ライフサイエンスコーパス: parotid

1 uamous cell carcinoma, thyroid, adrenal, and parotid.

2 tivation, consistent with reports from mouse parotid.

3 tially to the deactivation of the current in parotid.

4 ts and acetylcholine-evoked secretion in the parotid.

5 anes of secretory granules isolated from rat parotids.

6 069 +/- 0.022 mSv/MBq), and salivary glands (parotids, 0.031 +/- 0.011 mSv/MBq; submandibular, 0.061

7 ges obtained after MSG administration in the parotids (24% +/- 14%, P = 0.001), submandibular glands

8 ates of unstimulated whole (35%), stimulated parotid (47%), unstimulated submandibular/sublingual (23

9 es including 14 stomach, 11 lung, 7 orbit, 7 parotid, 5 thyroid, 5 lacrimal gland, 3 small intestine,

10 In parotid, a rapid global Ca(2+) signal was invariably ind

11 The P2X7R-mediated current was measured in parotid acinar and duct cells of wild type and P2X7R-/-

12 Here we used parotid acinar and duct cells to reveal the unique cell-

13 tiated apical [Ca(2+)](i) clearance in mouse parotid acinar cells and apical PMCA activity in Par-C10

14 ased the rate of oxygen consumption (QO2) of parotid acinar cells and PC12 cells.

15 rpolarization-activated chloride currents in parotid acinar cells and, as described previously, displ

16 nnels in red blood cells, T lymphocytes, and parotid acinar cells are indeed encoded by the Kcnn4 gen

17 Ca(2+)](i) clearance in the apical region of parotid acinar cells because of a dynamic translocation

18 ctifier chloride channel (Cl(ir)) from mouse parotid acinar cells by external protons (H(+)(o)) using

19 dy-state activation of BK channels in native parotid acinar cells by only 6 mV.

20 Parotid acinar cells exhibit rapid cytosolic calcium sig

21 ases in oxygen consumption) were measured in parotid acinar cells exposed to tyrosine kinase inhibito

22 Parotid acinar cells express two types of Ca(2+)-activat

23 re of normal appearance and fertility, their parotid acinar cells expressed no IK channels, and their

24 However, parotid acinar cells from Clcn2(-/-) mice recovered volu

25 anide-sensitive Cl(-) influx was observed in parotid acinar cells from mice lacking NKCC1.

26 Parotid acinar cells from these animals lacked maxi-K ch

27 The BK channels in parotid acinar cells have a much more hyperpolarized vol

28 Thus, stimulation of parotid acinar cells in Ca2+ -free medium with 0.5 micro

29 CO(3)(-) exchanger activity was increased in parotid acinar cells isolated from knockout mice suggest

30 (+) exchanger expression was examined in the parotid acinar cells of Nhe1(-/-) and Nhe2(-/-) mice, bo

31 arly, immunofluorescence of acutely isolated parotid acinar cells showed that the regulatory subunit

32 Exposure of freshly isolated parotid acinar cells to rottlerin and FCCP reduced cellu

33 These findings show that parotid acinar cells use low capacity, high sensitivity

34 To address these concerns, we profiled mouse parotid acinar cells using live-cell imaging to follow t

35 on sites on the alpha1 subunit in native rat parotid acinar cells using tandem mass spectrometry and

36 Ca(2+) signaling and cAMP pathways in human parotid acinar cells was investigated.

37 P(3) receptors, the Cl(-) current density of parotid acinar cells was more than four-fold greater tha

38 muscarinic receptor-induced acidification in parotid acinar cells was of a similar magnitude (0.25 +/

39 dly affect a variety of nuclear processes in parotid acinar cells while facilitating efficient fluid

40 In human parotid acinar cells, carbachol stimulation evoked incre

41 These data indicate that in human parotid acinar cells, in addition to modulation of Ca(2+

42 - channels previously characterized in mouse parotid acinar cells, nor is it dependent on P2 nucleoti

43 gain further insight into I(ATPCl) in mouse parotid acinar cells, we investigated the effects of ATP

44 as localized to the basolateral membranes of parotid acinar cells, whereas expression was not detecte

45 study demonstrates CICR in the non-excitable parotid acinar cells, which resembles the mechanism desc

46 or ligand carbachol in freshly dispersed rat parotid acinar cells.

