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1  of the lesions showed lower uptake than the parotid glands).
2 t of SLPI isolated previously from the human parotid gland.
3 iated lymphoid tissue (MALT) lymphoma in the parotid gland.
4 astic hypertrophy and hyperplasia of the rat parotid gland.
5 ess mucous acinar genes not intrinsic to the parotid gland.
6 d found later in development surrounding the parotid gland.
7 ed in the superficial-deep lobe of the right parotid gland.
8  2 females, presented painless masses in the parotid gland.
9 retion and regulatory volume decrease in the parotid gland.
10 rted DNase I mRNA transcript cloned from rat parotid gland.
11 the various epithelial cell types within the parotid gland.
12  in the submandibular glands, but not in the parotid glands.
13  submandibular glands and rat sublingual and parotid glands.
14 ease characterized by the enlargement of the parotid glands.
15 ormalities in the parenchyma relative to the parotid glands.
16 istent uptake in the salivary, lacrimal, and parotid glands.
17 therapy (IMRT) can reduce irradiation of the parotid glands.
18 om the native pancreas and submandibular and parotid glands.
19 the in vivo functions of these exchangers in parotid glands.
20 revealed that AC5/6 and AC3 are expressed in parotid glands.
21 ncluding human gingiva and submandibular and parotid glands.
22 e submandibular glands (0.1479 mSv), and the parotid glands (0.1137 mSv/MBq).
23 ecules demonstrated a comparable dose to the parotid glands (0.5 Gy/GBq, P = 0.27).
24 cycle ranged from 0.19 to 0.4 Gy/GBq for the parotid gland, 0.14 to 0.28 Gy/GBq for the submandibular
25 d epidermal cyst (1), Warthin's tumor of the parotid gland (1), surgical wound inflammation (2), leio
26 11.2 vs. 11.9), small bowel (16.2 vs. 17.1), parotid gland (19.2 vs. 19.6), lacrimal gland (8.9 vs. 1
27 ubjects for several organ systems, including parotid gland (50% vs. 3%), tongue (53% vs. 10%), and lu
28 ypes of embryonic mouse salivary glands: the parotid gland (a serous gland) and the submandibular gla
29 dings included gross enlargement of the left parotid gland, a focal lesion in the right parotid gland
30 nd amylase secretion (16-93%) from, isolated parotid gland acinar cells.
31 in submandibular and minor AdCCs compared to parotid gland AdCCs, and NRF2 pathway expression is asso
32  IL14alphaTG mice, but they lacked the later parotid gland and lung injury.
33 ixty neutral sugars were identified from the parotid gland and many of these were additionally found
34                                  One or both parotid glands and at least two-thirds of the tongue wer
35 erian, lacrimal, mandibular, sublingual, and parotid glands and from liver, kidney, pancreas, testis
36 ons, BPIFA2 is expressed specifically in the parotid glands and is abundant in salivary secretions.
37 s, and lymphocytic infiltrates in the lungs, parotid glands and kidneys.
38 y in secretory endpieces isolated from sheep parotid glands and loaded with the pH-sensitive fluoropr
39  or bilateral nonsuppurative swelling of the parotid glands and neurological complications that can r
40  This hormone has been purified from porcine parotid glands and partially sequenced in our previous s
41 associated with high viral loads in infected parotid glands and that late viral protein expression is
42 an, lacrimal, submandibular, sublingual, and parotid glands and the liver.
43 35 +/- 0.78 and 0.80 +/- 0.41 Gy/GBq for the parotid gland, and 0.67 +/- 0.62 and 0.30 +/- 0.27 Gy/GB
44 iver, 2.35 0.78 and 0.80 0.41 Gy/GBq for the parotid gland, and 0.67 0.62 and 0.30 0.27 Gy/GBq for th
45 t parotid gland, a focal lesion in the right parotid gland, and cervical lymphadenopathy.
46  while DNase I is expressed in the pancreas, parotid gland, and kidney.
47 ritical structures were the spinal cord, the parotid glands, and the mandible.
48                       Uptake in bone marrow, parotid glands, and tonsils was slightly but statistical
49                                              Parotid glands appeared to exert a positive effect on de
50 hly fucosylated glycans that co-exist in the parotid gland are not fully known.
51  IgG and IgA repertoire within pSS patients' parotid glands are distinct from those in non-pSS contro
52                                              Parotid glands are one of the most common sites for sali
53  data for the largely understudied embryonic parotid gland as compared with the submandibular gland,
54  dynamic contrast-enhanced MR imaging of the parotid gland at 1.5 T.
55      The mean (SD) excretory function of the parotid gland at 12 months, measured by scintigraphy, im
56 inductive site of the mucosal immune system (parotid gland) become polyclonal in piglets reared germf
57 r players and pathways that are relevant for parotid gland biology.
58 very of novel molecular players important in parotid gland biology.
59 d focus of follicular lymphoma in the second parotid gland biopsy.
60 mRNA expression was detected in the gingiva, parotid gland, buccal mucosa, and tongue.
