.
280 p plans in additional intraparietal/superior parietal regions.
281 tical areas, particularly in the frontal and parietal regions.
282 uperior parietal (intraparietal and superior parietal) regions that show saccade-specific modulations
283 of genome-wide association studies (MTAG) on parietal resting-state theta (3-7 Hz) EEG coherence, whi
284 her future global (rho = 0.29; P = .008) and parietal (rho = 0.31; P = .005) amyloid beta and parieta
285 ttentional task showed that this rTMS on the parietal site hindered participants' ability to integrat
286 , whereas more posterior regions-such as the parietal, squamosal, and quadrate-exhibited high rates i
287 he posterior collateral sulcus, the inferior parietal sulcus, and superior parietal lobule.
288 we used fMRI to identify a group of inferior parietal (supramarginal gyrus) and superior parietal (in
289 erior nucleus) and visual cortex (occipital, parietal, suprasylvian, temporal and splenial visual reg
290 k and somatosensory/somatomotor hand, fronto-parietal task control, memory retrieval, and visual and
291 etal (rho = 0.31; P = .005) amyloid beta and parietal tau load (rho = 0.31; P = .005).
292                     We conclude that temporo-parietal tau pathology and anterior temporal neuroinflam
293 ts that neural activity spanning prefrontal, parietal, temporal, and visual areas supports the genera
294 ludes 234 priors from frontal, sensorimotor, parietal, temporal, occipital, cingular and subcortical
295 amus, connectivity to sensorimotor networks, parietal-temporal-occipital networks, putamen, and cereb
296                 Lastly, both the frontal and parietal theta-band power encoded the outcome when it wa
297  response, which consistently increased from parietal to prefrontal and cingulate cortex.
298 -deficient placentas exhibit an expansion of parietal trophoblast giant cells with a concomitant decr
299 ions of contralateral areas 3b, 3a, 1 and 2, parietal ventral (PV), secondary somatosensory cortex (S
300  observed in the temporal, suprasylvian, and parietal visual areas.

 
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