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1 he P400cz has its major source in the medial parietal region.
2 in the left and right amygdala and the right parietal region.
3 dent on computations implemented within this parietal region.
4  periventricular regions of interest and the parietal region.
5 erior temporal regions and the left inferior parietal region.
6 usions about the evolution of this posterior parietal region.
7 bal fluency scores to atrophy in frontal and parietal regions.
8 dent of the integrity of areas MT/V5, V3A or parietal regions.
9 x, posterior basal ganglia, and thalamic and parietal regions.
10  the motor regions, including prefrontal and parietal regions.
11 ions were observed predominantly in temporal-parietal regions.
12 x that most severely affects the frontal and parietal regions.
13 ortex and in the right premotor and inferior parietal regions.
14 r tracts that connect frontal, temporal, and parietal regions.
15 nt ERP or fMRI retinotopic memory effects in parietal regions.
16 glia, but not in the frontal, occipital, and parietal regions.
17 e, and another weighted to posterior temporo-parietal regions.
18  related to a loss of deactivation in medial parietal regions.
19  and midbrain as well as select temporal and parietal regions.
20 ctivation (FE>NE) in the midline frontal and parietal regions.
21 entral midline region, with a later onset in parietal regions.
22 p plans in additional intraparietal/superior parietal regions.
23 yrification in women than men in frontal and parietal regions.
24 ntrolled' system depends upon prefrontal and parietal regions.
25  from the phonological store in temporal and parietal regions.
26 ty in the lateral premotor and inferolateral parietal regions.
27 rsal and ventral portions of the LOTC and in parietal regions.
28 imilar areas of the brain in the frontal and parietal regions.
29 Stem model were significant in left occipito-parietal regions.
30 tempo improves PLV between the occipital and parietal regions.
31 nd premotor cortices but not in striatal and parietal regions.
32 ificantly higher in hand than tool-selective parietal regions.
33 ical thinning in medial temporal and frontal/parietal regions.
34 pectral tilt, suggesting inhibition of these parietal regions.
35 lectrodes and included frontal, central, and parietal regions.
36 ynchrony value (PLV) between the frontal and parietal regions.
37  pathology were in the precuneus and lateral parietal regions.
38 er-frequency metrics, such as delta power in parietal regions.
39 e electrodes were centered over premotor and parietal regions.
40 predicted evidence accumulation signals over parietal regions.
41 ine to ATP ratios (PCr/ATP) in precuneus and parietal regions.
42 ical network encompassing mostly frontal and parietal regions.
43 ced amygdala connectivity with occipital and parietal regions.
44 erformance via modulating neural activity in parietal regions.
45  engaging premotor, prefrontal and posterior parietal regions.
46 the left-temporal, right-temporal, and right-parietal regions.
47 ct category decoding in occipitotemporal and parietal regions.
48 edial and inferior temporal, prefrontal, and parietal regions.
49 ter hyperintensity growth in the frontal and parietal regions.
50 l, precuneus, left postcentral, and inferior parietal regions.
51  cortex, dorsolateral prefrontal cortex, and parietal regions.
52 l inference process with specific visual and parietal regions.
53 may indicate the functional role of specific parietal regions.
54 o-parietal cortex and more posterior temporo-parietal regions.
55 tical areas, particularly in the frontal and parietal regions.
56 teral temporal, lateral parietal, and medial parietal regions.
57 iated with cortical thinning in temporal and parietal regions.
58 e of posture on primary motor, premotor, and parietal regions.
59 ementia, the greatest change was seen in the parietal regions (5.22%; 95% CI, 3.95%-6.49%).
60 e CMRglc of the conscious brain (e.g., right parietal region, 99.6 +/- 10.2 mumol/100 g/min; n = 6) w
61           In inferior temporal, frontal, and parietal regions, a gradual buildup in activity peaking
62 ew effect-related activations in frontal and parietal regions, a pattern not seen in control subjects
63 wo different pathways connecting frontal and parietal regions: a corticocortical pathway and a subcor
64 n map, lesion sparing of right dorsal fronto-parietal regions, age, and anosognosia.