47 MCA4 was localized to the apical membrane of parotid acinar cells.

48 TPase (PMCA) in a Ca(2+)-dependent manner in parotid acinar cells.

49 atial regulation of [Ca(2+)](i) clearance in parotid acinar cells.

50 ways from the maturing secretory granules in parotid acinar cells.

51 role in the storage of secretory proteins in parotid acinar cells.

52 mpaired in Kcnn4 null mice but was normal in parotid acinar cells.

53 -trisphosphate receptors (InsP(3)R) in mouse parotid acinar cells.

54 2X(4)R and P2X(7)R mRNA and protein in human parotid acinar cells.

55 temporal properties of [Ca2+]i signaling in parotid acinar cells.

56 icted [Ca2+]i signals were never observed in parotid acinar cells.

57 cAMP regulates Ca(2+) clearance pathways in parotid acinar cells.

58 oupling in a variety of cell types including parotid acinar cells.

59 This hypothesis was tested in mouse parotid acinar cells.

60 her TMEM16A or TMEM16B as well as from mouse parotid acinar cells.

61 and P2X(7)R activation evokes exocytosis in parotid acinar cells.

62 reatic lipase knockouts are lethal, exocrine parotid acini lacking lipases were used to verify the re

63 levels is due to activation of AC8 in mouse parotid acini, and strongly support a role for AC5/6 in

64 2+) entry stimulates cAMP synthesis in mouse parotid acini, suggesting that one of the Ca(2+)-sensiti

65 coupling Ca(2+) signals to cAMP synthesis in parotid acini.

66 Water permeability decreased by 65% in parotid and 77% in sublingual acinar cells from Aqp5(-)/

67 ffects the salivary glands and in particular parotid and cervical lymph nodes.

68 Ca(2+) sensitivity of the Cl(-) channels in parotid and pancreas was determined from the [Ca(2+)]-cu

69 strated the presence of ARC channels in both parotid and pancreatic acinar cells and shown that, agai

70 In parotid and pancreatic acinar cells, changes in [Ca(2+)]

71 (SMSL) saliva as well as citrate-stimulated parotid and SMSL saliva were measured in 399 subjects.

72 In contrast, the water permeability in parotid and sublingual acinar cells isolated from Aqp5(-

73 dy was to determine variations in stimulated parotid and submandibular flow rates over 6 hours and to

74 the immature acinar cells in developing rat parotid and submandibular glands and are also products o

75 imed to report a RCC case with metastasis to parotid and submandibular glands that has the same sonog

76 ulated whole and stimulated glandular (i.e., parotid and submandibular) saliva flow rate and composit

77 ve properties to blots of SDS-PAGE-separated parotid and submandibular-sublingual (SM-SL) saliva.

78 ed pattern observed for protein secretion in parotid and submandibular/sublingual glands, and that th

79 Parotid and submandibular/sublingual saliva samples and

80 tes and antimicrobial proteins in stimulated parotid and submandibular/sublingual saliva were determi

81 ns of appropriate agonists (carbachol in the parotid, and both carbachol and cholecystokinin in the p

82 by placing regions of interest on lacrimal, parotid, and submandibular glands; left ventricle; liver

83 Parotid biopsies from four non-pSS patients served as co

84 NA transcripts expressing IGHV3 genes in all parotid biopsies.

85 minimal effect on steady-state activation of parotid BK channels, it produced an approximate 2-fold s

86 This was not because the parotid BK isoform (parSlo) is inherently insensitive to

87 roteases by all agents was also inhibited in parotid C5 cells expressing PKCdeltaKD.

88 DNA fragmentation was blocked up to 71% in parotid C5 cells infected with the PKCdeltaKD adenovirus

89 Induction of apoptosis in rat parotid C5 cells produced catalytic domain polypeptides

90 arotid cells from PKCdelta(-/-) cells and in parotid C5 cells, which express a dominant inhibitory mu

91 ree cDNA clones were isolated from a porcine parotid cDNA library.

92 uced activation of p53 is similar in primary parotid cells and parotid glands from PKCdelta(+/+) and

93 amino-terminal kinase is reduced in primary parotid cells from PKCdelta(-/-) cells and in parotid C5

94 Primary parotid cells from PKCdelta(-/-) mice are defective in m

95 Notably, in native parotid cells isoproterenol enhanced the carbachol-promo

96 8 in Ca(2+) stimulation of cAMP synthesis in parotid cells, acini were isolated from AC1 mutant (AC1-

97 In parotid cells, this was distinct from the effects of the

98 We propose that mouse parotid Cl(ir) current has a bimodal dependence on the e

99 bsorbed dose (r = 0.62), and SUV(max) of the parotids correlated with absorbed dose (r = 0.67).