61 ter mRNA increased dramatically in Nhe1(-/-) parotid glands but not in those of Nhe2(-/-) or Nhe3(-/-
62        Stimulation of amylase secretion from parotid glands by beta-adrenergic agonists is mediated b
63          Mumps viral antigen was detected in parotid glands by immunohistochemistry (IHC).
64  volumes for head and neck tumors as well as parotid glands can be well-defined on cross-sectional CT
65  uterine leiomyosarcoma, and 1 had oncocytic parotid gland carcinoma and continued receiving treatmen
66 at the stimulation of amylase secretion from parotid gland cells by NaF may be mediated by an increas
67 timulate amylase secretion from isolated rat parotid gland cells.
68 y against bands of 140-190 and 120 kd in the parotid gland, colon, and stomach, sites where the secre
69 rall survival, was overexpressed in AdCCs of parotid gland compared to minor and submandibular glands
70            Saliva from the submandibular and parotid glands contained higher concentrations of adreno
71                                          The parotid gland contributes a variety of secretory protein
72 ls were assessed in 77 ACC and 11 unaffected parotid gland control samples by immunohistochemistry an
73 that ACh-evoked HCO3- secretion in the sheep parotid gland differs from that in many other salivary g
74              In the present work we used the parotid gland duct which expressed Slc26a4 and Slc26a6 a
75                                Patients with parotid gland enlargement accompanied by sicca symptoms
76                                 They all had parotid gland enlargement and lymphocytic interstitial p
77 gnosis of DILS was established in those with parotid gland enlargement by minor salivary gland biopsy
78 ere found in secondary end points except for parotid gland excretory function, as assessed by scintig
79  that Ca++ and calmodulin may play a role in parotid gland exocytosis by modulating the interaction b
80      The fluid-secreting acinar cells of the parotid gland express both IK and BK channels, raising q
81 ssory protein, LRRC26, which is expressed in parotid glands: expressed parSlo + LRRC26 channels were
82       The similar neonatal submandibular and parotid gland expression patterns observed for these gen
83                          For normal tissues, parotid gland factors were 6.7, 9.4, 13.3, and 19.3 Gy p
84                          For normal tissues, parotid gland factors were 6.7, 9.4, 13.3, and 19.3 Gy p
85                               The stimulated parotid gland fluid secretion rate was normal, but the s
86              Systems biology analyses of the parotid glands from patients with primary SS and those w
87  p53 is similar in primary parotid cells and parotid glands from PKCdelta(+/+) and PKCdelta(-/-) mice
88 on is suppressed in vivo in gamma-irradiated parotid glands from PKCdelta(-/-) mice.
89 st that with the use of 3-DTP, contralateral parotid gland function can be partially preserved for at
90 l solute movement led us to hypothesize that parotid gland function(s) may have a role in regulating
91 cute time points (30 min to 2 h), irradiated parotid glands had significantly decreased levels of the
92                                            A parotid gland hormone that stimulates intradentinal flui
93 ed in the inhibition of beta-agonist-induced parotid gland hypertrophy and hyperplasia.
94 A transfer to a single previously irradiated parotid gland in 11 subjects using an open label, single
95  and to probe its specific roles, we studied parotid glands in mice with the K(Ca)1.1 gene ablated.
96  salivary gland dysfunction to contralateral parotid glands in patients with head and neck cancers.
97         Nevertheless, due to the size of the parotid gland, increased elastographic resolution is req
98 of the lesions showed higher uptake than the parotid glands) intermediate (neither low nor high), and
99      Acetylcholine-evoked secretion from the parotid gland is substantially potentiated by cAMP-raisi
100 esting secretion of salivary proteins by the parotid gland is sustained in situ between periods of ea
101                                          The parotid gland is the mostly affected site among major sa
102                              In perfused rat parotid glands, isoproterenol induced staining of both a
103 f the salivary glands, adrenal gland, colon, parotid gland, kidney, thyroid gland, thymus, or uterus
104             Based on these observations, the parotid gland kinase may be related to other Golgi-local
105               The absorbed doses to kidneys, parotid glands, lacrimal glands, and red marrow were 23,
106  absorbed dose to kidneys, submandibular and parotid glands, liver, spleen, and bone marrow was 0.39,
107  the production of type 1 IFN; injury to the parotid glands, lungs, and kidneys is seen; 3) progressi
108               The follicular lymphoma of the parotid gland lymph node and the follicular lymphoma of
109   We propose that follicular lymphoma in the parotid gland lymph node may have resulted from coloniza
110 ulness in the quantitative assessment of the parotid glands, making it difficult to differentiate bet
111 y and accurately differentiate epitopes from parotid gland N-glycans and milk oligosaccharides based
112 mong healthy organs were in the lacrimal and parotid glands-not, however, resulting in any significan
113 rotection analyses indicated the presence in parotid gland of mRNA homologous to ClC-2.