65 correction), and less grey matter in several parietal regions (all p<0.002 uncorrected and corrected
66 lvian (pMS) sulcus in the posterior inferior parietal region also results in an equally severe impair
67                                 Premotor and parietal regions also exhibited changes in the fine-grai
68 al, working memory in ventral prefrontal and parietal regions, although they showed less verbal super
69  activation in the left prefrontal and right parietal regions, an effect not observed at any time poi
70  in particular, the activity in the inferior parietal region and in the anterior-medial cerebellum wa
71 with white matter volume in the left temporo-parietal region and that white matter volume influenced
72 on at the perceptual level occurs within the parietal region and the interaction at categorical decis
73 riod involved direction-specific activity in parietal regions and both dorsal and ventral sensory reg
74  groups showed reduced glucose metabolism in parietal regions and in middle and superior temporal reg
75 ficantly reduced connectivity in the PCC and parietal regions and increased frontal connectivity arou
76 ile mathematical processing recruits frontal-parietal regions and reading frontal-temporal regions, b
77 mporal regions and some thinning in inferior parietal regions and the posterior cingulate (P < 0.001)
78 bjects between activation in the frontal and parietal regions and the rate of correct responses on th
79 UVR (P < .05 for medial temporal and lateral parietal regions) and gray matter volumes (all 4 ROIs; P
80 edial temporal, lateral temporal, and medial parietal regions) and gray matter volumes (P < .05 for m
81 he first trimester (eyes, midface, chin, and parietal region), and binge-level exposure in the first
82 EM delta power especially in the frontal and parietal regions, and (iii) progressive increases in ind
83 ted with retinotopic activity in frontal and parietal regions, and assessed whether retinotopic activ
84 ral prefrontal cortex and medial and lateral parietal regions, and between the medial prefrontal cort
85 as increased gamma band power in frontal and parietal regions, and decreased spectral power in theta,
86 ral activity in hippocampal, prefrontal, and parietal regions, and that age-related memory impairment
87 nd neighboring motor, premotor, and inferior parietal regions, and tonic components, centered on oper
88 ncy of around 14 Hz, occurring in the centro-parietal region; and slow spindles, with a frequency of
89 trophy rates in the left medial-temporal and parietal regions; and (ii) in contrast, increased amyloi
90  orienting, which may be mediated by lateral parietal regions; and the experience of joint attention
91           Dorsal striatum, insula, posterior parietal regions, anterior and posterior cingulate, and
92                                 In addition, parietal regions appear most relevant for quantifying ar
93                  Disconnections in bilateral parietal regions are associated with lower hand temperat
94  level occur along a network where posterior parietal regions are connected to homologous premotor re
95 dings are discussed in the context that left parietal regions are critical for the modification of st
96 us, our results show that left but not right parietal regions are critical for visuomotor adaptation.
97                   The frontal, temporal, and parietal regions are heteromodal association cortices th
98                                   If certain parietal regions are involved in action understanding, t
99                   These results suggest that parietal regions are part of a network of brain areas th
100 information content, a number of frontal and parietal regions are thought to be domain- and process-g
101                                       In the parietal region, area 45 is connected with the angular g
102 ness, delta waves propagated from frontal to parietal regions as a traveling wave.
103 further indicated several common frontal and parietal regions as being involved in the control of bot
104 m, extending laterally into adjacent temporo-parietal regions as well as splenium and fornix.
105 vely with activity in several prefrontal and parietal regions (as measured by fMRI).
106 n the right parietal and left prefrontal and parietal regions, as early as acute phase I.
107 pattern similarity in lateral prefrontal and parietal regions, as well as in anterior and posterior m
108 he posterior thalamus and bilateral inferior parietal regions, associated with a lower electrical pai
109 th smaller volumes in primarily temporal and parietal regions at baseline.