100 The parotid dose also reduced with increasing body mass and

101 The mean parotid dose was also reduced with increasing body mass

102 These results suggest that in parotid duct cells apical NHE2 and NHE3 do not play a ma

103 RNA sequencing to map the transcriptomes of parotid epithelial cells.

104 initiate fluid secretion in salivary gland (parotid) epithelial cells.

105 ve Car2.Ae2 HCO(3)(-) transport metabolon in parotid exocrine cell function.

106 he correlation between any pair of the three parotid fat content measurement methods.

107 The parotid fat contents measured with the three methods wer

108 The means of measured parotid fat contents revealed significant differences (P

109 t squares (IDEAL) method were used to derive parotid fat contents.

110 Success rates in parotid fat measurements by using T1, T2, and IDEAL meth

111 DEAL method provided a high success rate for parotid fat measurements, even in subjects with metallic

112 neity of fat saturation to determine whether parotid fat quantification was successful, with the succ

113 afe and transfer of the hAQP1 cDNA increased parotid flow and relieved symptoms in a subset of subjec

114 negative effect of a high-sucrose diet on a parotid function involved in the control of intradentina

115 play an important role in the regulation of parotid function.

116 ubjects for several organ systems, including parotid gland (50% vs. 3%), tongue (53% vs. 10%), and lu

117 mice following delivery of the vector to the parotid gland (PTG), the submandibular gland (SMG) or to

118 nd amylase secretion (16-93%) from, isolated parotid gland acinar cells.

119 IL14alphaTG mice, but they lacked the later parotid gland and lung injury.

120 data for the largely understudied embryonic parotid gland as compared with the submandibular gland,

121 dynamic contrast-enhanced MR imaging of the parotid gland at 1.5 T.

122 r players and pathways that are relevant for parotid gland biology.

123 very of novel molecular players important in parotid gland biology.

124 at the stimulation of amylase secretion from parotid gland cells by NaF may be mediated by an increas

125 timulate amylase secretion from isolated rat parotid gland cells.

126 The parotid gland contributes a variety of secretory protein

127 In the present work we used the parotid gland duct which expressed Slc26a4 and Slc26a6 a

128 The fluid-secreting acinar cells of the parotid gland express both IK and BK channels, raising q

129 For normal tissues, parotid gland factors were 6.7, 9.4, 13.3, and 19.3 Gy p

130 For normal tissues, parotid gland factors were 6.7, 9.4, 13.3, and 19.3 Gy p

131 The stimulated parotid gland fluid secretion rate was normal, but the s

132 l solute movement led us to hypothesize that parotid gland function(s) may have a role in regulating

133 A parotid gland hormone that stimulates intradentinal flui

134 A transfer to a single previously irradiated parotid gland in 11 subjects using an open label, single

135 Acetylcholine-evoked secretion from the parotid gland is substantially potentiated by cAMP-raisi

136 esting secretion of salivary proteins by the parotid gland is sustained in situ between periods of ea

137 The parotid gland is the mostly affected site among major sa

138 Based on these observations, the parotid gland kinase may be related to other Golgi-local

139 y and accurately differentiate epitopes from parotid gland N-glycans and milk oligosaccharides based

140 ultiple compensatory changes in the exocrine parotid gland of Nhe1(-/-) mice that together attenuate

141 ound (US) with painful swelling in the right parotid gland region.

142 hDF-EpiSC-loaded native de-cellularized rat parotid gland scaffolds into the renal capsule of nude m

143 correlated with decreased submandibular and parotid gland sizes.

144 Parotid gland swelling appeared to be the sine qua non o

145 l transcripts correlated with viral loads in parotid gland tissue and saliva.

146 systems biology approach was used to analyze parotid gland tissue samples obtained from patients with

147 Furthermore, the ability of the parotid gland to conserve NaCl was abolished in NKCC1-de

148 vation of p53 is required and sufficient for parotid gland transformation in the presence of activate

149 In contrast to the dramatic effect of p53 in parotid gland transformation, p53 loss has little effect

150 thyroid carcinoma, basal cell carcinoma, and parotid gland tumor, and 68.5% +/- 6.4% for those with n

151 [SIR] 52.3), non-Hodgkin lymphoma (SIR 8.3), parotid gland tumors (SIR 33.4), thyroid cancer (SIR 13.