114 ultiple compensatory changes in the exocrine parotid gland of Nhe1(-/-) mice that together attenuate
115                                 The skin and parotid glands of certain toads accumulate a variety of
116         Similar results were observed in the parotid glands of mice pretreated with imatinib prior to
117 n vitro functioning of acinar cells from the parotid glands of mice with targeted disruption of Na(+)
118                                  Indeed, the parotid glands of Nhe1(-/-) mice expressed higher levels
119 sis is suppressed by greater than 60% in the parotid glands of PKCdelta(-/-) mice.
120 ping the Ig-producing cell repertoire in the parotid glands of primary Sjogren's syndrome (pSS) patie
121 culate that B cell hyperproliferation within parotid glands of pSS patients may result from Ag-indepe
122 ts in a robust induction of apoptosis in the parotid glands of wild type mice, whereas apoptosis is s
123       There was no significant difference in parotid gland or splenic SUV(max) or lesion-to-parotid g
124                           Involvement of the parotid gland (OR, 0.39; 95% CI, 0.18-0.85) and segment
125 ptors are expressed in axons innervating the parotid gland, paralleling our findings in the submandib
126  ice packs on (177)Lu-PSMA-617 uptake by the parotid glands (PGs).
127 andibular glands (PRP, 53%; mean SUV, 2.11), parotid glands (PRP, 51%; mean SUV, 1.90), and vocal cor
128 mice following delivery of the vector to the parotid gland (PTG), the submandibular gland (SMG) or to
129 rotid gland or splenic SUV(max) or lesion-to-parotid gland ratios between the 2 tracers although ther
130                                      Treated parotid glands received an average dose of 5745 cGy, whi
131                                              Parotid glands received higher doses (1.3 mGy/MBq) than
132 ound (US) with painful swelling in the right parotid gland region.
133                                The adjoining parotid gland releases saliva in response to olfactory o
134  hDF-EpiSC-loaded native de-cellularized rat parotid gland scaffolds into the renal capsule of nude m
135  correlated with decreased submandibular and parotid gland sizes.
136 epresent important players that could impart parotid gland-specific biological properties.
137  our detailed analysis has revealed a unique parotid gland-specific gene signature that may represent
138 us, ESE-2, but not ESE-1, transactivates the parotid gland-specific PSP promoter and the prostate-spe
139 -tissue metastases) as well as normal liver, parotid gland, spleen, and mediastinal blood-pool SUV(ma
140                Results: Tracer uptake by the parotid gland, submandibular gland, and spleen was moder
141 metrically defined ipsilateral/contralateral parotid glands, submandibular glands, and oral cavity su
142                                              Parotid gland swelling appeared to be the sine qua non o
143 ound uptake than (68)Ga-HBED-PSMA-11 (in the parotid glands, the mean SUVmax for (68)Ga-THP-PSMA was
144 that IGF-1 promotes DNA repair in irradiated parotid glands through the maintenance and activation of
145 l transcripts correlated with viral loads in parotid gland tissue and saliva.
146 systems biology approach was used to analyze parotid gland tissue samples obtained from patients with
147              Furthermore, the ability of the parotid gland to conserve NaCl was abolished in NKCC1-de
148     We propose that CB1 receptors act at the parotid gland to inhibit stimulated salivation but also
149 on (amylase-positive area) within irradiated parotid glands to levels similar to untreated controls.
150  were obtained using Hermes software for the parotid gland, tongue, thyroid, lung, gastric wall, panc
151 vation of p53 is required and sufficient for parotid gland transformation in the presence of activate
152 In contrast to the dramatic effect of p53 in parotid gland transformation, p53 loss has little effect
153 thyroid carcinoma, basal cell carcinoma, and parotid gland tumor, and 68.5% +/- 6.4% for those with n
154    The etiology of Warthin's tumor, a benign parotid gland tumor, is unknown.
155 [SIR] 52.3), non-Hodgkin lymphoma (SIR 8.3), parotid gland tumors (SIR 33.4), thyroid cancer (SIR 13.
156 mice heterozygous for a p53 deletion develop parotid gland tumors and loose their wild type p53 allel
157 accompanied by the down regulation of p21 in parotid gland tumors but not breast tumors.
158 the risk of meningioma, acoustic neuroma, or parotid gland tumors in relation to mobile phone use.
159                                          The parotid gland tumors were aneuploid and demonstrated inc
160 th the risks of brain, acoustic neuroma, and parotid gland tumors.
161          AdhAQP1 vector delivery to a single parotid gland was safe and transfer of the hAQP1 cDNA in
162 t Raf-1 protein kinase activity in the mouse parotid glands was induced by chronic isoproterenol admi
163  IK channels, activated fluid secretion from parotid glands was normal.
164 core (SUV(mean) ratio of whole-body tumor to parotid glands) was semiautomatically calculated on base
165 better elucidate the molecular nature of the parotid gland, we have performed RNA sequencing to gener
166    Homogenates and subfractions from macaque parotid glands were able to phosphorylate synthetic pept
167 ng demonstrated that the acinar cells of the parotid glands were the primary location for both the pa
168 ells among AdCC of minor, submandibular, and parotid glands while plasma cells were enriched in norma
169                                  Sparing the parotid glands with IMRT significantly reduces the incid

 
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