110 ing working memory in the medial frontal and parietal regions bilaterally and in the right dorsolater
111  not only adjoining prefrontal, temporal and parietal regions but also bilateral caudate and left put
112 fects in posterior cingulate, precuneus, and parietal regions but also significant positive associati
113 se in activation (right prefrontal and right parietal regions) but less in regions that showed a satu
114 , ventral and dorsal system in the posterior parietal regions, but no systematic causal description o
115 ckout mice, particularly in both rostral and parietal regions, but not caudal cortex.
116 ttern analysis showed that only premotor and parietal regions, but not primary motor cortex (M1), dev
117                    Cortical thickness in the parietal regions by risk status.
118  several areas of the human brain, including parietal regions, can respond multimodally.
119 edicted fear included superior-occipital and parietal regions, cerebellum and prefrontal cortex.
120 and 11C-PK11195 were derived from 15 temporo-parietal regions characteristically affected by Alzheime
121 orsomedial frontal, and insular and inferior parietal regions closely similar to the human counterpar
122 wed reduced activation in bilateral temporal-parietal regions compared to the other groups, which did
123 us and widespread frontal, frontolimbic, and parietal regions compared with HC subjects.
124                                       Fronto-parietal regions connected by this tract were activated
125 , the intraparietal sulcus, and the inferior parietal region), consistent with neural circuits that h
126 nd stronger midfrontal connectivity with the parietal region contralateral to, rather than ipsilatera
127  where distinct pathways between frontal and parietal regions contribute to multiple spatial represen
128                We addressed how human fronto-parietal regions control visuospatial attention on a fin
129                            First, visual and parietal regions coordinated with sensorimotor and premo
130                              Lower FA in the parietal region correlated significantly with higher Yal
131 o the subgenual cingulate and prefrontal and parietal regions correlated with BDI scores and [(18)F]P
132                          Although a superior parietal region demonstrated the retrieval success patte
133               These results suggest that DMN parietal regions directly supported memory retrieval, wh
134 d with enhancement of gamma power in a right parietal region during movement execution as well as gam
135 increase of theta power over that same right parietal region during movement planning, which is corre
136 ateralized network of frontal, temporal, and parietal regions during a variety of semantic processing
137 ly, lateralization of alpha-band activity in parietal regions during attentional orienting in expecta
138 k-induced decrease in BOLD signal) in medial parietal regions during successful compared with failed
139 teral activation of prefrontal and posterior parietal regions during successful identification of old
140 y the task (prefrontal, medial temporal, and parietal regions) during encoding were similar to those
141 vision of trigeminal nerve, neck, nose, jaw, parietal region, ear, teeth, eyebrow, shoulder (ipsilate
142 memory encoding task-related deactivation in parietal regions (eg, mean [SD] parameter estimates for
143 at the retrosplenial complex (RSC) and other parietal regions encode panoramas of views observed from
144 ivation in the dyslexic group, only the left parietal region exhibited reduced gray matter volume rel
145 oinsular, thalamus, cerebellum and bilateral parietal regions for C9P FTLD relative to C9N FTLD, and
146 of memory-related activations in frontal and parietal regions for retrieval of scenes and the absence
147                       In bilateral posterior parietal regions, greater activation was associated with
148                                 Whereas some parietal regions have specific motor functions, others a
149  the left amygdala and clusters in bilateral parietal regions; higher maternal anxiety was associated
150  Evidence from lesion patients suggests that parietal regions house supramodal representations of spa
151 cerebellum has connections with multisensory parietal regions; however, it is unknown if force adapta
152 s located predominantly in left temporal and parietal regions (i.e. the superior temporal sulcus, inf
153 r theta or alpha frequency to prefrontal and parietal regions identified using functional MRI.
154 mapping demonstrated that damage to temporal-parietal regions impacted the ability to retrieve words
155 differential connectivity with occipital and parietal regions implicated in the processing of low-lev
156 i, high attentional demands suppress ventral parietal regions important for veridical remembering.