152 mice heterozygous for a p53 deletion develop parotid gland tumors and loose their wild type p53 allel

153 accompanied by the down regulation of p21 in parotid gland tumors but not breast tumors.

154 the risk of meningioma, acoustic neuroma, or parotid gland tumors in relation to mobile phone use.

155 The parotid gland tumors were aneuploid and demonstrated inc

156 th the risks of brain, acoustic neuroma, and parotid gland tumors.

157 AdhAQP1 vector delivery to a single parotid gland was safe and transfer of the hAQP1 cDNA in

158 inductive site of the mucosal immune system (parotid gland) become polyclonal in piglets reared germf

159 dings included gross enlargement of the left parotid gland, a focal lesion in the right parotid gland

160 t parotid gland, a focal lesion in the right parotid gland, and cervical lymphadenopathy.

161 f the salivary glands, adrenal gland, colon, parotid gland, kidney, thyroid gland, thymus, or uterus

162 were obtained using Hermes software for the parotid gland, tongue, thyroid, lung, gastric wall, panc

163 better elucidate the molecular nature of the parotid gland, we have performed RNA sequencing to gener

164 epresent important players that could impart parotid gland-specific biological properties.

165 our detailed analysis has revealed a unique parotid gland-specific gene signature that may represent

166 the various epithelial cell types within the parotid gland.

167 retion and regulatory volume decrease in the parotid gland.

168 rted DNase I mRNA transcript cloned from rat parotid gland.

169 t of SLPI isolated previously from the human parotid gland.

170 ed in the superficial-deep lobe of the right parotid gland.

171 2 females, presented painless masses in the parotid gland.

172 d found later in development surrounding the parotid gland.

173 e submandibular glands (0.1479 mSv), and the parotid glands (0.1137 mSv/MBq).

174 ice packs on (177)Lu-PSMA-617 uptake by the parotid glands (PGs).

175 andibular glands (PRP, 53%; mean SUV, 2.11), parotid glands (PRP, 51%; mean SUV, 1.90), and vocal cor

176 One or both parotid glands and at least two-thirds of the tongue wer

177 erian, lacrimal, mandibular, sublingual, and parotid glands and from liver, kidney, pancreas, testis

178 ons, BPIFA2 is expressed specifically in the parotid glands and is abundant in salivary secretions.

179 s, and lymphocytic infiltrates in the lungs, parotid glands and kidneys.

180 or bilateral nonsuppurative swelling of the parotid glands and neurological complications that can r

181 This hormone has been purified from porcine parotid glands and partially sequenced in our previous s

182 associated with high viral loads in infected parotid glands and that late viral protein expression is

183 an, lacrimal, submandibular, sublingual, and parotid glands and the liver.

184 Parotid glands appeared to exert a positive effect on de

185 IgG and IgA repertoire within pSS patients' parotid glands are distinct from those in non-pSS contro

186 ter mRNA increased dramatically in Nhe1(-/-) parotid glands but not in those of Nhe2(-/-) or Nhe3(-/-

187 Mumps viral antigen was detected in parotid glands by immunohistochemistry (IHC).

188 Saliva from the submandibular and parotid glands contained higher concentrations of adreno

189 Systems biology analyses of the parotid glands from patients with primary SS and those w

190 p53 is similar in primary parotid cells and parotid glands from PKCdelta(+/+) and PKCdelta(-/-) mice

191 on is suppressed in vivo in gamma-irradiated parotid glands from PKCdelta(-/-) mice.

192 cute time points (30 min to 2 h), irradiated parotid glands had significantly decreased levels of the

193 and to probe its specific roles, we studied parotid glands in mice with the K(Ca)1.1 gene ablated.

194 n vitro functioning of acinar cells from the parotid glands of mice with targeted disruption of Na(+)

195 Indeed, the parotid glands of Nhe1(-/-) mice expressed higher levels

196 sis is suppressed by greater than 60% in the parotid glands of PKCdelta(-/-) mice.