157 n imaging studies have implicated a specific parietal region in symbolic number processing, leading t
158  These findings indicate a critical role for parietal regions in action planning when there is respon
159 xtension of cortical thinning in the temporo-parietal regions in addition to frontal atrophy could be
160 onization of both FP and MF networks in left parietal regions in all mediation styles, and (2) only t
161  shift in the associated patterns from right parietal regions in awake, to right frontoparietal durin
162 as analyzed across the frontal, central, and parietal regions in both hemispheres.
163 l recalibration, and reveal a role of medial parietal regions in linking present and previous multise
164 EG theta activity over frontal, temporal and parietal regions in response to negative stimuli in SAD
165          Our results posit a central role of parietal regions in shaping multisensory spatial recalib
166 nently in the inferior temporal and inferior parietal regions in the full cohort, with florbetapir po
167 l temporal areas as well as lateral temporal-parietal regions in the left hemisphere.
168 oke patients with focal damage to frontal or parietal regions in the left or right brain hemispheres
169 atter in left superior temporal and inferior parietal regions in the PPA group.
170 ed at recording locations over the posterior parietal regions in the vertical direction.
171 nnectivity was reduced between S1 and fronto-parietal regions, in both the TMSR and non-TMSR patients
172 cially the orbital frontal cortex and medial parietal regions, in these mnemonic abilities.
173  one proceeds from area 30 toward the medial parietal regions, including areas 3, 1, 2, 5, 31, and th
174 re associated with activation in frontal and parietal regions, including bilateral dorsal premotor co
175 ward value and uncertainty in prefrontal and parietal regions, including frontopolar cortex, and para
176  to pop-out) were observed in several fronto-parietal regions, including IPS, FEF, MFG and IFG, in ad
177 es depend on a shared network of frontal and parietal regions, including white matter association tra
178 y a shared network of frontal, temporal, and parietal regions, including white matter association tra
179 rtical volumes and thickness in temporal and parietal regions independently of Abeta.
180 the topography and response profile of human parietal regions inside and outside the DMN, independent
181 ning, goal-directed attention-involving left parietal regions-integrates "what" and "when" stimulus i
182  related to recollection and a more superior parietal region involved in familiarity.
183                        Our data suggest that parietal regions involved in multisensory and spatial me
184                     That grey matter loss in parietal regions is a part of healthy aging suggests tha
185 ional and structural connectivity in temporo-parietal regions known to have high MET expression, part
186 and S1 and of both these regions with fronto-parietal regions, known to be important for multisensory
187 ained by event-related potentials around the parietal regions: low performers showed increased wavefo
188 mi-spatial neglect suggest that the inferior parietal region may lie at the junction of the visual an
189                                   The medial parietal region may, then, be conceived of as a nodal st
190 The aim of this work was to demonstrate that parietal regions may mediate selective attention to moti
191 alyses indicated that lateral prefrontal and parietal regions may mediate the relation between abilit
192 task, and they thus suggest that frontal and parietal regions may play a general role in visual aware
193 c resonance (ER-fMRI), we show that distinct parietal regions mediated these different attentional pr
194  under distraction, higher-order frontal and parietal regions might contribute to content-specific wo
195 e matter diffusion anisotropy in the temporo-parietal region of the left hemisphere was significantly
196  matter was thinner in frontal, temporal and parietal regions of both brain hemispheres.
197 dominance based on either frontal or temporo-parietal regions of interest (ROIs) defined for the enti
198 n entorhinal, inferior temporal, and lateral parietal regions of interest and an AD meta-region of in
199 re significantly higher in the occipital and parietal regions of the adults with Down's syndrome than
200 production are depressed in the temporal and parietal regions of the cortex in patients with AD, we d
201 nectivity between frontoparietal systems and parietal regions of the dorsal attention network involve
202 elation between the default mode network and parietal regions of the dorsal attention network is cons
203  resting state connectivity with frontal and parietal regions of the dorsal attention network, encode
204  the remembered stimulus, as did frontal and parietal regions of the dorsal attention network.
205 ttern of widespread thinner cortex in fronto-parietal regions of the left hemisphere in both DSZ and
206 multisensory integration within temporal and parietal regions, only medial superior parietal activity
207 t, TFCE) and in FH +group (left temporal and parietal regions p<0.01, TFCE).
208 roup (left cingulate and lateral frontal and parietal regions p<0.01, threshold-free cluster enhancem
209 ng metabolism in the prefrontal and inferior parietal regions (P < 0.001).
210 eas connections originating from frontal and parietal regions peaked at beta frequency.
211 antic enhancement effects within frontal and parietal regions, perhaps reflecting downstream attempts
212 tion of the posterior cingulate and inferior parietal regions persisted in the remitted subjects, sug
213  absent, whereas activation in occipital and parietal regions persisted.
214 an also be used to highlight the role fronto-parietal regions play in the maintenance of covert task-
215 udies suggests that a network of frontal and parietal regions plays a crucial role.
216 ferior-frontal, middle-frontal, and inferior-parietal regions preceded by high-gamma attenuation in t
217 hat ongoing alpha band activity in occipital-parietal regions predicts the quality of visual informat
218 that interactions between primary visual and parietal regions predominantly influenced activity in fr
219  higher MPL precursor levels in the inferior parietal region (primarily right side) were noted in the
220 ts with damage to either the temporal or the parietal regions provide support for this functional dis
221 th the largest effects in frontotemporal and parietal regions; psychotic bipolar probands had small r
222 e effect on the form of BOLD response in the parietal region reflecting imagined transformations to t
223 rked connectome changes in bilateral temporo-parietal regions, reflecting an increased segregation of
224             The left hemisphere temporal and parietal regions remained significant when converters we
225 that a set of superior temporal and inferior parietal regions respond more strongly to conditions con
226 representational similarity analysis in this parietal region revealed that beneficial recurrent retri
227 related to alterations of neural activity in parietal regions seen over the course of MCI and AD.
228 ly, many cells in association and motor-like parietal regions show increasingly regular spike trains
229                    Furthermore, the inferior parietal region showed nonspecific tactile and motor res
230                                          The parietal region showed significantly greater left latera
231 nd theta/beta ratio at the frontocentral and parietal regions showed a graded correlation with 12-mon
232     Whereas beta-band activity (18-30 Hz) in parietal regions showed body-centered spatial selectivit
233                                    All three parietal regions showed comparable delay-period response
234                              The two sets of parietal regions showed different resting-state function
235 r cingulate, posterior inferior frontal, and parietal regions showed extended activation for all type
236                         During eCFT, lateral parietal regions showed progressively more distinct acti
237 maging data with prespecified prefrontal and parietal regions showed that, although both regions were
238 these results suggest that the left inferior parietal region subserves subword orthographic-to-phonol
239 s represented within both ventral-stream and parietal regions, suggesting that communication between
240 back layer-to-layer connectivity in occipito-parietal regions, suggesting that sensory plasticity gat
241  matter revealed lower FA bilaterally in the parietal region (supramarginal gyri), right posterior ci
242 d the prefrontal and between the SMA and the parietal regions tended to decrease after training.
243 as with spread to contralateral temporal and parietal regions than in simple partial seizures.
244                Two seeds were used: a medial parietal region that contributes to the default network
245 nses relative to non-switch trials in fronto-parietal regions that appeared to be left lateralized, i
246  These clusters are linked by prefrontal and parietal regions that are hub nodes in the underlying st
247 rted by a distributed network of frontal and parietal regions that enable complex, goal-directed beha
248  to increased activity in medial and lateral parietal regions that have been implicated in attention
249  and the uncinate fasciculus and in the left parietal regions that include the fiber bundle of the su
250            Paradoxically, damage to specific parietal regions that lack spatial maps can cause patien
251 -temporal regions and decreases in posterior-parietal regions that largely recovered by two weeks pos
252 on of structurally intact dorsal and ventral parietal regions that mediate related attentional operat
253 right inferior parietal and bilateral medial parietal regions that support egocentric movement throug
254 lar gyrus, but also more anterior and dorsal parietal regions that were anatomically separate from th
255 uperior parietal (intraparietal and superior parietal) regions that show saccade-specific modulations
256 components I, II and III; from the occipital-parietal region, the fronto-occipital fasciculus; from t
257 In the human brain, a network of frontal and parietal regions, the "multiple demand" (MD) system, is
258 vation in a network including prefrontal and parietal regions, the amygdala, caudate, and mid-brain.