197 ping the Ig-producing cell repertoire in the parotid glands of primary Sjogren's syndrome (pSS) patie

198 culate that B cell hyperproliferation within parotid glands of pSS patients may result from Ag-indepe

199 ts in a robust induction of apoptosis in the parotid glands of wild type mice, whereas apoptosis is s

200 Parotid glands received higher doses (1.3 mGy/MBq) than

201 that IGF-1 promotes DNA repair in irradiated parotid glands through the maintenance and activation of

202 IK channels, activated fluid secretion from parotid glands was normal.

203 Homogenates and subfractions from macaque parotid glands were able to phosphorylate synthetic pept

204 ng demonstrated that the acinar cells of the parotid glands were the primary location for both the pa

205 Sparing the parotid glands with IMRT significantly reduces the incid

206 ritical structures were the spinal cord, the parotid glands, and the mandible.

207 Uptake in bone marrow, parotid glands, and tonsils was slightly but statistical

208 In perfused rat parotid glands, isoproterenol induced staining of both a

209 absorbed dose to kidneys, submandibular and parotid glands, liver, spleen, and bone marrow was 0.39,

210 the production of type 1 IFN; injury to the parotid glands, lungs, and kidneys is seen; 3) progressi

211 metrically defined ipsilateral/contralateral parotid glands, submandibular glands, and oral cavity su

212 ound uptake than (68)Ga-HBED-PSMA-11 (in the parotid glands, the mean SUVmax for (68)Ga-THP-PSMA was

213 therapy (IMRT) can reduce irradiation of the parotid glands.

214 om the native pancreas and submandibular and parotid glands.

215 the in vivo functions of these exchangers in parotid glands.

216 revealed that AC5/6 and AC3 are expressed in parotid glands.

217 ncluding human gingiva and submandibular and parotid glands.

218 istent uptake in the salivary, lacrimal, and parotid glands.

219 ssory protein, LRRC26, which is expressed in parotid glands: expressed parSlo + LRRC26 channels were

220 G uptake in the cerebral cortex, cerebellum, parotid grand, myocardium, and bowel mostly reflected th

221 In addition, the 30-amino acid parotid hormone was synthesized.

222 lands were the primary location for both the parotid hormone-related mRNAs and the translation produc

223 with the amino acid sequence of the isolated parotid hormone.

224 cell lines from the submandibular (HSG) and parotid (HSY) salivary glands.

225 Our findings show that malignant parotid lesion was the stiffest lesion according to elas

226 RESENT THE ROLE OF ELASTOSONOGRAPHY IN THREE PAROTID LESIONS: a case of benign pleomorphic adenoma, a

227 By using parotid lobules from ad libitum fed rats stimulated with

228 the livers, kidneys, spleens and superficial parotid lymph nodes of the mice.

229 urgical oncology team, who believed that the parotid mass and cervical adenopathy were technically re

230 and showed that ectopic expression in either parotid or pulmonary MEC tumor cell lines containing the

231 eristics when compared with the proteomes of parotid or submandibular/sublingual secretions.

232 ndard and high-sucrose diets, removal of the parotid or the submandibular/sublingual glands, and diet

233 essed by the MSCs of human major SGs, namely parotid (PAG), sublingual (SLG) and submandibular (SMG)

234 , we achieved relative quantification of the parotid peptides at four time points.

235 he thymus as a source of immature T cells in parotid (PG) and submandibular salivary glands (SMG) wer

236 secreted by the 3 major salivary glands: the parotid (PG), the submandibular (SMG), and the sublingua

237 AG10 and AG18 blocked parotid phosphorylation events only at concentrations th

238 eased abundance of Car2.Ae2 complexes in the parotid plasma membranes of Nhe1(-/-) mice.

239 (+)-K(+)-2Cl(-) cotransporter (NKCC1) in rat parotid plasma membranes was studied using the reversibl

240 nomas, myeloma, melanoma, colonic carcinoid, parotid pleomorphic adenoma, and teratoma.

241 The increased potency of InsP(3) in parotid probably results from a four-fold higher number

242 on between tumor volume and mean dose to the parotids (r = -0.41) and kidneys (r = -0.43).

243 hole saliva (r = 0.10, p < 0.05), stimulated parotid saliva (r = 0.13, p < 0.02), and stimulated SMSL

244 le saliva (r = 0.24, p < 0.0001), stimulated parotid saliva (r = 0.13, p < 0.03), unstimulated SMSL (

245 however, it was 37%, 18% lower than Db from parotid saliva (reported previously).

246 Proline-rich proteins were enriched from parotid saliva and found to increase binding of anthocya

247 We collected parotid saliva as the ductal secretion from three human

248 ntified as the major iron-binding protein in parotid saliva by 59Fe autoradiography and Western blott

249 Iron binding was determined in parotid saliva by addition of nonlabeled and 59Fe labele

250 No iron was found in LF from parotid saliva in either group.

251 agglutinin blocked the inhibitory effect of parotid saliva on exogenously applied human leukocyte el

252 rnal effects on the composition of the human parotid saliva peptidome.

253 ptides in the < or =10-kDa fraction of human parotid saliva that included many novel species.

254 Whole and parotid saliva was collected from LAgP patients and matc

255 Parotid saliva was collected from subjects into separate

256 ed that the low-molecular-weight fraction of parotid saliva was peptide-rich, with novel fragments of

257 ated and stimulated whole saliva, stimulated parotid saliva, and stimulated labial minor gland saliva

258 Parotid saliva, nasal wash, and serum were collected pri

259 When iron was added to parotid saliva, the LAgP saliva bound 20 to 30 times les

260 o prepare a low-molecular-weight fraction of parotid saliva, which was analyzed directly or after rev

261 e majority of glucan made by GTF adsorbed to parotid saliva-coated HA remained attached to the surfac

262 requency was 14%, similar to Db protein from parotid saliva.

263 several other components from both SM-SL and parotid saliva.

264 l saliva, but it was essentially absent from parotid saliva.

265 uence, we isolated the agglutinin from human parotid saliva.

266 he wild-type strains adhered better to human parotid-saliva- and amylase-coated hydroxyapatite than d

267 efficacy was evaluated with measurements of parotid salivary flow rate.

268 ly shown that the IK1 and maxi-K channels in parotid salivary gland acinar cells are encoded by the K

269 Sequential parotid salivary gland biopsies were taken before RTX, a

270 n double-stranded breaks (DSB) in the DNA of parotid salivary gland cells immediately after treatment

271 Using rat parotid salivary gland cells, we examined multiple pathw

272 ells from embryonic day 12 submandibular and parotid salivary glands to characterize their molecular

273 I fold containing family A member 2 (BPIFA2)/parotid salivary protein (PSP), and lactoperoxidase, 3 s

274 ults indicate that proteolytic processing of parotid salivary proteins differs among individuals who

275 is high and these compounds localize to the parotid, salivary, and lacrimal glands as well as to the

276 n of sodium chloride to dialyzed, fungicidal parotid secretion abolished this activity, indicating th

277 genes, respectively, and in vivo stimulated parotid secretion is severely reduced in double-null mic

278 the fungicidal activities of some proteins, parotid secretion was subjected to dialysis with membran

279 Os of >=1000 promoted fungicidal activity of parotid secretion, and this activity was dose dependent.

280 Protein samples from parotid secretion, submandibular/sublingual secretion, w

281 the major candidacidal capacity of dialyzed parotid secretion.

282 Parotid secretory protein (PSP) and palate-lung-nasal ep

283 re, we report that the soluble cargo protein Parotid Secretory Protein (PSP) is bound to the membrane

284 1), anti-carbonic anhydrase 6 (CA6) and anti-parotid secretory protein (PSP), which occur early in th

285 ncluding amylase, proline-rich proteins, and parotid secretory protein (PSP)-to these functions.

286 1 (SP1), anti-carbonic anhydrase 6 (CA6) and parotid secretory protein (PSP).

287 ng family A member 2 (BPIFA2), also known as parotid secretory protein, which we identified via a mul

288 serine kinases with proteolytic activity for parotid secretory protein.

289 Parotid secretory proteins are stored in large dense-cor

290 preponderance was seen except in the case of parotid space lesions where female predominance was seen

291 In the parotid space, pleomorphic adenoma and in the prestyloid

292 We assessed the hypothesis that parotid-sparing IMRT reduces the incidence of severe xer

293 red conventional radiotherapy (control) with parotid-sparing IMRT.

294 als consists of 3 major pairs of glands: the parotid, submandibular, and sublingual glands.

295 Immunofluorescence of parotid tissue slices revealed that PMCA1 was distribute

296 ozygous p53 deletion have increased rates of parotid tumor onset suggesting that inactivation of p53

297 adenoma, a Wharthin's tumour and a malignant parotid tumour.

298 from the epithelial component of preexisting parotid Warthin tumors (WT).

299 InsP(3) was more effective in parotid, where [Ca(2+)](c) signals initiated with shorte

300 A muscle cluster was prominent in the parotid, which was not myoepithelial or vascular smooth