259                         Similarly, in medial parietal regions, the posterior cingulate was related to
260 l magnetic resonance imaging, allowing these parietal regions to be routinely and reliably identified
261 ing of cortical primary motor, premotor, and parietal regions to motor sequences: from the low-level
262 ifted from sensory modality in the posterior parietal regions to presentation format in the anterior
263 ound increasing involvement of occipital and parietal regions together with caudal-rostral recruitmen
264 re seen across the cortex, the occipital and parietal regions undergo the greatest rate of cortical a
265 he hypothesis that a single, domain-specific parietal region underlies both symbolic and nonsymbolic
266            Cortical responses in the frontal/parietal region underlying categorization of faces by sp
267 ong temporal, inferior frontal, and inferior parietal regions, underpinning the verb's modification o
268 in dorsal premotor cortex, with no effect in parietal regions until 150 ms post-perturbation.
269 ortical activation in frontal, temporal, and parietal regions using multiple regression models, adjus
270 articipants indicated that the left inferior parietal region was active during both action execution
271 ) and no retrieval success-sensitive lateral parietal region was insensitive to cueing.
272 ge, greater beta oscillatory activity in the parietal region was observed during right motor imagery
273 ollowing damage to the right fronto-temporal-parietal region was slow and sometimes unable to find ta
274 eta burden in bilateral precuneus/cuneus and parietal regions was associated with increased brain atr
275                      Deactivation in lateral parietal regions was equivalent across groups; in medial
276 work comprising the cerebellum, temporal and parietal regions was increased and localized to the medi
277 ors have different impacts on prefrontal and parietal regions, we designed a dual route decision-maki
278 elation (ISC) with age in sensory and medial parietal regions, we used a novel measure (between-group
279 ilk servings (P </= 0.013), and those in the parietal region were also correlated with cheese serving
280 fferences in the topological organization of parietal regions were found between phenotypically diffe
281                        Precuneus and lateral parietal regions were more activated for True and False
282 sial temporal, middle temporal, and inferior parietal regions were more active in the patients during
283  supported memory retrieval, whereas non-DMN parietal regions were more involved in postretrieval pro
284 ocampus, along with cerebellar, temporal and parietal regions were more substantial in major depressi
285 ases in the BOLD signal in right frontal and parietal regions when compared with valid trials.
286 embered spatial position in early visual and parietal regions when the required response was known ve
287 tion in the circuitry connecting frontal and parietal regions, where direct frontoparietal connection
288 ed with activation in frontal, temporal, and parietal regions, whereas both implicit regulation and e
289 ressed for IC sources in central midline and parietal regions, whereas mean beta band power increased
290 al gyrus is not connected with the posterior parietal region, which lies outside the confines of the
291 showed vasogenic edema in the right temporal parietal region, which prompted her transfer to our inst
292           In SD patients only, this inferior parietal region, which was not atrophied, was also activ
293 ities (hubs) in the primary sensorimotor and parietal regions, which formed a commonly shared core hu
294 ided into functionally distinct temporal and parietal regions, which have been implicated in feature-
295 l cortex and decreased in the right inferior parietal region with decreasing self-reference.
296 rior and middle temporal gyri, left inferior parietal region with postcentral gyrus, and right superi
297 econd component was maximal over more dorsal parietal regions with a longer latency (approximately 26
298 y-related deactivation in medial and lateral parietal regions with greater deactivation in less-impai
299 the skull itself (e.g., 47% mismatch for the parietal region), with decreased performance in the base
300 up x age interaction in the PFC and inferior parietal region, with relatively older psychostimulant-n

 
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